ライフサイエンスコーパス: matrix protein

1 ession of scleraxis and cartilage oligomeric matrix protein.

2 forming growth factor-beta1 or extracellular matrix protein.

3 sialoprotein (DSP) is a dentin extracellular matrix protein.

4 aled a significant increase in extracellular matrix proteins.

5 nase) family, known to process extracellular matrix proteins.

6 to function as a receptor for extracellular matrix proteins.

7 signaling and up-regulation of extracellular matrix proteins.

8 er unexpected of typical substrate-dependent matrix proteins.

9 asma membrane, cell surface or extracellular matrix proteins.

10 collagen I secretion without affecting other matrix proteins.

11 to collagen and other exposed subendothelial matrix proteins.

12 grity and the release of soluble peroxisomal matrix proteins.

13 aracterized by accumulation of extracellular matrix proteins.

14 expression of cartilage tissue extracellular matrix proteins.

15 tegrin subtypes and sensing of extracellular matrix proteins.

16 d in a decrease in adhesion to extracellular matrix proteins.

17 in the degradation of various extracellular matrix proteins.

18 TS1/PTS2) identified 51 putative peroxisomal matrix proteins.

19 racellular vesicles containing extracellular matrix proteins.

20 d after treatment with various extracellular matrix proteins.

21 llagen type I (COL1) and other extracellular matrix proteins.

22 interacts with a multitude of extracellular matrix proteins.

23 to myofibroblasts that secrete extracellular matrix proteins.

24 the Ser-x-Glu/pSer motifs in several enamel matrix proteins.

25 pread arthropod gene family, the peritrophic matrix proteins.

26 rin without altering adhesion to fibronectin matrix proteins.

27 otifs present in loops in many extracellular matrix proteins.

28 hat had large (14)N/(15)N ratios were mostly matrix proteins.

29 fibrin clots and cross-linking extracellular matrix proteins.

30 components, and proteases processing enamel matrix proteins.

31 proliferation and secretion of extracellular matrix proteins.

32 oline supports TGFbeta-induced production of matrix proteins.

33 al and neuronal cells, and the extracellular matrix proteins.

34 ian rhabdoviruses based on the viral P or M (matrix) protein.

35 aled more abundant osteopontin (OPN), dentin matrix protein 1 (DMP1), and matrix extracellular phosph

36 en (procollagen I) and a reduction in dentin matrix protein 1 (DMP1), which is partially responsible

37 ncluding osteocyte/cementocyte marker dentin matrix protein 1 (Dmp1).

38 ession of mineralized tissue markers, dentin matrix protein 1 (Dmp1/DMP1), osteopontin (Spp1/OPN), an

39 This drives association with viral matrix protein 1 (M1) at the chromatin, important for vR

40 neath the viral envelope of influenza virus, matrix protein 1 (M1) forms an oligomeric layer critical

41 Matrix protein 1 (M1) is a well-conserved internal prote

42 ost abundant protein in influenza virions is matrix protein 1 (M1), which mediates virus assembly by

43 nd CD8+ peptides from the internal proteins (matrix protein 1 [M1], nucleoprotein [NP], polymerase ba

44 The altered levels of dentin matrix protein 1 and osteopontin in Fam20C-deficient bon

45 Matrix protein 1(M1) is a well-conserved internal protei

46 ator of the dentinogenic marker DMP1 (dentin matrix protein 1) expression by hDPSCs.

47 Dmp1 (dentin matrix protein 1), Dkk1 (Dickkopf WNT signaling pathway

48 C-deficient bone had a lower level of dentin matrix protein 1, and higher levels of osteopontin and b

49 rylated full-length human recombinant dentin matrix protein-1 (17-513 AA), this bioinspired approach

50 osteopontin-, bone sialoprotein-, and dentin matrix protein-1-enriched fractions by anion-exchange ch

51 inidase (NA) and the extracellular domain of matrix protein 2 (M2e) fused to a virus-like particle (V

52 The influenza A virus matrix protein 2 ectodomain (M2e) is a universal influen

53 her viral proteins, including neuraminidase, matrix protein 2 or nucleoprotein.

54 rins, osteopontin, and related extracellular matrix proteins; (2) clastic cell fusion and activation

55 Here, we show that the mitochondrial matrix proteins 4-Nitrophenylphosphatase domain and non-

56 duced NOX4 expression, oxidative stress, and matrix protein accumulation in renal epithelial cells; t

57 ation to spur mesangial cell hypertrophy and matrix protein accumulation.

