ライフサイエンスコーパス: liver sinusoid

1 ectly accessible to sporozoites entering the liver sinusoid.

2 olling portal blood flow and pressure within liver sinusoids.

3 ominant IL-17A(+) T cell subset in the human liver sinusoids.

4 ent engulfment of B cells circulating in the liver sinusoids.

5 esulted in enlarged hepatocytes and narrowed liver sinusoids.

6 or the overall vascular architecture in the liver sinusoids.

7 a large intravascular macrophage bed in the liver sinusoids.

8 ion/aggregation and subsequent entrapment in liver sinusoids.

9 gh both vessels are largely derived from the liver sinusoids.

10 d that 56% of portal blood flow bypasses the liver sinusoids.

11 nd transformed by hepatic glutathione in the liver sinusoids.

12 alveoli and bronchioli, kidney tubules, and liver sinusoids.

13 gher levels of serum IgA and IgA deposits in liver sinusoids.

14 d can profoundly contribute to remodeling of liver sinusoids.

15 erved in portal vein radicles, as well as in liver sinusoids, albeit integration of cells in the live

16 finity binding of circulating HCV within the liver sinusoids allowing subsequent transfer of the viru

17 transplanted hepatocytes immediately entered liver sinusoids, along with attenuation of portal vein r

18 ce also showed significant dilatation of the liver sinusoids and an increase in inflammatory cells su

19 ial cells in the perfused rBELs colonize the liver sinusoids and express sinusoidal endothelial marke

20 tained tissue-resident macrophages that line liver sinusoids and play an important role on host defen

21 initial arrest, CD8 TE actively crawl along liver sinusoids and probe sub-sinusoidal hepatocytes for

22 trophils promote cancer cell adhesion within liver sinusoids and, thereby, influence metastasis.

23 g to collagen type IV (highly exposed in the liver sinusoids) and collagen type IV-dependent activati

24 e gastrointestinal tract, renal tubules, and liver sinusoids, and their application to modeling organ

25 t that LPS-induced structural changes in the liver sinusoid are mediated by an LPS-induced Kupffer ce

26 After transplantation, hepatocytes entering liver sinusoids are engrafted, whereas cells entrapped i

27 lymphocytes travel via lung capillaries and liver sinusoids at an extremely rapid rate with the aver

28 e show that circulating CD8 TE arrest within liver sinusoids by docking onto platelets previously adh

29 transplanted cells are rapidly cleared from liver sinusoids by proinflammatory cytokines/chemokines/

30 dothelial cell signaling for angiogenesis or liver sinusoid capillarization, the mechanism for initia

31 Liver sinusoid diameters decreased significantly with an

32 In previous work, two anatomically distinct-liver sinusoid endothelial cells (LEC): LEC-1 and LEC-2,

33 ood stream, Plasmodium sporozoites cross the liver sinusoid epithelium, enter and exit several hepato

34 C-SIGNR, a type 2 C-type lectin expressed on liver sinusoids, has been shown to bind with high affini

35 cells typically crawl on the luminal side of liver sinusoids (i.e., are in the blood); simulating T c

36 ement in digital structures derived from the liver sinusoids illustrates that liver structure alone i

37 been associated with vascular injury in the liver sinusoids in clinical studies.

38 novel mechanism by which the destruction of liver sinusoids, induced by the Jo2-mediated co-engageme

39 Gata4 caused transformation of discontinuous liver sinusoids into continuous capillaries.

40 em able to micropattern the self-assembly of liver sinusoid-like structures with micrometer resolutio

41 tablishes an inductive vascular niche in the liver sinusoids of the Id1(-/-) mice, initiating and res

42 lation of receptor-mediated cell adhesion in liver sinusoids or the manipulation of blood flow-depend

43 related receptor found on the endothelium of liver sinusoids, placental capillaries, and lymph nodes,

44 ort here that neutrophils accumulated in the liver sinusoids suppress cytokine and chemokine mRNA exp

45 fluoresence shows high protein expression in liver sinusoids, the venous sinuses of the red pulp in s

46 must penetrate cell barriers in the skin and liver sinusoid to reach their target cell, the hepatocyt

47 electron-microscopic examination of the rat liver sinusoid was performed in this study after in vivo

48 In addition, early adhesion within liver sinusoids was inhibited in the absence of neutroph

49 The liver sinusoids were the only capillaries in which EPCR

50 is largely expressed on endothelial cells in liver sinusoids, whereas DC-SIGN is expressed on dendrit

51 ycerine increased transplanted cell entry in liver sinusoids, whereas labetalol, nifedipine, CGRP, an

52 trations of both glutathione and cysteine in liver sinusoids, which transforms the nanoparticle surfa

53 Coating of cells or of liver sinusoids with natural collagen, natural laminin,

54 d shortly after deposition of hepatocytes in liver sinusoids, with clearance of a significant fractio