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1 o directly affect GC T cell migration within lymphoid follicles.
2 ty and functional activity of GCs in ectopic lymphoid follicles.
3 e thymocytes, and formation of thymic B cell lymphoid follicles.
4 to apoptosis that was localized to tonsillar lymphoid follicles.
5  inversely related to acute inflammation and lymphoid follicles.
6  are a subset of CD4(+) T cells in secondary lymphoid follicles.
7  of gastric inflammation, or the presence of lymphoid follicles.
8 n follicular structures resembling secondary lymphoid follicles.
9 capitulate many features of normal secondary lymphoid follicles.
10 und to be required for B-cell migration into lymphoid follicles.
11 assive numbers of apoptotic cells within the lymphoid follicles.
12 idermis, hair follicles, and subconjunctival lymphoid follicles.
13 ng cells, strategically located over mucosal lymphoid follicles.
14 , with impairments in B-cell accumulation in lymphoid follicles.
15 the presence of inflammatory fibroblasts and lymphoid follicles.
16  we resolved fine immune cell populations in lymphoid follicles.
17 s limited in rectal tissue and negligible in lymphoid follicles.
18 ar aggregates that resemble human intestinal lymphoid follicles.
19 ral replication is concentrated in secondary lymphoid follicles.
20  around both immune and stromal cells within lymphoid follicles.
21 o promote dissemination of infected cells to lymphoid follicles.
22 associated with an accumulation of pulmonary lymphoid follicles.
23 is and lymphoplasmacytic infiltrates without lymphoid follicles.
24 CR6 deficiency and remained clustered within lymphoid follicles.
25 f CXCL13 was observed within B-cell areas of lymphoid follicles.
26  progressive luminal occlusion, and tertiary lymphoid follicles.
27 ls, which accumulated in T-cell areas of the lymphoid follicles.
28 st immune response organized, remarkably, as lymphoid follicles.
29 lymphoid tissue, cryptopatches, and isolated lymphoid follicles.
30  followed by destruction of the integrity of lymphoid follicles.
31 o educate the immune cells of the intestinal lymphoid follicles.
32 of lymph node and were largely absent within lymphoid follicles.
33 is (present in 42% of sections), presence of lymphoid follicles (32%), a plasma cell-enriched inflamm
34 s of CXCR5(+)CD8(+) T cells that can home to lymphoid follicles, a key site of antibody production.
35 ugh potentially Ag-reactive cells within the lymphoid follicle accumulate following antigenic challen
36 -1, which correlated with greater degrees of lymphoid follicle adherence and lesser degrees of ileal
37 ns demonstrated some degree of Peyer's patch lymphoid follicle adherence, ranging from diffuse covera
38 ion, we examined the changes in the appendix lymphoid follicle (ALF) and Peyer's patches (PP) in thes
39 cytes and displayed morphologically in large lymphoid follicles along the intestine.
40 crease in gastric B cells, plasma cells, and lymphoid follicles, along with enhanced H. pylori-specif
41 ic architecture in A/WySnJ, with few primary lymphoid follicles and a second abnormal phenotype, mast
42 on at the boundary of T cell:B cell areas in lymphoid follicles and ability to express IL-4 and CD40L
43  dysfunction as measured by the formation of lymphoid follicles and alteration of the thymocyte subse
44                        Third eyelids contain lymphoid follicles and are easier to collect.
45 ack of Nkx2.3 impairs maturation of isolated lymphoid follicles and attenuates dextran sodium sulfate
46 as characterized by morphometric analysis of lymphoid follicles and by differentiating lymphocyte pop
47 atoid arthritis (RA) synovium often resemble lymphoid follicles and contain clonally related Ig trans
48 , we aimed at interrogating the link between lymphoid follicles and development of pulmonary inflamma
49 ls and marginal zone B cells may relocate to lymphoid follicles and differentiate into cytokine and a
50 l disorganization, with reduction of defined lymphoid follicles and expansion of red pulp, a greater
51 anization of immune cell types and states in lymphoid follicles and extrafollicular zones.
52 rely ill mice was characterized by disrupted lymphoid follicles and fragmented nuclei, while the sple
53 mph nodes and had a defect in forming normal lymphoid follicles and germinal centers in spleen and ly
54  cells near the corticomedullary junction in lymphoid follicles and in the subcapsular region.
