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1  (treatment with cuprizone and injections of lysolecithin).
2 nd spinal cord lesions of mice injected with lysolecithin.
3  was overcome by exposing cells to palmitoyl lysolecithin.
4 ndividually immediately before ICSI by using lysolecithin, a hydrolysis product of membrane phospholi
5 bition, leading to localized accumulation of lysolecithin, a known demyelinating agent and receptor-m
6 mary, age-exacerbated acute injury following lysolecithin administration is mediated in part by micro
7 TPase molecules from isolated membranes with lysolecithin, all behaved similar to the native enzyme w
8                                              Lysolecithin also activated cytosolic phospholipase A2 (
9                                              Lysolecithin also causes "unwinding" of paranodes: The s
10 increased ChAT-GFP expression following both lysolecithin and CPZ demyelination.
11 sceptible when sufficient reaction products (lysolecithin and fatty acid) accumulate in the membrane.
12 s to examine the individual contributions of lysolecithin and palmitic acid to the susceptibility of
13 odels (induced by proinflammatory cytokines, lysolecithin, and cuprizone) are associated with strongl
14 er demyelination by intraneural injection of lysolecithin, and during remyelination, the subcellular
15 identified on the intact spinal cord in both lysolecithin- and EAE-mediated demyelination models.
16 ion of TLR2 also enhances remyelination in a lysolecithin animal model.
17 e a type of lysophospholipase (LysoPLA) with lysolecithin as its physiological substrate.
18 ion of this enzyme attenuated the ability of lysolecithin (but not ionomycin) to induce susceptibilit
19                       Intraneurally injected lysolecithin causes both segmental and paranodal demyeli
20 tion of TSC in the remyelination of a focal, lysolecithin-demyelinated lesion in adult male mice.
21 loside treatment to reduce CSPG synthesis in lysolecithin-demyelinated mice increased numbers of OPCs
22                                    Following lysolecithin demyelination in middle-aged mice, indapami
23                   We demonstrate that in the lysolecithin demyelination model in young and middle-age
24  together at low calcium, and the effects of lysolecithin dominated at high calcium.
25 te lineage cells and their susceptibility to lysolecithin exposure could be captured by live imaging.
26 ate substrate of the standard NTE assay with lysolecithin for an "NTE-LysoPLA" assay with four import
27 ng a model of focal demyelination induced by lysolecithin in the corpus callosum of adult mice.
28                                              Lysolecithin increased bilayer polarity and the rate of
29 accelerated oligodendroglial regeneration in lysolecithin-induced corpus callosum demyelinative lesio
30                                        Using lysolecithin-induced demyelinating injury to the mouse s
31 OLs but was critical for remyelination after lysolecithin-induced demyelinating injury.
32 res, we investigated whether myelination and lysolecithin-induced demyelination affect axonal mitocho
33  adult mice and improves remyelination after lysolecithin-induced demyelination and cognitive functio
34 nist antibody promotes remyelination in both lysolecithin-induced demyelination and experimental auto
35  mimics enhance myelin restoration following lysolecithin-induced demyelination as well as experiment
36 x17 overexpression prevented cell loss after lysolecithin-induced demyelination by increasing Olig2+
37 emyelination in the toxic nonimmune model of lysolecithin-induced demyelination can be enhanced by ma
38  of central nervous system (CNS) axons after lysolecithin-induced demyelination in the spinal cord.
39                                        After lysolecithin-induced demyelination of corpus callosum, h
40 lial cell maturation and remyelination after lysolecithin-induced demyelination of organotypic cerebe
41  astrocyte activation on remyelination after lysolecithin-induced demyelination of spinal cord white
42                                        After lysolecithin-induced demyelination of the male mouse ven
43                  When mice were subjected to lysolecithin-induced demyelination of the spinal cord, s
44 y and robust upregulation of CSPGs following lysolecithin-induced demyelination was cleared during re
45 ifferentiation of oligodendrocytes following lysolecithin-induced demyelination, although apparently
46 luronidase inhibited remyelination following lysolecithin-induced demyelination.
47  endogenous PEDF in the corpus callosum upon lysolecithin-induced demyelination.
48 ed and uninjured axons following intraspinal lysolecithin-induced demyelination.
49                                     Applying lysolecithin-induced focal demyelinating spinal cord les
50 n mouse central nervous system lesions after lysolecithin-induced focal demyelination.
