ライフサイエンスコーパス: lung lavage

1 ory resistances were tested in 10 pigs after lung lavage.

2 epletion model of ARDS was created by saline lung lavage.

3 ss syndrome was induced by repetitive saline lung lavage.

4 and is also available in saliva, plasma, and lung lavage.

5 -6) reacted with a double band of 290 kDa in lung lavage.

6 Acute lung injury was induced by saline lung lavage.

7 ased concentrations of the chemokine CCL2 in lung lavage.

8 e lung injury was induced by repeated saline lung lavage.

9 w) settings in five piglets before and after lung lavage.

10 n at the same mean airway pressure as before lung lavage.

11 isotype antibodies in the serum and mucosal lung lavages.

12 s syndrome model was established by repeated lung lavages.

13 Lung injury was created by repeated saline lung lavages.

14 c DNA in C3H/HeBFEJ mice and performed whole lung lavage 4 h after the exposure.

15 Lung lavages also were obtained from 10 lungs at autopsy

16 unimodal after pulmonary embolism and saline lung lavage and bimodal after bronchoconstriction.

17 phatidylcholine was extracted from the total lung lavage and from the pulmonary parenchyma.

18 repeated after lung injury induced by whole-lung lavage and injurious ventilation in four rabbits.

19 lary endothelium and cytokine levels in both lung lavage and supernatants from cultured lymph node ly

20 Lung injury was induced by lung lavages and 210 minutes of injurious mechanical ven

21 ry distress syndrome was induced by repeated lung lavages and injurious mechanical ventilation.

22 antibodies specific for B. melitensis LPS in lung lavages and specific IgG and IgA antibody-secreting

23 wed that CRP was present in the lung tissue, lung lavage, and alveolar macrophages.

24 IgA LPS-specific antibodies were detected in lung lavages, and specific antibody-secreting cells were

25 lude that dilute surfactants administered by lung lavage are effective in reversing pulmonary dysfunc

26 more rapidly and to a greater extent in the lung lavage as compared with naive cells.

27 tocopherol were inhibitory and reversible in lung lavage but, importantly, were proinflammatory in lu

28 N-gamma-producing CD4+ and CD8+ cells in the lung lavages but no change in the number of IL-4-produci

29 sues (by in situ hybridization) and isolated lung lavage cells (by RT-PCR).

30 onchoalveolar lymphocytes and the ability of lung lavage cells to produce proinflammatory cytokines w

31 Lung lavage cells were recovered from each bronchoscopy

32 reased lung neutrophil accumulation, but not lung lavage cytokine-induced neutrophil chemoattractant

33 s in lung lavage neutrophil accumulation and lung lavage cytokine-induced neutrophil chemoattractant

34 mproving symptoms and quality of life; whole-lung lavage effectively removes excessive surfactant.

35 pite the fact that 22% had CMV isolated from lung lavage fluid and 32% had CMV isolated from blood.

36 r 3, was up-regulated significantly in mouse lung lavage fluid and plasma at postnatal day 14 in resp

37 Lung lavage fluid and tissue samples were obtained immed

38 rleukin-8 (IL-8), and GRO were detectable in lung lavage fluid at 4 h and declined by 24 h in animals

39 sponse in the lung as determined by assay of lung lavage fluid by enzyme-linked immunosorbent assay a

40 We further show that lung lavage fluid collected from both A. fumigatus-chall

41 pies per microliter and influenza A virus in lung lavage fluid containing 2.08 x 10(6) viral copies p

42 Furthermore, lung lavage fluid from endotoxemic, ethanol-fed rats had

43 CSF protein levels were observed in sera and lung lavage fluid from superinfected animals, suggesting

44 niae organisms are frequently present in the lung lavage fluid from this cohort of predominantly asth

45 Importantly, lung lavage fluid in severe pH1N1 disease is predominant

46 Following administration of bleomycin, lung lavage fluid of WT mice demonstrated an approximate

47 Percent neutrophils in lung lavage fluid post-infection are significantly highe

48 Analysis of lung lavage fluid revealed the presence of a leukocytic

49 Reduction in inflammatory cytokine levels in lung lavage fluid samples correlated with the clinical o

50 Myeloperoxidase activity in lung lavage fluid samples, an indicator of neutrophil in

51 ats given IL-1 intratracheally had increased lung lavage fluid tumor necrosis factor (TNF) levels, an

52 Lung lavage fluid was assayed for measurements of total

53 vated tocopherols on tissue inflammation and lung lavage fluid were reversible in a second phase of A

54 hione levels in the plasma, lung tissue, and lung lavage fluid, and increased (P < 0.05) oxidized glu

55 creased numbers of inflammatory cells in the lung lavage fluid, and polyclonal Ig activation.

56 ens, such as turbinate tissue homogenate and lung lavage fluid, as well as antemortem samples, such a

57 can assay GSH in whole blood, plasma, serum, lung lavage fluid, cerebrospinal fluid, urine, tissues a

58 n proinflammatory cytokines in perfusate and lung lavage fluid, compared to control.

59 cytokines interleukin-5 (IL-5) and IL-13 in lung lavage fluid, decreased regulatory T cell-associate

60 tal protein and proinflammatory cytokines in lung lavage fluid, enhanced LPS-mediated signaling in lu

61 In contrast to the lung lavage fluid, highly elevated levels of gamma-tocop

62 In lung lavage fluid, mice treated with Ad-cIKK1 or Ad-cIKK

63 oglobulin A (IgA) and IgG in the nasal wash, lung lavage fluid, saliva, and fecal extract.

64 hil infiltration and interleukin-5 (IL-5) in lung lavage fluid.

