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1 a Ni-NTA column to interact in Streptomyces lividans .
2 e of the KcsA K(+) channel from Streptomyces lividans.
3 mutant genes were expressed in Streptomyces lividans.
4 verexpression of their genes in Streptomyces lividans.
5 le in the chromosome of S. coelicolor and S. lividans.
6 ts was recently identified from Streptomyces lividans.
7 f neomycin resistance in both E. coli and S. lividans.
8 ted mitomycins when cloned into Streptomyces lividans.
9 hromosomal and plasmid genes in Streptomyces lividans.
10 us expression in the model host Streptomyces lividans.
11 and expressed heterologously in Streptomyces lividans.
12 n Streptomyces albus but not in Streptomyces lividans.
13 heterologous hosts, such as S. albus and S. lividans.
14 mes and ribosomal subunits from Streptomyces lividans.
15 e of a K(+) channel (KcsA) from Streptomyces lividans.
16 uced expression from the tylS promoter in S. lividans.
17 h, TpgR proteins for pSLA2 maintenance in S. lividans.
18 fusca into Escherichia coli and Streptomyces lividans.
19 xpressed in Escherichia coli or Streptomyces lividans.
20 l K(+) channel: i.e., KcsA from Streptomyces lividans.
23 he pentalenolactone nonproducer Streptomyces lividans 1326 resulted in production of pentalenic acid.
26 973 that allows the nonpathogen Streptomyces lividans 66 TK24 to necrotize and colonize potato tuber
28 shown by Southern blotting that Streptomyces lividans, a close relative of S. coelicolor and naturall
29 ter was cloned and expressed in Streptomyces lividans, a genetically well developed heterologous host
30 in Streptomyces avermitilis or Streptomyces lividans allows for production of significant amounts of
31 anslational start codon in both Streptomyces lividans and E. coli; cells expressing the unleadered vp
32 athways that regulate ACT biosynthesis in S. lividans and further demonstrate that the production of
33 were each expressed in and purified from S. lividans and had very low catalytic activity on swollen
34 We report that the Streptomyces species S. lividans and S. coelicolor, morphologically complex gram
39 sely related bacterial species, Streptomyces lividans and Streptomyces coelicolor, it normally is exp
40 avendulae) were introduced into Streptomyces lividans and transformants were selected for resistance
41 teria (Mycobacterium smegmatis, Streptomyces lividans, and Rhodococcus jostii) each exhibited a diffe
42 ad organic-acid metabolic capabilities of S. lividans, and sheds light into the control of the activi
43 L-arabinofuranosidase gene from Streptomyces lividans are readily identified by visual inspection on
44 s, and the potassium channel of Streptomyces lividans) are studied to address the role of glycine in
45 K(+) channel from the bacteria Streptomyces lividans) as a surrogate because it lacks a VSD and exhi
47 pression of AMO was achieved in Streptomyces lividans by cloning the AMO genes into the thiostrepton-
48 urprising that bacteria such as Streptomyces lividans can activate many of the available organic acid
49 acterial K(+) channel KcsA from Streptomyces lividans can be used to help elucidate questions about c
50 ctors in which doxA was poorly expressed, S. lividans catalyzed the reduction of daunomycin and other
54 on in the absence of glucose increased as S. lividans cells entered stationary phase, but unlike ACT
55 ar form and also prevented propagation of S. lividans cells that contain linear, but not circular, ch
56 ackground of AMO activity was detected in S. lividans cells without amoABCD and expression of AMO act
57 evelopment of pIJ101-containing Streptomyces lividans cells, with the concentration of KilB increasin
58 nsition to a circular form, we isolated a S. lividans chromosomal gene (tpgL) that we found specifies
59 rtion of the kilB gene of pIJ101 into the S. lividans chromosome in cells lacking the pIJ101 KorB pro
61 of the spore-forming bacterium Streptomyces lividans contains a regulatory gene, korB, whose product
62 ve expression of fdmR1; FDM production in S. lividans could be enhanced further by overexpressing fdm
63 n (CsoR) has been identified in Streptomyces lividans (CsoR(Sl)) and found to regulate copper homeost
64 turally characterized CsoR from Streptomyces lividans (CsoR(Sl)) together with three specific operato
65 eletion of csoR has only minor effects on S. lividans development when grown under high copper concen
68 erologous expression of fkbM in Streptomyces lividans established that fkbM encodes an O-methyltransf
71 initiate transcription correctly from the S. lividans galP1 and galP2 promoters, and the Bacillus sub
72 that the production of this antibiotic in S. lividans grown on agar can be modulated by carbon source
74 Since introduction of drrC into Streptomyces lividans imparted a DNR resistance phenotype, this gene
75 was able to confer the Pgl+ phenotype to S. lividans implying that these four genes constitute the w
76 the tetrameric K+ channel from Streptomyces lividans in a lipid bilayer environment was studied by p
77 fsR and afsR2, activate ACT production in S. lividans, indicating that this streptomycete encodes a f
79 t the intermycelial transfer of pIJ101 in S. lividans is complete by the onset of cellular differenti
80 hat the occurrence of ACT biosynthesis in S. lividans is determined conditionally by the carbon sourc
81 pglYZ and that introduction of pglYZ into S. lividans is not sufficient to confer a Pgl+ phenotype.
