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1 pathogenesis of amoebic colitis and amoebic liver abscess.
2 immunodeficient (SCID) mouse model of amebic liver abscess.
3 ansplant with 1 death, and 1 (20%) died of a liver abscess.
4 linical suspicion of an incompletely treated liver abscess.
5 al computed tomography showed a 2.2 x 2.0 cm liver abscess.
6 inate to other organs such as liver, causing liver abscess.
7 hat manifests as colitis and, in some cases, liver abscess.
8 n parasite that causes amoebic dysentery and liver abscess.
9 tobiliary infections include cholangitis and liver abscess.
10 o a new generation of patients with pyogenic liver abscess.
11 multiloculated liver lesion, suggestive of a liver abscess.
12 tion are at an increased incidence of amebic liver abscess.
13 d to the diagnostic armamentarium for amebic liver abscess.
14 site that causes amoebic colitis and amoebic liver abscess.
15 d differential diagnostics in the context of liver abscesses.
16 bae may exacerbate the damage seen in amebic liver abscesses.
17 es between cultured amoebae and amoebae from liver abscesses.
18 ng from gastrointestinal disorders to severe liver abscesses.
19 lore if JPDD could be related to cryptogenic liver abscesses.
20 countries and characterized by dysentery and liver abscesses.
21 a vaccine antigen to protect against amebic liver abscesses.
22 in do not sequester manganese from S. aureus liver abscesses.
23 stolytica, which causes amebic dysentery and liver abscesses.
24 ause of potentially fatal amebic colitis and liver abscesses.
25 n intestinal ameba that causes dysentery and liver abscesses.
26 a protozoan parasite that causes colitis and liver abscesses.
27 gastric stenosis (3%), hepatic failure (3%), liver abscesses (3%), paroxysmal atrial tachycardia (3%)
28 blood pressure, (5) jaundice, (6) rupture of liver abscess, (7) endophthalmitis, , and (8) multiple o
32 South Africa, were recently cured of amebic liver abscess (ALA) with or without concurrent Entamoeba
35 discuss a case of an 82 year old female with liver abscess and bacteremia from lactobacillus after us
38 ashington State with a magA(+) K. pneumoniae liver abscess and describe a simple approach for recogni
39 cal trial recruited hospitalized adults with liver abscess and K. pneumoniae isolated from blood or a
45 ic trophozoites cause intestinal disease and liver abscess, and have expanded our understanding of th
46 ions, neonatal sepsis and community-acquired liver abscess, and is associated with chronic intestinal
49 the protist that causes amebic dysentery and liver abscess, are of great interest for multiple reason
50 ted animals have significantly larger amebic liver abscesses at early stages of infection and that ab
52 virulence in the SCID mouse model of amebic liver abscess, but E. histolytica trophozoites that do n
53 same symptoms and signs as classic pyogenic liver abscess, but further distinguished by the presence
54 ly included in cattle feed for prevention of liver abscesses, but concerns regarding antimicrobial re
55 streptococcus in a patient with lymphoma, a liver abscess caused by Campylobacter curvus and an alph
56 Taiwan has witnessed an emerging syndrome of liver abscess caused by Klebsiella pneumoniae carrying t
58 monomicrobial Klebsiella pneumoniae pyogenic liver abscess, characterized by the same symptoms and si
60 eumoniae causing community-acquired pyogenic liver abscess complicated with metastatic meningitis and
61 raumatic liver abscess, post cholecystectomy liver abscess, concurrent acute cholecystitis, and hepat
62 stolytica causes amebic dysentery and amebic liver abscess, diseases associated with significant morb
63 ith susceptibility to infectious colitis and liver abscess due to Entamoeba histolytica as well as to
66 es cerevisiae fermentation product (SCFP) on liver abscesses, fecal microbiomes, and resistomes in ca
67 amsters reduced development of severe amebic liver abscesses following intrahepatic injection of E. h
