ライフサイエンスコーパス: magnetic stimulation

1 racortical inhibition (SICI) by transcranial magnetic stimulation.

2 scle was stimulated paired with transcranial magnetic stimulation.

3 MRI, magnetoencephalography and transcranial magnetic stimulation.

4 h motor-evoked potentials under transcranial magnetic stimulation.

5 with theta versus beta rhythmic transcranial magnetic stimulation.

6 ectrodes or noninvasively using transcranial magnetic stimulation.

7 citability were performed using transcranial magnetic stimulation.

8 overcoming the limitations of electrical or magnetic stimulation.

9 ics, with a particular focus on transcranial magnetic stimulation.

10 noninvasive approaches, such as transcranial magnetic stimulation.

11 evoked potentials elicited with transcranial magnetic stimulation.

12 ion and neuro-navigation of the transcranial magnetic stimulation.

13 via twitch P(di) (P(di,tw) ) using cervical magnetic stimulation.

14 ology [5], neuroimaging [6-11], transcranial magnetic stimulation [12, 13], single-unit and lesion st

15 Transcranial magnetic stimulation also induced anticorrelated connect

16 Transcranial magnetic stimulation altered size perception earlier ove

17 A new study using transcranial magnetic stimulation and a virtual reality navigation ta

18 In the current study, we used transcranial magnetic stimulation and demonstrated that temporary dis

19 vity, as measured by concurrent transcranial magnetic stimulation and EEG.

20 tions, as can be assessed using transcranial magnetic stimulation and electroencephalography (TMS-EEG

21 We used a combination of transcranial magnetic stimulation and electroencephalography (TMS/EEG

22 erve stimulation (nVNS), single-transcranial magnetic stimulation and external trigeminal nerve stimu

23 Concurrent transcranial magnetic stimulation and fMRI in healthy participants de

24 cked in vivo using single pulse transcranial magnetic stimulation and further highlight a novel thala

25 multiple parameters of repetitive cerebellar magnetic stimulation and have described the optimal sett

26 ssues, we combined single-pulse transcranial magnetic stimulation and motor-evoked potentials while h

27 ophysiological method involving transcranial magnetic stimulation and peripheral nerve stimulation.

28 s (MEPs) evoked by single-pulse transcranial magnetic stimulation and short-interval intracortical in

29 r physiology was performed with transcranial magnetic stimulation and somatosensory physiology with v

30 Using transcranial magnetic stimulation and tasks designed to assess differ

31 icospinal responses elicited by transcranial magnetic stimulation and the magnitude of maximal volunt

32 non-invasive such as repetitive transcranial magnetic stimulation and transcranial direct current sti

33 m extensor carpi radialis using transcranial magnetic stimulation, and fractional anisotropy (FA) in

34 s an updated form of repetitive transcranial magnetic stimulation, and it is an effective add-on inte

35 probed by the onset latency of transcranial magnetic stimulation applied to primary motor cortex (M1

36 Finally, single-pulse transcranial magnetic stimulation applied to the human PFC between su

37 Therefore, motor responses to transcranial magnetic stimulation are larger when a cortical input ar

38 Neurology Grand Rounds, we use transcranial magnetic stimulation as a model to explore the principle

39 s that had been identified with transcranial magnetic stimulation-based functional localization, phon

40 reatment including medications, transcranial magnetic stimulation, biofeedback, target-specific botul

41 -cortical spreading depression, transcranial magnetic stimulation blocked the propagation of cortical

42 rimentally and by physical calculations that magnetic stimulation can induce electric fields in the p

43 orm spatial sampling procedure, transcranial magnetic stimulation can produce cortical functional map

44 Transcranial magnetic stimulation can show changes in focal excitabil

45 tic arm positioned a repetitive transcranial magnetic stimulation coil over a subject-specific dorsal

46 ve connectivity, as assessed by transcranial magnetic stimulation combined with electroencephalograph

47 Transcranial magnetic stimulation combined with electroencephalograph

48 erve electrical stimulation and transcranial magnetic stimulation) combined with electroencephalograp

49 , we use continuous theta-burst transcranial magnetic stimulation (cTBS) to test this model causally.

