ライフサイエンスコーパス: low-grade

1 elated adverse reactions were graded 1 or 2 (low-grade).

2 y were 3.0% and 4.6%.Most complications were low grade.

3 Toxicity was predominantly low grade.

4 ILA areas were classified as high or low grade.

5 ns], WHO III: 17, WHO IV: 13, without biopsy low-grade: 1, high-grade: 1) were investigated with a hy

6 orty-three patients with cerebral gliomas (9 low-grade, 34 high-grade; 9 primary tumors, 34 recurrent

7 However, the implications of surviving low-grade ACLF in terms of risk of subsequent high-grade

8 in infants with newly diagnosed SCID-X1 had low-grade acute toxic effects and resulted in multilinea

9 current porphyria attacks resulted in mainly low-grade adverse events, reductions in induced ALAS1 mR

10 These effects are frequently low grade and are treatable and reversible; however, som

11 from anaemia, toxicities with olaparib were low grade and manageable.

12 FC1, a new candidate susceptibility gene for low-grade and borderline serous EOC.

13 On the molecular level, low-grade and high-grade DCIS have different molecular a

14 able methodology for differentiating between low-grade and high-grade DCIS.

15 of primary and metastatic cancers, including low-grade and high-grade gliomas and brain metastases (B

16 ave limited value in distinction between the low-grade and high-grade intraepithelial melanocytic pro

17 Most toxicities were low-grade and likely related to the treatment procedure,

18 wing that DACH1 was higher in normal breast, low-grade and luminal-type cancer in comparison with bre

19 Adverse effects were mainly low-grade and transient.

20 Es with nivolumab monotherapy were primarily low grade, and most resolved with established safety gui

21 hree patients had a transient, asymptomatic, low-grade atrioventricular block that resolved spontaneo

22 tinal B-cell lymphoma (DLBCL/PMBCL; n = 28), low-grade B-cell lymphoma (n = 8), or chronic lymphocyti

23 Follicular lymphoma (FL) is a low-grade B-cell malignancy that transforms into a highl

24 PD patients experience low-grade bacteremias with oral microbes implicated in t

25 ificantly higher than those of patients with low-grade BC.

26 ion syndrome are prone to the development of low-grade brain tumors (gliomas) within the optic pathwa

27 ears) with residual or progressive benign or low-grade brain tumors at a single center between April

28 s with residual and/or progressive benign or low-grade brain tumors requiring radiotherapy for long-t

29 with neurofibromatosis type 1 (NF1) develop low-grade brain tumors throughout the optic pathway.

30 s with residual and/or progressive benign or low-grade brain tumors treated with SCRT and ConvRT tech

31 nd a three-class classification into normal, low grade cancer, and high grade cancer.

32 d classification correctly graded 76% of the low-grade cancers and 71% of the high-grade cancers acco

33 Here, we present a mouse model of low-grade candidemia to determine the effect of dissemin

34 , including germ cell tumors, high-grade and low-grade carcinomas and benign tissues.

35 could be associated with the development of low-grade carcinomas.

36 Low grade cartilage degeneration, predominantly loss of

37 l microRNA profiles, immunosenescence, and a low-grade chronic inflammation (inflammaging).

38 Disturbances in glucose homeostasis and low-grade chronic inflammation culminate into metabolic

39 Obesity is associated with low-grade chronic inflammation promoting insulin-resista

40 etic foot ulcers (DFUs) are characterized by low-grade chronic inflammation, both locally and systemi

41 e-associated secretory phenotype, leading to low-grade chronic inflammation, which further drives sen

42 immune cell composition and likely promotes low-grade chronic inflammation, which has been proposed

43 Osteoarthritis (OA) is a low-grade chronic inflammatory joint disease.

44 ersisted for at least 5 months, suggesting a low-grade chronic inflammatory state.

