ライフサイエンスコーパス: lumbar spine

1 ervertebral disc and other structures of the lumbar spine.

2 nations showing no relevant pathology of the lumbar spine.

3 led also focal tenderness in the area of the lumbar spine.

4 method in the lower cervical, thoracic, and lumbar spine.

5 tative computed tomography of the trabecular lumbar spine.

6 se and reproducible injection throughout the lumbar spine.

7 women with SLE had lower BMD at the hip and lumbar spine.

8 as the change in bone mineral density at the lumbar spine.

9 ined as a Z score -1.0 or less at the hip or lumbar spine.

10 thy subjects) had osteoporosis at the hip or lumbar spine.

11 ctive against loss of trabecular bone at the lumbar spine.

12 RP) on BMD at the forearm, femoral neck, and lumbar spine.

13 Similar results were seen at the lumbar spine.

14 by g-ratio analysis within the thoracic and lumbar spine.

15 ugh much less frequently, in the thoracic or lumbar spine.

16 cium group (P for time-by-group interaction: lumbar spine, 0.002; total hip, 0.03; whole body, 0.03).

17 ter first scan) was average for age and sex (lumbar spine, +0.7 +/- 1.6; femoral neck, -0.1 +/- 1.1;

18 , mean bone density Z scores have increased (lumbar spine, -0.2 +/- 1.6; femoral neck, -0.6 +/- 1; to

19 elow average for age and sex (mean Z scores: lumbar spine, -0.4 +/- 1.6; femoral neck, -0.7 +/- 1.1;

20 n hip BMD: 0.83 (women), 0.95 (men) g/cm(2); lumbar spine: 0.86 (women), 0.93 (men) g/cm(2)].

21 e interval [95% CI] -0.70, -0.13) and at the lumbar spine (-1.03 versus 0.10; group difference -1.13;

22 (-7.24%) compared with the tamoxifen group (lumbar spine, +2.77%; total hip, +0.74%).

23 r Zic1 was the most up-regulated gene in the lumbar spine (202-fold; P<10(-7)) in comparison with the

24 At the lumbar spine, 3 year mean BMD change for the 77 women re

25 omen who received placebo (342 women) at the lumbar spine (-4.0% [-4.5 to -3.4] vs -1.2% [-1.7 to -0.

26 se in median BMD from baseline to 5 years in lumbar spine (-6.08%) and total hip (-7.24%) compared wi

27 /- 1.0%; whole body, -3.6 +/- 0.5%) and F52 (lumbar spine, -6.2 +/- 0.9%; total hip, -10.3 +/- 1.4%;

28 ased from extension baseline by 16.5% at the lumbar spine, 7.4% at total hip, 7.1% at femoral neck, a

29 rsisted at NPNL and F52 (P </= 0.001): NPNL (lumbar spine, -7.5 +/- 0.7%; total hip, -10.5 +/- 1.0%;

30 reased from FREEDOM baseline by 21.7% at the lumbar spine, 9.2% at total hip, 9.0% at femoral neck, a

31 bone mineral density of 13.7 percent at the lumbar spine (95 percent confidence interval, 12.0 to 15

32 It was unaccompanied by substantial lumbar spine abbreviation, an adaptation restricted to v

33 was strongly associated with low BMD at the lumbar spine (adjusted odds ratio 4.42; 95% CI 2.19, 8.9

34 ted with loss of BMD at the femoral neck and lumbar spine after 3 years of treatment.

35 ip, femoral neck, and trabecular bone of the lumbar spine also differed significantly between groups

36 one mineral density T scores are -2.6 at the lumbar spine and -1.9 at the total hip, and spine imagin

37 m(3) (2.7%; 95% CI 2.0-3.4; p<0.0001) at the lumbar spine and 0.025 g/cm(3) (1.4%; 0.8-1.9; p<0.0001)

38 Of the 374 SN, 153 (41%) were in the lumbar spine and 221 (59%) were in the thoracic spine.

39 ensive bone marrow metastases throughout the lumbar spine and a soft tissue mass in the lower sacral

40 rabecular bone mineral density (vBMD) of the lumbar spine and coronary artery calcium (CAC) and abdom

41 With a broad thorax, long lumbar spine and extended hips and knees, as in bipeds,

42 sing dual-energy X-ray absorptiometry at the lumbar spine and femoral neck (FN).

