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1 e to up to 31 drugs within 5 days after bone marrow biopsy.
2 ith tryptase in estimating the need for bone marrow biopsy.
3 aggregates, and atypical mast cells on bone marrow biopsy.
4 94% and 100% for PET-CT and 40% and 100% for marrow biopsy.
5 ent accuracy to replace routine staging bone marrow biopsy.
6 2, t[8;21][q22;q22.1]) was diagnosed on bone marrow biopsy.
7 e of biopsy site than the usual random iliac marrow biopsy.
8 ne and serum immunoelectrophoresis, and bone marrow biopsy.
9 diagnosis usually depends on results of bone marrow biopsy.
10 mastocytosis) and thus candidates for a bone marrow biopsy.
11 the potential to reduce the need for staging marrow biopsy.
12 re red cell aplasia (PRCA) confirmed by bone marrow biopsy.
13 with hematologic response criteria and bone marrow biopsies.
14 ot density compared with normal control bone marrow biopsies.
15 relevance of expression in 55 archival bone marrow biopsies.
16 loid leukemia (AML) routinely undergo a bone marrow biopsy 7-10 days after induction chemotherapy to
18 Response was assessed by weekly CBC and bone marrow biopsy after cycle 2 and after each subsequent cy
21 tochemically by factor VIII staining of bone marrow biopsies and quantified by assessment of microves
22 s of blood vessels were measured in 145 bone marrow biopsies and the levels of vascular endothelial g
24 nts with low-risk MGUS can safely defer bone marrow biopsy and advanced imaging, and should undergo p
26 and negligible gene marking, diagnostic bone marrow biopsy and aspirate were performed at day 88.
29 iate- and high-risk MGUS should trigger bone marrow biopsy and bone imaging to detect overt MM and sh
31 ischemia in Tc-99m SPECT who underwent bone marrow biopsy and were allocated to cells (n=16) or plac
32 ischemia in Tc-99m SPECT who underwent bone marrow biopsy and were allocated to cells (n=16) or plac
33 up consisted of a complete blood count, bone marrow biopsy, and immunohistochemical and histochemical
34 is, in the selection of those needing a bone marrow biopsy, and in the documentation of disease progr
36 o identify those patients who require a bone marrow biopsy, and whether the pathogenesis of IA involv
37 suspicious lymphocytic infiltrates in a bone marrow biopsy as the sole suggestion of residual disease
39 ing, as well as follow-up allograft and bone marrow biopsies at 3 and 9 months, including analyses of
40 he diagnosis of 661 PMF patients with a bone marrow biopsy at presentation was revised according to m
43 ll lymphoma (DLBCL), the sensitivity of bone marrow biopsy (BMB) for the detection of bone marrow inv
45 an IgG kappa monoclonal gammopathy, and bone marrow biopsy confirmed smoldering multiple myeloma with
46 A percutaneous biopsy of the mass and bone marrow biopsy confirmed the diagnosis of primary adrenal
49 transformed plasma cells from the same bone marrow biopsy enables discovery of patient-specific tran
50 ide therapy should undergo surveillance bone marrow biopsy every 2 to 3 years and that those who show
52 icaria pigmentosa or the characteristic bone marrow biopsy finding of multifocal mast-cell aggregates
56 comprehensive proteogenomic analysis of bone marrow biopsies from 252 uniformly treated AML patients
58 n factor (MITF), is highly expressed in bone marrow biopsies from 9 of 10 patients with systemic mast
65 taneous analysis of WM patient sera and bone marrow biopsies identified a set of dysregulated cytokin
66 tochemical paraffin section staining of bone marrow biopsies in the staging of B-cell malignant lymph
69 only used test to estimate the need for bone marrow biopsy in patients suspected to have indolent sys
71 r DNA (ctDNA) is directly comparable to bone marrow biopsy in representing the genomic heterogeneity
78 TA-anti-CD45 antibody (BC8) imaging and bone marrow biopsy measurements to ascertain the biodistribut
79 se activity in the plasma isolated from bone marrow biopsies of 100 patients reveals 86 positive for
80 dominant clonotypes in blood and in historic marrow biopsies of 35 AA, 37 MDS, and 21 paroxysmal noct
81 quantify IDO-1 expression on diagnostic bone marrow biopsies of AML patients in order to facilitate i
82 cutoff correctly classifies diagnostic bone marrow biopsies of MPN,U patients specified upon follow-
85 alth record; review of laboratory data; bone marrow biopsy pathology analysis; and in vitro enzymatic
86 features with the exception that their bone marrow biopsy pathology revealed abundant neutrophils co
87 ased risk of identifying pathology on a bone marrow biopsy performed for a low WBC count (odds-ratio
89 post-treatment assessments were serial bone-marrow biopsy, peripheral blood collections, staging, se
90 Cr corresponded with the high degree of bone marrow biopsies positive for atypical mast cells, the pr
95 ine model of Ph+ B-ALL as well as human bone marrow biopsy samples to assess the fundamental nature o
96 /mL, <15% infiltration by mast cells in bone marrow biopsy-sections, and diagnosis of ISM were identi
106 TMA was constructed using pretreatment bone marrow biopsy specimens from 64 adult patients with ALL.
107 e were detected on peripheral smear and bone marrow biopsy specimens, and PCR amplified Ehrlichia ewi
118 onoclone of IgG1, the patient underwent bone marrow biopsy which revealed Monoclonal Gammopathy of re
119 has historically been limited by infrequent marrow biopsies, which increase the risk of infections a