50 ptation, continuous theta-burst transcranial magnetic stimulation (cTBS) was delivered to block reten

51 (CBI): a conditioning pulse of transcranial magnetic stimulation delivered to the cerebellum before

52 close temporal contiguity with transcranial magnetic stimulation delivered to the contralateral prim

53 Dual-site transcranial magnetic stimulation (dsTMS) has highlighted the timing

54 Dual site transcranial magnetic stimulation (dsTMS) has revealed interhemispher

55 ated beneficial effects of deep transcranial magnetic stimulation (dTMS) targeting the medial prefron

56 of the hippocampal network via transcranial magnetic stimulation during concurrent fMRI.

57 We combined transcranial magnetic stimulation, electroencephalography, electromyo

58 A subsequent fMRI-guided transcranial magnetic stimulation experiment confirmed dissociable ro

59 rther support was obtained by a transcranial magnetic stimulation experiment, where subjects whose fr

60 his is further supported by our transcranial magnetic stimulation experiment: subjects whose right in

61 Transcranial magnetic stimulation focused on either the left anterior

62 Here, we use 180 pairs of transcranial magnetic stimulation for approximately 30 min over the h

63 humans, we applied theta-burst transcranial magnetic stimulation, guided by subject-specific connect

64 More generally, our novel transcranial magnetic stimulation-guided lesion-deficit mapping appro

65 ants, we show how damage to our transcranial magnetic stimulation-guided regions affected performance

66 e for the contribution that the transcranial magnetic stimulation-guided regions make to language tas

67 classification accuracy of the transcranial magnetic stimulation-guided regions was validated in a s

68 th and without damage to these 'transcranial magnetic stimulation-guided' regions remained highly sig

69 A single pulse of transcranial magnetic stimulation has been shown to be effective for

70 ysiology of the brain employing transcranial magnetic stimulation has convincingly demonstrated a pre

71 Recently, single pulses of cerebellar magnetic stimulation have been shown to directly evoke r

72 ephalography (EEG) responses to transcranial magnetic stimulation in 22 participants during 29 h of w

73 ode with continuous theta-burst transcranial magnetic stimulation in a randomized, sham-controlled de

74 ngs from functional imaging and transcranial magnetic stimulation in healthy participants, we show ho

75 onduction failure assessed with transcranial magnetic stimulation in the right upper limb (Spearman r

76 ry capacity that may explain the efficacy of magnetic stimulation in the treatment of migraine with a

77 stimulation (cPAS) is a form of transcranial magnetic stimulation in which paired pulses can induce p

78 eas neuronal activation using high-intensity magnetic stimulation increases barrier permeability and

79 magnetic resonance imaging and transcranial magnetic stimulation indicated the involvement of the le

80 ndicated by specific markers of transcranial magnetic stimulation-induced muscle and brain responses

81 A combination of transcranial magnetic stimulation-induced muscle relaxation, muscle f

82 gms, we report that noninvasive transcranial magnetic stimulation interference with a reactivated mot

83 individualized therapies (e.g., transcranial magnetic stimulation, intracerebral stem/progenitor cell

84 Neuromodulation using transcranial magnetic stimulation is one of the most promising techni

85 h as electroconvulsive therapy, transcranial magnetic stimulation, ketamine infusions, and, more rece

86 pinal excitability at rest with transcranial magnetic stimulation, local concentrations of basal GABA

87 n (STN-DBS) with motor cortical transcranial magnetic stimulation (M1-TMS) at specific times can indu

88 Overall baclofen did not alter transcranial magnetic stimulation-measured GABA(B) inhibition, althou

89 onotherapy (n = 35), repetitive transcranial magnetic stimulation monotherapy (n = 35), or sham stimu

90 iomarkers were assessed using a transcranial magnetic stimulation multiparadigm approach in 13 presym

91 Transcranial magnetic stimulation normalized depression-related subge

92 articipants, whether repetitive transcranial magnetic stimulation of a frontal midline node of the ci

93 ns are delivered to the Receiver's brain via magnetic stimulation of the occipital cortex.

