ライフサイエンスコーパス: magnetic resonance spectroscopy

1 n both the dorsal and ventral striatum using magnetic resonance spectroscopy.

2 lipoprotein subclass profiling from nuclear magnetic resonance spectroscopy.

3 uid chromatography and identified by nuclear magnetic resonance spectroscopy.

4 tu X-ray diffraction and solid-state nuclear magnetic resonance spectroscopy.

5 were assessed by MRI and single voxel proton magnetic resonance spectroscopy.

6 ure spectroscopy, and solution (31)P nuclear magnetic resonance spectroscopy.

7 covery half-times (PCr t(1/2)) on phosphorus magnetic resonance spectroscopy.

8 ysis, thermogravimetric analysis and nuclear magnetic resonance spectroscopy.

9 cterized using X-ray diffraction and nuclear magnetic resonance spectroscopy.

10 mass spectrometry, and infrared and nuclear magnetic resonance spectroscopy.

11 as measured by ex vivo (1)H-[(13)C]-nuclear magnetic resonance spectroscopy.

12 equential (2)H and (31)P solid-state nuclear magnetic resonance spectroscopy.

13 ate of glutamate and glutamine) levels using magnetic resonance spectroscopy.

14 ned using (1)H and (7)Li solid-state nuclear magnetic resonance spectroscopy.

15 ing solid-state magic angle spinning nuclear magnetic resonance spectroscopy.

16 igh resolution mass spectrometry and nuclear magnetic resonance spectroscopy.

17 vivo lowered Glu levels as measured by (1)H magnetic resonance spectroscopy.

18 roscopic resolution using zero-field nuclear magnetic resonance spectroscopy.

19 ulating amino acids were assessed by nuclear magnetic resonance spectroscopy.

20 electron paramagnetic resonance, and nuclear magnetic resonance spectroscopy.

21 ssed with the use of high-throughput nuclear magnetic resonance spectroscopy.

22 try and lipidome analysis using (1)H nuclear magnetic resonance spectroscopy.

23 ime-resolved fluorescence, and (19)F nuclear magnetic resonance spectroscopy.

24 ortical nuclei/regions with 1.5-tesla proton magnetic resonance spectroscopy.

25 re compared via 4-tesla proton single volume magnetic resonance spectroscopy.

26 determinants that are identified by nuclear magnetic resonance spectroscopy.

27 by using in vivo saturation transfer [(31)P]-magnetic resonance spectroscopy.

28 ometry, tandem mass spectrometry and nuclear magnetic resonance spectroscopy.

29 ultrahigh-field zinc-67 solid-state nuclear magnetic resonance spectroscopy.

30 echniques such as crystallography or nuclear magnetic resonance spectroscopy.

31 the left putamen using ultra-high-field (7T) magnetic resonance spectroscopy.

32 scopy, UV-vis spectroscopy, and (1)H nuclear magnetic resonance spectroscopy.

33 semblies were fully characterized by nuclear magnetic resonance spectroscopy.

34 n/deuterium fractionation factors by nuclear magnetic resonance spectroscopy.

35 atography with mass spectrometry and nuclear magnetic resonance spectroscopy.

36 ential scanning calorimetry and (2)H nuclear magnetic resonance spectroscopy.

37 mox, using hyperpolarized Carbon-13 ((13) C) magnetic resonance spectroscopy.

38 , molecular dynamics simulations and nuclear magnetic resonance spectroscopy.

39 nd ultraviolet visible, infrared and nuclear magnetic resonance spectroscopies.

40 oholic fatty liver disease (NAFLD) by proton magnetic resonance spectroscopy ((1) H-MRS) was establis

41 r 1:1 binding properties by means of nuclear magnetic resonance spectroscopy ((1)H and (31)P NMR), is

42 BA+/Creatine (Cr) concentrations with proton magnetic resonance spectroscopy ((1)H MRS) in the pregen

43 C)) in bipolar disorder using in vivo proton magnetic resonance spectroscopy ((1)H MRS), especially i

44 and 20 mg/L) was evaluated by proton nuclear magnetic resonance spectroscopy ((1)H NMR) and gas chrom

45 Nuclear magnetic resonance spectroscopy ((1)H NMR) was used to m

46 Proton nuclear magnetic resonance spectroscopy ((1)H NMR), hydrophilic

47 ctrometry (native nESI-MS), and (1)H-nuclear magnetic resonance spectroscopy ((1)H NMR).