58 ic gene expression, release of extracellular matrix proteins, activation of hepatic myofibroblasts, a

59 ls netrin-4 as a non-enzymatic extracellular matrix protein actively disrupting pre-existing BMs.

60 Within the mitochondrial matrix, protein aggregation activates the mitochondrial

61 eported that a fragment of the extracellular matrix protein agrin promotes cardiac regeneration after

62 enhanced glycosylation of the extracellular matrix protein, alpha-dystroglycan, all consistent with

63 ent of Trap1, an Hsp90-related mitochondrial matrix protein and a member of the mitochondrial unfolde

64 ripts: an unspliced mRNA that encodes the M1 matrix protein and a spliced transcript that encodes the

65 specific molecule targeting an extracellular matrix protein and delivering a TGF-beta mAb resulted in

66 ELX) is the predominant extracellular enamel matrix protein and plays an essential role in enamel for

67 high glucose-induced expression of NOX4 and matrix protein and whether H2S and NO pathways are integ

68 Virtually all mitochondrial matrix proteins and a considerable number of inner membr

69 ric followed by cell adhesion, extracellular matrix proteins and a few pro-inflammatory proteins.

70 osis is associated with an overproduction of matrix proteins and a pathological increase of liver sti

71 The main extracellular matrix proteins and associated cytokines were evaluated.

72 ssical structural roles, but rather regulate matrix proteins and cell-matrix interactions to influenc

73 hat is responsible for binding extracellular matrix proteins and certain arenaviruses.

74 tivated to lay a foundation of extracellular matrix proteins and glycosaminoglycans that alter tumor

75 healing with discussion on how extracellular matrix proteins and hypoxia can be utilized to improve M

76 s to MSC transplantation using extracellular matrix proteins and hypoxia preconditioning.

77 alpha-smooth muscle actin and extracellular matrix proteins and is dependent on metabolic reprogramm

78 ons as a receptor for multiple extracellular matrix proteins and its dysfunction leads to a form of m

79 c molecules, including several extracellular matrix proteins and myofibroblast and cell contractility

80 y cytokines, immune mediators, extracellular matrix proteins and oncogene expression.

81 shows extensive alterations in extracellular matrix proteins and pathways related to fibrogenesis, in

82 tion and adhesion to different extracellular matrix proteins and regulate cellular stiffness and cyto

83 zed by increased deposition of extracellular matrix proteins and scar formation in the lung, resultin

84 -held mechanistic dichotomy between adhesive matrix proteins and soluble growth factors and uncovers

85 folds increase in secretion of extracellular matrix proteins and the reorganization of the matrix fib

86 eat-derived HSA hydrogels with extracellular matrix proteins and these may be used as a xeno-free pla

87 ients by recruiting fibroblasts that produce matrix proteins and thicken the lamina reticularis.

88 ble formulation of native ECM (extracellular matrix) proteins and used this bioink to 3-dimensionally

89 MAD3 activation, production of extracellular matrix proteins, and cell contractility.

90 rotein responses, secretion of extracellular matrix proteins, and cell proliferation.

91 lease correlates with rapid deacetylation of matrix proteins, and SIRT3 is required for recovery of m

92 Extracellular matrix proteins are biosynthesized in the rough endoplas

93 , where excessive collagen and extracellular matrix proteins are deposited within the wound area, res

94 Matrix proteins are encoded by many enveloped viruses, i

95 s, osteoclasts and osteocytes, and that bone matrix proteins are present in vessel-associated calcifi

96 ay active enzymes that promote extracellular matrix protein assembly.

97 tegrin beta 1 dissociates from extracellular matrix proteins, becoming internalized with CD63, which

98 is issue, Huang et al. show that the spindle matrix protein BuGZ is sufficient to form micron-scale c

99 ammation not only by degrading extracellular matrix proteins but also by controlling the influx of ch

100 ly through direct proteolytic degradation of matrix proteins, but also through activation of M inflam

101 ortality that results from the deposition of matrix proteins by an activated mesenchyme.

