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1  cell (HSC) activation, which contributes to liver fibrogenesis.
2 tes the deposition of fibrillary collagen in liver fibrogenesis.
3 ANGO1 in procollagen I secretion in HSCs and liver fibrogenesis.
4 ndependent determinant of chemically induced liver fibrogenesis.
5 dative stress, which play important roles in liver fibrogenesis.
6 transcription 3 (STAT3) had been involved in liver fibrogenesis.
7 , CYLD ameliorates hepatocellular damage and liver fibrogenesis.
8 nalyze whether microbiota may interfere with liver fibrogenesis.
9  give rise to HSCs and myofibroblasts during liver fibrogenesis.
10 dentify potential key regulatory proteins of liver fibrogenesis.
11 ering yet unidentified mechanisms underlying liver fibrogenesis.
12 es the synthesis of extracellular matrix and liver fibrogenesis.
13 th IL-10R2 and IL-22R1, thereby ameliorating liver fibrogenesis.
14 pigenetic regulatory mechanisms impacting on liver fibrogenesis.
15 HSC) activation is an essential event during liver fibrogenesis.
16 A levels, suggesting a direct role of HIV in liver fibrogenesis.
17 GF-beta1) production play important roles in liver fibrogenesis.
18 4alpha knockdown enhanced Ctgf induction and liver fibrogenesis.
19 al insight into intrahepatic immunity during liver fibrogenesis.
20 erates hepatic myofibroblasts, which promote liver fibrogenesis.
21 rgo myofibroblastic trans-differentiation in liver fibrogenesis.
22 tellate cell activation is a main feature of liver fibrogenesis.
23 gy for inducing HSC apoptosis and inhibiting liver fibrogenesis.
24  (alphaSMA) and collagen I is a key event in liver fibrogenesis.
25 s to myofibroblastic cells to participate in liver fibrogenesis.
26 (HSC), the primary cellular event underlying liver fibrogenesis.
27 tivation regulated by a specific cytokine in liver fibrogenesis.
28 ation called "myofibroblastic activation" in liver fibrogenesis.
29  Ang II on HSCs and plays a critical role in liver fibrogenesis.
30 patocyte injury is mechanistically linked to liver fibrogenesis.
31 tic stellate cells (HSC), a pivotal event in liver fibrogenesis.
32  polarization and infiltration, and enhanced liver fibrogenesis.
33 atic stellate cells (HSCs) is a key event in liver fibrogenesis.
34 s with high signal amplification for imaging liver fibrogenesis.
35 l (HSC) activation, which is responsible for liver fibrogenesis.
36 -based hydrazine-equipped probes for imaging liver fibrogenesis.
37 tion, and survival of HSCs and protects from liver fibrogenesis.
38 t plays a pivotal role in the progression of liver fibrogenesis and carcinogenesis.
39              HSC activation is a key step in liver fibrogenesis and has a crucial role in collagen de
40             Amphiregulin (AR) involvement in liver fibrogenesis and hepatic stellate cells (HSC) regu
41 e EGF receptor (EGFR) inhibitor erlotinib on liver fibrogenesis and hepatocellular transformation in
42 ng liver disease, we observed development of liver fibrogenesis and necroptotic cell death.
43 fector T-cell responses, which may encourage liver fibrogenesis and progression to end-stage liver di
44        In conclusion, hyperammonemia induces liver fibrogenesis and RIPK1-mediated cell death, which
45 r mechanisms underlying activation of HSC in liver fibrogenesis and the potential therapeutic value o
46 g-term TCS exposure, especially on enhancing liver fibrogenesis and tumorigenesis, and the relevance
47 ced mouse models, a cholestasis rat model of liver fibrogenesis, and in human fibrotic liver tissues.
48           Stellate cells are key elements in liver fibrogenesis, and previously we have demonstrated
49  in the processing of type I collagen during liver fibrogenesis, and that TGF-beta1 and TNF-alpha reg
50  mice were protected against CCl(4)-mediated liver fibrogenesis, as evidenced by reduced collagen typ
51 usal factor of HSC activation and subsequent liver fibrogenesis, but the mechanism is not fully under
52          Thus, leptin has a direct action on liver fibrogenesis by stimulating TIMP-1 production in a
53                       It points to increased liver fibrogenesis during acute COVID-19 with possible l
54 f Fas-mediated hepatocyte apoptosis promotes liver fibrogenesis during extrahepatic cholestasis.
55                                           In liver fibrogenesis, hepatic stellate cells (HSCs) are th
56 senchymal transition (EndMT) and spontaneous liver fibrogenesis in EC-specific constitutive hemi-defi
57 HSCs), the predominant cell type involved in liver fibrogenesis in response to liver damage.
58 ase, plays a key role in this process and in liver fibrogenesis in vivo.
59 may participate in the pathologic process of liver fibrogenesis in vivo.
60 upon liver injuries and functions to inhibit liver fibrogenesis induced by either carbon tetrachlorid
61 negative endotoxin producers, may accelerate liver fibrogenesis, introducing dysbiosis as a cofactor
62                                              Liver fibrogenesis is accompanied by upregulation of lys
63                                              Liver fibrogenesis is associated with excessive producti
64                                              Liver fibrogenesis is associated with the transition of
65 n of pathologic angiogenesis, attenuation of liver fibrogenesis partly mediated through inhibition of
66 ollectively demonstrate that CHI3L1 promotes liver fibrogenesis through a direct effect on HSCs and s
67                    S1P participates in mouse liver fibrogenesis via a paracrine manner.
68 ere loss of ERG causes endothelial-dependent liver fibrogenesis via regulation of SMAD2/3.
69 y to cholangiocyte proliferation but also to liver fibrogenesis via the coordinate activation of GalR
70 n vitro or in vivo but are important because liver fibrogenesis was attenuated in db/db mice.
71           The ability of the probe to detect liver fibrogenesis was then demonstrated in vivo in CCl(
72 the potential function of E-type cyclins for liver fibrogenesis, we repetitively treated constitutive