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1 ues have been identified to date in Ustilago maydis.
2 ichrome and ferrichrome A biosynthesis in U. maydis.
3 nstrate that KP4 affects (45)Ca uptake in U. maydis.
4 heromone-responsive MAP kinase cascade in U. maydis.
5 f filamentous growth and pathogenicity in U. maydis.
6 rol of morphogenesis and pathogenicity in U. maydis.
7 is necessary for DNA replication in Ustilago maydis.
8 and gene targeting in the corn smut Ustilago maydis.
9 ein required for DNA replication in Ustilago maydis.
10 onal repair gene from the corn smut Ustilago maydis.
11 ogenicity of the corn smut pathogen Ustilago maydis.
12 ator of siderophore biosynthesis in Ustilago maydis.
13 ly, for siderophore biosynthesis in Ustilago maydis.
14 risea and the basidiomycete fungus, Ustilago maydis.
15 randed DNA gaps was investigated in Ustilago maydis.
16 ng during pathogenic development of Ustilago maydis.
17 e key RNA-binding protein Rrm4 from Ustilago maydis.
18 e TER from the basidiomycete fungus Ustilago maydis.
19 endent reduced susceptibility of maize to U. maydis.
20 omoting effector of the smut fungus Ustilago maydis.
21 s cerevisiae, Candida albicans, and Ustilago maydis.
22 or cell proliferation in the fungus Ustilago maydis.
23 odel of early endosome transport in Ustilago maydis.
24  infection by the pathogenic fungus Ustilago maydis.
25 ndosome (EE) motility in the fungus Ustilago maydis.
26 raction of maize with the fungal pathogen U. maydis.
27 nthases (CHSs) in the corn pathogen Ustilago maydis.
28 icient homologous recombination system in U. maydis.
29 rom a tryptophan (Trp) precursor in Ustilago maydis.
30 f Brh2, the BRCA2 family protein in Ustilago maydis.
31 getative and sporulating cultures of C. zeae-maydis.
32 s for the extreme radiation resistance in U. maydis.
33 Rec2 is the single Rad51 paralog in Ustilago maydis.
34 secreted by the P4 killer strain of Ustilago maydis.
35 le checkpoint protein Rad17 and the Ustilago maydis 3' --> 5' exonuclease, Rec1.
36   We inserted a rhodopsin gene from Ustilago maydis,(7) which encodes a proton pump localized to the
37 tors and two membrane proteins from Ustilago maydis, a biotrophic fungus causing smut disease in corn
38 proteins to telomere maintenance in Ustilago maydis, a fungus that bears strong resemblance to mammal
39 air-defective mutants in the fungus Ustilago maydis, a gene encoding a BRCA2 family member, designate
40 ergosterol biosynthesis inhibitors, Ustilago maydis alters the ratio of linoleic to oleic acid bound
41                                     Ustilago maydis, an edible mushroom growing on maize (Zea mays),
42                                Studies in U. maydis and Aspergillus nidulans reveal a complex interpl
43 t an ortholog of DSS1 is present in Ustilago maydis and associates with Brh2, the BRCA2-related prote
44 used by the fungal pathogens Cercospora zeae-maydis and Cercospora zeina, is a major foliar disease o
45  a recombinational repair gene from Ustilago maydis and contain functional domains to hRAD51 and hLIM
46 yme1 in the plant pathogenic fungus Ustilago maydis and demonstrate that the UPR is tightly interlink
47  expressed sequence tags (ESTs) from C. zeae-maydis and evaluate their expression during vegetative,
48  a switch of subjects to the fungus Ustilago maydis and how it causes disease in maize.
49 rogress has been made especially in Ustilago maydis and Magnaporthe oryzae.
50 ant pathogens Zymoseptoria tritici, Ustilago maydis and Magnaporthe oryzae.
51  as the phytopathogenic smut fungi, Ustilago maydis and Microbotryum violaceum, must switch from a ye
52                        The two genes from U. maydis and one of the genes from M. violaceum were expre
53 pores of the phytopathogenic fungus Ustilago maydis and spores of the social amoeba Dictyostelium dis
54 related, maize-infecting smut fungi Ustilago maydis and Sporisorium reilianum but has a larger repeat
55 ora sorghi in cluster I, five isolates of P. maydis and three isolates of P. sacchari in cluster II a
56  in the recombinational repair pathway in U. maydis, and imply that it plays a similar key role in th
57     One contains the S. cerevisiae, Ustilago maydis, and Trypanosoma brucei enzymes, which have a COO
58     Here we show that in the fungus Ustilago maydis approximately 95% of POs and LDs undergo diffusiv
59 oteins reported to influence virulence in U. maydis as the singular divergence that could explain its
60                                  In Ustilago maydis, Aspergillus nidulans, and Saccharomyces cerevisi
61 ut the regulation of pathogenesis in C. zeae-maydis at the molecular level.