102 und that proteolysis of extracellular enamel matrix proteins by MMP20 is not required for the initial

103 or an evolutionarily conserved mitochondrial matrix protein called Mam33 in Saccharomyces cerevisiae

104 t structural determinants on a mitochondrial matrix protein can govern its mitophagic fate, and that

105 primary amino acid sequence of extracellular matrix proteins can have profound effects on the biomine

106 his study, we demonstrate that extracellular matrix proteins can mediate interactions between gp120 a

107 The nuclear matrix protein Cip1-interacting zinc finger protein 1 (C

108 ed with gene expression of the extracellular matrix proteins, collagen (Col)1a1, Col1a2, and fibronec

109 Histologic analyses of four matrix proteins-collagen I and IV, laminin, and fibronec

110 Cartilage oligomeric matrix protein (COMP) functions as a structural componen

111 Cartilage oligomeric matrix protein (COMP) is a large, multifunctional extrac

112 region of the gene for cartilage oligomeric matrix protein (COMP) leads to pseudoachondroplasia, a s

113 The gene for cartilage oligomeric matrix protein (COMP) was the most significantly upregul

114 shut homolog (EYS), a secreted extracellular matrix protein containing multiple laminin globular (LG)

115 The mitochondrial matrix protein cyclophilin D (CypD) is an essential comp

116 etic deletion (Ppif-/-) of the mitochondrial matrix protein cyclophilin D (CypD) prevents perinatal K

117 ow that mice deficient for the mitochondrial matrix protein cyclophilin D (CypD) show robust protecti

118 rt through upregulation of the extracellular matrix protein decorin.

119 n of division and protein import, pexophagy, matrix protein degradation, solute transport, signaling,

120 re, we identified the secreted extracellular matrix protein Del-1 as a component and regulator of the

121 n to decrease TGF-beta-induced extracellular matrix protein deposition in the kidney in Ang-II induce

122 plied to in vitro cell-based models in which matrix protein deposition reflects the underlying biolog

123 ascades regulates glomerular hypertrophy and matrix protein deposition, which are early features of g

124 te molecules revealed that the extracellular matrix protein dermatopontin (Dpt) is involved in HSC ma

125 In this study, we show that mitochondrial matrix proteins display surprisingly different dependenc

126 We used dentin matrix protein (Dmp)-1-mediated Ghr knockout (DMP-GHRKO)

127 itro Thus, Fgfbp1 is a crucial extracellular matrix protein during BBB maturation that regulates cell

128 Bacterial adherence to extracellular matrix proteins (ECMp) plays important roles during host

129 e tissue fibrosis by producing extracellular matrix proteins, efficiently engulf dead cells.

130 ation of proteins, such as the extracellular matrix protein elastin, have not been reported.

131 psis (Arabidopsis thaliana), a mitochondrial matrix protein (EMB1793, At1g76060), which we term COMPL

132 tif, the molecular mechanism by which enamel matrix proteins (EMPs) assemble into the organic matrix

133 Supramolecular assemblies of enamel matrix proteins (EMPs) play a key role as the structural

134 n between cancer cells and the extracellular matrix proteins, enhancing cell detachment and promoting

135 ctive adhesion to a variety of extracellular matrix proteins, especially laminin-1 and fibronectin, s

136 nravel a mechanism by which an extracellular matrix protein evokes stress signaling in endothelial ce

137 The Ebola virus (EBOV) VP40 matrix protein (eVP40) orchestrates assembly and budding

138 Here, we report that Tiggrin, a matrix protein expressed in the LG, is a specific regula

139 ucose-induced mesangial cell hypertrophy and matrix protein expression.

140 ant impact on inflammatory and extracellular matrix protein expression.

141 ADD-4, a member of the ADAMTSL extracellular matrix protein family, was identified as an anterograde

142 t mimic the nanoscale order of extracellular matrix protein fibers and yield suitable electrical char

143 FBN1 encodes the extracellular matrix protein fibrillin 1, which is a major structural

144 ons in FBN1, which encodes the extracellular matrix protein fibrillin-1.

145 e mechanism of assembly of the extracellular matrix protein fibronectin (FN) into elastic, insoluble

146 atation, we show here that the extracellular matrix protein fibronectin also contributes to the respo

147 to mimic the binding domain of extracellular matrix protein fibronectin and selectively bind to a sub

148 oteolytic cleavage of the host extracellular matrix protein fibronectin by peritoneal cell-derived ma

149 ntal factor, aggregates of the extracellular matrix protein fibronectin perturb the maturation of oli

150 heir migration depends on C-cadherin and the matrix protein fibronectin.

151 ascular smooth muscle cells to extracellular matrix proteins fibronectin and collagen.

