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1 to blockade of inhibitory M2Rs by eosinophil major basic protein.
2  antagonism of the M2 receptor by eosinophil major basic protein.
3 n macrophage function through the release of major basic protein.
4 ed in these secondary granules (by mass) are major basic protein 1 (MBP-1) and eosinophil peroxidase
5 Concomitantly, eosinophil mediators, notably major basic protein 1 (MBP-1), released during cytolysis
6  deficient in eosinophil peroxidase (EPO) or major basic protein 1 (MBP-1), suggesting that eosinophi
7  not dependent upon eosinophil peroxidase or major basic protein 1 and did not correlate with activit
8                      EST-captured eotaxin-3, major basic protein 1, EDN, eosinophil peroxidase, and C
9 e reliability of plasma and urine eosinophil major basic protein 1, eosinophil cationic protein, eosi
10 in (EDN), eosinophil cationic protein (ECP), major basic protein-1 (MBP-1), galectin-10 (CLC/GAL-10),
11 r RNase 2), eosinophil peroxidase (EPO), and major basic protein-1 (MBP1) intradermally into guinea p
12 ficient for the secondary granule component, major basic protein-1 (mMBP-1).
13                                              Major basic protein-1 activation of CBMC primed with fib
14                                              Major basic protein-1-induced CBMC activation is mediate
15 rived granule protein double knock-out mice (major basic protein-1/eosinophil peroxidase dual gene de
16 dditionally, the eosinophil granule proteins major basic protein and eosinophil peroxidase were more
17 ophilia was determined by immunostaining for major basic protein and flow cytometry for cell-surface
18                      There was deposition of major basic protein and nitrotyrosine at the base of the
19 of peribronchial cells staining positive for major basic protein and TGF-beta.
20 ain cytoplasmic granules with immunoreactive major basic protein and they express surface Siglec F an
21 inic receptors from antagonism by eosinophil major basic protein, and this protective mechanism appea
22 ion of Epx (eosinophil peroxidase) and Prg2 (major basic protein) as well as lower interleukin 1beta
23 s, such as the eosinophil peroxidase and the major basic protein, but did not express basophil/mast c
24 ss surface Siglec F and transcripts encoding major basic protein, eosinophil peroxidase, and GATA-1,
25 robial host defense peptides, neuropeptides, major basic protein, eosinophil peroxidase, and many US
26 ing a marker for eosinophil precursor cells (major basic protein gene expression) suggest that the pe
27  of a cDNA with 64% identity to human prepro-major basic protein (hprepro-MBP).
28 We assessed eosinophil numbers by using anti-major basic protein immunostaining, goblet cell hyperpla
29 2 muscarinic receptor function by eosinophil major basic protein in antigen-challenged guinea pigs.
30 ntrations of cytokines and compounds such as major basic protein in BAL fluids, only the cellular ele
31 rized by impingement of eosinophilic rods of major basic protein into the muscle cell membrane.
32 s, such as eosinophil-derived neurotoxin and major basic protein, into the extracellular milieu and o
33                Eosinophils were labeled with major basic protein (MBP) Ab to visualize granules and a
34    RNA silencing of EGO results in decreased major basic protein (MBP) and eosinophil derived neuroto
35 deficient in the eosinophil granule products major basic protein (MBP) and eosinophil peroxidase (EPO
36 s investigation, we find that the release of major basic protein (MBP) by eosinophils is a prominent
37              The cDNA for eosinophil granule major basic protein (MBP) encodes a prepromolecule with
38                                   Eosinophil major basic protein (MBP) is an effective stimulus for n
39       To test the hypothesis that eosinophil major basic protein (MBP) is an important regulator of f
40                             Human eosinophil major basic protein (MBP) is strongly implicated as a me
41 re immunostained with either anti-eosinophil major basic protein (MBP) or with anti-neutrophil Ab.
42 s of eosinophil-derived neurotoxin (EDN) and major basic protein (MBP) were elevated in patients with
43          Among soluble mediators, eosinophil major basic protein (MBP), a hallmark of allergy, is par
44  the expression of eosinophil granule genes, major basic protein (MBP), and eosinophil peroxidase (EP
45 ted the in vitro effects of human eosinophil major basic protein (MBP), eosinophil cationic protein (
46 using antibodies directed against eosinophil major basic protein (MBP), however, revealed massive ext
47                      Release of eosinophilic major basic protein (MBP), IL-4, and IL-5 to the periton
48  of a model eosinophil granulocyte gene, the major basic protein (MBP).
49 tors by the endogenous antagonist eosinophil major basic protein (MBP).
50 pendent stimulus, eosinophil-derived granule major basic protein (MBP).
51 cytotoxic proteins, including the eosinophil major basic protein (MBP-1).
52  to the 2 other eosinophil granule proteins (major basic protein [MBP] and eosinophil-derived neuroto
53 ncentrations of eosinophil granule proteins (major basic protein [MBP] and eosinophil-derived neuroto
54                     Human eosinophil granule major basic protein (MBP1) is an exceedingly basic (isoe
55    Additionally, IL-13, IL-10 and eosinophil major basic protein mRNA levels were greater in patients
56                     Specific staining of the major basic protein of eosinophils showed peribronchiola
57 and their degranulation/activation products (major basic protein [p < 0.001, r = 0.7353] and eosinoph
58 ith rabbit antibody to guinea pig eosinophil major basic protein prevented hyperresponsiveness, and p
59                                  The protein major basic protein, primarily expressed by eosinophils,
60 ogenous inhibitor, the proform of eosinophil major basic protein (proMBP), may also play a role in th
61                                              Major basic protein released from eosinophils to airway
62 tochemical staining for the granule protein, major basic protein, revealed that eosinophils accumulat
63                                              Major basic protein secretion from eosinophils parallele
64 d eosinophils (bmEos) express immunoreactive major basic protein, Siglec F, IL-5R alpha-chain, and tr
65                                              Major basic protein staining demonstrated eosinophil deg
66 ficantly reduced the number of peribronchial major basic protein(+)/TGF-beta(+) cells, suggesting tha
67 hil granule proteins (eosinophil peroxidase, major basic protein, the ribonucleases) together with ar
68                                Extracellular major basic protein was present on the surface of airway
69   A rabbit antibody to guinea pig eosinophil major basic protein was used to determine whether M2 mus
70       Eosinophils and the eosinophil product major basic protein were absent from the skin of sham an
71   Immunohistochemistry analyses of CCL26 and major basic protein were done on bronchial biopsy specim
72 d nasal levels of IL-6, IL-5, and eosinophil major basic protein were observed in CRSwNP patients.
73 y nerves where, when activated, they release major basic protein, which binds to and blocks the M2Rs.
74                Activated eosinophils release major basic protein, which binds to M2 receptors and pre
75                Activated eosinophils release major basic protein, which blocks inhibitory M2 muscarin
76  EETs are decorated with the granule protein major basic protein, which causes platelet activation by
77 receptor dysfunction is caused by eosinophil major basic protein, which is an allosteric antagonist a
78 of peribronchial cells staining positive for major basic protein, which was paralleled by a similar r