ライフサイエンスコーパス: lobule

1 ich facilitates migration within the hepatic lobule.

2 obular region and finally replace the entire lobule.

3 ting at a size much smaller than a choroidal lobule.

4 rior hippocampus and right inferior parietal lobule.

5 from activity in the left inferior parietal lobule.

6 ls adjacent to the central vein in the liver lobule.

7 cant decrease in the right inferior parietal lobule.

8 uscular fatty structures typical of the alar lobule.

9 sion of non-melanoma skin cancer on the alar lobule.

10 tion (rTMS) applied to the inferior parietal lobule.

11 s and intraparietal sulcus/inferior parietal lobule.

12 rrelations observed in the inferior parietal lobule.

13 with the identity of the injected cerebellar lobule.

14 frontal and parietal lobes, and paracentral lobule.

15 eral prefrontal cortex and inferior parietal lobule.

16 rea and bilaterally in the inferior parietal lobule.

17 erior parietal sulcus, and superior parietal lobule.

18 or temporal gyri, and left superior parietal lobule.

19 emporal gyrus and the left inferior parietal lobule.

20 onsidered to be broadly dispersed across the lobule.

21 ed by the oxygen gradient present across the lobule.

22 had only minor effects on stellate cells in lobules.

23 al decrease in the dimensions of the hepatic lobules.

24 signal generation in normal mouse pancreatic lobules.

25 occipital lobe and of the superior parietal lobules.

26 Fibrous septae separated adipocyte lobules.

27 ent forms of sensory input across cerebellar lobules.

28 of hexagonal prisms represented the hepatic lobules.

29 basement membrane, primarily in the anterior lobules.

30 ally used for functional restoration of alar lobules.

31 erior lobules and vestibular input in caudal lobules.

32 asymmetrical inferior and superior parietal lobules.

33 atures of APAP hepatotoxicity within hepatic lobules.

34 r Ca(2+) waves that propagated across entire lobules.

35 gyrus, the putamen and the superior parietal lobules.

36 re widespread throughout multiple cerebellar lobules.

37 ted by Cav3-Kv4 interactions proves to allow lobule 2 granule cells to respond more effectively to ta

38 nse of granule cells to mossy fiber input in lobules 2 and 9 of the rat cerebellum.

39 acute perturbation of the cerebellar vermis (lobule 4/5) or simplex produced reliable and specific ef

40 ely to tactile stimulus-like burst input and lobule 9 cells to slow shifts in input frequency charact

41 pressed at a substantially higher density in lobule 9 cells, acting to increase A-type current availa

42 ta-burst stimulation of mossy fiber input in lobule 9 granule cells lowered the current threshold to

43 or surgical reconstruction of the nasal alar lobule after two-layer excision of non-melanoma skin can

44 otor cortex (M1), anterior inferior parietal lobule (aIPL)-M1, and dorsal inferior parietal lobule (d

45 representation in the left inferior parietal lobule and a significant decrease in the right inferior

46 The paracentral lobule and cuneus had the highest resting metabolic stat

47 lcium waves that spread throughout the liver lobule and elicited a synergistic increase in hepatic gl

48 d DR expanded as biliary epithelium into the lobule and established new junctions with the canaliculi

49 ral vein or distributed throughout the liver lobule and exhibiting active WNT signaling or high telom

50 creased diffusivity in the superior parietal lobule and increased diffusivity in the hippocampus.

51 hat was localized to the right intraparietal lobule and left Brodmann area 9 (BA9).

52 ignal in the left anterior inferior parietal lobule and posterior inferior temporal gyrus and sulcus

53 e the left insula, cuneus, inferior parietal lobule and prefrontal regions.

54 activity patterns in right superior parietal lobule and premotor cortex, and also left frontopolar co

55 m to start at the periphery of the pulmonary lobule and progressively extend toward the core of this

56 actional anisotropy in the superior parietal lobule and reduced mean diffusivity in the thalamus in t

57 fied decreased MTR in left inferior parietal lobule and right superior parietal lobule in suicide att

58 x differences in the right inferior parietal lobule and superior marginal gyrus, and displayed revers

59 nstruction of the 3D architecture of a liver lobule and the development of an experimental model of t

60 cortex and both the right superior parietal lobule and the left lateral occipital cortex) included t

61 ight postcentral gyrus, the left paracentral lobule and the precentral gyrus antidepressant dose-asso