45 Low-grade, chronic inflammation has been associated with

46 Prespecified outcome measures included low-grade CIN (grade 1 [CIN1]) and high-grade CIN (grade

47 rder of the gastrointestinal tract is a rare low-grade clonal lymphoid proliferation, included as a p

48 Controllers (n = 16): spontaneously resolved low-grade CMV DNAemia without antiviral therapy; and (3)

49 Pathologic diagnoses were low-grade conjunctival melanocytic intraepithelial lesio

50 in unexpected genes; permitted detection of low-grade constitutional, somatic, and revertant mosaici

51 ed ultrasound was judged semiquantitatively; low-grade contrast enhancement (CE) suggested its absenc

52 h the prevalence round; conversely, rates of low-grade DCIS and, less markedly, intermediate-grade DC

53 us contrast enhancement of vertebral bodies, low-grade destruction of vertebral bodies, hyperintense/

54 disease may have a recurrence in the form of low-grade disease after immunochemotherapy, and those wi

55 logic grade and underlying pathobiology with low-grade disease hypothesized to be immune-dependent an

56 ase after immunochemotherapy, and those with low-grade disease may progress to high-grade disease aft

57 patients with T1N0 breast cancer was 10% for low-grade disease, 13% for moderate-grade disease, and 1

58 containing EAC, high grade dysplasia (HGD), low grade dysplasia (LGD), Barrett's esophagus (BE), col

59 00 person-years among patients with baseline low-grade dysplasia (95% CI 1.5-7.2), and 7.3 per 100 pe

60 nts with inflammatory bowel disease and flat low-grade dysplasia (fLGD) in the colon.

61 s with Barrett's esophagus, the diagnosis of low-grade dysplasia (LGD) is subjective, and reported ou

62 s) are common lesions that may progress from low-grade dysplasia (LGD) through high-grade dysplasia (

63 r for invasive progression but some cases of low-grade dysplasia also progressed to cancer.

64 ale sex, smoking, length of BE, and baseline low-grade dysplasia that identified patients with BE at

65 Low-grade dysplasia was a risk factor for progression bu

66 moking, length of BE, and baseline-confirmed low-grade dysplasia were significantly associated with p

67 GD or EAC included age, caffeine intake, and low-grade dysplasia while colonic adenomas trended towar

68 utcomes were occurrence of CRN (inclusive of low-grade dysplasia) and colectomy.

69 TICE ADVICE 1: In BE patients with confirmed low-grade dysplasia, a repeat examination with high-defi

70 ection of SSP without dysplasia, with any or low-grade dysplasia, and with high-grade dysplasia were

71 th a cascade of gastric lesions that include low-grade dysplasia, high-grade dysplasia, and adenocarc

72 was classified as strong while evidence for low-grade dysplasia, strictures, primary sclerosing chol

73 of BE patients with confirmed and persistent low-grade dysplasia.

74 idental hyperplastic polyps or adenomas with low-grade dysplasia.

75 was further able to differentiate tumor from low-grade dysplasia.

76 nually thereafter; and baseline diagnosis of low-grade dysplasia: at 1 and 3 years.

77 ugh a multistep carcinogenesis process, from low-grade dysplastic lesions to carcinoma in situ and ev

78 The upstream genetic causes of low-grade elevations in cTnI and cTnT appear distinct, a

79 isease recurrence in women with early-stage, low-grade endometrial carcinoma.

80 The diagnoses of low-grade epilepsy associated neuroepithelial tumour (LE

81 vents and quantify the burden of continuous, low-grade events.

82 and six EHRs (age, hemoglobin, weight loss, low-grade fever, calcification detected by computed tomo

83 d with the transformation and progression of low-grade fibrillary astrocytoma to high-grade anaplasti

84 We aimed to differentiate low-grade (Fuhrman I-II) from high-grade (Fuhrman III-IV

85 onferred high-grade maribavir resistance and low-grade ganciclovir resistance.

86 no dose-limiting toxicity was observed, but low-grade gastrointestinal toxicity was common.

87 ratio to distinguish between high-grade and low-grade glial tumors.

88 Here, we find that Nudt21 is reduced in low grade glioma (LGG) and all four subtypes of high gra

89 TCGA, Lung Adenocarcinoma (LUAD) dataset and Low Grade Glioma (LGG), and the model stratified the LUA

90 rformance for IDH gene mutation detection in low-grade glioma (AUC, 0.818) and MTI in high-grade glio

91 sion kurtosis imaging (DKI) to differentiate low-grade glioma (LGG) (World Health Organization [WHO]

92 orrelation between the increased survival in low-grade glioma (LGG) and complementarity of IDH1 mutan

93 interact to govern Neurofibromatosis-1 (NF1) low-grade glioma (LGG) growth.

94 ractice-changing study for patients with WHO low-grade glioma (LGG, grade II), as it was the first to

95 istological types of childhood brain cancer: low-grade glioma (n = 93), ependymoma (32), high-grade g

96 romatosis type 1 (NF1)-associated paediatric low-grade glioma (WHO grades I and II).

