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1 1 cm), with higher hematocrit showing faster margination.
2 hat hematocrit plays a role in the degree of margination.
3 2 interactions in regulating lung neutrophil margination.
4 r n-3 fatty acids affect fasting and fed TRL margination.
5 al cell activation as evidenced by leukocyte margination.
6 antly altering blood neutrophil half-life or margination.
7 .58 vs. 3.4 +/- 0.20; p < 0.0001), chromatin margination (14.2 +/- 0.57 vs. 6.5 +/- 0.37; p < 0.0001)
8                                              Margination and adhesion should be independently address
9 l NPs in the flow play a dominant role in NP margination and cell interaction, compared to Brownian m
10 -3-positive neurons also exhibited chromatin margination and condensation, chromatin balls, and nucle
11     In this paper, we demonstrate that early margination and emigration of neutrophils originate in t
12      The roles of selectins in the pulmonary margination and emigration of neutrophils were investiga
13 fficacy because of their effects on particle margination and interactions with various cells in vivo.
14 red blood cell (RBC) interaction on platelet margination and near-wall dynamics in a shear flow.
15 models of whole blood to understand platelet margination and near-wall platelet dynamics.
16 ction was associated with reduced neutrophil margination and pulmonary myeloperoxidase activity.
17  support hemostasis by facilitating platelet margination and releasing platelet-activating factors su
18                    However, the influence of margination and shear rate on occlusive clot formation i
19 es from 1 hour of reperfusion revealed dense margination and substantial emigration of neutrophils as
20 ocyte defects, such as decreased endothelial margination and tissue recruitment, are rate-limiting st
21 ctivated endothelial cells, mononuclear cell margination) and interstitial hemorrhage, but not the ex
22 ng (zeiosis), and chromatin condensation and margination, and decrease in cellular DNA content.
23                   Sub-G1 fraction, chromatin margination, and phosphatidylserine exposure were eviden
24               Although particle adhesion and margination are related, adhesion also depends on other
25 tration distribution profile and observe its margination at 10%, 20%, and 30% red blood cell hematocr
26 nular, or smudgy chromatin without prominent margination at the nuclear membrane; they exhibited smal
27 nce in multiple ways, including i) increased margination behavior in blood vessel-like flow chambers,
28 ing (zeiosis) and chromatin condensation and margination, both of which are hallmarks of apoptosis.
29  requirements-microparticles are optimal for margination, but nanoparticles are better for intracellu
30 e crucial role of the vessel geometry in the margination by calculations when the blood is seen as vi
31 tive particles can strongly accelerate their margination by moving against the flow direction: partic
32 ur results imply that a significantly faster margination can be achieved either technically by the ap
33 ate convective transport in the bloodstream, margination, cell adhesion, selective cellular uptake, a
34 imply alter leukocyte stiffness to fine tune margination/demargination and therefore leukocyte traffi
35 n the red blood cell-free layer and platelet margination due to an increase in stiffened RBCs present
36 s in flow, suggesting a decrease of platelet margination due to an increased fraction of stiffened RB
37 tumor via the folic acid-mediated target and margination effect.
38          These simulations also confirm that margination from an initially uniform distribution of sp
39                                     Although margination has beenmodeled by numerical simulations and
40           Experimental results indicate that margination has largely occurred before particles travel
41 eficiency normalized both LPL levels and TRL margination in BAT at 30 degrees C.
42 metal NPs likely results from their distinct margination in laminar blood flow, which opens up a new
43  selectively blocked IL-8-induced neutrophil margination in rabbits.
44 lular spaces with chromatin condensation and margination in the upper stratum spinosum in lesional sk
45 the first data demonstrating that neutrophil margination in uninfected pulmonary capillaries does not
46 tical physical property prescribing particle margination in vitro and in vivo blood flows.
47 is responsible for neutrophil chemotaxis and margination induced by IL-8.
48 BC) deformability in whole blood on platelet margination is investigated using confocal microscopy me
49                The vessel-geometry-regulated margination is then confirmed by in vitro experiments in
50                                              Margination occurs when blood borne particles attach to
51 tures characteristic of apoptosis, including margination of chromatin and crenated nuclei of cells in
52 pleted preOLs included nuclear condensation, margination of chromatin, and mitochondrial swelling.
53 he H5N1 infection also resulted in prolonged margination of circulating T lymphocytes and notable apo
54 heir progeny can induce the condensation and margination of host chromatin during productive infectio
55  is marked by inflammatory immune responses, margination of leukocytes, and parasitized erythrocytes
56                                   Deliberate margination of lysosomes is associated with reduced acid
57  allograft recipients indicated perivascular margination of monocytes and neutrophils, vascular endot
58 s neutrophilia was associated with increased margination of neutrophils within pulmonary capillaries
59 apoptotic changes including condensation and margination of nuclear chromatin, DNA fragmentation, and
60                          Here, we report the margination of stiffened RBCs in vivo, and reveal the cr
61                                              Margination of stiffened red blood cells has been implic
62  admixed with normal RBCs, leads to aberrant margination of the less-deformable RBC subpopulations to
63       GPIHBP1-bound LPL is essential for the margination of triglyceride-rich lipoproteins (TRLs) alo
64   The LPL*GPIHBP1 complex is responsible for margination of triglyceride-rich lipoproteins along capi
65 llary LPL levels were accompanied by reduced margination of TRLs along capillaries.
66                                          The margination parameter, M, is defined as the total number
67 nent peritubular capillary inflammatory cell margination; patchy interstitial hemorrhage; interstitia
68             In this study, we quantified the margination propensity of particles of varying diameters
69          Most previous studies evaluated the margination propensity of these particles via an adhesio
70                                              Margination refers to the migration of particles toward
71 tudy was undertaken to determine whether TRL margination reflects in vivo LpL activity and whether n-
72 sion pointing to a specific and rapid vessel margination response to cardiac injury.
73 d through a multistep paradigm that includes margination, selectin-mediated rolling, beta 2 integrin-
74 fic in the lungs, particularly in neutrophil margination, sequestration, and emigration, using L-sele
75 sions, with a nuclear phenotype of chromatin margination similar to that described for wild-type embr
76 ody expression may have been aided by forced margination through collision with erythrocytes.
77      Here, we evaluate the effect of RBCs on margination through microfluidic studies in vitro and by
78 efit of optimizing microparticles' efficient margination to enhance nanocarriers' transport to the va
79 tly used to establish the degree of monocyte margination to the coronary vasculature that could poten
80 l of acute cardiac injury and involves rapid margination to the coronary vasculature.
81 ation, such as plasma skimming and leukocyte margination, to separate leukocytes directly from whole
82 ity and use it as an express lane for a fast margination toward the wall.
83  of mature neutrophils from the bone marrow, margination, trafficking and transmigration through the
84 istone administration resulted in neutrophil margination, vacuolated endothelium, intra-alveolar hemo
85                                              Margination volume (MV) was calculated by subtracting tr
86                                    Leukocyte margination was differentially impaired in these mice du
87 t has long been known that platelets undergo margination when flowing in blood vessels, such that the
88 om blood flow to vesselwalls, also known as "margination",which promotes particle contact and adhesio
89 g their circadian oscillations and pulmonary margination, which contributes toward lung injury and se
90 n this study, an M-value of 0.2 indicated no margination, which was observed for all particle sizes i
91    Understanding the mechanisms that lead to margination will aid in tailoring the attributes of drug
92                      No defect in neutrophil margination within either capillaries or arterioles and
93 00 red blood cell profiles) but no change in margination within noncapillary pulmonary microvessels.