55 umbers and prevented CS-induced formation of lymphoid follicles and increases in immunoglobulin level
56 y which the TSE agent initially localizes to lymphoid follicles and interacts with FDCs are unknown.
57 st splenic NK cells were associated with the lymphoid follicles and marginal zone.
58 inal fluid, and presence of EBV in meningeal lymphoid follicles and perivenular infiltrates in the wh
59 ymphocytes is known to block cell entry into lymphoid follicles and promote accumulation in T cell zo
60 -derived leukocytes to areas of pre-existing lymphoid follicles and replacement of graft-resident don
61 he more inducible lymphoid tissues, isolated lymphoid follicles and tertiary lymphoid tissues, is unc
62 igration of Ag-transporting cells (ATC) into lymphoid follicles and the phenotypic similarity between
63 g high endothelial venules within intragraft lymphoid follicles and the recruitment of B cells, but n
64  VIPR1 would stabilize T cell populations in lymphoid follicles and tissue infiltrates.
65 ophy of intestinal villus and colon-resident lymphoid follicle, and degeneration and atrophy of brain
66             By contrast, though PP, isolated lymphoid follicle, and villous M cells are all derived f
67 ponses in the skin, lack intestinal isolated lymphoid follicles, and cannot control intestinal bacter
68            HIV is known to be sequestered in lymphoid follicles, and CD8+CXCR5+ T cell frequency is a
69  kinase (NIK) is highly active in intestinal lymphoid follicles, and is required for M-cell maintenan
70 g intestinal Peyer's patches (PPs), isolated lymphoid follicles, and nasal-associated lymphoid tissue
71 on, and B-cell memory formation in secondary lymphoid follicles; and (iii) antigen recognition.
72  cells; the major stromal cell components in lymphoid follicle are the follicular dendritic cells (FD
73                                              Lymphoid follicles are B-cell-rich compartments of lymph
74                                      Mucosal lymphoid follicles are covered by a specialized follicle
75 and IL-8 and marked hyperplasia of secondary lymphoid follicles are early consequences of H. pylori i
76                                      Ectopic lymphoid follicles are hallmarks of chronic autoimmune i
77 hanisms underlying the exclusion of CTL from lymphoid follicles as well as the role of lymphoid folli
78 he rectal mucosa, specialized M cells of the lymphoid follicle-associated epithelium conduct vesicula
79 y not correlate with the formation of thymic lymphoid follicles but may correlate with the expansion
80 B cells in the germinal centers of secondary lymphoid follicles, but not in the mantle zones.
81  node may have resulted from colonization of lymphoid follicles by MALT lymphoma cells, following whi
82 A class switching in systemic and intestinal lymphoid follicles by shuttling Nef from infected macrop
83 ated with emphysema severity; (4) there were lymphoid follicles (CD20(+)IgM(+)) with active B cells (
84 rved ectopic lymphoid structures, defined as lymphoid follicles comprising clusters of B lymphocytes
85 P in the small intestine; in both instances, lymphoid follicles covered by surface epithelium (dome-f
86         It comprises isolated and aggregated lymphoid follicles, cryptopatches (CPs) and tertiary lym
87 occurs only in the epithelium over organized lymphoid follicles) deliver samples of foreign material
88 shown that advanced COPD is characterized by lymphoid follicles, drawing attention to immunological m
89  along with the generation of colon-isolated lymphoid follicles driving increased fecal and serum IgA
90 ssociated osteopontin covary with changes in lymphoid follicles during acute and late stages of infec
91      Germinal centers (GCs) that form within lymphoid follicles during antibody responses are sites o
92                             Within secondary lymphoid follicles, follicular helper T (TFH) cells have
93 ry B cells and IL-27-mediated suppression of lymphoid follicle formation are all involved in governin
94 on recruits plasma cells and induces ectopic lymphoid follicle formation beneath the mucosal epitheli
95 ected against Mtb infection, and facilitated lymphoid follicle formation.
96 chimeric mice was associated with diminished lymphoid follicle formation.
97 ferentially involved germinal centers of the lymphoid follicles, forming confluent aggregates.