48 we address this need by presenting a proton magnetic resonance spectroscopy ((1)H-MRS) acquisition s

49 fat can be non-invasively measured by proton magnetic resonance spectroscopy ((1)H-MRS) and fibrosis

50 Here, we applied (1)H magnetic resonance spectroscopy ((1)H-MRS) at ultra-high

51 PET) and GABA concentrations by using proton magnetic resonance spectroscopy ((1)H-MRS) in 28 adults

52 ed case-control studies of high-field proton magnetic resonance spectroscopy ((1)H-MRS) investigating

53 Proton magnetic resonance spectroscopy ((1)H-MRS) studies have

54 on density fat fraction (PDFF), using proton magnetic resonance spectroscopy ((1)H-MRS), instead of c

55 ment in chronically-stressed mice using (1)H-magnetic resonance spectroscopy ((1)H-MRS).

56 and healthy full-term newborns using proton magnetic resonance spectroscopy ((1)H-MRS).

57 IHTG was determined by proton magnetic resonance spectroscopy ((1)H-MRS).

58 xperience of preclinical and clinical proton magnetic resonance spectroscopy ((1)HMRS) studies conduc

59 lucidated by one and two-dimensional nuclear magnetic resonance spectroscopy (1D and 2D NMR) and high

60 Using proton magnetic resonance spectroscopy (1H MRS), this study ass

61 In this regard, proton magnetic resonance spectroscopy (1H-MRS) has been used a

62 of affects HFC measured in vivo using proton magnetic resonance spectroscopy (1H-MRS) in healthy subj

63 Phosphorus magnetic resonance spectroscopy ((31)P-MRS), in particul

64 Phosphorus-31 magnetic resonance spectroscopy (31P MRS) allows for the

65 usion tensor imaging with diffusion-weighted magnetic resonance spectroscopy, a powerful tool capable

66 and changes in chemical shifts from nuclear magnetic resonance spectroscopy also support the above r

67 Nuclear magnetic resonance spectroscopy analysis of potent cycli

68 lar dichroism, thermal denaturation, nuclear magnetic resonance spectroscopy, analytical ultracentrif

69 sing macromolecular crystallography, nuclear magnetic resonance spectroscopy and 3D electron microsco

70 articipants undertook ultra-high field (7 T) magnetic resonance spectroscopy and a stop-signal task o

71 sex-matched healthy controls, using nuclear magnetic resonance spectroscopy and a validated ex vivo

72 ng, cross-linking mass spectrometry, nuclear magnetic resonance spectroscopy and computational modeli

73 Here we show, using nuclear magnetic resonance spectroscopy and density functional t

74 1)P solid-state magic-angle-spinning nuclear magnetic resonance spectroscopy and differential scannin

75 Pre-mortem, non-invasive magnetic resonance spectroscopy and diffusion tensor ima

76 ted and analysed using proton ((1)H)-nuclear magnetic resonance spectroscopy and direct infusion mass

77 was synthesized and characterized by nuclear magnetic resonance spectroscopy and Fourier transform in

78 n of the combined fractions was performed by Magnetic Resonance Spectroscopy and Gas Chromatography-M

79 ls and in stroke-affected C57-BL6 mice using magnetic resonance spectroscopy and GC-MS.

80 ics from 93 paired tissue-serum samples with magnetic resonance spectroscopy and identified tissue an

81 e we report operando (1)H and (23)Na nuclear magnetic resonance spectroscopy and imaging experiments

82 n, are observed and mapped by (23)Na nuclear magnetic resonance spectroscopy and imaging, and their t

83 VAT, and subcutaneous fat were determined by magnetic resonance spectroscopy and imaging.

84 argeted metabolic profiling using 1H-nuclear magnetic resonance spectroscopy and liquid chromatograph

85 le quantum coherence (2D (1)H-(13)C) nuclear magnetic resonance spectroscopy and mass spectrometry.

86 ted by column chromatography and analyzed by magnetic resonance spectroscopy and mass spectrometry.