94 ivity of brain regions, such as transcranial magnetic stimulation or rapid-acting antidepressants lik

95 Here, we applied transcranial magnetic stimulation over four frontoparietal cortex loc

96 Transcranial magnetic stimulation over primary motor cortex provided

97 We used paired-pulse transcranial magnetic stimulation over primary motor cortex to measur

98 d potentials (MEPs) elicited by transcranial magnetic stimulation over the arm representation of the

99 d potentials (MEPs) elicited by transcranial magnetic stimulation over the ipsilateral motor cortex w

100 SICI was obtained by delivering transcranial magnetic stimulation over the left motor cortex.

101 d potentials (MEPs) elicited by transcranial magnetic stimulation over the leg representation of the

102 Transcranial magnetic stimulation over the PPC is used to study cogni

103 Using transcranial magnetic stimulation over the primary motor cortex (M1)

104 corticospinal volleys evoked by transcranial magnetic stimulation over the primary motor cortex arriv

105 r evoked potentials elicited by transcranial magnetic stimulation, paired-pulse intracortical inhibit

106 Using a dual-site transcranial magnetic stimulation paradigm, we examined the age-relat

107 cuss how recent methods such as transcranial magnetic stimulation, positron emission tomography, MRI,

108 ied by twitch P(di) (P(di,tw) ) via cervical magnetic stimulation post-PTL.

109 A targeted pulse of transcranial magnetic stimulation produced a brief reemergence of the

110 ask with a real-time repetitive transcranial magnetic stimulation protocol, we provide evidence indic

111 sticity induction by repetitive transcranial magnetic stimulation protocols such as intermittent thet

112 ing target for novel repetitive transcranial magnetic stimulation protocols.

113 ortex target, and 50 repetitive transcranial magnetic stimulation pulses were delivered at 10 Hz (exc

114 Results showed that transcranial magnetic stimulation reduced classification accuracy com

115 A causal intervention with transcranial magnetic stimulation revealed clear specialization along

116 While repetitive magnetic stimulation (rMS) has been shown to induce plas

117 Here, we used repetitive transcranial magnetic stimulation (rTMS) and fMRI to determine the sp

118 ily inhibited PPC by repetitive transcranial magnetic stimulation (rTMS) and hypothesized that the mo

119 ed waveforms such as repetitive transcranial magnetic stimulation (rTMS) and transcranial alternating

120 imaging, and online repetitive transcranial magnetic stimulation (rTMS) applied during an individual

121 Repetitive transcranial magnetic stimulation (rTMS) applied over the right poste

122 low frequency (1 Hz) repetitive transcranial magnetic stimulation (rTMS) as an inhibitory noninvasive

123 Repetitive transcranial magnetic stimulation (rTMS) can alter neuronal activity

124 rowing evidence that repetitive transcranial magnetic stimulation (rTMS) can be used as a treatment f

125 suggest efficacy of repetitive transcranial magnetic stimulation (rTMS) for the treatment of negativ

126 everal strategies of repetitive transcranial magnetic stimulation (rTMS) have been investigated as tr

127 gnitive responses to repetitive transcranial magnetic stimulation (rTMS) in bipolar II depressed pati

128 peutic potential for repetitive transcranial magnetic stimulation (rTMS) in swallowing rehabilitation

