ライフサイエンスコーパス: marcescens

1 rocessive endochitinase, ChiC, from Serratia marcescens.

2 a TagJ homologue as shown here with Serratia marcescens.

3 ia burhodogranaria, is gram negative like S. marcescens.

4 fection with the bacterial pathogen Serratia marcescens.

5 by a Gram-negative entomopathogen, Serratia marcescens.

6 e positive for P. aeruginosa also yielded S. marcescens.

7 undii group, Enterobacter spp., and Serratia marcescens.

8 D efflux systems in biocide resistance in S. marcescens.

9 ors from Pseudomonas aeruginosa and Serratia marcescens.

10 rs from some strains preferred E. coli to S. marcescens.

11 anism for the second step of secretion in S. marcescens.

12 gy-dependent and Sec-dependent pathway in S. marcescens.

13 cetylneuraminic acid, and n-butanol using S. marcescens.

14 y any environmental or staff reservoir of S. marcescens.

15 eptomyces coelicolor and SmAA10A of Serratia marcescens.

16 ively controlled prodigiosin synthesis in S. marcescens.

17 re E. cloacae complex, K. pneumoniae, and S. marcescens.

18 teria, including the human pathogen Serratia marcescens.

19 d T6SS-mediated antibacterial activity in S. marcescens.

20 uginosa, Staphylococcus aureus, and Serratia marcescens.

21 losely related clinical isolates of Serratia marcescens.

22 dengue virus infection than those free of S. marcescens.

23 n intrinsic multidrug resistance in Serratia marcescens.

24 example is serratin, isolated from Serratia marcescens.

25 the processive chitinase ChiA from Serratia marcescens.

26 ecorina and the chitinase ChiA from Serratia marcescens.

27 uce SlpB as a new cytotoxic protease from S. marcescens.

28 onized with provisionally matching GBS or S. marcescens.

29 tance test using the live bacterium Serratia marcescens.

30 , including Clostridium species and Serratia marcescens.

31 m the opportunistic human pathogen, Serratia marcescens.

32 insect survival after oral infection with S. marcescens.

33 ecies (3%), Proteus mirabilis (2%), Serratia marcescens (0.6%), and Pseudomonas aeruginosa (0.5%).

34 ), Pseudomonas aeruginosa (10), and Serratia marcescens (1), were included; and 123 (98.4%) were accu

35 ), Klebsiella pneumoniae (18%), and Serratia marcescens (12%).

36 21 Enterobacter cloacae complex, 18 Serratia marcescens, 12 Proteus mirabilis, 10 Citrobacter koseri,

37 rance of a bacterial infection with Serratia marcescens, 3 Acps significantly reduced the bacterial c

38 (9.1%), Acinetobacter spp. (6.2%), Serratia marcescens (5.5%), Enterobacter aerogenes (4.4%), Stenot

39 th Acinetobacter spp., P. aeruginosa, and S. marcescens, 5/6 with Citrobacter spp., 13/14 with Entero

40 Finally, recombinant expression of the S. marcescens 56-kDa metalloprotease conferred a cytotoxic

41 Secretomes from 95% of Serratia marcescens, 71% of Pseudomonas aeruginosa, 29% of Staphy

42 Serratia marcescens, a member of the carbapenem-resistant Enterob

43 Serratia marcescens, a member of the Enterobacteriaceae family, i

44 of anthranilate synthase (AS) from Serratia marcescens, a mesophilic bacterium, has been solved in t

45 lity of the tigecycline Etest for testing S. marcescens, Acinetobacter spp., and S. pneumoniae is war

46 one acetyltransferase 1) and SmAAT (Serratia marcescens aminoglycoside 3-N-acetyltransferase), sugges

47 related N-acetyltransferase (GNAT), Serratia marcescens aminoglycoside 3-N-acetyltransferase, bound t

48 ast histone acetyltransferase 1 and Serratia marcescens aminoglycoside 3-N-acetyltransferase.

49 ive means to control the dissemination of S. marcescens, an in-depth analysis of the population struc

50 (R)-(-)-mellein and micromolide, against S. marcescens and a Gram-positive bacterium, Staphylococcus

51 An outbreak of S. marcescens and E. cloacae bacteremia in a surgical inten

52 infusions from two case patients yielded S. marcescens and E. cloacae.

53 crobial pigment, was produced using Serratia marcescens and encapsulated with beta-cyclodextrin (BCD)

54 , Acinetobacter baumannii, E. coli, Serratia marcescens and Enterobacter cloacae complex.