51 stently, excess Gal3 accumulated in OxPC and lysolecithin-induced focal spinal cord white matter (SCW
52                   Moreover, myelin repair of lysolecithin-induced lesions is delayed in PIKE(-/-) bra
53 odendrocytes and enhances remyelination in a lysolecithin-induced mouse model of focal demyelination.
54 th HGF markedly accelerated remyelination in lysolecithin-induced rat dorsal spinal cord lesions and
55 m of hyaluronan inhibits remyelination after lysolecithin-induced white matter demyelination.
56             Together these data suggest that lysolecithin induces susceptibility through both cPLA2-d
57      We evaluated the expression of CSPGs in lysolecithin-injected mouse spinal cord, an animal model
58  increased significantly 3 and 5 weeks after lysolecithin injection in the spinal cord.
59 del of central nervous system demyelination, lysolecithin injection into the spinal cord white matter
60 gand-mediated muscarinic signaling following lysolecithin injection, we administered neostigmine, a c
61 ion of focal demyelinated lesions induced by lysolecithin injections.
62      Induction of membrane susceptibility by lysolecithin involved an increase in cytosolic calcium a
63 ogical inhibition of Cdk5 inhibits repair of lysolecithin lesions.
64 s and rheology of adsorbed layers containing lysolecithin (LL), monoolein (MG), or glyceryl polyethyl
65 nic high fat consumption, after focal toxin [lysolecithin (LL)]-mediated demyelinating injury, and in
66 in reactive gliosis in corpus callosum after lysolecithin (LPC)-induced focal demyelination and in cu
67 Following a focal demyelination induced with lysolecithin, many of the BAG-labeled cells differentiat
68                                    Moreover, lysolecithin-mediated demyelination in mice deficient in
69    In contrast, in the efficiently repairing lysolecithin model of demyelination (astrocyte-free), ne
70 ferent types of MS lesions and in the murine lysolecithin model of demyelination.
71  during remyelination were performed using a lysolecithin model, coupled with lentiviral misexpressio
72 roglial response during remyelination in the lysolecithin mouse model using single-cell RNA sequencin
73                                         In a lysolecithin neuroinflammation mouse model, we detected
74                       The effect of OxLDL or lysolecithin on endothelial PG was abolished in the pres
75  models of myelin injury and repair, that is lysolecithin or cuprizone-mediated demyelination, showed
76                                              Lysolecithin or ionomycin caused concurrent hydrolysis o
77 tly released from the cells upon addition of lysolecithin or ionomycin.
78                                  Finally, in lysolecithin-permeabilized cells, the synthesis of full-
79 as labeled with bromouridine triphosphate in lysolecithin-permeabilized MHV-infected cells.
80    In contrast, un-ionized palmitic acid and lysolecithin promoted hydrolysis by augmenting a step di
81                       Palmitic acid, but not lysolecithin, promoted the binding of phospholipase A2 t
82                                              Lysolecithin reduced the ability of fatty acid to enhanc
83 dothelial cells with oxidized LDL (OxLDL) or lysolecithin resulted in decreased matrix proteoglycans
84 g agents in both chronic cuprizone and acute lysolecithin rodent animal models.
85  Finally, experimental distances between the lysolecithin spin and each single spin site on SBL1 were
86  on soybean seed lipoxygenase-1 (SBL1) and a lysolecithin spin-labeled on choline were measured by pu
87 from the basolateral than the apical side of lysolecithin-stimulated polarized endothelial cells.
88      In coculture experiments, lipolysis and lysolecithin stimulation of endothelial cells increased
89 ecombinant protein preferentially hydrolyzes lysolecithin, suggesting that this enzyme may be a type
90 sal columns of adult rats were injected with lysolecithin to induce a local demyelinating lesion.
91                                      We used lysolecithin to induce demyelination in white matter of
92  localized the polar-end and the spin of the lysolecithin to the region between the two domains in th
93 , Sandhoff and Niemann-Pick Type-C1, and the lysolecithin toxin model of focal demyelination.
94 emyelinating injuries triggered acutely with lysolecithin, TREM2 agonist antibodies unexpectedly disr
95                     This kinetic response to lysolecithin was calcium-dependent.
96 -) mice using stereotactic microinjection of lysolecithin were larger than in controls, and remyelina
97  product was observed when palmitic acid and lysolecithin were present together at low calcium, and t