129 Repetitive transcranial magnetic stimulation (rTMS) is a commonly- used treatmen

130 Repetitive transcranial magnetic stimulation (rTMS) is a noninvasive neuromodula

131 Repetitive transcranial magnetic stimulation (rTMS) is an effective treatment fo

132 ain stimulation like repetitive transcranial magnetic stimulation (rTMS) is an increasingly popular s

133 Repetitive transcranial magnetic stimulation (rTMS) is used as a therapeutic too

134 ts of high-frequency repetitive transcranial magnetic stimulation (rTMS) of the right dorsolateral pr

135 Critically, repetitive transcranial magnetic stimulation (rTMS) on participants' peak of act

136 ting depression with repetitive transcranial magnetic stimulation (rTMS) remains unknown.

137 e imaging (fMRI) and repetitive transcranial magnetic stimulation (rTMS) to examine the role of S1 in

138 after low-frequency repetitive transcranial magnetic stimulation (rTMS) to the right PPC or to the s

139 Using repetitive transcranial magnetic stimulation (rTMS), we have recently shown a fu

140 for retreatment with repetitive transcranial magnetic stimulation (rTMS).

141 Transcranial magnetic stimulation selectively modulates functional co

142 ion-specific therapies, such as transcranial magnetic stimulation.SIGNIFICANCE STATEMENT By estimatin

143 Single pulse transcranial magnetic stimulation significantly inhibited both mechan

144 Additionally transcranial magnetic stimulation significantly inhibited the spontan

145 of phonological processing, or transcranial magnetic stimulation sites that did not use functional l

146 ning the borders of each of the transcranial magnetic stimulation sites to include areas that were co

147 Here, we used a dense grid of transcranial magnetic stimulation spots covering the whole premotor c

148 Transcranial magnetic stimulation studies have highlighted that corti

149 ent with functional imaging and transcranial magnetic stimulation studies in human Parkinson's diseas

150 nctional assessments along with transcranial magnetic stimulation studies were taken on 15 patients w

151 ased cortical excitability in a transcranial magnetic stimulation study in healthy volunteers.

152 ortex by delivering theta-burst transcranial magnetic stimulation (TBS) concurrent with fMRI, as an i

153 Theta burst transcranial magnetic stimulation (TBS) is a potential new treatment

154 estion by combining theta burst transcranial magnetic stimulation (TBS) with fMRI to test the predict

155 d with left stellate ganglion transcutaneous magnetic stimulation (TCMS) to reduce cardiac sympatheti

156 r and sensory cortices by using transcranial magnetic stimulation techniques.

157 We finally confirm, using transcranial magnetic stimulation, that the fMRI-identified medial wa

158 stimulation techniques such as transcranial magnetic stimulation, the therapeutic efficacy of these

159 also predict responsiveness to transcranial magnetic stimulation therapy (n = 154).

160 o different forms of repetitive transcranial magnetic stimulation therapy for MDD.

161 rupting cerebellar activity via transcranial magnetic stimulation (TMS) abolished the adaptation of M

162 l cortex (DLPFC) using combined transcranial magnetic stimulation (TMS) and electroencephalography (E

163 By combining transcranial magnetic stimulation (TMS) and electroencephalography (E

164 Here, we combined transcranial magnetic stimulation (TMS) and fMRI to test the role of

165 left speech motor cortex using transcranial magnetic stimulation (TMS) and measured the impact of th

166 Here we used fMRI-guided transcranial magnetic stimulation (TMS) and simultaneous electroencep

167 sess this, we used single-pulse transcranial magnetic stimulation (TMS) applied to visual cortical ar

168 This fMRI study used concurrent transcranial magnetic stimulation (TMS) as a causal perturbation appr

169 merging from the application of transcranial magnetic stimulation (TMS) as a research and clinical to

170 Transcranial magnetic stimulation (TMS) at beta frequency has previou

171 TUS transducer was coupled to a transcranial magnetic stimulation (TMS) coil.

172 p (N=17) underwent single-pulse transcranial magnetic stimulation (TMS) concurrent with fMRI to exami