97 rrent, refractory, or progressive paediatric low-grade glioma after at least one standard therapy wer

98 in patients with newly diagnosed paediatric low-grade glioma both with and without NF1.

99 e, phase 2 study in patients with paediatric low-grade glioma in 11 hospitals in the USA.

100 Transformation of low-grade glioma into a higher tumor grade is typically

101 Paediatric low-grade glioma is the most common CNS tumour of childh

102 ved phenomena, such as a lack of symptoms in low-grade glioma patients versus a rapid onset of sympto

103 ally, when applied to an ex vivo sample of a low-grade glioma resection margin, SM-OCT is able to res

104 data from 335 adult patients with high- and low-grade glioma to form a replicable tumour frequency m

105 CMRO2 was decreased (P = .037) in low-grade glioma with a mutated IDH gene, and MTI was si

106 the most common childhood brain tumor, is a low-grade glioma with a single driver BRAF rearrangement

107 cluded (EL_INST: 17 patients, 17 lesions, 10 low-grade glioma, 3 cavernoma, 4 focal cortical dysplasi

108 ts with lung adenocarcinoma, bladder cancer, low-grade glioma, and thyroid carcinoma.

109 AF aberrations and NF1-associated paediatric low-grade glioma.

110 ates with significantly inferior survival in low-grade glioma.

111 Infiltrating low grade gliomas (LGGs) are heterogeneous in their beha

112 Paediatric low-grade gliomas (also known as pLGG) are the most comm

113 Diffuse low-grade gliomas (LGG) have been reclassified based on

114 These pediatric low-grade gliomas (LGGs) are fundamentally different fro

115 urprisingly given recent technical advances, low-grade gliomas (LGGs), which arise from the glia (the

116 For low-grade gliomas (N = 50), low observer confidence in t

117 Pediatric low-grade gliomas (pLGG) are frequently driven by geneti

118 Pediatric low-grade gliomas (PLGGs) are commonly associated with B

119 olecularly characterized cohort of pediatric low-grade gliomas (pLGGs) published to date.

120 70-90% of low-grade gliomas and secondary glioblastomas are charac

121 Low-grade gliomas cause significant neurological morbidi

122 ue available for detection of enlargement of low-grade gliomas in the clinical setting; subjective ev

123 children with NF1 are at risk for developing low-grade gliomas of the optic pathway and brainstem, in

124 In general, paediatric low-grade gliomas show clinical and biological features

125 is study presents a non-invasive analysis of low-grade gliomas using imaging features based on the up

126 Low-grade gliomas with favorable characteristics are slo

127 n, and TERT mutations) prediction methods of low-grade gliomas with imaging.

128 ogical features that are distinct from adult low-grade gliomas, and the developing paediatric brain i

129 th significant delays in detecting growth of low-grade gliomas.

130 od helps physicians detect earlier growth of low-grade gliomas.

131 , in bladder and renal carcinomas as well as low-grade gliomas.

132 ations for response assessment in paediatric low-grade gliomas.

133 e 1 and the use of Ras pathway inhibitors in low-grade gliomas.

134 option in patients diagnosed with high-risk low-grade gliomas.

135 Cardiac CT demonstrated low-grade HAT in 9 (3.6%) cases at 8 weeks; and 13 cases

136 Low-grade HAT resolved spontaneously over time.

137 ted with thromboembolism in 2 cases, whereas low-grade HAT was not related to embolic events.

138 addition, cardiac CT demonstrates cases with low-grade HAT, not visualized by TEE.

139 rget expressed in human control subjects and low-grade HD patients.

140 HE) is a promising technology for converting low grade heat to electricity.

141 d on these unique phase behaviors to convert low grade heat to work or electricity.

142 into electrical power, can harvest waste or low-grade heat in an economical and continuous approach

143 Efficient low-grade heat recovery can help to reduce greenhouse ga

144 rson-years were 1.09 (95% CI, 0.85-1.33) for low-grade/high-grade dysplasia and 0.14 (95% CI, 0.06-0.

145 a more favorable prognosis, and tumors with low-grade histology especially tend evolve slowly.

146 ffered a survival benefit across all grades (low-grade, HR 0.38, P = 0.002 and high-grade, HR 0.62, P

147 transmural-ileitis on day 7, microscopically low-grade ileitis on day 22 and VH at days 7-22.

