ライフサイエンスコーパス: manic episode

1 otic agent following remission from an acute manic episode.

2 ive episode, and the other two experienced a manic episode.

3 every suicide attempt made before the index manic episode.

4 had negative correlations with the number of manic episodes.

5 ssociation between KYNA levels and number of manic episodes.

6 ite polarity symptom levels in depressive or manic episodes.

7 t that impaired sleep can induce and predict manic episodes.

8 was associated with a higher number of prior manic episodes.

9 are associated with nonremission from mixed manic episodes.

10 ive symptoms and treatment response in acute manic episodes.

11 fy individuals who will develop future (hypo)manic episodes.

12 ammatory markers in BD patients experiencing manic episodes.

13 ng was observed in association with frequent manic episodes.

14 After a first manic episode, 1 year of randomized treatment with lithi

15 attributable to the familial specificity of manic episodes after adjusting for both proband and rela

16 The preventive effect is clear for manic episodes, although it is equivocal for depressive

17 s were 17 patients hospitalized for an acute manic episode and 12 group-matched comparison subjects.

18 n view of the efficacy in prevention of both manic episode and depressive episode relapse or recurren

19 mong subjects 18.0 years or older, 44.4% had manic episodes and 35.2% had substance use disorders.

20 al focus is warranted on connections between manic episodes and anxiety disorders.

21 n between the rise of new-onset psychotic or manic episodes and national incidence of COVID19 cases,

22 between Wave 1 major depressive episode with manic episodes and other psychiatric disorders.

23 the strength of associations between Wave 1 manic episodes and Wave 2 depression, anxiety and substa

24 agnostic Criteria and DSM-III-R criteria for manic episodes and were rated before institution of phar

25 may have more depressive episodes (and fewer manic episodes) and may be more likely to suffer from mi

26 der, defined by the presence of a syndromal, manic episode, and bipolar II disorder, defined by the p

27 orders (major depressive episode, dysthymia, manic episode), anxiety (panic, separation anxiety, over

28 sodes significantly increased risk of Wave 2 manic episodes (AOR: 2.2; 1.7-2.9) and anxiety disorders

29 Manic episodes are one of the major diagnostic symptoms

30 -IV bipolar I disorder experiencing an acute manic episode (baseline Young Mania Rating Scale score >

31 ducation, numbers of previous depressive and manic episodes, baseline scores on the Hamilton Rating S

32 reatment outcomes in bipolar disorder during manic episodes (BDM).

33 ear interval, the longer they had spent in a manic episode before baseline.

34 y hospital in Sweden and receiving ECT for a manic episode between 2012 and 2019 were considered for

35 Patients who experienced hypomanic or manic episodes between time points showed abnormal thinn

36 isorder can be diagnosed on the basis of one manic episode); bipolar disorder type II (depressive and

37 ike the number and duration of depressive or manic episodes both during the two-year interval and bef

38 polar disorder patients who had had repeated manic episodes, but it does not appear to be secondary t

39 d TNF-a were elevated in both depressive and manic episodes, but not in euthymia, while IL-6 remained

40 These data suggest that manic episodes can be naturalistically classified as cla

41 BD patients with at least one depressive or manic episode during the two-year interval, BD patients

42 Manual of Mental Disorders, Fourth Edition) manic episodes during the study's 3-year follow-up perio

43 for IL-6 during euthymia, and higher during manic episodes for CRP, IL-6, and TNF-a.

44 ted in BD-I compared with BD-II and MDD, and manic episodes had focused progressive effect on the CA2

45 Adults with manic episodes have an approximately equivalent relative

46 Manic episodes have been clinically classified a posteri

47 pathological mechanisms of manic syndrome or manic episodes in bipolar disorder remain poorly charact

48 ian gene disruption and the precipitation of manic episodes in bipolar disorder.

49 recently been approved for stabilization of manic episodes in patients with bipolar disorder.

50 ts capable of triggering both depressive and manic episodes in patients with BPD.

51 y greater number of depressive and hypomanic/manic episodes in the prior year.

52 A substantial number of manic episodes include conspicuous depressive symptoms.

53 ipramine, an antidepressant that can trigger manic episodes, increased synaptic expression of GluR1 i

54 ty that there might be a natural division of manic episodes into clinical types.

55 convulsive therapy (ECT) in the treatment of manic episodes is based on clinical experience, but empi

56 d unequivocal evidence for efficacy in acute manic episodes, lithium in acute depressive episodes and

57 The presence of depressive symptoms during manic episodes may be associated with poor response to p

58 ar disorder who were experiencing a mixed or manic episode (n = 100) and healthy youth (n = 63).

59 ria within 3 months of the onset of an index manic episode (n = 372) were randomized to maintenance t

60 episodes and negatively associated with any manic episodes (odds ratio=0.76, 95% CI=0.69-0.84).

61 zophrenia was positively associated with any manic episodes (odds ratio=1.13, 95% CI=1.01-1.27), but

62 disorder was positively associated with any manic episodes (odds ratio=1.23, 95% CI=1.09-1.38).

63 modulation, have been reported during acute manic episodes of bipolar disorder (BD).

64 5, all patients with a diagnosis of a single manic episode or bipolar disorder between January 1, 199

65 had both a major mental disorder (psychosis, manic episode, or major depressive episode) and a substa

66 compared to HC (P = 0.008) especially during manic episodes (P = 0.0002).

67 as associated with psychotic features during manic episodes (P=0.003).

68 was clustered in later adult age groups for manic episode, phobias, alcohol use disorders, and gener

69 When considering euthymic, depressive, and manic episodes separately, CRP and TNF-a were elevated i

70 .89, 1.45-2.48, p<0.0001), greater number of manic episodes (seven studies, 3909 participants; 1.26,

71 nce of treating bipolar patients, for rating manic episode severity).

72 Individuals with a manic episode showed the greatest reductions in tryptoph

73 In multivariable models, Wave 1 manic episodes significantly increased the odds of Wave

74 ic condition characterized by depressive and manic episodes that affect 2% of the world population.

75 te extensive studies on the mechanism of the manic episodes, the molecular targets that control manic

76 ude: bipolar disorder type I (depressive and manic episodes: this disorder can be diagnosed on the ba

77 Secondary measures were time to a manic episode, time to a depressive episode, average cha

78 tance of long-term prophylaxis after a first manic episode to lessen episode recurrence, allow cognit

79 Immediately following remission of a manic episode treated with the combination of a typical

80 ects with child BP-I, the 44.4% frequency of manic episodes was 13 to 44 times higher than population

81 ed relapse to mania, and more weeks ill with manic episodes was predicted by low maternal warmth and

82 More (hypo)manic episodes were associated with faster cortical thin

83 d study, 179 patients hospitalized for acute manic episodes were randomized to receive divalproex sod

84 circadian rhythm can trigger transitions to manic episodes, whereas depressive states are reversed.

85 had proven effective in managing most of her manic episodes, while her two most severe episodes had b

86 xed anxiety depression; replacement of mixed manic episodes with a 'mixed features' specifier applica

87 ity was greater for depressive episodes than manic episodes, with approximately 74.0% of respondents

88 e monotherapy displayed an increased rate of manic episodes within 3 months of medication initiation