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1      CNN generated imaging features from the liver parenchyma.
2 s, and the rare occurrence of mitoses in the liver parenchyma.
3  could repopulate a mean of 32% of recipient liver parenchyma.
4 foci eventually become confluent and replace liver parenchyma.
5 um within the arteries and iodine within the liver parenchyma.
6 tions of cells in perivascular areas and the liver parenchyma.
7 cement was compared with that of surrounding liver parenchyma.
8  cell proliferation and rapid restoration of liver parenchyma.
9 th increased histological alterations in the liver parenchyma.
10 earer views of the abscess pouch, septa, and liver parenchyma.
11  their LDHA-expressed subunits that dominate liver parenchyma.
12 at of a region of interest in the nontumoral liver parenchyma.
13 (BS)-mediated toxic injury of the "upstream" liver parenchyma.
14 igated the physiological role of HOIP in the liver parenchyma.
15 slet allografts can survive long term in the liver parenchyma.
16 tion of tolerance to islet allografts in the liver parenchyma.
17 the largest area of the tumor and background liver parenchyma.
18 ding cGAS-driven IRF3 activation through the liver parenchyma.
19  mean fluorescence intensity in the adjacent liver parenchyma.
20 tify tissue attenuation properties of ICG in liver parenchyma.
21  bile ducts developed in the Prox1-deficient liver parenchyma.
22  HCC tissues compared with the nonneoplastic liver parenchyma.
23 ogram parameters were quantified in HCCs and liver parenchyma.
24 ficient (D*), and perfusion fraction (PF) in liver parenchyma.
25 unique tissue architecture of the developing liver parenchyma.
26  carcinoma cases arise within a noncirrhotic liver parenchyma.
27 te interactions between cancer cells and the liver parenchyma.
28 al liver metastases and nontumorous-adjacent liver parenchyma.
29 synchronous liver metastases and/or adjacent liver parenchyma.
30 stinct signal voids dispersed throughout the liver parenchyma.
31 n the donor hepatocyte mass engrafted in the liver parenchyma.
32 hrough the recruitment of lymphocytes to the liver parenchyma.
33  the surgical resection clips and the normal liver parenchyma.
34 arameters between primary HCC and background liver parenchyma.
35 rotein in either the lymphoid compartment or liver parenchyma.
36 tion and tend to deposit iron throughout the liver parenchyma.
37  new cells are added and incorporated to the liver parenchyma.
38 ence of numerous cysts spread throughout the liver parenchyma.
39  with ascites), and lesion isointensity with liver parenchyma.
40 epatocyte hyperplasia and removal of injured liver parenchyma.
41  area of transgene expression throughout the liver parenchyma.
42 and integration of transplanted cells in the liver parenchyma.
43  contrary to the concentrations found in the liver parenchyma (0.5 +/- 0.3 mg/mL versus 4.2 +/- 0.3 m
44  splenically injected hepatocytes engraft in liver parenchyma; 2) a mean of 6.6% of splenically injec
45    In the presence of the primary tumor, the liver parenchyma adjacent to the synchronous liver metas
46 tion selectively and quantitatively into the liver parenchyma after inducing apoptosis.
47 large liver vessels (at 24 hours) and in the liver parenchyma (after day 3).
48 on (PVL), and in situ splitting (ISS) of the liver parenchyma along the falciform ligament were perfo
49  liver tumors while minimizing the impact on liver parenchyma and avoiding systemic effects.
50 tic adenocarcinoma with those in surrounding liver parenchyma and healthy subjects.
51 tary disorder caused by fatty changes in the liver parenchyma and hepatocytes without alcohol consump
52        In this work, analysis of CHB patient liver parenchyma and in vitro HBV infection models shows
53 hich resulted in the atrophy of the affected liver parenchyma and induction of hyperplasia in the tra
54 ted into hepatocytes when engrafted into the liver parenchyma and into bile epithelial cells when eng
55         Transplanted cells integrated in the liver parenchyma and liver architecture were preserved n
56 e direct effect of ethanol byproducts on the liver parenchyma and neurobehavioral mechanisms.