87 GCG was carefully characterized with nuclear magnetic resonance spectroscopy and mass spectrometry.

88 cetylated by mono- and bidimensional Nuclear Magnetic Resonance spectroscopy and mass spectrometry.

89 on spectroscopic techniques, such as nuclear magnetic resonance spectroscopy and mass spectrometry.

90 s, Isothermal Titration Calorimetry, Nuclear Magnetic Resonance spectroscopy and molecular modeling.

91 lomics on two mass spectrometry, one nuclear magnetic resonance spectroscopy and one fluxomics study.

92 MDAR hypofunction affects the brain, we used magnetic resonance spectroscopy and resting-state functi

93 rescence cell wall using solid-state nuclear magnetic resonance spectroscopy and small-angle neutron

94 Nuclear magnetic resonance spectroscopy and thioacidolysis revea

95 cts were analysed using 600 MHz (1)H Nuclear Magnetic Resonance spectroscopy and Ultra-Performance Li

96 on and speciation of Tc using (99)Tc nuclear magnetic resonance spectroscopy and X-ray absorption spe

97 LFAT (by proton magnetic resonance spectroscopy) and clinical characteri

98 y water into glucose), hepatic triglyceride (magnetic resonance spectroscopy), and hepatic fat oxidat

99 tolic function, myocardial energetics ((31)P-magnetic resonance spectroscopy), and lipid content ((1)

100 tation index, lipoprotein status (by nuclear magnetic resonance spectroscopy), and nitric oxide (NO)-

101 ion (flow-mediated dilation), liver fat (MRI/magnetic resonance spectroscopy), and secondary outcomes

102 IHCL concentrations were measured by proton magnetic resonance spectroscopy, and a dual intravenous

103 rpolarized in vivo pyruvate studies, nuclear magnetic resonance spectroscopy, and carbon-13 feeding s

104 ospray ionization mass spectrometry, nuclear magnetic resonance spectroscopy, and density functional

105 Using positron emission tomography, magnetic resonance spectroscopy, and electroencephalogra

106 ephalography, magnetoencephalography, proton magnetic resonance spectroscopy, and functional magnetic

107 ins using microscale thermophoresis, nuclear magnetic resonance spectroscopy, and isothermal titratio

108 tional photophysical investigations, nuclear magnetic resonance spectroscopy, and kinetic studies to

109 easured at the start and end of the study by magnetic resonance spectroscopy, and liver fibrosis was

110 e, using vibrational and solid-state nuclear magnetic resonance spectroscopy, and molecular dynamics

111 tabolomics were evaluated using (1)H nuclear magnetic resonance spectroscopy, and multivariate analys

112 aman spectroscopy, (27)Al and (35)Cl nuclear magnetic resonance spectroscopy, and pair distribution f

113 ion, local concentrations of basal GABA with magnetic resonance spectroscopy, and RT with a behaviora

114 pport for the idea of measuring choline with magnetic resonance spectroscopy as a noninvasive way of

115 constant using (31)P magnetization transfer magnetic resonance spectroscopy as described previously.

116 itration calorimetry and solid-state nuclear magnetic resonance spectroscopy as well as bacterial kil

117 gulate cortex (ACC) were measured using 1[H]-magnetic resonance spectroscopy at 3 Tesla and analyzed

118 late cortex at 1 and 24 h post infusion with magnetic resonance spectroscopy at 7 T.

119 Proton magnetic resonance spectroscopy at 7T was performed in 2

120 in patients with schizophrenia using proton magnetic resonance spectroscopy at 7T, which allows sepa

121 We perform (1)H and (19)F nuclear magnetic resonance spectroscopy at room temperature in m

122 With the use of a (1)H nuclear magnetic resonance spectroscopy-based metabolic profilin

123 yzed using in vivo 13C/31P/1H and ex vivo 2H magnetic resonance spectroscopy before and during hyperi

124 approach thus has implications not only for magnetic resonance spectroscopy but also for the design

125 sphocreatine pool after exercise using (31)P magnetic resonance spectroscopy can provide an in vivo m

126 ere we employed high-resolution (1)H nuclear magnetic resonance spectroscopy coupled with advanced mu

127 tasets, and single volume diffusion-weighted magnetic resonance spectroscopy data from the anterior b

128 ts, and 19 unmedicated patients) contributed magnetic resonance spectroscopy data.