62 irror neurons (MNs) in the inferior parietal lobule and ventral premotor cortex (PMv) can code the in

63 nal connectivity with left inferior parietal lobule and ventral premotor cortex, indicating that each

64 pmental volume trajectories of 10 cerebellar lobule and vermis tissue constituents in 548 no/low drin

65 cated in fibrotic septa between the exocrine lobules and adjacent to the ductal system of the pancrea

66 onnected to prefrontal-projecting cerebellar lobules and anterior prefrontal cortex, forming circuits

67 TFF3 is expressed in normal breast lobules and ducts, at higher levels in areas of fibrocys

68 xamined were identified throughout the liver lobules and in portal tracts, although portal tracts wer

69 ry differences from normal involved anterior lobules and vermis of youths who initiated substantial d

70 accelerated gray matter decline in anterior lobules and vermis, accelerated vermian white matter exp

71 ile receptor afferents prevalent in anterior lobules and vestibular input in caudal lobules.

72 perimental models: ex vivo (mouse pancreatic lobules) and in vitro (human cells).

73 ri, middle occipital lobe, inferior parietal lobule, and also cingulate, paracentral, and precentral

74 lateral prefrontal cortex, inferior parietal lobule, and cerebellum.

75 onal connectivity with the inferior parietal lobule, and children with ASD showed atypical functional

76 superior occipital gyrus, superior parietal lobule, and dorsal premotor area) was relevant for monit

77 eft middle temporal gyrus, inferior parietal lobule, and inferior frontal gyrus as videos were rated

78 transverse temporal gyrus, superior parietal lobule, and paracentral, lateral orbitofrontal, and late

79 frontal cortex, posterior inferior parietal lobule, and parahippocampus.

80 tion sensitive area MT/V5, superior parietal lobule, and primary visual cortex, while showing decreas

81 group (left temporal pole, inferior parietal lobule, and superior temporal gyrus) corresponded to reg

82 of the prefrontal cortex, inferior parietal lobule, and temporoparietal junction, as well as the ins

83 the cerebellum, area Spt, inferior parietal lobule, and the anterior cingulate cortex.

84 icularly the right and left lateral parietal lobule, and the Language Network, including the left inf

85 aparietal sulcus, anterior superior parietal lobule, and the ventral object-specific lateral occipita

86 ., middle occipital gyrus, inferior parietal lobule, and ventral premotor area) was specifically invo

87 ght frontal pole, the right lateral parietal lobules, and the left posterior cingulate cortex.

88 de network, precuneus, and inferior parietal lobule; and, within the dorsal attention network, intrap

89 er metabolic values in the inferior parietal lobule, anterior cingulate, inferior temporal lobule, th

90 motor cortex face area and inferior parietal lobule are both implicated in the cortical mirror-neuron

91 distinct anatomic locations within the liver lobule are replenished under homeostasis and injury-indu

92 structed by enlarged hepatic lobules; no new lobules are formed during this process.

93 In macaques, superior parietal lobule area 5 has been described as occupying an extensi

94 onded to activation in the inferior parietal lobule, as well as to activation around the inferior fro

95 ble selection approach, was used to identify lobules associated with motor function, language, execut

96 or area, premotor area and superior parietal lobule, based on the anatomic location of the hand-motor

97 ast map included bilateral superior parietal lobule, bilateral dorsolateral prefrontal cortex (DLPFC)

98 In liver lobules, blood flows from portal triads that are situate

99 duced, or metabolic) in which DR invaded the lobule but not in biliary diseases (obstruction or chola

100 genitors destined to form the posterior-most lobule causes this unique phenotype.

101 eased to 10% (p < 0.001), at which point the lobule ceased to expand further and was counterbalanced

102 representation within the inferior parietal lobule changes, with a decrease of the ipsilateral hemif

103 ross the functional syncytium of the hepatic lobule, co-ordinating and amplifying the metabolic respo

104 erior intraparietal sulcus/superior parietal lobule (consistent with sensorimotor output).

105 pically of repeating units of various sizes (lobules) consisting of CD34-postive, GLUT-1-negative end

106 ounterbalanced by the number of contributing lobules containing microspheres that increases as r(2).

107 that activity in the human inferior parietal lobule correlates with the divergence of such outcome di

108 eral prefrontal cortex and superior parietal lobule, corresponded to the decision variables resulting

109 e found significant activation of cerebellar lobules Crus I and VI bilaterally related to the CS+ com

110 Most importantly, significant activation of lobules Crus I and VI was also present during the unexpe

111 Breast ducts and lobules demonstrated increased decorin in the extracellu

112 nsory-related information to the cortex in a lobule-dependent manner.