173 patterns of rhythmic or random transcranial magnetic stimulation (TMS) delivered to the right Fronta

174 er high-frequency, non-invasive transcranial magnetic stimulation (TMS) delivered twice a week over a

175 elbow flexors was assessed via transcranial magnetic stimulation (TMS) during maximum voluntary cont

176 timing of beta events preceding transcranial magnetic stimulation (TMS) each significantly predicted

177 vated us to conduct a series of transcranial magnetic stimulation (TMS) experiments to examine in det

178 , safety, and efficacy of 10-Hz transcranial magnetic stimulation (TMS) for adolescents with TRD.

179 ts who received left prefrontal transcranial magnetic stimulation (TMS) for treatment of depression (

180 Transcranial magnetic stimulation (TMS) has been shown to modulate mu

181 Transcranial magnetic stimulation (TMS) has been suggested as a relia

182 Transcranial magnetic stimulation (TMS) has emerged as an important t

183 cillations, we applied rhythmic transcranial magnetic stimulation (TMS) in either theta or alpha freq

184 Previous studies have used transcranial magnetic stimulation (TMS) in humans to demonstrate the

185 The development of the use of transcranial magnetic stimulation (TMS) in the study of psychological

186 est by combining the FCM with a transcranial magnetic stimulation (TMS) intervention that transiently

187 as become a particular focus of transcranial magnetic stimulation (TMS) investigational studies.

188 Transcranial magnetic stimulation (TMS) is a noninvasive method to st

189 Transcranial magnetic stimulation (TMS) is a widely used non-invasive

190 Transcranial magnetic stimulation (TMS) is an accessible, non-invasiv

191 Transcranial magnetic stimulation (TMS) is an effective treatment for

192 Transcranial magnetic stimulation (TMS) is widely used in clinical in

193 Transcranial magnetic stimulation (TMS) measures of corticospinal exc

194 We demonstrate that paired transcranial magnetic stimulation (TMS) near ventral premotor cortex

195 ween trigeminal stimulation and transcranial magnetic stimulation (TMS) of fM1 was 15-30 ms.

196 nd neural plasticity often used transcranial magnetic stimulation (TMS) of hand motor cortex (M1) as

197 Transcranial magnetic stimulation (TMS) of human occipital and poster

198 d potentials (MEPs) obtained by transcranial magnetic stimulation (TMS) of M1 using an online MRI-gui

199 Repetitive transcranial magnetic stimulation (TMS) of the dorsolateral prefronta

200 muscle at the motor point with transcranial magnetic stimulation (TMS) of the motor cortex generated

201 corticospinal excitability via transcranial magnetic stimulation (TMS) of the primary motor cortex a

202 equential task while undergoing transcranial magnetic stimulation (TMS) of the RLPFC versus two prefr

203 e shown asymmetrical effects of transcranial magnetic stimulation (TMS) on task performance, but it i

204 in combination with directional transcranial magnetic stimulation (TMS) over M1.

205 succeeds lbif) while undergoing transcranial magnetic stimulation (TMS) over the cortical motor repre

206 ed this possibility by means of transcranial magnetic stimulation (TMS) over the hand area of the pri

207 scheme, we tested the effect of transcranial magnetic stimulation (TMS) over the hand area of the pri

208 We used transcranial magnetic stimulation (TMS) over the occipital cortex to

209 found that applying theta-burst transcranial magnetic stimulation (TMS) over the PPC, but not the dor

210 By applying transcranial magnetic stimulation (TMS) over the primary motor cortex

211 sterior-anterior (PA) pulses of transcranial magnetic stimulation (TMS) over the primary motor cortex

212 ntrols, we applied paired-pulse transcranial magnetic stimulation (TMS) protocols to evaluate the exc

213 Transcranial magnetic stimulation (TMS) represents a novel approach t

214 forefront of advancing clinical transcranial magnetic stimulation (TMS) since the mid-1990s, shortly

215 Transcranial magnetic stimulation (TMS) studies in humans have shown

216 ic Resonance Imaging (fMRI) and Transcranial Magnetic Stimulation (TMS) study.