148 int tissues from people with OA with high or low grade inflammation and non-arthritic post-mortem con

149 long-term changes in neuronal function, and low grade inflammation of the bowel have been hypothesiz

150 rrier functions play a major role in chronic low-grade inflammation (CLGI)-associated obesity, but th

151 effects on transcriptomic markers, decreased low-grade inflammation (e.g., as alpha(1)-acid glycoprot

152 eration, leading to microbiota encroachment, low-grade inflammation (LGI), and metabolic syndrome.

153 Obesity is associated with low-grade inflammation and elevated levels of circulatin

154 abetes are associated with increased chronic low-grade inflammation and elevated plasma glucose level

155 ich encompasses genetic alterations, chronic low-grade inflammation and gut dysbiosis, has led to imp

156 blood lipopolysaccharides and its initiated low-grade inflammation and increased oxidative phosphory

157 vel insight into the development of chronic, low-grade inflammation and oxidative stress in age-relat

158 besity is associated with a chronic state of low-grade inflammation and progressive tissue infiltrati

159 sity and type 2 diabetes are associated with low-grade inflammation and specific changes in gut micro

160 connectivity among brain networks related to low-grade inflammation and stress exposure using two lar

161 itive network system as neural correlates of low-grade inflammation and stress exposure, and suggest

162 ve networks were reduced in association with low-grade inflammation and stress exposure.

163 d cellular and molecular processes including low-grade inflammation are major players in the pathogen

164 in school-aged children suggesting systemic low-grade inflammation as a phenotypic characteristic of

165 Evidence suggests low-grade inflammation as the cause of metabolic syndrom

166 The bacteremia, endotoxemia, and systemic low-grade inflammation associate periodontitis with syst

167 While low-grade inflammation could impair immune response, it

168 etabolic disorder are accompanied by chronic low-grade inflammation has fundamentally changed our vie

169 ysis and HIF-1alpha activation as drivers of low-grade inflammation in obesity.

170 challenge the common assumption that central low-grade inflammation in schizophrenia is mirrored by i

171 sis may contribute to insulin resistance and low-grade inflammation in the mother, placenta, or fetus

172 Obesity fosters low-grade inflammation in white adipose tissue (WAT) tha

173 Obesity induces chronic low-grade inflammation in white adipose tissue (WAT).

174 Chronic, low-grade inflammation increases the risk for atheroscle

175 uggest that the metabolic demands of chronic low-grade inflammation induce a reduction of striatal DA

176 ancreatic islet inflammation, while systemic low-grade inflammation is a feature of obesity and type

177 Persisting low-grade inflammation is suggested to play a role in po

178 tors have hypothesized that chronic systemic low-grade inflammation may contribute to greater risk of

179 While low-grade inflammation may impair immune response, it is

180 Obesity is associated with chronic low-grade inflammation of adipose tissue (AT) and an inc

181 Thus, microglia and low-grade inflammation of myelinated tracts emerged as t

182 OME is defined as a chronic low-grade inflammation of the middle ear (ME), without a

183 Chronic, low-grade inflammation of VAT, and eventually systemical

184 Obesity is associated with chronic low-grade inflammation of visceral adipose tissue (AT) c

185 e underlying cause of catatonic signs is the low-grade inflammation of white matter tracts, which mar

186 The development of a chronic, low-grade inflammation originating from adipose tissue i

187 richt Study (n = 685), an increased systemic low-grade inflammation profile was specifically related

188 being excessive oxidative stress and chronic low-grade inflammation superimposed on the limited cardi

189 teristic of aging is the presence of chronic low-grade inflammation that is characterized by elevated

190 Low-grade inflammation was defined according to equally

191 Chronic low-grade inflammation, a hallmark of obesity, involves

192 mation and stress exposure, and suggest that low-grade inflammation, alongside with stress, may rende

193 isk of weight gain, cardiovascular diseases, low-grade inflammation, and cancer.

194 Obesity may represent a chronic low-grade inflammation, but there is a lack of long-term

195 g the relationship between oxidative stress, low-grade inflammation, carcinogenesis, obesity and phys

196 implication of processes related to chronic low-grade inflammation, including a network involving me

197 an atypical, metabolically induced, chronic low-grade inflammation, plays an important role in the d

198 the intestinal microbiota and induce chronic low-grade inflammation, ultimately leading to metabolic

199 A condition more common than IBD is low-grade inflammation, which correlates with altered gu

200 Aging is characterized by a chronic, low-grade inflammation, which is a major risk factor for

201 Low-grade inflammation, which is related to obesity and

202 rt failure and are characterized by chronic, low-grade inflammation, which promotes adverse cardiac r

203 plicated in diseases associated with chronic low-grade inflammation, yet homeostatic signaling mechan

204 rgeted in a personalized approach of chronic low-grade inflammation.