55 s of the cheA loci from isolates of Serratia marcescens and Enterobacter cloacae, demonstrating the p

56 tate transcarbamoylases (ATCase) of Serratia marcescens and Escherichia coli differ in both regulator

57 tate transcarbamoylases (ATCase) of Serratia marcescens and Escherichia coli have distinct allosteric

58 o the natural product isolated from Serratia marcescens and from overexpression of the biosynthetic g

59 hit operons previously reported for Serratia marcescens and Haemophilus influenzae, respectively, and

60 screen for loss of virulence of ingested S. marcescens and identified FliR, a structural component o

61 of TLM on the ecFabB homologues in Serratia marcescens and Klebsiella pneumonia is an important fact

62 ormaechei, Acinetobacter baumannii, Serratia marcescens and Leclercia adecarboxylata are dominant; ST

63 n of bronchoscopes with P. aeruginosa and S. marcescens and possible infection of patients at a commu

64 digiosin has implications in virulence of S. marcescens and promising clinical applications.

65 acterise the tripartite SmhABC toxin from S. marcescens and propose a mechanism of pore assembly.

66 tracellular polysaccharides production in S. marcescens and provides important clues for future studi

67 The rare gut bacteria Serratia marcescens and Pseudomonas protegens contributed to atra

68 es and in the ants of two bacteria, Serratia marcescens and S. entomophila, which are involved in the

69 late in midgut cells in response to Serratia marcescens and Sindbis virus or when the native microbio

70 gans against the bacterial parasite Serratia marcescens and tested for a correlation between them.

71 noted among Acinetobacter spp. and Serratia marcescens and, to a lesser extent, with Streptococcus p

72 s spp., Pseudomonas aeruginosa, and Serratia marcescens) and 6 antimicrobial resistance determinants

73 tion by a focal non-core bacterium (Serratia marcescens) and its consequences for bee health.

74 R-flagellins from Serratia marcescens (S. marcescens) and Salmonella muenchen (S. muenchen) do not

75 xidase activity, resistance against Serratia marcescens), and for the life history traits, age and si

76 lomerance., Microbacterium sp., and Serratia marcescens), and their nine mixture treatments in tripli

77 homologues, a homologue of OmpF of Serratia marcescens, and a locus (designated rscBAC) with similar

78 r roggenkampii, Klebsiella oxytoca, Serratia marcescens, and Citrobacter farmeri.

79 ividuals exhibited reduced preference for S. marcescens, and dauers from some strains preferred E. co

80 eradication of Pseudomonas species, Serratia marcescens, and Enterobacter aerogenes in most of the tr

81 ion including Citrobacter freundii, Serratia marcescens, and Klebsiella aerogenes.

82 freundii, Yersinia enterocolitica, Serratia marcescens, and Morganella morganii) and two nonenteric

83 a gonorrhoeae and N. meningitidis), Serratia marcescens, and other gram-negative bacteria utilize a p

84 egative bacteria (Escherichia coli, Serratia marcescens, and Pseudomonas aeruginosa).

85 faciens, Agrobacterium radiobacter, Serratia marcescens, and Pseudomonas aureofaciens) and fungi (Phy

86 ent was observed, excluding A. baumannii, S. marcescens, and S. pneumoniae, for which >/=4-fold diffe

87 r to biotin synthases from E. coli, Serratia marcescens, and Saccharomyces cerevisiae (about 50% sequ

88 holderia cepacia, Escherichia coli, Serratia marcescens, and Stenotrophomonas maltophilia isolates.

89 rst reports applying PFGE to the study of S. marcescens, and this method was a useful marker of strai

90 iotic-treated mosquitoes identified Serratia marcescens as a commensal bacterium critical for efficie

91 Using Serratia marcescens as a model organism, we identify here a stage

92 ify a common fecal enterobacterium, Serratia marcescens, as the causal agent of white pox.

93 tic collection of antimicrobial-resistant S. marcescens associated with bloodstream infections in hos

94 stimulates the catalytic activity of the S. marcescens ATCase and CTP/UTP inhibitory synergism has b

95 s, the temperature response of the native S. marcescens ATCase suggests a strong entropic effect that

96 The mechanisms by which S. marcescens attacks enterocytes and damages the intestina

97 A case was defined as the occurrence of S. marcescens bacteremia in any patient in the surgical int

98 ing to univariate analysis, patients with S. marcescens bacteremia stayed in the surgical intensive c

99 Twenty-six patients with S. marcescens bacteremia were identified; eight (31 percent

100 y risk factors, we compared patients with S. marcescens bacteremia with randomly selected controls.