113 tanding of the mechanisms by which ducts and lobules develop is derived from model organisms and thre

114 Other cerebellar lobules did not relate to temporal variability.

115 bule (aIPL)-M1, and dorsal inferior parietal lobule (dIPL)-M1 before and after inducing a long term d

116 bility gave for the less irradiated tissue a lobule dose distribution centered around 103 Gy (full wi

117 can replace all hepatocytes along the liver lobule during homeostatic renewal.

118 ated BOLD responses in the superior parietal lobule during WM encoding, and the bilateral hippocampus

119 GM volume decrease in posterior lobules (especially vermis VI) was associated with reduc

120 Atrophy of specific cerebellar lobules explains different aspects of motor and cognitiv

121 us, cuneus, precuneus, and superior parietal lobule [F=19.04-28.51, df=1, 189, partial eta squared=0.

122 03), and a decrease in NAc-inferior parietal lobule FC relative to controls (p < 0.001).

123 tomic force microscopy analysis across liver lobules from normal and fibrotic livers.

124 thickness in the pars orbitalis, paracentral lobule, fusiform gyrus and inferior temporal gyrus was l

125 ending on hepatocyte position in the hepatic lobule, gene expression and metabolism are differently a

126 from 103 down to 50 Gy, and about 17% of the lobules got a dose lower than 40 Gy to their different s

127 ggest vulnerability of the superior parietal lobule, hippocampus, and thalamus to glycemic extremes d

128 ipsilateral cerebellar cortex in cerebellar lobule HVI and in lobule I.

129 ociated with the anterior lobe and posterior lobule HVI.

130 ellar cortex in cerebellar lobule HVI and in lobule I.

131 S, FTD and PSP (Crus I/II), and MSA and PSP (lobules I-IV), respectively.

132 mulation over right medial superior parietal lobule impaired target discrimination after a shift of a

133 to establish the functional significance of lobules implicated in cognitive and motor functions in n

134 gular gyrus, precuneus and superior parietal lobule in carriers compared to non-carriers, with trend-

135 le frontal gyrus and right inferior parietal lobule in ECN, as well as increased RSFC between the rig

136 parietal lobule and right superior parietal lobule in suicide attempters relative to both non-attemp

137 iddle frontal gyrus to the superior parietal lobule in the right hemisphere in healthy controls, at-r

138 ry temporal region and the inferior parietal lobule in the right hemisphere.

139 g of clinical dysfunctions to the cerebellar lobules in disease populations is necessary to establish

140 mparison) and between PwMS and PwCIS for all lobules in subregions VI and left crus I (p<0.05).

141 ructural alterations in individual posterior lobules, in which cognitive functioning seems prepondera

142 in both the left and right inferior parietal lobule, including the supramarginal and angular gyri.

143 y somatosensory cortex and superior parietal lobule influences brain networks associated with tactual

144 erior intraparietal sulcus/inferior parietal lobule interfered with perceptual conflict processing.

145 paradigm, we show that the inferior parietal lobule (IPL) (corresponding to the supramarginal gyrus)

146 emporal network; bilateral inferior parietal lobule (IPL) activity was larger in HC versus SZ and HC

147 he contribution of rostral inferior parietal lobule (IPL) regions, in particular PFt, and the parieta

148 premotor cortex (PMd), and inferior parietal lobule (IPL) were modulated by prior belief on unexpecte

149 ior temporal gyrus (pSTG), inferior parietal lobule (IPL), and ventral central sulcus (vCS) that were

150 tronger signal in the left inferior parietal lobule (IPL), bilateral precuneus (PCN), bilateral premo

151 posterior cingulate gyrus, inferior parietal lobule (IPL), postcentral gyrus) areas.

152 premotor cortex (PMv) and inferior parietal lobule (IPL), presumably consisting of motor-related are

153 was possible in the right inferior parietal lobule (IPL).

154 cortex (PCC) via the right inferior parietal lobule (IPL).