57                          Average CNR between liver parenchyma and segmental veins were measured in ea
58 mutant mice presented with severely necrotic liver parenchyma and significantly larger hypoxic areas,
59 rats, transplanted cells integrated into the liver parenchyma and survived in vascular spaces.
60 troglitazone for 3.5 months, massive loss of liver parenchyma and symptoms of liver failure developed
61  differences in SH U 508A uptake between the liver parenchyma and the lesion for metastases and chola
62 requency of five morphologic findings of the liver parenchyma and two intrahepatic biliary findings i
63 gnificantly enhanced the number of DC in the liver parenchyma and within the liver metastases, as wel
64 d more transplanted cells to be deposited in liver parenchyma, and decreased hepatic ischemia and end
65 t of multiple cysts scattered throughout the liver parenchyma, and include different entities, classi
66 factant proteins in lung alveoli, albumin in liver parenchyma, and lipase in the stomach lining.
67 mage models, the extent of the injury to the liver parenchyma, and potential species-specific differe
68 rtal vein or tail vein, or directly into the liver parenchyma, and the forms of rAAV vector DNA extra
69 were hypoenhancing compared with surrounding liver parenchyma, and they reached their nadir signal in
70 giogenic factors was encountered in adjacent liver parenchyma as compared to the metastases.
71 rge region of interest, inclusive of as much liver parenchyma as possible in the right lobe while avo
72 period of replication and expansion into the liver parenchyma, as well as penetration into the centra
73 ce of finding the metal stent outside of the liver parenchyma at the time of transplantation is repor
74 ment (the ratio of signal intensities of the liver parenchyma before and 20 minutes after intravenous
75 atio of signal intensity measurements of the liver parenchyma before and 20 minutes after intravenous
76 nosis, and one biopsy specimen showed benign liver parenchyma; both ablated lesions showed no residua
77 evealed VWF-specific staining throughout the liver parenchyma but not in endothelial cells.
78              Presence of core protein in the liver parenchyma but not in lymphoid cells protects from
79 /immunohistochemistry combination within the liver parenchyma but not within hepatocytes.
80 itopes were not uniformly distributed in the liver parenchyma but preferentially located in distinct
81 epatic cryoablation of 30% to 35% or more of liver parenchyma, but this complication has not been rep
82 is, which is associated with a change in the liver parenchyma called ductular reaction.
83 A) represents a pus-filled cavity within the liver parenchyma caused by the invasion and multiplicati
84                          Histologically, the liver parenchyma contained randomly distributed, occasio
85 howed that the hepatocytes that comprise the liver parenchyma do not activate RNase L when infected w
86 innate and adaptive immune components of the liver parenchyma during acute viral hepatitis.
87  changes in perfusion to HCC and surrounding liver parenchyma during MR-IR-monitored TACE.
88                        Any disruption of the liver parenchyma during transplantation should be secure
89 dard model to score precisely bile ducts and liver parenchyma features.
90 s increased nearly 2-fold in the recipients' liver parenchyma for 20 days after hepatocyte transplant
91 n of Tc-99m-RBC in hemangiomas and in normal liver parenchyma (HEM/liv), and to verify, whether the q
92 lthough being classically located inside the liver parenchyma, hemangiomas may occasionally develop o
93 limited roles for pericentral hepatocytes in liver parenchyma homeostasis.
94 1.029, P=0.012) and only partial drainage of liver parenchyma (HR 4.158, P=0.040).
95  SH U 508A uptake between the lesion and the liver parenchyma (ie, subjective conspicuity score [SCS]
96 R-2 gene expression was abundant in adjacent liver parenchyma in all 3 groups.
97 nt correlated with pronounced improvement in liver parenchyma in BDL/SIRT(oe) mice.
98                                              Liver parenchyma in both patient groups enhanced signifi
99 ent of necroinflammation and fibrosis in the liver parenchyma in chronic HCV infection.
100 ration and more prominent iron deposition in liver parenchyma in vivo, retained iron export function
101 g-4/VCAM-1 interactions to transmigrate into liver parenchyma in vivo.