217 Repetitive transcranial magnetic stimulation (TMS) therapy can modulate patholog

218 rrent study, we used MRI-guided transcranial magnetic stimulation (TMS) to assess whether temporary d

219 Here we employed fMRI-guided transcranial magnetic stimulation (TMS) to assess whether temporary d

220 Here, by using transcranial magnetic stimulation (TMS) to block consolidation, we re

221 ttentional modulations, we used transcranial magnetic stimulation (TMS) to briefly alter cortical exc

222 derwent MRI-guided single-pulse transcranial magnetic stimulation (TMS) to co-localise pharyngeal and

223 EG) to record brain signals and transcranial magnetic stimulation (TMS) to deliver information noninv

224 Here, we used transcranial magnetic stimulation (TMS) to evaluate the causal role o

225 Here, we used transcranial magnetic stimulation (TMS) to examine the physiological

226 l lesion methodology offered by transcranial magnetic stimulation (TMS) to explore the impact of pre-

227 e to use MI in conjunction with transcranial magnetic stimulation (TMS) to induce plasticity in the h

228 participants using single-pulse transcranial magnetic stimulation (TMS) to interfere with postmovemen

229 To test this idea, we used transcranial magnetic stimulation (TMS) to interrupt processing in th

230 Here, we used transcranial magnetic stimulation (TMS) to measure cortical inhibitio

231 l nerve stimulation paired with transcranial magnetic stimulation (TMS) to primary motor cortex (M1)

232 e) brain noninvasively, we used transcranial magnetic stimulation (TMS) to probe the excitability of

233 Here we used fMRI-guided transcranial magnetic stimulation (TMS) to shed light on the role of

234 Here, we used transcranial magnetic stimulation (TMS) to test the effects of a sing

235 ated with 5 days of twice-daily transcranial magnetic stimulation (TMS) to the cerebellar midline.

236 Here, transcranial magnetic stimulation (TMS) was used to establish a causa

237 We delivered double-pulse transcranial magnetic stimulation (TMS) while moving a single tactile

238 the process by combining online transcranial magnetic stimulation (TMS) with computational modeling o

239 asuring the brain's response to transcranial magnetic stimulation (TMS) with electroencephalography (

240 We employed transcranial magnetic stimulation (TMS) with simultaneous electroence

241 d (FEF) by combining repetitive transcranial magnetic stimulation (TMS) with subsequent magnetoenceph

242 we hypothesized that inhibitory transcranial magnetic stimulation (TMS) would reduce appetitive neuro

243 Using transcranial magnetic stimulation (TMS), 25 motor-evoked potentials (

244 using fMRI, psychophysics, and transcranial magnetic stimulation (TMS), all within the same human pa

245 orticospinal volley elicited by transcranial magnetic stimulation (TMS), by interacting TMS with stim

246 time polymerase chain reaction, transcranial magnetic stimulation (TMS), functional magnetic resonanc

247 ting motor threshold (RMT) with transcranial magnetic stimulation (TMS), is known to be associated wi

248 ion of response to both ECT and transcranial magnetic stimulation (TMS), offering a new framework for

249 Using transcranial magnetic stimulation (TMS), we applied a novel cortico-c

250 ity of the cortical response to transcranial magnetic stimulation (TMS)--an approach that has proven

251 is for contagious yawning using transcranial magnetic stimulation (TMS).

252 ics of relevant processes using transcranial magnetic stimulation (TMS).

253 tic resonance imaging-navigated transcranial magnetic stimulation (TMS).

254 was investigated with fMRI and transcranial magnetic stimulation (TMS).