155 ocial and moral cognition (inferior parietal lobule [IPL], prefrontal cortex [PFC], and cingulate), a

156 a specific sector of left inferior parietal lobule is devoted to tool use in humans, but not in monk

157 signal processing across multiple cerebellar lobules is controlled differentially by postsynaptic ion

158 the shape of the liver elemental systems-the lobules-is discovered, while their permeability is also

159 s) and SCA (contraction of total cerebellar, lobule IV, and Crus I volumes with additional X- or Y-ch

160 ults demonstrate that simple spike firing in lobules IV-VI is significantly correlated with position,

161 pyridine-3-ol hydrochloride, into cerebellar lobules IV-VI, in vivo, significantly reduced their alco

162 acingulate gyri (ACG/ApCG), left cerebellum (lobules IV/V and VIII), bilateral superior frontal gyrus

163 ule, the dentate nucleus, and the cerebellar lobules IV/V, VI, and VIII bilaterally corresponding to

164 in the vestibular part of the caudal vermis (lobules IX and X) to understand their role in this compu

165 in the right insula, left inferior parietal lobule, left dorsolateral prefrontal cortex/superior fro

166 igured periportal-to-CV gradients to exhibit lobule-location dependent behaviors.

167 permatids are released into the seminiferous lobule lumen (SLL), where they develop into spermatozoa

168 on of hepatocytes distributed throughout the lobule maintains the hepatocyte mass and that most hepat

169 erior intraparietal sulcus/inferior parietal lobule may resolve perceptual conflicts selectively when

170 ietal sulcus and bilateral superior parietal lobule, met our criteria for transsaccadic orientation i

171 ady visualization of features such as ducts, lobules, microcysts, blood vessels, and arterioles and t

172 gular nuclear shapes characterized by blebs, lobules, micronuclei, or invaginations are hallmarks of

173 s, while the 3D matrix with a modified liver lobule microstructure allows toxins to be trapped effici

174 tream, specifically in the inferior parietal lobule, middle frontal gyrus, and dorsal parts of the in

175 mic activity in the medial superior parietal lobule (mSPL), previously implicated in voluntary (as op

176 between locations [medial superior parietal lobule (mSPL)].

177 eactive metabolite) formation within hepatic lobules (NAPQI zonation) are necessary and sufficient pr

178 ted liver is constructed by enlarged hepatic lobules; no new lobules are formed during this process.

179 membrane of central hepatocytes in the liver lobule of control mice.

180 presence of rare Thy1(+) cells in the liver lobule of normal animals, occasionally forming small col

181 parse cerebrocerebellar projections to every lobule of the cerebellum.

182 terminals were significantly larger in both lobules of CB (1) -KO with no changes in PC dendritic sp

183 transfection of CFs in the caudal cerebellar lobules of rats.

184 Histological features included lobules of small vessels within the dermis, resembling a

185 ging picture in which increasingly posterior lobules of the anterior cerebellar cortex are associated

186 Gray matter atrophy of the superior lobules of the cerebellum (IV, V, VI), and lobules VIII

187 ways, and small vessels within the secondary lobules of the lung.

188 ly conserved and represented across multiple lobules of the rodent vermis.

189 nfiltrative growth that replaced the hepatic lobule or histologic nodular growth in the portal triad

190 gregation of mossy fibers across 10 distinct lobules over the rostrocaudal axis, with tactile recepto

191 a decrease in the number of hepatocytes per lobule (p = 0.029).

192 ts suggest a significant impact of posterior lobules pathology in corticocerebellar loop disruption r

193 Hepatocyte area (HA) and lobule radius (LR) were also measured.

194 ey system in the precuneus/superior parietal lobule region with reduced functional connectivity, whic

195 cantly reduced MTR in left inferior parietal lobule relative to controls, as well as an MTR reduction

196 rospinal fluid volumes expansion of anterior lobules relative to youths who remained no/low drinkers.

197 tilage grafts allow safe and functional alar lobule restoration.

198 mentary motor area (SMA), bilateral parietal lobule, right hippocampus, bilateral middle frontal gyru

199 nterior hippocampus, right inferior parietal lobule, right posterior cingulate cortex, and right vent

200 es, temporoparietal junction and paracentral lobule, right superior temporal and parietal lobes, temp

201 mice is that necrosis begins adjacent to the lobule's central vein (CV) and progresses outward.

202 lyses with hippocampal and inferior parietal lobule seed regions and the rest of the brain also revea

203 olded along the anterior-posterior axis into lobules separated by fissures, allowing the large number

204 active during lick-related movement across a lobule-specific region of the cerebellum showing high te

205 Superior and inferior parietal lobule (SPL and IPL) possessed both types of structure.

206 etal sulcus (IPS) and left superior parietal lobule (SPL) differing in time and sign for recognized o

207 d the critical role of the superior parietal lobule (SPL) in shifting spatial attention, a finding no

208 frontal eye fields (FEF), superior parietal lobule (SPL), and right supramarginal gyri (SMG).