43 PD patients had lower hip, lumbar spine and femoral neck BMD levels compared with h

44 ith the use of random-effects models for the lumbar spine and femoral neck for all studies providing

45 avone therapies for treating BMD loss at the lumbar spine and femoral neck in estrogen-deficient wome

46 Bone mineral density (BMD) of lumbar spine and femoral neck was measured, and tryptase

47 ry bone resorption markers (n = 2929) at the lumbar spine and femoral neck were performed in perimeno

48 nd logistic regression, respectively, at the lumbar spine and femoral neck, stratified by male, preme

49 vely assessed standardized BMD (sBMD) at the lumbar spine and femoral neck, World Health Organization

50 mulant use had lower DXA measurements of the lumbar spine and femur compared with nonusers.

51 roved bone mass and microarchitecture in the lumbar spine and femur in F508del mice.

52 Mean bone mineral density z scores (lumbar spine and femur) remained stable and were maintai

53 puted tomography, as well as with BMD of the lumbar spine and hip at dual x-ray absorptiometry.

54 -old women with or without low BMD underwent lumbar spine and hip bone densitometry and a complete pe

55 S-986001 groups showed a smaller decrease in lumbar spine and hip bone mineral density but greater ac

56 Substantial loss of BMD in the lumbar spine and hip was seen in patients who discontinu

57 Bone mineral density was measured at the lumbar spine and hip, and hip geometry was extracted fro

58 (CAL) and bone mineral density (BMD) at the lumbar spine and hip, lifestyle, smoking, sociodemograph

59 and 0.8% (0.3-1.4; p=0.003) in year 2 at the lumbar spine and hip, respectively.

60 was associated with greater BMD loss at both lumbar spine and hip.

61 sion in human bone biopsy samples taken from lumbar spine and iliac crest, sites that experience high

62 coordinates that assist in targeting of the lumbar spine and instructional videos.

63 utative somatosensory representations of the lumbar spine and leg.

64 of 64 y underwent (18)F-fluoride PET of the lumbar spine and measurements of biochemical markers of

65 ineral density (rs3736228, p=6.3x10(-12) for lumbar spine and p=1.9x10(-4) for femoral neck) and an i

66 nsity (top SNP, rs4355801: p=7.6x10(-10) for lumbar spine and p=3.3x10(-8) for femoral neck) and incr

67 al examination, and general knowledge of the lumbar spine and pelvic anatomy relevant to the child in

68 Bone mineral density (BMD) of the lumbar spine and proximal femur (by DXA), liver function

69 BMD of the lumbar spine and proximal femur were measured at entry a

70 BMD of the posteroanterior lumbar spine and proximal femur were measured by dual-en

71 ified disease starting simultaneously in the lumbar spine and sacroiliac joints in a proportion of pa

72 ages obtained by magnetic resonance scans of lumbar spine and the clinical symptoms of the disease in

73 to increase the bone mineral density at the lumbar spine and the femoral neck in men.

74 Bone mineral density was measured at lumbar spine and the hip.

75 changes in bone mineral density (BMD) in the lumbar spine and total hip between patients treated with

76 A + R resulted in a significant increase in lumbar spine and total hip BMD compared with A + P treat

77 In the H stratum, lumbar spine and total hip BMD increased significantly (

78 Lumbar spine and total hip BMD were assessed at baseline

79 The primary endpoints were changes in lumbar spine and total hip bone mineral densities (BMDs)

80 Lumbar spine and total hip bone mineral density (BMD) we

81 ndpoints were the mean percentage changes in lumbar spine and total hip bone mineral density at week

82 Bone mineral density was measured at the lumbar spine and total hip by dual-energy X-ray absorpti

83 nts were percent change of BMD at 2 years in lumbar spine and total hip for both groups.

84 an increase of bone mineral density in both lumbar spine and total hip sites, with a significant pos

85 Lumbar spine and whole body BMD z-scores remained below

86 neral density (BMD) and content (BMC) at the lumbar spine, and (2) focal lesions in x-rays of long bo

87 ft tibia determined by pQCT, and whole-body, lumbar spine, and femoral neck measurements by DXA.