98 of DCs that migrate rapidly into the primary lymphoid follicles from marginal zone after immunization
99  is expressed in the alveolar epithelium and lymphoid follicles from patients with IPF, and AKAP13 mR
100            Imaging mass cytometry identifies lymphoid follicles from week 16 onwards in a villus-like
101 lthy intestine, Peyer's patches and isolated lymphoid follicles generate protective and homeostatic i
102 hronic obstructive pulmonary disease (COPD), lymphoid follicles have been associated with disease sev
103                 Increased numbers of ectopic lymphoid follicles have been observed in lungs of patien
104  in the murine small intestine, the isolated lymphoid follicle (ILF).
105 le to initiate development of CP or isolated lymphoid follicles (ILF) after transfer to CD132-null mi
106 mal cells in cryptopatches (CP) and isolated lymphoid follicles (ILF) in the small intestine of C57BL
107                                     Isolated lymphoid follicles (ILFs) are organized lymphoid structu
108                                     Isolated lymphoid follicles (ILFs) are recently appreciated membe
109          Colonic patches (CLPs) and isolated lymphoid follicles (ILFs) are two main lymphoid structur
110 ues such as cryptopatches (CPs) and isolated lymphoid follicles (ILFs) constitute steady-state activa
111         The intestine is lined with isolated lymphoid follicles (ILFs) that facilitate sampling of lu
112 tally 'imprinted' cryptopatches and isolated lymphoid follicles (ILFs), but not embryonically 'imprin
113 uce cryptopatches to transform into isolated lymphoid follicles (ILFs), which subsequently act as sit
114 ass of intestinal lymphoid tissues, isolated lymphoid follicles (ILFs).
115                     In summary, the types of lymphoid follicle in lesions from HIV-positive women wer
116 tion response of Ag delivery by B cells into lymphoid follicles in an autoimmune condition has not be
117          CXCL12 was also highly expressed in lymphoid follicles in COPD lungs, and the pulmonary expr
118 ive Th1 response, the formation of secondary lymphoid follicles in granulomas and the role of Th1 res
119 tic antral gastritis and formation of antral lymphoid follicles in H. pylori-infected animals.
120 vented the CS-induced formation of pulmonary lymphoid follicles in mice.
121 on and with the presence of plasma cells and lymphoid follicles in more severe cases.
122 tes within the germinal centers of secondary lymphoid follicles in normal and reactive nodes were TRA
123 om lymphoid follicles as well as the role of lymphoid follicles in perpetuating other chronic pathoge
124  identified in conjunctival tissue overlying lymphoid follicles in rabbits and guinea pigs.
125 ) DCs, expressing CXCR5, localize to primary lymphoid follicles in response to CXC ligand 13 (B lymph
126 2 or GPR183) directs B-cell migration in the lymphoid follicles in response to its endogenous ligands
127 ent stimulation of B cells and activation of lymphoid follicles in secondary lymphoid tissues.
128  MS, Th17 cells specifically induced ectopic lymphoid follicles in the central nervous system (CNS).
129 ceptor 2 and are mainly restricted to innate lymphoid follicles in the colon.
130 +) CD4 T cells, which were induced in innate lymphoid follicles in the colon.
131 ity of CD4+ T cells, and reduced fibrosis of lymphoid follicles in the colon.
132  cells in the submucosa and ectopic tertiary lymphoid follicles in the ectocervix and vagina; and 3)
133 of cells in the epithelium overlying mucosal lymphoid follicles in the fornix region.
134 , for the first time, we illustrate isolated lymphoid follicles in the large intestine, consisting of
135 ties such as pleuritis and the effacement of lymphoid follicles in the regional lymph nodes and splee
136 o play an important role in the formation of lymphoid follicles in the spleen.
137 model to study the formation and function of lymphoid follicles in the thyroid.