65 -tocopherol reduced leukocyte numbers in the lung lavage fluid.

66 er of CD4(+) and CD8(+) T cells recovered in lung lavage fluid.

67 vironments, such as lung surfactant or mouse lung lavage fluid.

68 ytopenia were observed in blood, dermis, and lung lavage fluid.

69 tion of macrophage inflammatory protein-2 in lung lavage fluid.

70 aluate surfactant by biochemical analysis of lung lavage fluid.

71 f the 7S fragment of type IV collagen in the lung lavage fluid.

72 P < 0.05) oxidized glutathione levels in the lung lavage fluid.

73 e correlated the quantity of leukotrienes in lung lavage fluids of infected mice with respiratory rat

74 ulin A antibody in saliva, nasal washes, and lung lavage fluids.

75 Methods: ARDS was induced by saline lung lavage followed by injurious mechanical ventilation

76 Lung injury was induced with saline lung lavage followed by injurious ventilation in supine

77 inin peptides were identical to sequences in lung (lavage) gp-340, a member of the scavenger receptor

78 lar lavage (BAL) in human subjects and whole-lung lavage in mice following a single inhalation exposu

79 ed after acute lung injury induced by saline lung lavage in neonatal piglets.

80 led very high percentages of Th17.1 cells in lung lavage in sarcoidosis compared with controls in two

81 P, and may represent an alternative to whole-lung lavage in treating the disease.

82 nificantly more monocytes and neutrophils in lung lavage, increased CD4+/CD8+ T-lymphocyte ratio, and

83 curs following the use of open suctioning in lung lavage injured sheep and whether the baseline PaCO2

84 y, IL-17A-secreting cells have been found in lung lavage, invoking Th17 immunity in sarcoidosis.

85 mmatory response, as observed from recovered lung lavage leukocytes and histology.

86 Finally, the blood and lung lavage levels of CD40L are significantly elevated i

87 Whole-lung lavage may be a potentially useful modality of trea

88 In a lung lavage model of acute lung injury, both variable pr

89 e protein-containing surfactant, in a saline lung lavage model of ARDS in neonatal piglets.

90 pulmonary embolism (n = 3) and after saline lung lavage (n = 3).

91 Lung lavage (n = 6/group) 24 h after a first phosgene ex

92 Fbp intravenously had the same elevations in lung lavage neutrophil accumulation and lung lavage cyto

93 The average total lung lavage neutrophil count was 5.5 x 10(6) with endoto

94 xhibited a twofold increase in the number of lung lavage neutrophil level whereas NGFR knockout mice

95 activation, concentration of KC and MIP-2 in lung lavage, neutrophil influx, and lung edema measured

96 We assayed lung lavage nitrotyrosine (NT) and chlorotyrosine (CT) b

97 Interleukin-1 (IL-1) is increased in lung lavages obtained from patients with acute respirato

98 of all classes of leukocytes recovered from lung lavages of infected neonates and adults were simila

99 ng IFNgamma than those producing IL-4 in the lung lavages of mice given either syngeneic or allogenei

100 We performed multiple whole lung lavages on a patient with PAP, and cultured BAL cel

101 We found that lung PLA2 activity, lung lavage phospholipid content, lung leak index, lung

102 sures of lung injury including gas exchange, lung lavage protein and lactate dehydrogenase (LDH), lun

103 Total lung lavage protein concentration, TUNEL staining, lipid

104 nitrogen oxides, and phosgene increase both lung lavage protein concentrations and neutrophils.

105 tent, lung leak index, lung weight gain, and lung lavage protein concentrations were increased in rat

106 lymphocyte precursor frequency, NO levels in lung lavage, rates of virus clearance, and anti-RSV Ab t

107 Although whole-lung lavage remains the first-line therapy, inhaled GM-C

108 at each level of mean airway pressure after lung lavage, respiratory system compliance and functiona

109 Lung lavage revealed 0.41 +/- 0.03 mL of plasma in the l

110 (inactive) forms of IL-33 can be detected in lung lavage samples from mice challenged with Alternaria

111 CD8(+) T cells in lung lavage samples from SIV- and P. carinii-coinfected

112 Particle sizing of the final lung lavages showed preservation of large surfactant agg

113 Lung lavage significantly lowered respiratory system com

114 Lung lavage Th17.1-cell percentages were also higher tha

115 Lung lavage was collected on Day 3 (pre-study) and Day 7

116 The TNF-alpha concentration in lung lavage was increased by AdRelA treatment and benefi

117 on eight septic rats and on seven controls; lung lavage was performed on three septic rats and three

118 were anesthetized and intubated, and saline lung lavage was performed.

119 y of the patient's symptoms, bilateral whole-lung lavage was undertaken, leading to symptomatic impro

120 Lung lavage was used to create acute respiratory distres

121 nts with PAP who underwent therapeutic whole-lung lavage were compared with those of 10 healthy volun

122 Hsp70 levels in lung tissue and in cell-free lung lavage were increased compared with mice exposed to

123 nterferon, or tumor necrosis factor alpha in lung lavages were found between CCL3(+/+) and CCL3(-/-)

124 Two sequential series of lung lavages were performed to lower PaO2 < 100 mm Hg, e

125 g injury was induced in 23 sheep by repeated lung lavage with warmed saline until the PaO2/FIO2 ratio

126 stress syndrome was induced combining saline lung lavages with injurious mechanical ventilation.

127 were determined in noninjured (n = 9) and in lungs lavaged with saline (n = 8) at quasi-static (low f

128 s (4-4.5 mg phospholipid/ml) administered by lung lavage would be equally effective in reversing pulm