88 one mineral density (BMD) at the total body, lumbar spine, and hip (total and femoral neck) were eval

89 BMC of the total body, lumbar spine, and hip and dietary phylloquinone intake w

90 orptiometry (DXA) bone outcomes (whole body, lumbar spine, and hip), controlling for known determinan

91 DXA was used to determine BMD of the radius, lumbar spine, and hip.

92 BMC, or bone area for the total-body radius, lumbar spine, and total hip were observed between subjec

93 moral neck; -0.09, 95% CI -0.15 to -0.03 for lumbar spine; and -0.05, 95% CI -0.07 to -0.03 for total

94 or lower at the total hip, femoral neck, or lumbar spine; and a history of fracture.

95 ooked for if abnormalities in the MRI of the lumbar spine are not found.

96 microRNA MIR196A2 gene that associates with lumbar spine area (P = 2.3 x 10(-42), beta = -0.090) and

97 The mean increase in lumbar spine areal BMD after 1 year was 16.3% in the ris

98 y efficacy endpoint was percentage change in lumbar spine areal bone mineral density (BMD) at 1 year.

99 from baseline in bone mineral density at the lumbar spine at 12 months.

100 ercent change in bone mineral density at the lumbar spine at 24 months.

101 neral density (BMD) at the femur, tibia, and lumbar spine at 3 months and at the lumbar spine at 4 mo

102 bia, and lumbar spine at 3 months and at the lumbar spine at 4 months, with full normalization of the

103 ident, with higher lumbar spine BMC (13.9%), lumbar spine BA (6.2%), and lumbar spine BMD (10.6%) in

104 Supplemented mothers had higher lumbar spine BA (6.7%; P = 0.002) and lumbar spine BMC (

105 etween groups were more evident, with higher lumbar spine BMC (13.9%), lumbar spine BA (6.2%), and lu

106 higher lumbar spine BA (6.7%; P = 0.002) and lumbar spine BMC (7.9%, P = 0.08) than did mothers who c

107 Mean lumbar spine BMC was significantly lower in stimulant us

108 ients (n = 98) had lower rates of decline in lumbar spine BMD (-0.004 +/- 0.003 vs. -0.015 +/- 0.003

109 e L stratum showed a significant decrease in lumbar spine BMD (-2.1%; P = .0109) and a numerical decr

110 l hip (0.029 +/- 0.006 g/cm2, P <0.001), and lumbar spine BMD (0.025 +/- 0.007 g/cm2, P = 0.001).

111 r 3 months or longer had significantly lower lumbar spine BMD (0.89 g/cm2; 95% CI, 0.85-0.93 g/cm2 vs

112 95% CI, 13.26-13.51 g; P = .02), as was mean lumbar spine BMD (0.90 g/cm2; 95% CI, 0.87-0.94 g/cm2 vs

113 ine BMC (13.9%), lumbar spine BA (6.2%), and lumbar spine BMD (10.6%) in the supplemented group (P </

114 ctors of incident fracture were the baseline lumbar spine BMD (for each 1-point decrease in T score,

115 tly greater femoral neck BMD (P = 0.008) and lumbar spine BMD (P = 0.007) than did those who never co

116 between SNPs in the Osterix region and adult lumbar spine BMD (P = 9.9 x 10(-11)).

117 al hip BMD (r=-0.33, P<0.0001), but not with lumbar spine BMD (r=-0.09, P=0.27).

118 combined OA phenotype (hip and/or knee) and lumbar spine BMD (rg=0.18, P = 2.23 x 10-2), which may b

119 n of previously reported common variants for lumbar spine BMD (rs11692564(T), MAF = 1.6%, replication

120 ly beer had a positive significant effect on lumbar spine BMD after adjustment for lifestyle (P = 0.0

121 lts were seen for change in femoral neck and lumbar spine BMD and across a range of subgroup analyses

122 r ALN trial for followup measurements of the lumbar spine BMD and hip BMD, and retrospective informat

123 ual-energy x-ray absorptiometry to determine lumbar spine BMD and total-body BMD.