101 ve care unit of a hospital acquired Serratia marcescens bacteremia.

102 roswimmer system driven by multiple Serratia marcescens bacteria, we quantify the chemotactic drift o

103 ogenes, K. pneumoniae, P. aeruginosa, and S. marcescens) became more susceptible.

104 Experiments showed that Serratia marcescens better colonizes the gut when bees are inocul

105 which OxyR contributes to early stages of S. marcescens biofilm formation by influencing fimbrial gen

106 In March 2011, Serratia marcescens bloodstream infections (BSIs) were identified

107 Cases were defined as S. marcescens BSIs in patients receiving PN from the pharma

108 g standards contributed to an outbreak of S. marcescens BSIs.

109 ter they fed on the insect pathogen Serratia marcescens but not after feeding on the Leishmania that

110 t species like B. thailandensis and Serratia marcescens, but also a majority of Gram-negative and Gra

111 Purified LPS from S. marcescens, but not from Escherichia coli or S. marcesce

112 observed with another CGD pathogen, Serratia marcescens, but not with Escherichia coli.

113 Serratia marcescens can cause a range of severe infections and co

114 l antibiotic resistance enzyme from Serratia marcescens catalyzes adenylation and acetylation of amin

115 acterium and opportunistic pathogen Serratia marcescens causes ocular infections in healthy individua

116 plicable to emerging clinical isolates of S. marcescens causing bacteremia.

117 rmined that the addition of a chi-induced S. marcescens cell lysate to an uninfected culture causes a

118 Here, the development and evaluation of a S. marcescens cgMLST scheme is reported with the goal of en

119 It has been reported that a S. marcescens chimera, SM : rS5'ec, in which five divergent

120 ha + beta domain similar to that of Serratia marcescens chitinases A and B.

121 asured differences in processivity of the S. marcescens chitinases.

122 tion methods are insufficient to identify S. marcescens complex and laboratory reporting should be mo

123 ultiple members of the recently described S. marcescens complex causing hospital- or community-associ

124 Between-patient transmission of S. marcescens complex outside of outbreaks is likely rare.

125 re is no standardized analytic scheme for S. marcescens core genome multilocus sequence typing (cgMLS

126 of E. coli have been replaced with their S. marcescens counterpart, lost both heterotrophic and homo

127 Serratia marcescens culture filtrates have been reported to be cy

128 treatments abrogated the cytotoxicity of S. marcescens culture filtrates towards HeLa cells, suggest

129 o cytotoxic activity commonly observed in S. marcescens culture filtrates.

130 ve detection of Escherichia coli or Serratia marcescens cultures from 1 to 10(3) CFU mL(-1).

131 The hemophore protein HasA from Serratia marcescens cycles between two states as follows: the hem

132 t putative RND efflux system genes in the S. marcescens Db10 genome that included the previously char

133 g effectors, exemplified by Ssp4 of Serratia marcescens Db10.

134 genomic sequence of NCBI reference strain S. marcescens Db11 (NZ_HG326223.1) as a starting point-all

135 Bioinformatic analysis of the S. marcescens Db11 genome revealed three additional open re

136 etabolites based on genomic annotation of S. marcescens Db11.

137 ized secretomes from wild-type and mutant S. marcescens derivatives.

138 We hypothesize that S. marcescens detects the threat of phage-mediated cell dea

139 S. marcescens does not normally colonize human skin, but ar

140 and prodigiosin concentration changes for S. marcescens during cultivation in batch culture.

141 Serratia marcescens, E. cloacae, and Enterobacter kobei presented

142 Fis are 100% identical in K. pneumoniae, S. marcescens, E. coli, and S. typhimurium and 96 to 98% id

143 One crystal form of the S. marcescens enzyme displays a bound pyruvate as well as a

144 r97) of the regulatory polypeptide of the S. marcescens enzyme have been replaced with their E. coli

145 etained 455 out of 460 amino acids of the S. marcescens enzyme, it possessed characteristics similar

146 ric bacteria Klebsiella pneumoniae, Serratia marcescens, Erwinia carotovora, and Proteus vulgaris but

147 terobacteria Klebsiella pneumoniae, Serratia marcescens, Erwinia carotovora, and Proteus vulgaris, st

148 thms, especially in differentiating Serratia marcescens, Escherichia coli, and Yersinia enterocolitic