205 a-cells from proapoptotic UPR during chronic low-grade inflammation.

206 which was strongly associated with systemic low-grade inflammation.

207 racterized by insulin resistance and chronic low-grade inflammation.

208 oxygen species (ROS) production and chronic low-grade inflammation.

209 increase in intestinal permeability leads to low-grade inflammation.

210 bacterial LPS synthesis and host markers of low-grade inflammation; transcriptome databases identifi

211 Histologic analysis showed absent to low-grade inflammatory infiltrates without cardiomyocyte

212 ficient in exosome secretion have a chronic, low-grade inflammatory phenotype characterized by elevat

213 whether TSPO imaging can accurately capture low-grade inflammatory processes such as those present i

214 e injuries were more likely to progress than low-grade injuries (HR, 3.3; P = .005).

215 olve (hazard ratio [HR], 0.2; P < .001) than low-grade injuries.

216 Low-grade intestinal inflammation and alterations of gut

217 levels in patients with active FPIES suggest low-grade intestinal mast cell activation or increased m

218 inous neoplasms (IPMN), 2 adenocarcinomas, 1 low-grade intraepithelial pancreatic neoplasia, and 1 ca

219 The results suggest that low-grade intraocular inflammation may play an important

220 gher levels of phospho-MNK1 and NODAL versus low-grade (invasion-free) DCIS.

221 )F-PSMA-1007, however, may detect additional low-grade lesions of limited clinical relevance.

222 Low-grade lesions provided the greatest interpretative c

223 the histologic and molecular features of the low-grade lesions.

224 The overall evidence provides a low-grade level of the evidence supporting the efficacy

225 Whereas low-grade levels of plasma cytokine/chemokine were appar

226 epithelial lesions or malignancy (NILM), and low-grade (LSIL) and high-grade (HSIL) squamous intraepi

227 er Anogenital Squamous Terminology (LAST) in low-grade (LSIL) and high-grade squamous intraepithelial

228 of augmenting the immune response to EBV in low-grade LYG include treatment with interferon-alpha2b,

229 treated within phase 3 trials of the German Low-Grade Lymphoma Study Group, were comparatively analy

230 or DLBCL/PMBCL, 63% (95% CI, 25% to 92%) for low-grade lymphoma, and 50% (95% CI, 16% to 84%) for CLL

231 Low-grade lymphomas (EMZL and FL) were most commonly tre

232 a process typically thought to occur during low-grade metamorphism.

233 s (blueschists) from subduction zones and in low-grade metamorphosed mudstones (phyllites and schists

234 sions of Enterobacteriaceae that exacerbated low-grade mucosal inflammation, suggesting that remediat

235 polyposis of the gastrointestinal tract and low grade neuroendocrine tumor as part of the TSC syndro

236 m by which subtle myelin abnormalities cause low-grade neuroinflammation and catatonic behavior.

237 Approximately 50% of low-grade NF1-gliomas displayed an immune signature, T l

238 inary data seem to confirm their activity on low-grade NHL.

239 d that the adult mammalian heart undergoes a low grade of cardiomyocyte turnover.

240 T) on the device, which was subclassified as low grade or high grade.

241 sk groups were: low (non-metastatic R0 or R1 low-grade, or <=5 cm R1 high-grade tumour); intermediate

242 Low-grade persistent inflammation is a feature of diabet

243 the activation of resident immune cells, in low-grade pre-invasive lesions; (3) the activation of im

244 Patients with untreated low-volume low-grade prostate cancer (clinical stage T2a or lower;

245 inal cord glioblastoma (SC-GBM) patients and low grade SCG (L-SCG) patients.

246 Higher rates of low-grade screen-detected tumors were observed in the co

247 ntracellular autoactivation and constitutive low-grade secretion of activated FXII.

248 tissue from patients with high grade but not low grade serous ovarian cancer.

249 some less common ovarian cancer histotypes (low grade serous, mucinous, and clear cell carcinomas),

250 ed chemotherapy in women with stage II to IV low-grade serous carcinoma of the ovary or peritoneum.