124 Low and very low BMD were defined as lumbar spine BMD and/or total-body BMD z scores of -1 or

125 The ZOL arm had an 8% higher lumbar spine BMD at 12 weeks relative to the placebo arm

126 We measured femoral neck and lumbar spine BMD at baseline and after 1 and 3 years, an

127 influence of all protein supplementation on lumbar spine BMD but showed no association with relative

128 Lumbar spine BMD decline was also less with MVC (median

129 ponse parameter was the percentage change in lumbar spine BMD from the end of year 1 to the followup

130 n evidence of association with adult hip and lumbar spine BMD in an Icelandic population, as well as

131 ositive effect of protein supplementation on lumbar spine BMD in randomized placebo-controlled trials

132 nosis could reduce the likelihood of reduced lumbar spine BMD in these patients by prompting interven

133 At 12 and 24 months, lumbar spine BMD increased by 5.5% and 7.6%, respectivel

134 In the combined group, the lumbar spine BMD increased by 7.2%, and total hip BMD in

135 Lumbar spine BMD increased in the placebo group by 0.98%

136 At 12 months, posterior-anterior lumbar spine BMD increased more in the combination group

137 CD4(+)CD38(+)HLA-DR(+)) were associated with lumbar spine BMD loss.

138 No differences were observed in change in lumbar spine BMD or lean body mass.

139 ucocorticoid dose, neither total hip BMD nor lumbar spine BMD was significantly associated with focal

140 Hip and lumbar spine BMD were measured by dual-energy x-ray abso

141 ns between lycopene intake and 4-y change in lumbar spine BMD were significant for women (P for trend

142 At 48 weeks, lumbar spine BMD with ZOL was 11% higher than placebo (n

143 At 48 weeks, lumbar spine BMD with ZOL was 11% higher than placebo (n

144 ficantly higher than that in patients with a lumbar spine BMD Z score higher than -1.5 (P = 0.038).

145 In patients with a lumbar spine BMD Z score of -1.5 or lower, the RANKL:OPG

146 accretion and a subsequent reduction in the lumbar spine BMD Z score.

147 Lumbar spine BMD Z scores (mean +/- SD -0.13 +/- 1.19 [r

148 t 5 years from diagnosis, with whole-body or lumbar spine BMD z scores of -1.0 or lower.

149 ration of untreated juvenile DM have reduced lumbar spine BMD Z scores.

150 ineral density (BMD), with femoral neck BMD, lumbar spine BMD, and lumbar spine trabecular bone score

151 ing variables (including menstrual history), lumbar spine BMD, bone mineral content, and BMD z score

152 ied for soy protein or milk basic protein on lumbar spine BMD.

153 of DNA pools prepared from individuals with lumbar spine-BMD (LS-BMD) values falling into the top an

154 Baseline total hip, femoral neck, and lumbar spine BMDs were 1.016 +/- 0.160, 0.941 +/- 0.142,

155 amounts of alcohol was associated with less lumbar spine bone loss (P < 0.01 for quartile of alcohol

156 d that soy protein with isoflavones lessened lumbar spine bone loss in midlife women.

157 ts with low calcium intake results in higher lumbar spine bone mass and a reduced rate of femoral nec

158 sts that the observed deficits in height and lumbar spine bone mass may not be related to suboptimal

159 The lumbar spine bone mass was measured in 45 consecutive pa

160 ulant use and total femur, femoral neck, and lumbar spine bone mineral content (BMC) and bone mineral

161 inuation of football after 6 months, hip and lumbar spine bone mineral density (BMD), mental health s

162 rcentage change from baseline at month 12 in lumbar spine bone mineral density (BMD).

163 and superior to risedronate for increase of lumbar spine bone mineral density in both the treatment

164 Lumbar spine bone mineral density showed a mean increase

165 Total-body and lumbar spine bone mineral density were measured in adole

166 point was percentage change from baseline in lumbar spine bone mineral density.

167 cts were significantly shorter and had lower lumbar spine bone mineral density; the deficits were gre

168 decline in HIV-uninfected individuals at the lumbar spine but not at the hip.

169 pression fractures of the lower thoracic and lumbar spine by using the Genant visual semiquantitative

170 obtained for other reasons that include the lumbar spine can be used to identify patients with osteo

171 ssociated with less bone loss at the hip and lumbar spine compared with TDF.

