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1 tion is associated with moderate, multifocal lymphoplasmacytic ampullitis comprising clusters of B (C
2                    We demonstrate a systemic lymphoplasmacytic and histiocytic arteritis and periarte
3 erized by expansion of the lamina propria by lymphoplasmacytic and histiocytic infiltrates.
4 of mice is affected by AIH, characterized by lymphoplasmacytic and periportal hepatic infiltrates, au
5           A significant fraction of thoracic lymphoplasmacytic aortitis cases, about 40% of inflammat
6 immune pancreatitis, sclerosing cholangitis, lymphoplasmacytic aortitis, salivary gland involvement,
7 ma characterized by elevated serum IgM and a lymphoplasmacytic bone marrow infiltrate.
8 ortem lung examination of sibling 2 revealed lymphoplasmacytic bronchiolitis with intraluminal purule
9  variation in disease progression, including lymphoplasmacytic bronchiolitis, BOOP, and death.
10 least in part to the presence of a prominent lymphoplasmacytic cell infiltrate in the tumor stroma, w
11 east in part, to the presence of a prominent lymphoplasmacytic cell infiltrate in the tumor stroma.
12                             We studied the B lymphoplasmacytic cell infiltrates in MCB to determine t
13 pathy and bone marrow (BM) infiltration with lymphoplasmacytic cells (LPCs).
14 posed of both small B lymphocytes and larger lymphoplasmacytic cells and plasma cells: CD3(-), CD19(+
15                           Tumor-infiltrating lymphoplasmacytic cells are a key feature of medullary c
16 dy response produced by tumor-infiltrating B lymphoplasmacytic cells are autoimmune in nature and a c
17 terface hepatitis; (2) acidophil bodies with lymphoplasmacytic cells are seen in early recurrent AIH;
18                                 We genotyped lymphoplasmacytic cells from 175 WM patients and observe
19                  We sequenced sorted CD19(+) lymphoplasmacytic cells from 6 WM patients who progresse
20 k factors for progression were percentage of lymphoplasmacytic cells in the bone marrow, size of the
21 d confers prosurvival signaling in malignant lymphoplasmacytic cells in Waldenstrom macroglobulinemia
22 isorder characterized by the infiltration of lymphoplasmacytic cells into bone marrow and the presenc
23 ainst actin produced by tumor-infiltrating B lymphoplasmacytic cells is Ag-driven, affinity-matured,
24 braries were generated from MCB-infiltrating lymphoplasmacytic cells of two patients, and MCB-reactiv
25 he infiltration of the bone marrow by clonal lymphoplasmacytic cells that produce monoclonal immunogl
26 levels raises issues on interactions between lymphoplasmacytic cells, mast cells, and endothelial cel
27 ion of clonal immunoglobulin (Ig)M-secreting lymphoplasmacytic cells.
28 ne marrow, of clonally related IgM-secreting lymphoplasmacytic cells.
29 one marrow (BM) involvement of IgM secreting lymphoplasmacytic cells.
30 with CD20+, IgM+, and monotypic light chain+ lymphoplasmacytic cells.
31 otubules and with clinical associations with lymphoplasmacytic disorders.
32 phoid neoplasm characterised by a monoclonal lymphoplasmacytic expansion accompanied by a serum monoc
33 tion of the main stomach revealed multifocal lymphoplasmacytic gastritis with silver-stained spiral-s
34                                   Multifocal lymphoplasmacytic hepatitis was observed in 9 of 17, 15
35 ed with wild type exhibited moderate lobular lymphoplasmacytic hepatitis with hepatocytic coagulative
36 istortion of the crypts; (b) dense and basal lymphoplasmacytic infiltrate and (c) scarcity of eosinop
37 rane by electron microscopy and a pronounced lymphoplasmacytic infiltrate by histologic examination.
38                IgG4-RD is characterized by a lymphoplasmacytic infiltrate composed of IgG4(+) plasma
39 ated disease (IgG4-RD) is characterized by a lymphoplasmacytic infiltrate composed of IgG4(+) plasma
40 ined as a B-cell neoplasm characterized by a lymphoplasmacytic infiltrate in bone marrow (BM) and IgM
41  which affected organs have a characteristic lymphoplasmacytic infiltrate rich in IgG4-positive cells
42  At the time of aortic surgery, a transmural lymphoplasmacytic infiltrate was detected in the patient
43 sease is characterized by a dense polyclonal lymphoplasmacytic infiltrate, and is frequently associat
44 vealed moderate cervical lymphadenopathy and lymphoplasmacytic infiltrate, but these were less extens
45                    Human Abs cloned from MCB lymphoplasmacytic infiltrate-derived phage display libra
46  lobular hepatitis with acidophil bodies and lymphoplasmacytic infiltrate.