138                                    Secondary lymphoid follicles in uninfected cats were rare and posi
139  last of these exhibited elements of classic lymphoid follicles, including networks of follicular den
140 eceptor needed for lymphocyte migration into lymphoid follicles indicates that multiple chemoattracti
141                                      Mucosal lymphoid follicles, inductive sites for adaptive mucosal
142                                          The lymphoid follicle is a specialized microenvironment for
143 13, a critical chemokine for B-cell entry to lymphoid follicles, is one of the most highly up-regulat
144                                              Lymphoid follicles (LFs) in AATD and usual COPD were mar
145 racterize the transcriptomic signatures from lymphoid follicles (LFs) in ever-smokers without COPD an
146 is known about what drives the appearance of lymphoid follicles (LFs), which may function as lymphoid
147 at of Peyer's patches (PP), mucosal isolated lymphoid follicles (M-ILF), and submucosal ILF (SM-ILF).
148 replication is concentrated, suggesting that lymphoid follicles may be immune-privileged sites.
149 anced generation of IgA(+) cells in isolated lymphoid follicles of intestines offset defective intest
150                    CXCL13 is produced within lymphoid follicles of patients with COPD and is crucial
151 ression of BAFF has been demonstrated within lymphoid follicles of patients with severe COPD.
152 zed epithelia known as M cells overlying the lymphoid follicles of Peyer's patches are important in t
153           The amount of proliferation in the lymphoid follicles of the appendix estimated by in vivo
154 s, and they are located predominantly in the lymphoid follicles of the spleen and the lymph nodes.
155 neously in trigeminal ganglionic neurons and lymphoid follicles of tonsil.
156 issue and lead to the formation of inducible lymphoid follicles or aggregates that can mediate local
157 b2 as a nonredundant regulator that controls lymphoid follicle organization and germinal center react
158 ecursors was identified, suggesting that the lymphoid follicles represent newly formed, ectopic lymph
159                           In formula-fed the lymphoid follicle size (p < 0.01) and germinal centers (
160 ne system in submucosal and mucosal isolated lymphoid follicles (SM-ILFs and M-ILFs, respectively) as
161 e the formation and persistence of meningeal lymphoid follicles suggest persistence of antigens to dr
162 issue-specific antiviral immune responses in lymphoid follicles that limit SIV replication in this pa
163 sustains the production of germinal centers, lymphoid follicles that ordinarily are anatomical signat
164 ity maturation occurs in germinal centers in lymphoid follicles through a critical interaction betwee
165 te to disease progression and development of lymphoid follicles via activation of CXCL12.
166 ntly, another subset of T cells in secondary lymphoid follicles was described, follicular regulatory
167                             BAFF staining in lymphoid follicles was observed around B cells, CD4(+) c
168 ity, degree of inflammation, and presence of lymphoid follicles was observed.
169                       Development of ectopic lymphoid follicles was partly dependent on the cytokine
170                                              Lymphoid follicles were characterized by a 2- to 3-fold
171                                     Distinct lymphoid follicles were increased in the RAJ of vaccinat
172                                              Lymphoid follicles were present in 9.5% of normal cervix
173 obule leukocytes and numerous active mucosal lymphoid follicles were present in infected animals.
174 eaths (PALS) and subsequently migrate to the lymphoid follicle where they enter nascent germinal cent
175 phoid tissues, but fail to accumulate within lymphoid follicles where HIV-1 replication is concentrat
176      Over the course of HIV-1 infection, the lymphoid follicles where the humoral immune response is
177  these is a unique lymphoid tissue, isolated lymphoid follicles, which shares properties of both Peye
178                                              Lymphoid follicles with follicular colonization were see
179 ated and typically leads to the formation of lymphoid follicles with germinal center (GC) reactions.
180                            In seven samples, lymphoid follicles with germinal center formation were d
181 rs examined and clear evidence of extranodal lymphoid follicles with germinal center-like architectur
182  In a series of 64 synovial tissue biopsies, lymphoid follicles with germinal centers (GCs) were foun
183                  Lymphocytic infiltrates and lymphoid follicles with germinal centers are often detec
184 es in infection and in the reconstitution of lymphoid follicles with treatment and that mapping genes
185 ate immunity and the formation of protective lymphoid follicles within granulomas.
186 dtype hosts mediates B cell recruitment into lymphoid follicles within the allograft, resulting in a
187 issue inflammation, characterized by ectopic lymphoid follicles within the target organ.
188  selectively localized to the mantle zone of lymphoid follicles within the thyroid gland.

 
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