戻る
「早戻しボタン」を押すと検索画面に戻ります。 [閉じる]

コーパス検索結果 (１語後でソート)

通し番号をクリックするとPubMedの該当ページを表示します
1 se (the more severe case also presented with macrocytic anemia).
2 in SFXN4 as a cause of mitochondriopathy and macrocytic anemia.
3  of the MDS progenitors, which corrected the macrocytic anemia.
4 neutrophils and monocytes, associated with a macrocytic anemia.
5 ia, a disorder characterized by hypoplastic, macrocytic anemia.
6 are responsible for Pearsons syndrome with a macrocytic anemia.
7 characterized by vitamin B-12 deficiency and macrocytic anemia.
8 ly with multiple female individuals who have macrocytic anemia.
9 iculocytes, hence recapitulating features of macrocytic anemia.
10 ythroid differentiation because they develop macrocytic anemia.
11                    STM infection resulted in macrocytic anemia and enhanced splenomegaly with destruc
12  Tc1 mice do not develop leukemia, they have macrocytic anemia and increased extramedullary hematopoi
13 ications of vitamin B-12 deficiency, such as macrocytic anemia and neurological complications affecti
14 duced serum vitamin B12 concentrations, mild macrocytic anemia, and fecal loss of Na+ and K+, the lat
15 ion in deep brain nuclei, mild normocytic to macrocytic anemia, and gastrointestinal symptoms.
16 ad to many clinical abnormalities, including macrocytic anemia, cardiovascular diseases, birth defect
17  mechanism is underscored in 5q- syndrome, a macrocytic anemia caused by a large monoallelic deletion
18  is characterized by red blood cell aplasia, macrocytic anemia, clinical heterogeneity, and increased
19                    Sf3b1(K700E) mice develop macrocytic anemia due to a terminal erythroid maturation
20 cartilage, and pulmonary lesions, as well as macrocytic anemia, during the treatment process for feve
21 inal features of the 5q- syndrome, including macrocytic anemia, erythroid hypoplasia, and megakaryocy
22 unction could contribute to the emergence of macrocytic anemias, for instance, due to nutritional def
23 cking the heme exporter FLVCR1 have a severe macrocytic anemia; however, the mechanisms that underlie
24 mitochondrial respiration and development of macrocytic anemia in mtDNA-mutator mice.
25              A 47-year-old woman with severe macrocytic anemia markedly improved during the second an
26                                              Macrocytic anemia occurring in patients with fatigue sug
27        Classical 5q- syndrome is an acquired macrocytic anemia of the elderly.
28 s, was causally associated with a pernicious/macrocytic anemia pattern, i.e. decreased erythrocyte co
29 , encoding the ribosomal protein S14) caused macrocytic anemia, prominent erythroid dysplasia and mon
30  of the mtDNA polymerase, PolgA, and develop macrocytic anemia similar to that of patients with MDS.
31            The disease is characterized by a macrocytic anemia that remains poorly understood.
32                   In this group, the risk of macrocytic anemia was 63% lower than in the placebo grou
33 hat mutant animals develop an age-dependent, macrocytic anemia with abnormal erythroid maturation and
34                                              Macrocytic anemia with abnormal erythropoiesis is a comm
35  defect observed in both individuals and the macrocytic anemia with megaloblastic features of the mor
36 pc (del/+)) accelerated the development of a macrocytic anemia with monocytosis, early features of t-
37  that is deleted neonatally develop a severe macrocytic anemia with proerythroblast maturation arrest