152 expansion, accumulation of the extracellular matrix proteins fibronectin and type IV collagen, and lo

153 , the rejuvenated fibroblasts show increased matrix protein (fibronectin and laminin) deposition and

154 gion of anionic membranes in contrast to the matrix protein from Ebola virus.

155 he level of 3-bromotyrosine in extracellular matrix proteins from normally cultured cells was 1.1 mmo

156 Extracellular matrix proteins from the thrombospondin (TSP) family hav

157 We purified skeletal organic matrix proteins from this species and explored how these

158 nucleoprotein (N), P (phosphoprotein), or M (matrix protein) genes, or the N and P genes, were exchan

159 y impaired dimerization of the largest Golgi matrix protein, giantin.

160 collagenous and noncollagenous extracellular matrix proteins, growth factors, proteases, and cell-sur

161 Extracellular matrix proteins have been shown to modulate angiogenesis

162 alophosphoprotein (DSPP) is an extracellular matrix protein highly expressed by odontoblasts in teeth

163 The matrix proteins identified included components of acyl-C

164 ion stabilizes PEX5 and promotes peroxisomal matrix protein import, suggesting that mulibrey nanism i

165 n expression (COL1A1), a major extracellular matrix protein important in liver fibrosis, is investiga

166 ng evidence supports roles for extracellular matrix proteins in boosting synapse formation and functi

167 ce supports roles for secreted extracellular matrix proteins in boosting synaptogenesis, synaptic tra

168 fied widespread involvement of extracellular matrix proteins in DBL-1 regulation of body size.

169 C was superior amongst several extracellular matrix proteins in enhancing the sphere-forming capacity

170 deposition and remodelling of extracellular matrix proteins in fibrosis.

171 role of fibronectin and other extracellular matrix proteins in HIV-1 biology.IMPORTANCE Immune tissu

172 eins and a higher abundance of extracellular matrix proteins in SAGN.

173 ated proteins in squamous, and extracellular matrix proteins in sarcomatoid subtypes.

174 with other membrane transporters or cellular matrix proteins in specialized plasma membrane microdoma

175 ain collagen fibrils and other extracellular matrix proteins in tendon.

176 ory cytokines, chemokines, and extracellular matrix proteins in the heart.

177 ecular analyses revealed that the removal of matrix proteins in the mutant enamel was drastically del

178 the expression of a subset of extracellular matrix proteins in the skin, including upregulation of e

179 icular, mineralization-specific and eggshell matrix proteins in the uterus and in purified EVs.

180 sis involves the production of extracellular matrix proteins in tissues and is often preceded by inju

181 Elastin is an important ECM (extracellular matrix) protein in large and small arteries.

182 ain the observed enrichment of extracellular matrix proteins include both increased protein secretion

183 copy number alterations in key extracellular matrix proteins including collagen 1 type 1 alpha 1 (COL

184 nserved AH motif is absent from other enamel matrix proteins, including amelogenin, enamelin, and ame

185 Multiple extracellular matrix proteins, including collagen 1 and 3, fibronectin

186 TGFbeta-dependent induction of extracellular matrix proteins, including collagen Ialpha1 (colIalpha1)

187 e linked by several additional extracellular matrix proteins, including nidogen and perlecan (Figure

188 ECM (Extracellular matrix) proteins, including basement-membrane proteins,

189 sulfide (H2S) inhibits high glucose-induced matrix protein increase by activating AMPK in renal cell

190 Tenascin-C, a matrix protein induced upon tissue damage and expressed

191 ctivation by FN, but not other extracellular matrix proteins, induces the release of the granules' co

192 s needed to further incorporate other enamel matrix proteins into the system, this study brings us on

193 rity, cell junction and apical extracellular matrix proteins involved in tracheal tube size control:

194 eplication in cell culture.IMPORTANCE The M1 matrix protein is critical for many stages of the influe

195 The EBOV VP40 (eVP40) matrix protein is the main driving force for virion asse

196 Import of yeast peroxisomal matrix proteins is initiated by cytosolic receptors, whi

197 y of the machinery for import of peroxisomal matrix proteins is that it can accept already folded pro

198 ies showed COL13A1, a synaptic extracellular-matrix protein, is involved in the formation and mainten

199 d Collagen XIX, a synaptogenic extracellular matrix protein, is required for the formation of GABAerg

200 Tenascin-C (TnC), an extracellular matrix protein, is transiently expressed during tissue i

201 g of mitochondrial membrane carrier, but not matrix proteins, is sufficient to induce cytosolic aggre

202 ective on how mutations in the extracellular matrix protein laminin cause severe consequences in glia

203 kininogen (HK), as well as the extracellular matrix proteins laminin and collagen V.