149 ample, for growth on N-acetylglucosamine, S. marcescens exhibits high pentose phosphate pathway activ

150 S. marcescens expresses prodigiosin, a bright red and cell-

151 The Serratia marcescens extracellular nuclease gene, nucA, is positiv

152 amily of mutants overexpressing the Serratia marcescens extracellular nuclease has been known for dec

153 The Serratia marcescens extracellular nuclease is a secreted protein

154 reted by the Gram-negative bacteria Serratia marcescens, extracts heme from host hemoproteins and shu

155 S. marcescens facilitates arboviral infection through a sec

156 a pneumoniae, Escherichia coli, and Serratia marcescens, for which the trend is >= 30% and therefore

157 igation, allowing better understanding of S. marcescens genomic epidemiology and facilitating interla

158 was refined by integrating RNAseq data of S. marcescens growth on three different carbon sources (glu

159 trains, 89% of K. pneumoniae, and half of S. marcescens had an extended-spectrum B-lactamase phenotyp

160 -), and TLR4/5(-/-) corneas infected with S. marcescens had significantly increased CFU, indicating i

161 Hemophores from Serratia marcescens (HasA(sm)) and Pseudomonas aeruginosa (HasA(p

162 We included first-time isolates of S. marcescens identified via culture and confirmed by matri

163 l-known chitinases and an LPMO from Serratia marcescens Importantly, comparison of the chitinolytic e

164 phenoloxidase activity and resistance to S. marcescens in both sexes, but these relationships were l

165 ven of these had Enterobacter cloacae and S. marcescens in the same culture.

166 ter initial developmental work with Serratia marcescens in water, Salmonella Typhimurium ATCC 14028 w

167 a pneumoniae, Escherichia coli, and Serratia marcescens, in a further nine of 11 (81%) cases where no

168 pairs of the trpEDCBA operator from Serratia marcescens indicated an obligate hierarchy of site occup

169 Tobramycin-killed S. marcescens induced corneal inflammation in C57BL/6 mice,

170 S. marcescens induced neutrophil recruitment to the corneal

171 interleukin-1 receptor type 1 (IL-1R1) in S. marcescens-induced corneal inflammation and infection.

172 ial therapeutic targets for inhibition of S. marcescens-induced corneal inflammation.

173 S. marcescens induces corneal inflammation by activation of

174 Several risk factors for S. marcescens infection were identified, but hospital and h

175 reaks of Pseudomonas aeruginosa and Serratia marcescens infections associated with bronchoscopy have

176 ant clones suggests that the treatment of S. marcescens infections will become increasingly difficult

177 nia hospital acquired postoperative Serratia marcescens infections, and 1 died.

178 Oral introduction of S. marcescens into field mosquitoes that lack this bacteriu

179 rated by the opportunistic pathogen Serratia marcescens involved in activation of autophagy.

180 Serratia marcescens is a bacterium frequently found in the enviro

181 Serratia marcescens is a chitinolytic bacterium that can potentia

182 Our results show that S. marcescens is a diverse species with a high level of gen

183 Chitinase B (ChiB) from Serratia marcescens is a family 18 exo-chitinase whose catalytic

184 Serratia marcescens is a Gram-negative bacterium of the Enterobac

185 Serratia marcescens is a gram-negative environmental bacterium an

186 Serratia marcescens is a red pigment (prodigiosin)-producing Gram

187 Serratia marcescens is a soil- and water-derived bacterium that s

188 Serratia marcescens is a well-known cause of nosocomial infection

189 Serratia marcescens is an extremely rare cause of necrotizing fas

190 Serratia marcescens is an opportunistic AmpC beta-lactamase-produ

191 Serratia marcescens is an opportunistic bacterium that infects a

192 Serratia marcescens is an opportunistic human pathogen involved i

193 Serratia marcescens is an opportunistic human pathogen that produ

194 The enteric bacterium Serratia marcescens is an opportunistic human pathogen.

195 Serratia marcescens is an opportunistic pathogen associated with

196 Serratia marcescens is frequently isolated from lenses of patient

197 The extracellular nuclease of Serratia marcescens is one of a wide variety of enzymes secreted

198 Currently, S. marcescens is poorly characterized and studies on intrac

199 T6SS of the opportunistic pathogen Serratia marcescens, is a toxin that forms ion-selective pores.

200 racterize the iron acquisition systems in S. marcescens isolate UMH9, which was recovered from a clin

201 A Serratia marcescens isolate was particularly efficient in coloniz

202 Pseudomonas aeruginosa isolates, 1 Serratia marcescens isolate, 1 Aeromonas hydrophila isolate, 1 Ae

203 Three bla(SME)-carrying Serratia marcescens isolates and one bla(NDM-1) carrying Providen

204 nt inactivating mutations, exclusively in S. marcescens isolates of clinical origin.