172 ethnicity was associated with low BMD at the lumbar spine controlling for relevant clinical covariate

173 eprogrammed for full cervical, thoracic, and lumbar spine coverage (combined 70-cm FOV, seven section

174 Reviewing the full-FOV images from lumbar spine CT examinations will result in the detectio

175 were present on images in 162 (40.5%) of 400 lumbar spine CT examinations; 59 (14.8%) patients had in

176 212 male and 188 female patients) undergoing lumbar spine CT for low back pain and/or radiculopathy.

177 ne mineral density of the posterior-anterior lumbar spine decreased by 2.5% +/- 0.5% in the leuprolid

178 Mean (+/- SE) BMD of the posteroanterior lumbar spine decreased by 3.1% +/- 1.0% in men assigned

179 group, the mean bone mineral density at the lumbar spine decreased by 3.2 percent (P=0.03 for the co

180 data exist concerning the natural history of lumbar spine disc degeneration and associated risk facto

181 y to examine the radiographic progression of lumbar spine disc degeneration over the course of 9 year

182 idual radiographic features of AO and DSN in lumbar spine disc degeneration.

183 with cervical, as compared to patients with lumbar spine disease.

184 MATERIALS AND Lumbar spine diskograms and prediskogram MR images of 73

185 ompared in 782 participants with symptomatic lumbar spine disorders who were referred to orthopedists

186 Lumbar spine dual X-ray absorptiometry does not consiste

187 enal stone protocols (26.2%) and thoracic or lumbar spine examinations (6.6%).

188 gy x-ray absorptiometry, was assessed at the lumbar spine, femoral neck, and total femur (grams per s

189 numerically greater decreases in BMD at the lumbar spine, femoral neck, and total hip from the end o

190 lower rib fractures, 7.6% (eight of 105) for lumbar spine fractures, and 5.2% (nine of 174) for pelvi

191 lted in a significant increase in BMD at the lumbar spine (from 0.875 +/- 0.025 to 0.913 +/- 0.026 g/

192 Thirty-four samples of three cadaveric lumbar spines (from subjects who died at ages 51, 57, an

193 For lumbar spine fusion, rhBMP-2 and iliac crest bone graft

194 At one year, the bone mineral density at the lumbar spine had decreased by a mean of 0.7 percent in t

195 At 24 months, bone mineral density of the lumbar spine had increased by 5.6% in the denosumab grou

196 the mean (+/-SE) bone mineral density at the lumbar spine had increased more in the teriparatide grou

197 Bone mineral density of the lumbar spine, hip, and total body was measured yearly fo

198 spondylodiscitis include: involvement of the lumbar spine, ill-defined paraspinal abnormal contrast e

199 To improve the accuracy of lumbar spine imaging-based marrow dosimetry, one can adj

200 he dual-energy x-ray absorptiometry scans of lumbar spine in 39 KTR and 77 controls.

201 be used to assess MAT content and BMD of the lumbar spine in a single examination and provides data t

202 phic (CT) trabecular texture analysis of the lumbar spine in patients with anorexia nervosa and norma

203 limitations regarding load-relaxation of the lumbar spine in response to flexion exposures and the in

204 nst 4-y loss in trochanter BMD in men and in lumbar spine in women.

205 ant increases in bone mineral density at the lumbar spine, including an increase of 11.3% with the 21

206 The bone mineral density at the lumbar spine increased significantly more in men treated

207 The bone mineral density of the lumbar spine increased significantly more in the combina

208 h as physical therapy, oral medications, and lumbar spine injections.

209 outcomes at 1 day, 1 week, and 1 month after lumbar spine injections.

210 Lumbar spine instability (LSI), or aging, induces spinal

211 The finding of a VT on MRI imaging of the lumbar spine is often incidental but may be found in pat

212 At the lumbar spine, isoflavone treatment was associated with a

213 rmed a cross-sectional audit of MRI scans of lumbar spine (L-spine) and sacroiliac (SI) joints.

214 ormed before treatment, and Z scores for the lumbar spine (L1-L4) were determined.