129 Solid-state (13) C nuclear magnetic resonance spectroscopy demonstrated that anaero

130 Nuclear magnetic resonance spectroscopy demonstrated that the N-

131 Nuclear magnetic resonance spectroscopy demonstrates the intrins

132 he CA1 hippocampal subregion and from proton magnetic resonance spectroscopy-derived hippocampal leve

133 Hyperpolarized magnetic resonance spectroscopy enables quantitative, no

134 t polymer chain ends is evidenced by nuclear magnetic resonance spectroscopy, end group analysis, and

135 volumes and function, whereas phosphorus-31 magnetic resonance spectroscopy evaluated ventricular en

136 ients with ESKD underwent phosphorus ((31)P) magnetic resonance spectroscopy examination during a 4-h

137 Phosphorus magnetic resonance spectroscopy examination of patients

138 vels at which circular dichroism and nuclear magnetic resonance spectroscopy fingerprinting, both gol

139 an electroencephalographic session for MMN, magnetic resonance spectroscopy for glutamate and GABA,

140 We used proton nuclear magnetic resonance spectroscopy for metabolic profiling

141 cardiac magnetic resonance imaging and (31)P magnetic resonance spectroscopy for myocardial energetic

142 ed by Nox1 deletion as determined by nuclear magnetic resonance spectroscopy, glucose tolerance tests

143 ative metabolomics platform based on nuclear magnetic resonance spectroscopy has found widespread use

144 While X-ray crystallography and nuclear magnetic resonance spectroscopy have revealed the struct

145 cal platforms, mass spectrometry and nuclear magnetic resonance spectroscopy, have been used to study

146 GABA levels in visual cortex, measured using magnetic resonance spectroscopy, have stronger perceptua

147 Hyperpolarized magnetic resonance spectroscopy (HP MRS) using dynamic n

148 spectrometry-solid-phase extraction-nuclear magnetic resonance spectroscopy (HR-bioassay/HPLC-HRMS-S

149 High-Resolution Magic Angle Spinning nuclear magnetic resonance spectroscopy (HR-MAS) and their progn

150 bolites, mostly amino acids, by (1)H-nuclear magnetic resonance spectroscopy in 157 white ever-smoker

151 -center study (four centers), we used proton magnetic resonance spectroscopy in 51 children with ASD,

152 Here, using (1)H- and (19)F-nuclear magnetic resonance spectroscopy in combination with deut

153 We used magnetic resonance spectroscopy in humans, to measure le

154 d glutamate and GABA levels using semi-LASER magnetic resonance spectroscopy in the right inferior fr

155 nance spectroscopy), and lipid content ((1)H-magnetic resonance spectroscopy) in the fasted state.

156 Nuclear magnetic resonance spectroscopy, in addition to nucleus-

157 sient HG bps, we used solution-state nuclear magnetic resonance spectroscopy, including measurements

158 Proton nuclear magnetic resonance spectroscopy indicates isopropoxy lig

159 equirement was assessed using hyperpolarized magnetic resonance spectroscopy indicating increased in

160 Hepatic fat was measured by proton magnetic resonance spectroscopy, insulin sensitivity ind

161 Dynamic phosphorus magnetic resonance spectroscopy is a noninvasive tool th

162 Nuclear magnetic resonance spectroscopy is a powerful tool for t

163 Mean baseline and end-of-study magnetic resonance spectroscopy liver fat percentage val

164 ts were structurally confirmed using nuclear magnetic resonance spectroscopy, matrix assisted laser d

165 GlycA is a novel proton nuclear magnetic resonance spectroscopy-measured biomarker of sy

166 e established 3-way correlations between the magnetic resonance spectroscopy measures of glutamate, e

167 Despite this, in vivo magnetic resonance spectroscopy measures of resting CK f

168 PET and magnetic resonance spectroscopy methods can also be used

169 ues used to study cerebral metabolism, (13)C magnetic resonance spectroscopy (MRS) allows following t

170 basis of noninvasive imaging tools, such as magnetic resonance spectroscopy (MRS) and magnetic reson

171 he role of magnetic resonance imaging (MRI), magnetic resonance spectroscopy (MRS) and transrectal ul

172 Metabolite levels assessed by magnetic resonance spectroscopy (MRS) are used as marker

173 r lipid and glycogen accumulation, employing magnetic resonance spectroscopy (MRS) at 7 T.

