ライフサイエンスコーパス: macrosomia

1 e, with implications for the pathogenesis of macrosomia.

2 loss, maternal glucose intolerance and fetal macrosomia.

3 th SGA(2SD) and did not decrease the odds of macrosomia.

4 d with SGA(2SD) but did decrease the odds of macrosomia.

5 yperinsulinemic hypoglycemia associated with macrosomia.

6 yperinsulinemic hypoglycemia associated with macrosomia.

7 t outcomes, including preeclampsia and fetal macrosomia.

8 ted fetal development with increased risk of macrosomia.

9 ia appears not to be the sole cause of fetal macrosomia.

10 syndrome (LOS/AOS) which is characterized by macrosomia.

11 nancy, preterm birth, cesarean delivery, and macrosomia.

12 < 0.001), PTB (1.02; 1.01-1.03; P < 0.001), macrosomia (1.07; 1.06-1.08; P < 0.001), SGA (1.06; 1.02

13 ies, 90 044 participants; tau(2)=0.009), and macrosomia (1.52, 1.33 to 1.73; 29 studies, 68 138 parti

14 tions were associated with decreased risk of macrosomia (25 trials, n = 13 990; RR, 0.77 [95% CI, 0.6

15 ), LGA (-2.3% [95% CI, -2.8% to -1.8%]), and macrosomia (-4.7% [95% CI, -5.3% to -4.0%]).

16 ood glucose concentrations may contribute to macrosomia, adiposity, and poorer vascular health in the

17 isk of all assessed outcomes, except LGA and macrosomia; American Indian individuals were at signific

18 llitus (GDM), caesarean section (C-section), macrosomia and large for gestational age (LGA) babies.

19 tational age, preterm and postterm birth, or macrosomia and LGA.

20 ic status) and the effects of exclusion (for macrosomia and maternal diabetes) to be examined.

21 Three infants presented with macrosomia and severe hypoglycemia with a positive famil

22 enhanced maternal glucose intolerance, fetal macrosomia, and a long-lasting transgenerational alterat

23 h, small or large for gestational age (SGA), macrosomia, and birth length or head circumference (HC)

24 ampsia, cesarean delivery, preterm delivery, macrosomia, and congenital defects.

25 l in mothers and birth weight, birth length, macrosomia, and large for gestational age in neonates.

26 ional diabetes, emergency cesarean delivery, macrosomia, and large for gestational age.

27 , low 5-minute Apgar score (<7) after birth, macrosomia, and shoulder dystocia.

28 comes: placental weight, head circumference, macrosomia, Apgar score, small for gestational age, larg

29 delivery (aRR, 1.09; 95% CI, 0.99-1.20) and macrosomia (aRR, 1.13; 95% CI, 0.93-1.37) were increased

30 .14, 0.78 per 1-mmol/L increase) and risk of macrosomia (birth weight >4000 g) (RR = 1.21; 95% CI: 1.

31 centile) had >50% sensitivity for predicting macrosomia (birthweight above 4,000 g or 90th centile) a

32 e-ethnicity-specific mean BW (SGA(2SD)), and macrosomia (BW ge 4500 g).

33 s of women with deflation had a high risk of macrosomia compared with controls (adjusted RR 0.40, p=0

34 rcent and 13 percent, respectively); who had macrosomia, defined as a birth weight of 4000 g or more

35 ciated with embryonal cancers, macroglossia, macrosomia, ear pits or ear creases, and midline abdomin

36 es included large for gestational age (LGA), macrosomia (>4000 g at birth), small for gestational age

37 weight, participants who had a sibling with macrosomia had a lower mean IQ score.

38 in less preeclampsia, shoulder dystocia, and macrosomia; however, current evidence does not show an e

39 Seven percent of neonatal macrosomia in all the population, and 13% in Black mothe

40 ases of preeclampsia, shoulder dystocia, and macrosomia in the treated group.

41 s during a singleton pregnancy could predict macrosomia in their newborns.

42 f long-term hyperglycemic exposure, predicts macrosomia in their newborns.

43 aternal exercise prevented fetal overgrowth (macrosomia) induced by MO.

44 e neonatal death, large for gestational age, macrosomia, infant birth injury, hypoglycemia, respirato

45 ery for ultrasonographically diagnosed fetal macrosomia is medically and economically unsound.

46 The effectiveness of screening for macrosomia is not well established.

47 luding admission to neonatal intensive care, macrosomia, low Apgar scores, and perinatal death.

48 (ii) Perinatal outcomes: macrosomia, low birth weight, admission to neonatal inte

49 and surrounding genes increases the risk for macrosomia, mild developmental delay and pervasive devel

50 Adverse perinatal outcomes included macrosomia (multiple thresholds), shoulder dystocia, and

51 The odds of macrosomia (odds ratio [OR] 0.59, 95% CI 0.46 to 0.77, p

52 .10-106.31, p = 0.02) with increased risk of macrosomia (odds ratio [OR] 1.38, 95% CI 1.01-1.89, p =

53 to -33.06 g, p < 0.001) with reduced risk of macrosomia (OR 0.60, 95% CI 0.45-0.79, p < 0.001) than i

54 aginal birth (OR, 0.87 [95% CI, 0.79-0.97]), macrosomia (OR, 0.66 [95% CI, 0.48-0.91]), and low 5-min

55 low birth weight, P < .05; 7.7% vs 14.6% for macrosomia, P < .05).

56 reported [1 study]; and 0% vs 2.6%-4.3% for macrosomia, P = not reported [1 study] and P = .28 [1 st

57 t gain, age, parity, smoking, and history of macrosomia resulted in an area under the curve (AUC) of

58 (PTB), fetal growth restriction (FGR) and/or macrosomia resulting from gestational diabetes (GDM).

59 < 0.001) with a nonsignificant reduction in macrosomia risk (OR 0.32, 95% CI 0.08-1.19, I2 = 0%, p =

60 a (RR, 0.42 [95% CI, 0.23-0.77]; ARD, 1.3%), macrosomia (RR, 0.53 [95% CI, 0.41-0.68]; ARD, 8.9%), la

61 en FPG levels and spontaneous abortion, PTB, macrosomia, SGA, and perinatal infant death (P for trend

62 tcomes, including spontaneous abortion, PTB, macrosomia, SGA, and perinatal infant death.

63 g spontaneous abortion, preterm birth (PTB), macrosomia, small for gestational age infant (SGA), birt

64 irth outcomes, i.e., low birth weight (LBW), macrosomia, small-for-gestational-age (SGA), large-for-g

65 Late gestational macrosomia was absent, apparently requiring a different

66 Macrosomia was defined as birth weight >=4,000 g and/or

67 tic regression, the relation between sAF and macrosomia was significant (odds ratio 4.13 for 1-AU inc

68 ents with midline abdominal-wall defects and macrosomia was significantly higher, 65% (41/63) and 60%

69 hypertension, waist circumference, and fetal macrosomia were significantly associated with T2D (p = 0

70 complications, including preeclampsia, fetal macrosomia (which can cause shoulder dystocia and birth

71 was significant heterogeneity at predicting macrosomia, which could reflect the different study desi

72 aevia, assisted reproductive technology use, macrosomia with a birthweight of more than 4500 g, and s

73 elivered at term via cesarean section due to macrosomia, with a reported birth weight of 11 lb 8.7 oz