251 Conclusion Women with stage II to IV low-grade serous carcinoma who received HMT after primar

252 Although the MEK Inhibitor in Low-Grade Serous Ovarian Cancer Study did not meet its p

253 Low-grade serous ovarian carcinomas (LGSC) are associate

254 Low-grade serous ovarian carcinomas (LGSOCs) have histor

255 d abnormal follicle structures and developed low-grade serous ovarian carcinomas with 100% penetrance

256 istically nonsignificantly increased risk of low-grade serous tumors (pOR = 1.48, 95% CI: 0.92, 2.38)

257 For participants with low-grade SIL (LSIL) at baseline, risk of progression to

258 For those with low-grade SILs (LSILs) at baseline, the risk of progress

259 were 51.9% (133/215) normal, 87.9% (20/232) low-grade SILs (LSILs), and 90.9% (149/164) high-grade S

260 lls of undetermined significance (ASC-US) or low-grade squamous intraepithelial lesions (LSIL) who we

261 We enrolled women with ICC, high- and low-grade squamous intraepithelial lesions, as well as,

262 but this proportion increased through ASCUS, low-grade squamous intraepithelial lesions, CIN1, and CI

263 Chronic low grade systemic inflammation is linked to impaired lu

264 milar to human sepsis, old mice demonstrated low-grade systemic inflammation 14 days after cecal liga

265 s, to autoinflammatory diseases, which cause low-grade systemic inflammation and contribute to severa

266 indicates a reciprocal relationship between low-grade systemic inflammation and stress exposure towa

267 Metaflammation refers to a chronic, low-grade systemic inflammation as opposed to the classi

268 Obese individuals with persistent low-grade systemic inflammation showed reduced leukocyte

269 Obesity is accompanied by low-grade systemic inflammation that etiologically contr

270 n glucose homeostasis and the development of low-grade systemic inflammation, which increase the risk

271 In addition, OSAS is considered as low-grade systemic inflammation, which is associated wit

272 Obesity is characterized by chronic low-grade, systemic inflammation, altered gut microbiota

273 Chronic, low-grade, systemic, and mucosal inflammation correlates

274 in the same grade category, i.e. the high or low grade, the survival stratification between races is

275 o design high efficient OHEs for recovery of low grade thermal energy to work or electricity.

276 nd ranges on the spectrum of malignancy from low grade to overtly malignant.

277 ival melanocytic intraepithelial lesion from low-grade to high-grade in 2 (4%) of 47 cases.

278 oangiogenic myeloid cells, and prevention of low-grade to high-grade transition.

279 rcatinib showed durable efficacy with mainly low-grade toxic effects in patients with medullary thyro

280 ancer, although it resulted in more frequent low-grade toxicities.

281 oma (n = 20), sarcoma (n = 20) and benign to low grade tumor (n = 6).

282 de tumor CMRO(2), 0.23 mumol/g/min +/- 0.07; low-grade tumor CMRO(2), 0.39 mumol/g/min +/- 0.16; over

283 report a detailed study of a musician with a low-grade tumor in the right temporal lobe.

284 Patient survival for benign to low grade tumors, sarcoma, and carcinoma was 100%/100%/1

285 Results Low-grade tumors (grade 0, 1, or 2) demonstrated a favor

286 tem, individuals with NF2 typically manifest low-grade tumors affecting the cranial nerves (vestibula

287 Low-grade tumors exhibited fewer mutations that were ove

288 or compared to those in children and adults: low-grade tumors have a higher mortality rate, while hig

289 ue) and high-grade tumors from other tissue (low-grade tumors or benign tissue).

290 ratio to discriminate between high-grade and low-grade tumors to be 2.21.

291 SAT1 target genes that distinguish high- and low-grade tumors, in support of the prognostic utility o

292 d) high- or intermediate-grade tumors versus low-grade tumors.

293 rs) with primary or recurrent biopsy-proven, low-grade upper tract urothelial cancer (measuring 5-15

294 Most patients with low-grade upper tract urothelial cancer are treated by r

295 Primary chemoablation of low-grade upper tract urothelial cancer with intracavita

296 ohistochemistry in a small cohort, including low-grade urothelial carcinoma samples.

297 ents in 60 patients were analyzed: 141 (89%) low grade versus 17 (11%) high grade.

298 f merit (or ZT), which can directly converts low-grade wasted heat (400 to 500 K) into electricity, h

299 such as histologic grade (G1, G2, or G3, for low-grade [well differentiated], intermediate-grade [mod

300 Results Low-grade (WHO grade II) glioma showed areas with increa