174 iable using multiple quantum coherence (MQC) magnetic resonance spectroscopy (MRS) at the expense of

175 l magnetic resonance imaging (fMRI) and (1)H magnetic resonance spectroscopy (MRS) data were obtained

176 Hyperpolarized (HP) (13)C magnetic resonance spectroscopy (MRS) has the unique abi

177 Hyperpolarized (13)C magnetic resonance spectroscopy (MRS) is a developing im

178 noninvasive neuroimaging techniques such as magnetic resonance spectroscopy (MRS) may cover anatomic

179 lementary information.SIGNIFICANCE STATEMENT Magnetic resonance spectroscopy (MRS) measures local glu

180 Magnetic resonance spectroscopy (MRS) measures of glutam

181 Hyperpolarized (13)C magnetic resonance spectroscopy (MRS) provides unprecede

182 s by quantifying signal reductions in proton magnetic resonance spectroscopy (MRS) resulting from the

183 We applied the novel (17)O magnetic resonance spectroscopy (MRS) technique on R6/2

184 This study used high-field proton magnetic resonance spectroscopy (MRS) to determine wheth

185 differences in this response in ASD, we used magnetic resonance spectroscopy (MRS) to measure glutama

186 resonance imaging (fMRI) with resting-state magnetic resonance spectroscopy (MRS) to measure task-re

187 This study used magnetic resonance spectroscopy (MRS) to quantify the co

188 We assessed whether Glu measured with magnetic resonance spectroscopy (MRS) was associated wit

189 ion in human brain and NAAG levels using 7-T magnetic resonance spectroscopy (MRS) were measured.

190 hearts, energetics were measured using (31)P magnetic resonance spectroscopy (MRS), and glycolysis an

191 cle in individuals living with ALS using 31P-magnetic resonance spectroscopy (MRS), the modality of c

192 of this study was to determine the value of magnetic resonance spectroscopy (MRS)-based metabolic ch

193 goal of this study was therefore to identify magnetic resonance spectroscopy (MRS)-detectable metabol

194 ng in vivo hyperpolarized [1-(13)C] pyruvate magnetic resonance spectroscopy (MRS).

195 as age-matched healthy control subjects with magnetic resonance spectroscopy (MRS).

196 e pool size assayed via high-resolution (1)H magnetic resonance spectroscopy (MRS).

197 c resonance imaging (fMRI) and resting-state magnetic resonance spectroscopy (MRS).

198 try and high resolution magic angle spinning magnetic resonance spectroscopy (MRS).

199 fMRI) and glutamate (Glu) concentration with magnetic resonance spectroscopy (MRS).

200 ng, brain diffusion tensor imaging (DTI) and magnetic resonance spectroscopy (MRS).

201 ft insula, and visual cortex using 7T proton magnetic resonance spectroscopy (MRS).

202 ncluding hyperinsulinemic-euglycemic clamps, magnetic resonance spectroscopy, muscle biopsies, and as

203 of N-acetylaspartate to creatinine levels (a magnetic resonance spectroscopy neuronal integrity bioma

204 l techniques including proton ((1)H) nuclear magnetic resonance spectroscopy (NMR) and electrospray i

205 Using (1)H Nuclear Magnetic Resonance spectroscopy (NMR) and Gas Chromatogr

206 Liquid-state, one-dimension (31)P nuclear magnetic resonance spectroscopy (NMR) has greatly advanc

207 Nuclear magnetic resonance spectroscopy (NMR) is a versatile too

208 Solid-state nuclear magnetic resonance spectroscopy (NMR) is potentially a p

209 elucidation of protein structures by Nuclear Magnetic Resonance spectroscopy (NMR) rely on distance r

210 Solid-state (31)P nuclear magnetic resonance spectroscopy (NMR) revealed the main

211 u by activated recombinant ERK2 with nuclear magnetic resonance spectroscopy (NMR) reveals phosphoryl

212 of Czech origin were analysed using nuclear magnetic resonance spectroscopy (NMR) with the aim of bu

213 tty acid methyl esters (FAMEs), (1)H nuclear magnetic resonance spectroscopy (NMR), employing a 700 M

214 Interactions were measured using Nuclear Magnetic Resonance spectroscopy (NMR), Isothermal Titrat

215 EM), Dynamic Light Scattering (DLS), Nuclear Magnetic Resonance Spectroscopy (NMR), Thermogravimetric

216 l X-ray crystallography, UV-vis-NIR, nuclear magnetic resonance spectroscopy (NMR), X-ray absorption

217 ecules is typically determined using nuclear magnetic resonance spectroscopy (NMR).