204 both receptor cleavage and the presence of a matrix protein, laminin, to activate GPR56, whereas coll

205 define the structure and arrangement of the matrix protein layer that mediates formation of filoviru

206 poptosis and the generation of extracellular matrix proteins leading to fibrosis/cirrhosis.

207 p55 processes several imported mitochondrial matrix proteins leading to their stabilization.

208 Extracellular matrix protein levels and contractile function were also

209 Because of their remarkably long half-life, matrix proteins, like type I collagen and elastin, are p

210 an spectral changes related to extracellular matrix proteins, lipids, and nucleic acids were tracked

211 presequence peptide degrading enzyme in the matrix.Protein localization plays an important role in t

212 L-1beta, consistent with our observations of matrix protein loss and mechanical property decrease dur

213 tural and nonstructural genes, we identified matrix protein (M) as the virus' most potent antihost pr

214 VSV-p53wt and VSV-p53-CC encode a VSV matrix protein (M) with a DeltaM51 mutation (M-DeltaM51)

215 For NiV the matrix protein (M), the fusion glycoprotein (F) and, to

216 Influenza A virus matrix protein M1 is involved in multiple stages of the

217 equence motif close to the amino terminus of matrix protein (M1) exposed during acid priming of the v

218 orresponding to amino acids 37-47 in the PVM matrix protein (M37-47).

219 , and deposit large amounts of extracellular matrix proteins maintaining the structural integrity of

220 metalloprotease MMP-10 and the extracellular matrix protein mindin (encoded by Spon2) in the diseased

221 can be caused by the deficiencies of enamel matrix proteins, molecules responsible for the transport

222 w that BMs are not static-surprisingly, many matrix proteins move within the laminin and collagen sca

223 he formation of cross-links in extracellular matrix proteins, namely, collagen and elastin, and is in

224 Neural crest cells produce the extracellular matrix protein nidogen: impairing nidogen function disru

225 Nipah virus (NiV) matrix protein (NiV M) plays a major role in virus assem

226 Proteomics revealed multiple matrix proteins not previously associated with injured s

227 In these studies, we identified the matrix protein of the deadly fish novirhabdovirus VHSV a

228 HIV-1 matrix protein p17 (p17) has been detected in autoptic b

229 proteins examined, only expression of HIV-1 matrix protein p17 was associated with leukemia/lymphoma

230 apillary density and increased deposition of matrix proteins, particularly in the vicinity of transfo

231 ncreased apoptosis and the deposition of the matrix protein periostin during the late stages of tendo

232 Extracellular matrix proteins play an integral role in modulating vasc

233 Periostin (POSTN), a secretory matricellular matrix protein, plays a multitude of biologic functions.

234 e the conventional designation of structural matrix proteins primarily as supporting scaffolds for re

235 CXCL16 deficiency attenuated extracellular matrix protein production and suppressed bone marrow-der

236 t a novel epigenetic regulatory mechanism of matrix protein production by corneal endothelial cells i

237 signaling pathways and reduced extracellular matrix protein production, and blocked myofibroblast dif

238 t activation and proliferation and increased matrix protein production.

239 ence that tenascin-C (TNC), an extracellular matrix protein prominent in malignant glioma, increases

240 Netrin-1, a chemorepulsant, laminin-like matrix protein, promotes inflammation by preventing macr

241 Coordinated cell movement required the matrix protein RbmA, without which cells expanded errati

242 Vibrio exopolysaccharides (VPS) and the matrix proteins RbmA, Bap1 and RbmC are required for the

243 o polysaccharide (VPS) and the extracellular matrix proteins RbmA, RbmC, and Bap1.

244 Pase facilitates recycling of the peroxisome matrix protein receptor PEX5 and is the most commonly af

245 l adhesion molecule L1 and the extracellular matrix protein Reelin play crucial roles in the developi

246 myofibroblasts, which secrete extracellular matrix proteins responsible for the fibrotic scar.

247 ls that secrete high levels of extracellular matrix proteins, resulting in fibrosis.

248 (FBLN5), an elastogenesis-promoting cellular matrix protein, results in prolapse in mice.