215 th low bone mineral density (BMD) (g/cm(2)), lumbar spine L2-L4 and femoral neck (T-scores) (P = 0.01

216 the femoral neck, trochanter, total hip, and lumbar spine (L2-L4) was associated with a 0.005-0.008-g

217 the femoral neck, trochanter, total hip, and lumbar spine (L2-L4) was measured by using dual-energy X

218 osteopenia at the trochanter, total hip, and lumbar spine (L2-L4) were lower by 14% (OR: 0.86; 95% CI

219 e femoral neck, trochanter, total femur, and lumbar spine (L2-L4) were measured by using dual-energy

220 revalence of osteoporosis at baseline at the lumbar spine (LS) and femoral neck (FN) was 17.6% and 7.

221 ears) perinatally infected with HIV with low lumbar spine (LS) BMD (Z score < -1.5) were randomized t

222 The primary outcome was percent change in lumbar spine (LS) BMD at 6 months.

223 The primary end point was the change in lumbar spine (LS) BMD from baseline to 1 year.

224 otein intake may have a protective effect on lumbar spine (LS) bone mineral density (BMD) compared wi

225 delayed group received zoledronic acid when lumbar spine (LS) or total hip (TH) T score decreased to

226 in areal bone mineral density (aBMD) of the lumbar spine (LS), as determined by dual-energy X-ray ab

227 Lumbar spine (LS), femoral neck (FN), and distal radius

228 -miRTS)-centric multistage meta-analysis for lumbar spine (LS)-, total hip (HIP)- and femoral neck (F

229 elected because of reduced BMD values at the lumbar spine (LS-BMD) or femoral neck (FN-BMD) in proban

230 t total femur (TFBMD), femoral neck (FNBMD), lumbar spine (LSBMD), and physician-diagnosed osteoporos

231 bdominal computed tomography (CT), brain and lumbar spine magnetic resonance (MR) imaging, and body p

232 Five cadaveric lumbar spines (mean age, 61 years +/- 11) were prepared

233 bone histology; the first carrier had normal lumbar spine measurements (L1-L4), as determined by dual

234 The interpretation of general lumbar spine MR characteristics has sufficient reliabili

235 ncomplicated degenerative changes on initial lumbar spine MR images were identified, 71 (30%) of whic

236 ar non-SLIP patients undergoing conventional lumbar spine MR imaging as usual care in calendar year 2

237 demiologic information was included in their lumbar spine MR imaging reports.

238 nt was routinely but arbitrarily included in lumbar spine MR imaging reports.

239 T and MR imaging procedures were head CT and lumbar spine MR imaging.

240 improvement initiatives include head CT and lumbar spine MR imaging.

241 a GWA study of DXA bone area of the hip and lumbar spine (N >= 28,954), we find thirteen independent

242 re of the sixth through 12th ribs (n = 216), lumbar spine (n = 105), or pelvis (n = 174).

243 ary statistics from the GEFOS consortium for lumbar spine (n = 31,800) and femoral neck (n = 32,961)

244 n an increase in bone mineral density at the lumbar spine of 3.0 to 6.7 percent (as compared with an

245 examinations within 2 months, comprising the lumbar spine of 40 patients, were included.

246 and Z scores) of the hip, femoral neck, and lumbar spine of IgE-CMA patients were significantly lowe

247 vertebral body fractures in the thoracic and lumbar spine on CT images with a high sensitivity and a

248 d MRI group vs 4 in the radiograph group had lumbar spine operations (risk difference, 0.34; 95% CI,

249 eral density (T score of -1.8 to -4.0 at the lumbar spine or -1.8 to -3.5 at the proximal femur).

250 z score of >/= 2.0 SDs below the mean at the lumbar spine or hip, was highly prevalent in all 3 group

251 less than -2.5 but not less than -4.0 at the lumbar spine or total hip.

252 Hematuria and fracture of the lower ribs, lumbar spine, or pelvis are objective predictors of miss

253 tly associated with systemic BMD loss at the lumbar spine (osteocalcin, bone-turnover biomarker, p =

254 No association between lumbar spine osteopenia/osteoporosis and radiographic sc

255 A were independently associated with risk of lumbar spine osteoporosis.

256 fter ZOL infusion, BMD did not change at the lumbar spine (P = .22) but declined at the hip (P = .04)

257 0(-7)) and lower bone mineral density at the lumbar spine (P = 0.038), but not the femoral neck.

258 f the thoracic spine (P <.05) but not in the lumbar spine (P =.09).