209 Atypical RCrusI-inferior parietal lobule structural connectivity was also evident in the P

210 e of BSEP from zone 1 to zone 3 of the liver lobule, suggesting that the mutation identified here may

211 fied a region in the right superior parietal lobule that responded to both types of visuomotor load a

212 lum, a structural separation emerges between lobules that are functionally connected to distinct, mai

213 cerebellum consists of an intricate array of lobules that arises during the process of foliation.

214 of the PF-PC synapses located in cerebellar lobules that differ in vulnerability to damage and motor

215 is region specific: it is most prominent in lobules that regulate eye movement and process vestibula

216 obule, anterior cingulate, inferior temporal lobule, the dentate nucleus, and the cerebellar lobules

217 ferior parietal sulcus and superior parietal lobule, the frontal eye-movement field, and the inferior

218 ntral and postcentral gyrus, the paracentral lobule, the superior temporal gyrus, the middle cingulat

219 This enables the liver lobules to respond as functional units to produce full-s

220 dermal and mesodermal origins in a hexagonal lobule unit.

221 ed this in the vermis of the spinocerebellar lobule V and the vestibulocerebellar lobule X of CB (1)

222 ynaptic vesicles close to the active zone in lobule V and X of CB (1) -KO was observed.

223 There were as many vesicles in lobule V of CB (1) -KO as in CB (1) -WT, but their distr

224 The PF terminals in lobule V of CB (1) -KO contained less synaptic vesicles

225 nd that the right cerebellar vermis and left lobule V of cerebellar anterior lobe were additionally a

226 Lobule V, but not lobule X, of CB (1) -KO had significan

227 cerebellar peduncles, cerebellar vermis and lobules V and VI, and corpus callosum.

228 inkers indicated that gray matter volumes of lobules V and VI, crus II, lobule VIIB, and lobule X dec

229 tate, the ipsilateral motor representations (lobules V/VI), and most interestingly the contralateral

230 PwMS and HS for the right (p<0.001) and left lobule VI (p<0.01), left crus I, right VIIb and entire c

231 ) = 0.3) and reduced GM volume in cerebellar lobule VI (R (2) = 0.35).

232 audal dorsal premotor cortex, and cerebellar lobule VI (t >/= 4.18, whole-brain familywise error corr

233 f SCA6 with greater loss of GM in cerebellum lobule VI exhibit temporal invariance and more severe at

234 invariance related to exacerbated atrophy of lobule VI of the cerebellum and exacerbated disease seve

235 that the anterior cerebellum, extending into lobule VI, and parts of lobule VIII were smaller than no

236 were most pronounced in, but not limited to, lobules VI and interposed nuclei.

237 Right cerebellar lobules VI and VII (including Crus I/II) are engaged dur

238 By using extracellular recordings from lobules VI through X in awake mice, we show that silenci

239 ct of grey matter (GM) volume alterations in lobules VI to VIIIb on IPS in persons with clinically is

240 s, with cerebellar lesion volume, cerebellar Lobules VI, Crus I and VIIIa atrophy being independent p

241 theta burst stimulation (iTBS) to the vermis lobule VII or right lateral cerebellar Crus I/II, subreg

242 The vermis of lobule VII receives a prominent input from the retrosple

243 erebral default mode network, whereas vermal lobule VII stimulation influences the cerebral dorsal at

244 ngly the contralateral posterior cerebellum (lobule VII) impede the benefits of motor execution on pe

245 on in ventral cerebellum, in the vicinity of lobules VII/VIII.

246 ctional connections between right cerebellar lobule VIIb and the posterior parietal cortex.

247 analysis revealed that the medial portion of lobule VIIb and to a lesser degree the lateral most port

248 e findings suggest that the right cerebellar lobule VIIb interacts with the posterior parietal cortex

249 matter volumes of lobules V and VI, crus II, lobule VIIB, and lobule X declined faster with age in ma

250 ated neural signal from the right cerebellar lobule VIIb, specifically during the late encoding phase

251 me in the occipital lobe and left cerebellar lobule VIIb, which is functionally connected with prefro

252 e present study demonstrates that cerebellar lobule VIIb/VIIIa activity patterns are selective for re

253 Cerebellar lobule VIIb/VIIIa delay-period activation accurately dec

254 red stimulus, while intermediate portions of lobule VIIb/VIIIa did not.