205 ted an investigation of P. aeruginosa and S. marcescens isolates related to bronchoscopy at a communi

206 S. marcescens isolates were compared using restriction-endo

207 All K. pneumoniae and S. marcescens isolates were resistant to ampicillin, and th

208 rity of the K. pneumoniae and half of the S. marcescens isolates were resistant to both cefotaxime an

209 lates, 5 Enterobacter cloacae isolates, 2 S. marcescens isolates, 1 Proteus mirabilis isolate, and 2

210 Together these data suggest that S. marcescens LPS is sufficient for inhibition of epithelia

211 2%), E. coli (<1%), K. oxytoca (<1%), and S. marcescens (<1%).

212 ectedly, fliR but not flhD is involved in S. marcescens-mediated damages of the intestinal epithelium

213 Literature pertinent to S. marcescens-mediated necrotizing fasciitis is also review

214 alpha-thrombin by only 10% whereas Serratia marcescens metalloprotease reduced the Ca2+ response by

215 Our results show that S. marcescens model iSR929 can provide reasonable predictio

216 in vitro cytotoxic activity revealed that S. marcescens mutant strains that are deficient in producti

217 ation in the opportunistic pathogen Serratia marcescens, mutations in an oxyR homolog and predicted f

218 main cause of neonatal sepsis, with Serratia marcescens (n = 151), Klebsiella michiganensis (n = 117)

219 = 23), Klebsiella oxytoca (n = 8), Serratia marcescens (n = 6), Citrobacter freundii (n = 4), and Kl

220 eruginosa (n = 2 and n = 5), two of Serratia marcescens (n = 9 and n = 7), five of Staphylococcus aur

221 Despite multiple clinical descriptions of S. marcescens nosocomial pneumonia, little is known regardi

222 tracellular nuclease (Nuc) from the Serratia marcescens nucA chromosomal locus is inhibited in cells

223 The Serratia marcescens NucC protein is structurally and functionally

224 Extracellular secretion of Serratia marcescens nuclease occurs as a two-step process via a p

225 NucC binding to the S. marcescens nuclease promoter P(nucA) and to the sequence

226 . coli for extracellular secretion of the S. marcescens nuclease.

227 birth, but gut colonization with GBS and S. marcescens occurred closer to time of bloodstream infect

228 al secondary metabolite produced by Serratia marcescens, on HSV infection.

229 tion and in infections with Gram-negative S. marcescens or Gram-positive E. faecalis bacteria, which

230 iming during infections with either Serratia marcescens or with Escherichia coli.

231 tained group B Streptococcus (GBS), Serratia marcescens, or Escherichia coli before their sepsis epis

232 t is observed in other bacterial species, S. marcescens OxyR is required for oxidative stress resista

233 S. marcescens oxyR mutants were severely impaired in biofil

234 that serratiochelin is required for full S. marcescens pathogenesis in the bloodstream.

235 3 of 15), and Enterobacter cloacae, Serratia marcescens, Pneumocystis carinii pneumonia, and unknown

236 aspiration model of lethal and sublethal S. marcescens pneumonia in BALB/c mice and extensively char

237 This study describes a model of S. marcescens pneumonia that mimics known clinical features

238 uginosa PAO1, Proteus mirabilis and Serratia marcescens, possibly by interfering with their QS system

239 hat is located upstream of NucC-dependent S. marcescens promoters and the late promoters of P2-relate

240 located upstream of NucC-dependent Serratia marcescens promoters and the late promoters of P2-relate

241 herichia coli, Salmonella muenchen, Serratia marcescens, Proteus mirabilis, and Proteus vulgaris).

242 re in complex with chitinase B from Serratia marcescens provide further insight into the mechanism of

243 we discover that the E2 enzyme from Serratia marcescens regulates cGAS by imitating the ubiquitinatio

244 ind secondary metabolites biosynthesis in S. marcescens remains limited.