47 nority of breast cancers is characterized by lymphoplasmacytic infiltrates that have been correlated
48 losed moderate intraparenchymal fibrosis and lymphoplasmacytic infiltrates without lymphoid follicles
49                                      Diffuse lymphoplasmacytic infiltrates, presence of abundant IgG4
50 he multivariate analysis demonstrated tissue lymphoplasmacytic infiltration (OR 88.38, 95% CI 7.98-97
51 obulin M 4,500 mg/dL or greater, bone marrow lymphoplasmacytic infiltration 70% or greater, beta2-mic
52 gM protein>/=3 g/dL and/or>/=10% bone marrow lymphoplasmacytic infiltration but no evidence of end-or
53 histopathological features of IgG4-RHP are a lymphoplasmacytic infiltration of IgG4-positive plasma c
54     WM is a B-cell neoplasm characterized by lymphoplasmacytic infiltration of the bone marrow and a
55 is characterized by a predominant polyclonal lymphoplasmacytic infiltration, and is frequently refrac
56 thology features of IgG4-related disease are lymphoplasmacytic infiltration, storiform fibrosis, and
57 c presentation was rectal ulcer (61%). Basal lymphoplasmacytic inflammation and mild crypt distortion
58                                   Sclerosing lymphoplasmacytic inflammation at almost any site can re
59                                     AIH-like lymphoplasmacytic inflammation with mild fibrosis was th
60 patitic" pattern of injury, characterized by lymphoplasmacytic inflammation with necroinflammatory ac
61                          Minimal to moderate lymphoplasmacytic inflammation, with a predilection for
62 ncommon and was seen in 42% of patients with lymphoplasmacytic lobular, portal, and interface hepatit
63                                              Lymphoplasmacytic lymphoma (LPL) is an incurable low-gra
64                                              Lymphoplasmacytic lymphoma (LPL) is characterized by t(9
65      CD52 is expressed on malignant cells in lymphoplasmacytic lymphoma (LPL), including IgM-secretin
66 oglobulinemia is an incurable, IgM-secreting lymphoplasmacytic lymphoma (LPL).
67  mutation in 28.2% (37/131) of patients with lymphoplasmacytic lymphoma (Waldenstrom macroglobulinemi
68  144 months after diagnosis, in 1 patient to lymphoplasmacytic lymphoma and in 2 patients to multiple
69 igration of chronic lymphocytic leukemia and lymphoplasmacytic lymphoma cells, two other B cell malig
70 aldenstrom macroglobulinemia (WM) is a rare, lymphoplasmacytic lymphoma characterized by hypersecreti
71  transformation and leveraged these to model lymphoplasmacytic lymphoma in mice, based on mutated MYD
72 ld Caucasian male with a history of relapsed lymphoplasmacytic lymphoma was admitted to the hospital
73 strom macroglobulinemia (WM) is an incurable lymphoplasmacytic lymphoma with limited options of thera
74 phoma (28), marginal-zone lymphoma (15), and lymphoplasmacytic lymphoma with or without Waldenstrom's
75 d tissue marginal zone lymphoma, and 38 with lymphoplasmacytic lymphoma) who were randomly assigned t
76 age in elderly individuals and patients with lymphoplasmacytic lymphoma, a singular disease for study
77 acroglobulinemia (WM), a distinct subtype of lymphoplasmacytic lymphoma, early data on limited sequen
78 om macroglobulinemia (WM), an IgM-associated lymphoplasmacytic lymphoma, has witnessed several practi
79  specifically, chronic lymphocytic leukemia, lymphoplasmacytic lymphoma, marginal zone lymphoma, and
80 L), 13 had follicular lymphoma (FL), six had lymphoplasmacytic lymphoma, six had Hodgkin's disease (H
81 ent of Waldenstrom macroglobulinemia (WM), a lymphoplasmacytic lymphoma.
82 oglobulinemia (WM) is an incurable low-grade lymphoplasmacytic lymphoma.
83 ), multiple myeloma (MM; RR = 2.9; 1.9-4.3), lymphoplasmacytic lymphoma/Waldenstrom macroglobulinemia
84 r a better definition of the new category of lymphoplasmacytic lymphoma; the variants of mantle-cell
85 ion was 37 marginal zone lymphomas (MZLs), 2 lymphoplasmacytic lymphomas, 2 follicular lymphomas, 4 C
86  mucosa-associated lymphoid tissue lymphoma, lymphoplasmacytic lymphomas, and small lymphocytic leuke
87  considered an obligate precursor to several lymphoplasmacytic malignancies, including immunoglobulin
88 sentative microscopic lesions were gastritis lymphoplasmacytic, moderate, multifocal, with perivascul
89 aracterized by cyclin D1 up-regulation and a lymphoplasmacytic morphology, but is also more frequentl
90  myeloma, where it is also associated with a lymphoplasmacytic morphology.
91  9 cats), lymphocytic hepatitis (in 6 cats), lymphoplasmacytic myocarditis (in 8 cats), and interstit
92 g follicular (n = 9), marginal zone (n = 4), lymphoplasmacytic (n = 1), or small lymphocytic lymphoma
93 ation) as having lymphoplasmacytoid (n =92), lymphoplasmacytic (n = 24), polymorphous (n = 9), or und
94 s type I [24 of 25 (96%)], with interstitial lymphoplasmacytic nephritis [23 of 25 (92%)], and glomer
95 ificant differences were observed in overall lymphoplasmacytic or mast cell densities, nor in B cell
96  by the dominant clones in the oligoclonal B lymphoplasmacytic response in both patients was not a ca
97 itis (AIP) has been divided into subtypes 1 (lymphoplasmacytic sclerosing pancreatitis) and 2 (idiopa
98  from patients with follicular, mantle cell, lymphoplasmacytic, small lymphocytic, or marginal zone l