82 rt the crystal structure of the Streptomyces lividans K(+) channel KcsA in its open-inactivated confo
83 vity filter is identical to the Streptomyces lividans K(+) channel within error of measurement (r.m.s
84 ology with the structure of the Streptomyces lividans K+ channel KcsA, suggested the existence of an
86 licheamicin cognate PKSE-TEs in Streptomyces lividans K4-114; and (iii) selected native producers of
89 row pore of the K+ channel from Streptomyces lividans (KcsA), suggesting that K+ ions might literally
91 ssion of the megalomicin PKS in Streptomyces lividans led to production of 6-deoxyerythronolide B, th
92 in with tra mRNA concentration during the S. lividans life cycle indicated that the disappearance of
95 her with the time of appearance of Tra in S. lividans membranes, indicate that the intermycelial tran
97 that rescue of such plasmids in Streptomyces lividans occurs by three distinct types of events: (i) r
101 ive sequence similarity between Streptomyces lividans plasmid pIJ101 and Streptomyces plasmid pSB24.2
103 distorted conformations of the Streptomyces lividans potassium channel (KcsA), corresponding to a ra
105 amily transcription factor from Streptomyces lividans, promotes antibiotic resistance by sequestering
106 iosynthesis genes encABCDLMN in Streptomyces lividans resulted in the formation of the rearranged met
107 Expression of these genes in Streptomyces lividans resulted in the production of ala(0)-actagardin
108 of these vector sets were introduced into S. lividans, resulting in strains producing a wide range of
109 simocyclinone D8 resistance on Streptomyces lividans, showing that simX encodes a simocyclinone effl
112 knockout were employed in the engineered S. lividans strain to identify the P450 monooxygenase GetJ
113 olor, Streptomyces avermitilis, Streptomyces lividans, Streptomyces tsukubaensis, Streptomyces venezu
114 trepton-induced protein TipA of Streptomyces lividans strongly suggest that Mta is an autogenously co
115 ced prodiginine production from Streptomyces lividans, suggesting differential regulation of pks gene
116 J101) is expressed as a 10-kDa protein in S. lividans that is immediately processed to a mature 6-kDa
117 ngineered gene was expressed in Streptomyces lividans, the strain produced 6-deoxyerythronolide B and
118 r-probe analysis carried out in Streptomyces lividans, the TylP protein powerfully inhibited reporter
119 its heterologous expression in Streptomyces lividans TK24 and Streptomyces venezuelae ATCC 10712, an
124 ient and necessary to confer on Streptomyces lividans TK24 the ability to convert rhodomycin D, the f
125 ransfer of the four clusters to Streptomyces lividans TK24, expression of one cluster from each organ
130 lasmid pSB24.1 is deleted upon entry into S. lividans to form pSB24.2, a nonconjugative derivative th
131 from Streptomyces coelicolor or Streptomyces lividans to Mycobacterium smegmatis mc2155 in plate cros
132 al activator in the response of Streptomyces lividans to the peptide antibiotic thiostrepton, and les
134 as purified to homogeneity from Streptomyces lividans transformed with a plasmid containing the Strep
136 his gene, named herein doxA, in Streptomyces lividans TY24 resulted in in vivo bioconversion of dauno
139 ant AMO activity in cell-free extracts of S. lividans was stimulated by the addition of NADH and prod
140 ng heterologous biosynthesis in Streptomyces lividans, we also determined that biosynthesis of the SF
141 ously, in engineered strains of Streptomyces lividans, we showed that TylP, a deduced gamma-butyrolac
142 itiation factors (IF1, IF2, and IF3) from S. lividans were isolated and included in toeprint and filt
143 neighboring PepN homologs from Streptomyces lividans were purified in E. coli but displayed ca.100-f
144 tached, mutant K+ channels from Streptomyces lividans were used to screen venom from Leiurus quinques
145 ts of KcsA, a K(+) channel from Streptomyces lividans, were expressed in Escherichia coli, and inner
146 in throughout cellular differentiation of S. lividans, which leads to maximum KilB concentrations dur
147 membrane fractions of both surface-grown S. lividans, which mate readily, and of cells grown in liqu
148 bacteria, Bacillus subtilis and Streptomyces lividans, while capable of specifically interacting with
149 ma pretiosum was coexpressed in Streptomyces lividans with the genes encoding the 6-deoxyerythronolid
150 e we identify SlPatA, a GNAT in Streptomyces lividans with unique domain organization, and a new acet
152 probe a genomic DNA library of Streptomyces lividans ZX7, whose linear chromosome can undergo transi