245 volution with a bacterial pathogen (Serratia marcescens) resulted in significantly more outcrossing i

246 R-flagellins from Serratia marcescens (S. marcescens) and Salmonella muenchen (S. m

247 oreskin fibroblasts was also inhibited by S. marcescens secretomes indicating that the effect is not

248 LPS depletion of S. marcescens secretomes with polymyxin B agarose rendered

249 d host survival in a mouse model of Serratia marcescens sepsis.

250 nella enterica serovar Typhimurium, Serratia marcescens, Shigella flexneri, Enterobacter aerogenes, K

251 , which, based on their homology to Serratia marcescens shlA and shlB genes, are believed to encode t

252 a targeted protease, Enhancin from Serratia marcescens (SmE), with ultraviolet photodissociation (UV

253 pneumoniae, Pseudomonas aeruginosa, Serratia marcescens, Staphylococcus aureus, and Stenotrophomonas

254 c bacteriophage chi (Chi) to a culture of S. marcescens stimulates a greater than fivefold overproduc

255 owth-promoting rhizobacteria (PGPR) Serattia marcescens strain 90-166 and Bacillus pumilus strain SE3

256 e clinically isolated multidrug-resistant S. marcescens strain and found that the sdeXY deletion muta

257 nous bacteria (Enterobacter sp. and Serratia marcescens strain Db11) and parasitic African trypanosom

258 binding protein, RsmA, in Ecc71 and Serratia marcescens strain SM274.

259 The VME was found for a single Serratia marcescens strain.

260 s transporter ShlB resulted in attenuated S. marcescens strains that failed to cause profound weight

261 cescens, but not from Escherichia coli or S. marcescens strains with mutations in the waaG and waaC g

262 chromosome of carbapenem-resistant Serratia marcescens strains.

263 lum mutants fliR and flhD in two distinct S. marcescens strains.

264 rthermophile Sulfolobus solfataricus, the S. marcescens structure shows similar subunit structures bu

265 y ingesting the pathogenic bacteria Serratia marcescens, suggesting that subdued has novel functions

266 oteins are the original substrates of the S. marcescens T6SS, before horizontal acquisition of other

267 s of the chest wall due to infection with S. marcescens that initially manifested as bilateral breast

268 e set of D. melanogaster lines with Serratia marcescens, the bacterium used in the previous study, an

269 hogens (the gram-negative bacterium Serratia marcescens, the gram-positive bacterium Bacillus cereus

270 Rhs1 effectors from two strains of Serratia marcescens, the model strain Db10 and clinical isolate S

271 m on prodigiosin (PG) production by Serratia marcescens TKU011 is examined.

272 Both genes are required for S. marcescens to escape the gut lumen into the hemocoel, in

273 We adsorbed swarmer cells of Serratia marcescens to polydimethylsiloxane or polystyrene.

274 to characterize the metabolic profile of S. marcescens to provide insight for metabolic engineering

275 When hasF, encoding the S. marcescens TolC ortholog, was expressed in KAM43, all of

276 a and experimental Tn-Seq data from Serratia marcescens transposon mutant library used to identify ge

277 the chitinase secretion pathway of Serratia marcescens uses an endopeptidase to facilitate secretion

278 udomonas spp., Salmonella enterica, Serratia marcescens, Vibrio vulnificus and Enterobacter nimipress

279 ection with Burkholderia cepacia or Serratia marcescens was caused by a new strain in 9 of 10 cases (

280 In summary, a novel cgMLST scheme for S. marcescens was developed and evaluated.

281 The activity of the fraction against S. marcescens was explained by (R)-(-)-mellein alone, and t

282 In addition, the clearance of S. marcescens was genetically correlated with the resistanc

283 S. marcescens was identified from a pharmacy water faucet,

284 knockout mice was abraded, and 1 x 10(7) S. marcescens were added in the presence of a silicone hydr

285 0 nM), and Enterobacter cloacae and Serratia marcescens were highly resistant (IC(50), >10,000 nM).

286 Field Aedes mosquitoes positive for S. marcescens were more permissive to dengue virus infectio

287 Four hundred ninety-one high-quality S. marcescens WGS data sets were extracted from public data

288 me the virulent bacterial parasite, Serratia marcescens, when given a choice between the parasite and

289 ases produced by P. fluorescens and Serratia marcescens, which comprise a second sequence family, be

290 e the structure of chitinase B from Serratia marcescens, which consists of a catalytic domain with a

291 sis except for Escherichia coli and Serratia marcescens, which could not be interdifferentiated using

292 solated the Gram-negative bacterium Serratia marcescens, which is a potent entomopathogen that can ra