249 Here, we show that the nuclear matrix protein SAFA (also known as HnRNPU) functions as

250 We previously found that the extracellular matrix protein secreted protein acidic and rich in cyste

251 onal scaffold that coordinates extracellular matrix proteins, secreted cues, and transmembrane recept

252 It is known that shell matrix proteins (SMPs) play important roles in shell for

253 The expression of extracellular matrix proteins such as Laminin and Fibronectin was also

254 Fibrogenesis encompasses the deposition of matrix proteins, such as collagen I, by hepatic stellate

255 Basement membrane matrix proteins, such as matrigel, are able to improve t

256 -held models of vesicular transport of large matrix proteins, such as procollagen, and are implicatin

257 The presence of extracellular matrix proteins suggested increased production by smooth

258 ion, reactive oxygen species generation, and matrix protein synthesis by inhibiting AMP-activated pro

259 n beta1 were also required for extracellular matrix protein synthesis in response to HG.

260 ow TGFbeta supports the bioenergetic cost of matrix protein synthesis is not fully understood.

261 yofibroblasts, responsible for extracellular matrix protein synthesis, and the macrophages infiltrati

262 last activation, inducing ECM (extracellular matrix) protein synthesis and promoting fibrosis and dia

263 KO increased tumor-associated extracellular matrix protein tenascin C (TNC) in xenografts, which was

264 Deposition of the extracellular matrix protein tenascin-C is part of the reactive stroma

265 Fibronectin (FN) is a core matrix protein that assembles to form a dynamic cellular

266 chondrial ferritin (FtMT) is a mitochondrial matrix protein that chelates iron.

267 que function for LecB and identifies it as a matrix protein that contributes to biofilm structure thr

268 a unique endothelial specific extracellular matrix protein that has been implicated in angiogenesis

269 n (ACLP) is a collagen-binding extracellular matrix protein that has important roles in wound healing

270 ced fibronectin expression, an extracellular matrix protein that is increased in diabetic nephropathy

271 Here, we identified an extracellular matrix protein that is released by these early-born SST(

272 e secretion of fibronectin, an extracellular matrix protein that regulates cell migration and invasio

273 logenin and ameloblastin are 2 extracellular matrix proteins that are essential for the proper develo

274 erythrocytes to interact with extracellular matrix proteins that are exposed within the splenic arch

275 ralized tissues is governed by extracellular matrix proteins that assemble into a 3D organic matrix d

276 The data illustrated matrix proteins that constitute the basement membranes i

277 roduced fibronectins and other extracellular matrix proteins that copurified with gp120.

278 ses, natural aGPCR ligands are extracellular matrix proteins that dissociate the NTF to reveal the te

279 synaptic formation by clearing extracellular matrix proteins that embed neurons.

280 tile functions and secrete the extracellular matrix proteins that form this fibrous scar.

281 show that starved human fibroblasts secrete matrix proteins that maintain the growth of starved mamm

282 factors including enzymes and extracellular matrix proteins that promote or inhibit hydroxyapatite c

283 igned an approach to target an extracellular matrix protein, the fibronectin extra domain A isoform (

284 (+),K(+)-ATPase alpha-isoforms with cellular matrix proteins, the cytoskeleton, or other membrane pro

285 F-1alpha) and the synthesis of extracellular matrix proteins through a mechanical mechanism that invo

286 lasts that excessively deposit extracellular matrix proteins, thus compromising lung architecture and

287 The extracellular matrix protein tropoelastin is classically regarded as a

288 12-mediated degradation of the extracellular matrix proteins tropoelastin and fibronectin in the tiss

289 r interacting with laminin, an extracellular matrix protein ubiquitously expressed in the respiratory

290 It is known that different mitochondrial matrix proteins undergo mitophagy with very different ra

291 The production and secretion of matrix proteins upon stimulation of fibroblasts by trans

292 and migration on periostin, an extracellular matrix protein upregulated in asthma by type 2 immunity

293 a receptor for the plasma and extracellular matrix protein vitronectin (Vn).

294 Ebola virus GP1,2, the Ebola virus matrix protein VP40, and BST2 are at least additive with

295 One of the seven MARV genes encodes the matrix protein VP40, which forms a matrix layer beneath

296 This process is achieved by the matrix protein VP40.

297 We found that the Ebola virus matrix protein, VP40, and envelope glycoprotein, GP, eac

298 s (wild-type and attenuated VSV with mutated matrix protein [VSVm] versions) that express either the

299 Agrin is a large extracellular matrix protein whose isoforms differ in their tissue dis

300 -mHsp10 assists the folding of mitochondrial matrix proteins without the negative ATP binding inter-r