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1 mutation frequencies at a specific site in a mammalian chromosome.
2 lp maintain the structural integrity of each mammalian chromosome.
3  genomic loci that show block patterns along mammalian chromosomes.
4 e for RNA in the topological organization of mammalian chromosomes.
5 ression levels of mini-genes integrated into mammalian chromosomes.
6  inappropriate repair at the natural ends of mammalian chromosomes.
7 s to study intrachromosomal recombination in mammalian chromosomes.
8 n can serve as a major DSB repair pathway in mammalian chromosomes.
9 (homeologous recombination) is suppressed in mammalian chromosomes.
10 , analogous to the fragile sites observed in mammalian chromosomes.
11 of telomere replication patterns of specific mammalian chromosomes.
12 ole in chromosome-wide replication timing of mammalian chromosomes.
13 iC31 integrase, can mediate integration into mammalian chromosomes.
14 large-scale variation in G + C content along mammalian chromosomes.
15 etroposons, integrate at staggered breaks in mammalian chromosomes.
16 tromeres, and telomeres, we propose that all mammalian chromosomes also contain "Inactivation/Stabili
17 l, functional and evolutionary basis for the mammalian chromosome and chromosomal banding patterns.
18 romosomes expedite the creation of synthetic mammalian chromosomes and genomes.
19 ing the mechanism of DNA damage tolerance in mammalian chromosomes and their connection to pathologic
20         The temporal order of replication of mammalian chromosomes appears to be linked to their func
21 select for loxP-specific genome targeting in mammalian chromosomes are also present.
22                  Several studies reveal that mammalian chromosomes are composed of topological domain
23                                              Mammalian chromosomes are organized into megabase-sized
24                               Centromeres of mammalian chromosomes are rich in repetitive DNAs that a
25                                              Mammalian chromosomes are three-dimensional entities sha
26 We show that the distribution of cohesins on mammalian chromosome arms is not driven by transcription
27          Such divergence is unprecedented in mammalian chromosome biology and has implications for mo
28  predictions of the arrangement of ancestral mammalian chromosomes, but the basis for these rearrange
29                                   Replicated mammalian chromosomes condense to segregate during anaph
30 during development and provide evidence that mammalian chromosomes consist of multiple independent un
31                                              Mammalian chromosome duplication progresses in a precise
32 ht into the effect of reduced DSB numbers on mammalian chromosome dynamics.
33 tion, revealing that compartmentalization of mammalian chromosomes emerges independently of proper in
34                             We conclude that mammalian chromosome ends are highly susceptible to homo
35                                              Mammalian chromosome ends contain long arrays of TTAGGG
36  telomeric DNA binding protein POT1 protects mammalian chromosome ends from the ATR-dependent DNA dam
37 ucleosomes are the key players that organize mammalian chromosome ends into the protective telomere c
38          Telomerase adds TTAGGG repeats onto mammalian chromosome ends, replenishing the terminal seq
39 vents DNA damage signaling and DNA repair at mammalian chromosome ends.
40 s functions in vertebrates and as a tool for mammalian chromosome engineering.
41 des an improved resource for the analysis of mammalian chromosome evolution, the identification of ca
42 nd should contribute to our understanding of mammalian chromosome evolution.
43 ther species provides an emerging picture of mammalian chromosome evolution.
44  demonstrated that in their compacted state, mammalian chromosomes form arrays of closely stacked con
45 een developed to address questions regarding mammalian chromosome function and for biotechnological a
46 to further define the elements necessary for mammalian chromosome function.
47 erstanding of the structure and evolution of mammalian chromosomes, including gene deserts, segmental
48  model in which homeologous recombination in mammalian chromosomes is suppressed by a nondestructive
49 nter-strand DNA cross-links are removed from mammalian chromosomes is unknown, these lesions are repa
50 eractions leading to kinetochore assembly on mammalian chromosomes is unknown.
51 ivalent to homogeneously staining regions in mammalian chromosomes) or extrachromosomal palindromic m
52                                  New data on mammalian chromosome organization across different condi
53 erated will be a cornerstone for elucidating mammalian chromosome phylogeny and the identification of
54 hromosome conformation capture revealed that mammalian chromosomes possess a rich hierarchy of struct
55 portant traits, the phylogenomic analysis of mammalian chromosomes, proofing of the BAC fingerprint m
56 e molecular mechanisms underlying folding of mammalian chromosomes remain poorly understood.
57 which may be critical for a fully functional mammalian chromosome, remains poor.
58                             Establishing how mammalian chromosome replication is regulated and how gr
59                                              Mammalian chromosome replication starts from distinct si
60 at-rich pericentromeric regions of plant and mammalian chromosomes results from their high level of D
61                    Replication time bands in mammalian chromosomes show overall congruency with struc
62  channel gene or gene cluster on a different mammalian chromosome, supporting the hypothesis that fou
63                                              Mammalian chromosomes terminate in stretches of repetiti
64                                              Mammalian chromosomes terminate with a 3' tail which con
65 breakpoint reuse are distinctive features of mammalian chromosomes that are not well understood in ev
66  leading to initiation of DNA replication in mammalian chromosomes, the time when hamster origin reco
67 rying defined site-specific DNA lesions into mammalian chromosomes, using phage integrase-mediated in
68 repair of DNA double-strand breaks (DSBs) in mammalian chromosomes, we designed DNA substrates contai
69 uble-strand break (DSB)-induced mutations in mammalian chromosomes, we stably transfected thymidine k
70 uble-strand break (DSB)-induced mutations in mammalian chromosomes, we transfected thymidine kinase (
71   Using an optical tweezers system, isolated mammalian chromosomes were held in a 1064 nm laser trap.
72 riants form a unique epigenetic landscape on mammalian chromosomes where the principal epigenetic het
73  systems and between bacterial nucleoids and mammalian chromosomes with respect to physical propertie
74        Here we introduce an approach to tile mammalian chromosomes with self-mapping fluorescent labe