68 immunodeficient (SCID) mouse model of amebic liver abscess formation and compared liver damage in neu
69 er, our findings define hallmarks of E. coli liver abscess formation and suggest that hyperactivation
71 r ability to delay the development of amebic liver abscess formation in an E. histolytica infected ha
76 disease, immunization with the CRD inhibited liver-abscess formation, yet in humans, a naturally acqu
79 the clinical presentation and management of liver abscess have evolved considerably, not only in the
81 t role in the host cell death seen in amebic liver abscess in a mouse model of disease and suggest th
82 port a peculiar case of left hepatic amoebic liver abscess in a patient without a well-known source o
91 Klebsiella pneumoniae causes pneumonia and liver abscesses in humans worldwide and contains virulen
92 ses of K. pneumoniae bacteremia with primary liver abscesses in patients with diabetes mellitus in Ta
94 proven to improve feed efficiency and reduce liver abscess incidence, how these products impact the g
95 merging population of patients with pyogenic liver abscess includes those with complications of aggre
96 at antibiotics fail to kill K. pneumoniae in liver abscesses, independently of resistance or spatial
98 human immunodeficiency virus/AIDS, pyogenic liver abscess, infectious endocarditis, cirrhosis, and d
99 report a 16-month-old male child with amebic liver abscess, initially misdiagnosed with pneumonia, wh
100 thalmitis secondary to Klebsiella pneumoniae liver abscess is a blinding infection that is being repo
102 n contrast to amebic liver abscess, pyogenic liver abscess is associated with greater morbidity and m
105 overall prognosis for patients with pyogenic liver abscess is improving, although poor outcomes are c
109 ca, the cause of amebic dysentery and amebic liver abscess, is an obligate anaerobe, and derives ener
111 ysis was conducted for Klebsiella pneumoniae liver abscess (KLA) patients enrolled in a randomized co
122 old man of asian origin who presented with a liver abscess of unknown origin, complicated by septic s
123 n the absence of antibiotic resistance, hvKp liver abscesses often respond poorly to treatment, somet
124 95% CI: 1.246-3.297, p = 0.008), rupture of liver abscess (OR = 5.167, 95% CI: 2.194-23.150, p = 0.0
125 epatitis (OR, 7.435 [95% CI, 1.397-39.572]), liver abscess (OR, 9.068 [95% CI, 1.747-47.061]), metast
126 amebic colitis and a hamster model of amebic liver abscess, oral auranofin markedly decreased the num
127 = 0.024), jaundice (p = < 0.001), rupture of liver abscess (p < 0.001), endophthalmitis (p = 0.003),
130 sting that patients who experienced pyogenic liver abscess (PLA) have higher CRC incidence rates, dat
131 though endophthalmitis secondary to pyogenic liver abscess (PLA) is becoming a globally emerging infe
135 diagnoses of amebic liver abscess, traumatic liver abscess, post cholecystectomy liver abscess, concu
139 9 X-linked CGD patients with staphylococcal liver abscesses refractory to conventional therapy succe
140 lays a role in the innate immunity to amebic liver abscess seen in SCID mice while NO is required for
142 As of 1999, the vast majority of pyogenic liver abscesses should be approached therapeutically by
143 h after amebic inoculation, reduced the mean liver abscess size by 70% at 24 h compared to a control
146 rium SREHP-MBP were protected against amebic liver abscess, the most common extraintestinal complicat
147 to highlighting the complications of amebic liver abscess, this case demonstrates the value of PCR t
151 An association of this allele with amebic liver abscess was also determined in an independent coho
154 a coli leads to bacterial replication within liver abscesses, while other organs such as the spleen c
155 that the prognosis of patients with pyogenic liver abscess, who do not have underlying comorbid condi
156 dophthalmitis, extra-hepatic metastasis from liver abscess with diabetes mellitus, could lead to a de
157 a, the causative agent of amebic colitis and liver abscess, would reduce childhood mortality in count