218 mass spectrometry (MS) and 1D and 2D nuclear magnetic resonance spectroscopy (NMR).

219 graphy mass spectrometry (GC-MS) and nuclear magnetic resonance spectroscopy (NMR).

220 etry (ESI-HRAM-MS/MS), and 1D and 2D nuclear magnetic resonance spectroscopy (NMR).

221 Here, we employ ultra-high field magnetic resonance spectroscopy of GABA to test whether

222 We acquired near-whole-brain magnetic resonance spectroscopy of N-acetyl compounds, g

223 readout capability, they have been used for magnetic resonance spectroscopy of nanoscale samples on

224 Nuclear magnetic resonance spectroscopy of products from the cop

225 thyl transverse relaxation-optimized nuclear magnetic resonance spectroscopy on a 450-kilodalton comp

226 PubMed comprised articles with search terms (magnetic resonance spectroscopy OR MRS) AND (glutamate O

227 ies measured by pulse field gradient nuclear magnetic resonance spectroscopy (PFG-NMR, which gives mo

228 (31)P magnetic resonance spectroscopy provides a noninvasive w

229 Using quantitative magnetic resonance spectroscopy (qMRS), we compared mPFC

230 mes strongly correlate with those from (31)P magnetic resonance spectroscopy (R = 0.813, p < 0.001, t

231 esolution mass spectrometry (MS) and nuclear magnetic resonance spectroscopy reveal abundant nitrogen

232 X-ray absorption fine structure and nuclear magnetic resonance spectroscopy reveal that a variety of

233 In vivo (1)H-magnetic resonance spectroscopy revealed an age-dependen

234 (1)H magnetic resonance spectroscopy revealed that greater re

235 Solution nuclear magnetic resonance spectroscopy revealed that HPMC distr

236 reatments (28 drug naive) underwent a proton magnetic resonance spectroscopy scan measuring glutamate

237 Following baseline proton magnetic resonance spectroscopy scans targeting the mPFC

238 groups were brought back to undergo outcome magnetic resonance spectroscopy scans, which were identi

239 Search terms included magnetic resonance spectroscopy, schizophrenia, psychosi

240 oscopic analysis performed by proton-nuclear magnetic resonance spectroscopy showed that the particul

241 e biophysical approach that includes nuclear magnetic resonance spectroscopy, small-angle x-ray scatt

242 Nuclear magnetic resonance spectroscopy studies and density func

243 However, most prior magnetic resonance spectroscopy studies had small sample

244 Here, we conducted a series of proton magnetic resonance spectroscopy studies in human volunte

245 ine, or choline compounds measured by proton magnetic resonance spectroscopy suggest that neuron or g

246 esent study employed state-of-the-art proton magnetic resonance spectroscopy techniques to measure ch

247 Here we show by nuclear magnetic resonance spectroscopy that inhibitors of LpxC-

248 ng, dynamic contrast enhanced sequences, and magnetic resonance spectroscopy that may provide insight

249 Here, we demonstrate by nuclear magnetic resonance spectroscopy that the Escherichia col

250 ined is limited to glucose uptake only.(13)C magnetic resonance spectroscopy theoretically has certai

251 ontent, left ventricular function, and (31)P magnetic resonance spectroscopy to assess phosphocreatin

252 tractile function and Phosphorus-31 ((31) P) magnetic resonance spectroscopy to demonstrate myocardia

253 ng solubility enhancement as well as nuclear magnetic resonance spectroscopy to demonstrate solution-

254 We used nuclear magnetic resonance spectroscopy to describe in atomic-le

255 trate the ability of hyperpolarized pyruvate magnetic resonance spectroscopy to detect metformin-indu