259 subjects had lost a mean of 2.4% BMD at the lumbar spine (P=0.003) but did not experience significan

260 fter ZOL infusion, BMD did not change at the lumbar spine (p=0.22), but declined at the hip (p=0.04)

261 Radiographs of the cervical spine, lumbar spine, pelvis, and hips were scored by using the

262 neral density were measured from total body, lumbar spine, proximal femur, and forearm with dual-ener

263 The bone mineral density of the lumbar spine, proximal femur, radial shaft, and total bo

264 th reduced whole-body (r=0.21, p=0.0088) and lumbar-spine (r=0.17, p=0.03) bone-mineral content in ch

265 tween fracture and BMD in patients with IBD (lumbar spine, r = -0.103, p = 0.17 and femoral neck, r =

266 Seven hundred ninety-six paired lumbar spine radiographs were read by a single reader fo

267 BMD), and BA-adjusted BMC of the whole-body, lumbar spine, radius, and hip were measured by dual-ener

268 dioactivity to the cumulated activity of the lumbar spine region of interest (ROI) from serial gamma-

269 ng a mean follow-up of 6.5 years, additional lumbar spine surgery was performed in 22% of the patient

270 l hip arthroplasty, hip fracture repair, and lumbar spine surgery.

271 ) and up to one year (Y1) after cervical and lumbar spine surgery.

272 Lumbar spine TBS significantly increased at 6 mo in the

273 d MRI sequences of the sacroiliac joints and lumbar spine that were scored for active bone marrow ede

274 ual-energy X-ray absorptiometry, we compared lumbar spine, total hip, and femoral neck bone mineral d

275 as well as bone mineral density (BMD) at the lumbar spine, total hip, femoral neck, and one-third rad

276 al density (a T score of -2.0 or less at the lumbar spine, total hip, or femoral neck and -3.5 or mor

277 Outcomes included lumbar spine, total proximal femur, femoral neck, and wh

278 with femoral neck BMD, lumbar spine BMD, and lumbar spine trabecular bone score (TBS) as secondary ou

279 al density was measured at the total hip and lumbar spine using dual-energy x-ray absorptiometry.

280 X-ray absorptiometry images at the L1 to L4 lumbar spine using TBS software.

281 seline BMD measurements (femoral neck and/or lumbar spine) using dual x-ray absorptiometry.

282 bone mineral density (BMD) loss at the L2-L4 lumbar spine vertebra (P < 0.05), femoral neck (P < 0.01

283 Subjects lost 1-2% BMD annually at lumbar spine vertebrae 2-4, the forearm, the femoral nec

284 trally and laterally within the thoracic and lumbar spine vertebral bodies.

285 After 18 months, DeltaBMD at the lumbar spine was 0.068 +/- 0.21 and 0.015 +/- 0.034 for

286 rcentage loss in bone mineral density in the lumbar spine was greater in the standard group than in t

287 Diffusion-weighted MR imaging of the lumbar spine was performed in 39 patients (all men; mean

288 BMD of the lumbar spine was significantly (P<or=0.05) and comparabl

289 using a specific exercise that isolated the lumbar spine, was efficacious in preventing steroid-indu

290 c resonance images of the lower thoracic and lumbar spine were analyzed in 516 healthy female twins (

291 disease, after which the cervical spine and lumbar spine were equally involved.

292 ual-energy x-ray absorptiometry scans of the lumbar spine were performed.

293 Vertebral deformities of the thoracic and lumbar spine were radiographically classified by using t

294 The patient initially had a CT scan of the lumbar spine which only revealed a protrusion of the L5-

295 of the pelvis and lateral radiographs of the lumbar spine, which were scored using the Stoke Ankylosi

296 cision of (18)F-fluoride PET measured at the lumbar spine, which will aid in the accurate interpretat

297 posture with correction at the cervical and lumbar spine with the 3D-printed padded collar being wor

298 g/d) had no significant effect on BMD at the lumbar spine (WMD: 0.74%; 95% CI: -0.10%, 1.59%; I2 = 47

299 Computed tomography, bone scintigraphy, and lumbar spine x-rays were performed at the beginning and

300 Lumbar spine z scores measured 19-44 d after delivery (n