102 zation; to reveal incomplete coverage of the liver parenchyma involved by the tumor, which may be rel
103 rvations show that sporozoite entry into the liver parenchyma involves a complex cascade of events, f
104           The accumulation of the fat in the liver parenchyma is accompanied by downregulation of gen
105 e course, whereas T cell infiltration of the liver parenchyma is associated with progressive liver in
106                                          The liver parenchyma is composed of hepatocytes and bile duc
107 ve bile ductular hyperplasia in which normal liver parenchyma is largely replaced with well-different
108 nized liver rodent models, in which the host liver parenchyma is repopulated by human hepatocytes, ha
109 ontaining small (<5 cm) cysts with preserved liver parenchyma is safe and appropriate.
110 that the distribution of microspheres in the liver parenchyma is uniform.
111 assessed lesions (HEM) and in the uninvolved liver parenchyma (liv).
112 tracellular pH, 6.78 +/- 0.09) compared with liver parenchyma (mean extracellular pH, 7.18 +/- 0.03)
113         Transplanted cells integrated in the liver parenchyma more rapidly in CP-treated animals, and
114 IgM in mouse blood, because IgM deposited in liver parenchyma most likely initiated complement activa
115 interfered with neither cell integrations in liver parenchyma nor secretory function of transplanted
116    While the biomechanical properties of the liver parenchyma (normal liver tissue) are known to affe
117 9 uveal melanoma cells was injected into the liver parenchyma of 105 CB17 SCID mice through a 1-cm ab
118 r injecting 2.5 million H4IIE cells into the liver parenchyma of ACI rats, they typically form a 1-cm
119 nd they constitute approximately 0.5-2.5% of liver parenchyma of all donor ages.
120 teria were identified in the portal vein and liver parenchyma of fatal YF cases along with elevations
121 m hESCs and hiPSCs could be engrafted in the liver parenchyma of immune-deficient transgenic mice car
122  were the only ErbB proteins detected in the liver parenchyma on embryonic day 19.
123 vasculature and in the kidney but not in the liver (parenchyma or vasculature).
124 er (90)Y uptake in the tumor relative to the liver parenchyma (P < 0.001).
125  neurotrophin receptor (p75NTR): HSCs in the liver parenchyma; perivascular mesenchymal cells express
126 in consensus) for the imaging pattern of the liver parenchyma, presence and grade of intrahepatic bil
127 ly, significantly different from surrounding liver parenchyma relaxation times of 840 msec +/- 113 an
128 tegration of transplanted hepatocytes in the liver parenchyma required cell membrane regenesis, with
129 crosphere accumulation and regions of normal liver parenchyma that demonstrated no apparent microsphe
130                         After integration in liver parenchyma, the position of transplanted cells was
131 etic resonance (MR) imaging with surrounding liver parenchyma to determine the relationship between t
132 thm was trained for the following: to detect liver parenchyma, to exclude the blood vessels and any a
133 planted cell survival and integration in the liver parenchyma was determined by histochemical analysi
134                       Objective noise in the liver parenchyma was measured, and the tube current was
135 ry or histology in patients whose background liver parenchyma was normal on SPIO-enhanced MRI.
136                                 The necrotic liver parenchyma was seen as high and low intensity on T
137 inusoids, albeit integration of cells in the liver parenchyma was slower in gadolinium-treated rats a
138  of the complex set of cues arising from the liver parenchyma, we cocultured adult rat liver SECs, id
139  which transplanted cells integrate into the liver parenchyma, we used dipeptidyl peptidase IV-defici
140 rtosis, skewness) MRI parameters] in HCC and liver parenchyma were compared using Wilcoxon signed-ran
141 ted by SOX9 are responsible for regenerating liver parenchyma when cell proliferation is impaired fol
142 d distribution of hepatocytes throughout the liver parenchyma, whereas an intraperitoneal injection r
143 arge, well-defined granulomas throughout the liver parenchyma, whereas CCR2-deficient mice had much f
144 es, and to accelerate cell integrations into liver parenchyma will advance applications of hepatocyte
145 n that results in replacement of the healthy liver parenchyma with fibrotic tissue and regenerative n
146 l hepatectomy in mice combine destruction of liver parenchyma with hepatocyte mitoinhibition.
147 latelets were intercalated throughout normal liver parenchyma, with ConA treatment activating residen

 
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