256 We used hyperpolarized [1-(13)C]pyruvate magnetic resonance spectroscopy to determine the effects

257 We used nuclear magnetic resonance spectroscopy to determine the solutio

258 Here, we use solid-state nuclear magnetic resonance spectroscopy to gain atomic level ins

259 multilocularis metacestodes by (1)H nuclear magnetic resonance spectroscopy to identify the unknown

260 This work uses nuclear magnetic resonance spectroscopy to investigate metabolic

261 Here, we used 7 T magnetic resonance spectroscopy to investigate the dynam

262 ery and neuroimaging that included optimized magnetic resonance spectroscopy to measure anterior cing

263 er and 51 healthy control subjects underwent magnetic resonance spectroscopy to measure glutamate, gl

264 We used pulmonary gas exchange and (31) P magnetic resonance spectroscopy to measure whole-body VO

265 ation of the ability of hyperpolarized (13)C magnetic resonance spectroscopy to noninvasively assess

266 ng, dynamic contrast enhanced sequences, and magnetic resonance spectroscopy to provide insight into

267 dults (N = 48; age range, 50-79 years) using magnetic resonance spectroscopy to quantify GABA levels

268 We used phosphorus magnetic resonance spectroscopy to quantify intracellula

269 his study, we used hyperpolarized (HP) (13)C-magnetic resonance spectroscopy to study the impact of a

270 innovative application of (13)C NMR (nuclear magnetic resonance) spectroscopy to determine the bulk (

271 ques, such as infrared spectroscopy, nuclear magnetic resonance spectroscopy, ultraviolet-visible spe

272 striatal glutamate, as measured using proton magnetic resonance spectroscopy, underlies the acute psy

273 metabolism, we performed phosphorous ((31)P) magnetic resonance spectroscopy using a 1.5-Tesla system

274 c content measured in lymphocytes by nuclear magnetic resonance spectroscopy was altered in septic pa

275 Nuclear Magnetic Resonance spectroscopy was employed to study th

276 Nuclear magnetic resonance spectroscopy was shown to be a rapid

277 Phosphorus nuclear magnetic resonance spectroscopy was used to quantify con

278 Finally, using in vivo proton magnetic resonance spectroscopy we found tissue signatur

279 Using Nuclear Magnetic Resonance spectroscopy we reveal how disease-ca

280 Using nuclear magnetic resonance spectroscopy, we first show that expo

281 Using time-resolved nuclear magnetic resonance spectroscopy, we found that ERK2 phos

282 igh-resolution mass spectrometry and nuclear magnetic resonance spectroscopy, we integrated them into

283 Using proton magnetic resonance spectroscopy, we recently showed that

284 tion imaging and in situ solid-state nuclear magnetic resonance spectroscopy, we reveal the underlyin

285 In this study, using nuclear magnetic resonance spectroscopy, we show that Kindlin-3

286 Using high-resolution solution nuclear magnetic resonance spectroscopy, we show that the drug i

287 Using proton magnetic resonance spectroscopy, we showed that choline

288 monary gas-exchange and intramuscular (31) P magnetic resonance spectroscopy, we tested the hypothese

289 r, molecular phenotypes, neuropathology, and magnetic resonance spectroscopy were done on all groups,

290 c fat fraction (HFF) of 5% or more by proton magnetic resonance spectroscopy were eligible.

291 onally, tandem mass spectrometry and nuclear magnetic resonance spectroscopy were used to elucidate r

292 antar flexion exercise fatigability test and magnetic resonance spectroscopy were used to probe the m

293 limited structural information, and nuclear magnetic resonance spectroscopy, which can achieves the

294 volumes suitable for high-resolution nuclear magnetic resonance spectroscopy while maintaining high c

295 Using magnetic resonance spectroscopy with hyperpolarized pyru

296 te-cycled, non-water suppressed (1)H cardiac magnetic resonance spectroscopy with prospective and ret

297 (7)Li, (19)F, and (13)C solid-state nuclear magnetic resonance spectroscopies, with the organics dom

298 ine recovery time constant (tauPCr) by (31)P-magnetic resonance spectroscopy, with higher tauPCr valu

299 The focus is on synchrotron nuclear magnetic resonance spectroscopy, X-ray diffraction, X-ra

300 ailure successfully underwent cardiovascular magnetic resonance spectroscopy, yielding data on intram