255 a variety of brain disorders is transcranial magnetic stimulation (TMS).

256 omotor interaction using paired transcranial magnetic stimulation (TMS).

257 entrain") these oscillations by transcranial magnetic stimulation (TMS).

258 tudy addressed this issue using transcranial magnetic stimulation (TMS).

259 nvestigative techniques such as transcranial magnetic stimulation (TMS).

260 spinal excitability (CSE) using transcranial magnetic stimulation (TMS).

261 tivity and is readily probed by transcranial magnetic stimulation (TMS).

262 o probe candidate regions using transcranial magnetic stimulation (TMS).

263 dence, using fMRI-guided online transcranial magnetic stimulation (TMS).

264 tatory or inhibitory repetitive transcranial magnetic stimulation to a subject-specific frontal-cingu

265 Using transcranial magnetic stimulation to alter brain function during retr

266 eripheral nerve stimulation and transcranial magnetic stimulation to alter functional responses in th

267 esonance imaging and repetitive transcranial magnetic stimulation to demonstrate the representation o

268 Using transcranial magnetic stimulation to inhibit the right frontopolar co

269 Applying rhythmic transcranial magnetic stimulation to interfere with early retrieval p

270 We used fMRI and transcranial magnetic stimulation to investigate the neural underpinn

271 We tested this idea using transcranial magnetic stimulation to measure corticospinal excitabili

272 This was achieved by applying a transcranial magnetic stimulation to the medial prefrontal cortex (Br

273 l stop-signal task, and applied transcranial magnetic stimulation to the motor cortex, to assess move

274 orce generation in response to femoral nerve magnetic stimulation, to assess leg strength before and

275 We used transcranial magnetic stimulation tools to investigate the mechanisms

276 ulation in addiction, including transcranial magnetic stimulation, transcranial direct current stimul

277 stimulation techniques such as transcranial magnetic stimulation, transcranial direct current stimul

278 Using a combined transcranial magnetic stimulation-transcranial alternating current st

279 potential target for repetitive transcranial magnetic stimulation treatment in OCD, these results sup

280 differentially to a repetitive transcranial magnetic stimulation treatment outcome.

281 motor-cortical excitability via transcranial magnetic stimulation up to 2 h after stimulation.

282 tential mechanisms of action of transcranial magnetic stimulation, using a transcortical approach, in

283 Single-pulse transcranial magnetic stimulation was applied 100 ms after visual pre

284 deoed partner, and double-pulse transcranial magnetic stimulation was applied around the turn-switch

285 Inhibitory theta-burst Transcranial Magnetic Stimulation was applied to the left DLPFC or to

286 Transcranial magnetic stimulation was delivered at 80, 240, and 400 m

287 articipants, threshold-tracking transcranial magnetic stimulation was used to assess cortical functio

288 Third, using transcranial magnetic stimulation, we demonstrate that the anterior r

289 Finally, using transcranial magnetic stimulation, we found that during the same time

290 Using transcranial magnetic stimulation, we investigated the role of the ea

291 th rTPJ activity through online transcranial magnetic stimulation, we showed that participants were l

292 c incentive motivation task and transcranial magnetic stimulation, we studied the motor cortical mech

293 muscle action potentials evoked by cervical magnetic stimulation were unaffected by tsDCS.

294 iffusion and perfusion MRI, and transcranial magnetic stimulation were used to study structural conne

295 Furthermore, the orientation-dependence of magnetic stimulation, which leads to specific changes in

296 otor cortical excitability with transcranial magnetic stimulation while female and male human partici

297 means of continuous theta-burst transcranial magnetic stimulation, while measuring effort perception

298 r-evoked potentials elicited by transcranial magnetic stimulation with an anterior-posterior (AP) ori

299 -evoked potentials generated by transcranial magnetic stimulation with an AP orientation over the lat

300 hibitory continuous theta-burst transcranial magnetic stimulation with model-based functional MRI, we