255 fMRI research has revealed that cerebellar lobule VIIb/VIIIa exhibits load-dependent activity that

256 gs of stimulus-specific coding of VWM within lobule VIIb/VIIIa indicate for the first time that the d

257 attention tasks robustly recruit cerebellar lobules VIIb and VIIIa, in addition to canonical cortica

258 a lesser degree the lateral most portion of lobules VIIb and VIIIa, which exhibit robust resting sta

259 tween task-responsive portions of cerebellar lobules VIIb/VIIIa and cortex.

260 nnectivity patterns revealed that cerebellar lobules VIIb/VIIIa group with cortical nodes of the dors

261 trates that a cerebellar subdivision (mainly lobules VIIb/VIIIa), which exhibits strong intrinsic fun

262 ork should be expanded to include cerebellar lobules VIIb/VIIIa.

263 rsal attention network to include cerebellar lobules VIIb/VIIIa.

264 minates as climbing fibres in lateral vermal lobule VIII (pyramis).

265 The C1 zone of lobule VIII receives a more prominent projection from th

266 llum, extending into lobule VI, and parts of lobule VIII were smaller than normal in children with au

267 r lobules of the cerebellum (IV, V, VI), and lobules VIII also correlated with worse posturometric va

268 ite matter volumes in crus I and crus II and lobules VIIIA and VIIIB expanded faster in female youths

269 of total cerebellum, flocculus, and Crus II-lobule VIIIB volumes in males) and SCA (contraction of t

270 romosomes; X-specific contraction of Crus II-lobule VIIIB).

271 Cerebellar lobule volumes were derived from a graph-cut based segme

272 nd the right IFG and right inferior parietal lobule was also significantly correlated with age of acq

273 In the liver, a structural model of the lobule was pioneered by Elias in 1949.

274 the postcentral gyrus and superior parietal lobule was sensitive to dot periodicity.

275 The presence of CD8+ cells in the lobules was associated with fibrosis.

276 ere present when different zones of the same lobule were injected.

277 odel of the testis was designed in which the lobules were approximated by a cross-section of seminife

278 The main cell types in the portal tracts and lobules were CD3+ and CD68+ cells.

279 atter displayed numerous aggregates, whereas lobules were mildly affected.

280 Cerebellum lobules were segmented using SUIT V.3.0 to estimate thei

281 e found bilaterally in the inferior parietal lobule when prisms, but not plain glasses, were used.

282 perfunctional islets are confined within few lobules, whereas hypofunctional islets are present throu

283 d to the adjacent acinar cells in pancreatic lobules; whereas taurolithocholic acid 3-sulfate primari

284 the presence of a partially formed posterior lobule which echoes the posterior vermis DW 'tail sign'

285 create an excitable medium across the liver lobule, which allows global propagation of calcium signa

286 s to form a progenitor pool in the posterior lobule, which is not seen in other organisms, not even i

287 generators within the left superior parietal lobule, which may reflect post-lexical activation of the

288 movement direction in the superior parietal lobule, which may underlie a transformation from the loc

289 It contains a large number of lobules, which in turn are composed of convoluted semini

290 tely around pericentral areas of the hepatic lobule, while there was no transgene expression in perip

291 or temporal gyrus, and the inferior parietal lobule, while those of patients with atypical language l

292 lobules V and VI, crus II, lobule VIIB, and lobule X declined faster with age in male youths than in

293 ebellar lobule V and the vestibulocerebellar lobule X of CB (1) -KO and wild-type (CB (1) -WT) mice b

294 cle density; by contrast, vesicle density in lobule X of CB (1) -KO remained unchangeable relative to

295 In lobule X of CB (1) -KO, less vesicles were found within

296 ellar vermis, inferior cerebellum (bilateral lobule X), and the right superior temporal sulcus.

297 Lobule V, but not lobule X, of CB (1) -KO had significantly less and longe

298 stimulus-driven responses of interneurons in lobules X (nodulus) and IXc,d (ventral uvula) of the cau

299 stimulus-driven responses of interneurons in lobules X (nodulus) and IXc,d (ventral uvula) of the mac

300 of the cerebellar nodulus and ventral uvula (lobules X and IXc,d of the vermis) for vestibular proces