62 5 transformants/micrograms linear DNA and U. maydis at up to 25 transformants/microgram circular DNA
63 ling cell fate in the basidiomycete Ustilago maydis, bE5 and bE6, allows cooperative DNA binding with
64 ized for Bipolaris zeicola and Stenocarpella maydis, but the identities of the proteases are not know
65 reported in Illinois and Indiana in 2015, P. maydis can now be found across much of the corn growing
66                                     Ustilago maydis causes common smut in maize, which is characteriz
67            The Basidiomycete fungus Ustilago maydis causes corn smut disease and alternates between a
68                   The basidiomycete Ustilago maydis causes smut disease in maize (Zea mays) by infect
69               The biotrophic fungus Ustilago maydis causes smut disease in maize with characteristic
70 om bovine protein standards, yeast, Ustilago maydis cell lysates, and Arabidopsis thaliana leaves.
71 ted in a surface-exposed form in cultured U. maydis cells.
72                    Here, we show that the U. maydis class VII chitin synthase and 1,3-beta-glucan syn
73    As the gall-inducing smut fungus Ustilago maydis colonizes maize (Zea mays) plants, it secretes a
74 iomycetous pathogens and mushrooms (Ustilago maydis, Coprinus cinereus, Schizophyllum commune), yet o
75                 In the fungal model Ustilago maydis, cytokinesis-dependent unconventional secretion m
76               Mutants of the fungus Ustilago maydis defective in the RecQ helicase Blm are highly sen
77                                Mutants of U. maydis deleted of DSS1 are extremely radiation sensitive
78                    In telomerase-positive U. maydis, deletion of rad51 and blm separately caused shor
79                                     While U. maydis Deltagls1 cells induce strong plant defense respo
80                                           U. maydis deploys many effector proteins to manipulate its
81 , we report that in the smut fungus Ustilago maydis detergent resistant core structures are enriched
82                                       For U. maydis, disruption of ump2 eliminated the filamentous ph
83 rmed a functional characterization of the U. maydis effector Jasmonate/Ethylene signaling inducer 1 (
84 9 is not induced after infection with the U. maydis effector mutant Deltapep1, which elicits massive
85                          We show that the U. maydis effector ROS burst interfering protein 1 (Rip1),
86                 Here, we characterize the U. maydis effector Sts2 (Small tumor on seedlings 2), which
87         Brh2, the BRCA2 ortholog in Ustilago maydis, enables recombinational repair of DNA by control
88 ally closely related plant pathogen Ustilago maydis encodes a different arsenal of extracellular hydr
89                    The REC2 gene of Ustilago maydis encodes a homologue of the Escherichia coli RecA
90 is work has improved the understanding of P. maydis epidemiology and provided the foundation for the
91                    The REC1 gene of Ustilago maydis functions in the maintenance of genome stability
92          Brh2, the BRCA2 homolog in Ustilago maydis, functions in recombinational repair of DNA damag
93 n isolated from Curvularia sp. and Bipolaris maydis fungi.
94 is evident by blast analysis of the Ustilago maydis genome database.
95 n genome defense, that are lacking in the U. maydis genome due to clean excision events.
96 ages of this pathway in maize smut (Ustilago maydis), glucosidase I (Gls1) and glucosidase II beta-su
97   The unusual C-terminal extension of the U. maydis Hac1 homolog, Cib1 (for Clp1 interacting bZIP1),
98                          For years, Ustilago maydis has stood out as the model system to study the ge
99 elongated hyphal cell of the fungus Ustilago maydis, Higuchi et al. now demonstrate that polysomes as
100 firm and extend earlier observations that U. maydis hyphae branch extensively on the leaf surface and
101                        We found that upon U. maydis infection of Z. mays, KWL1-b is expressed at sign
102 lignin biosynthesis are hypersensitive to U. maydis infection.
103          The biotrophic smut fungus Ustilago maydis infects all aerial organs of maize (Zea mays) and
104 ulators of G2/M cell cycle progression in U. maydis, interacts and controls the subcellular localizat
105                                     Ustilago maydis is a biotrophic fungus causing corn smut disease
106                                     Ustilago maydis is a biotrophic pathogen causing maize (Zea mays)
107                                     Ustilago maydis is a dimorphic fungus with a yeast-like non-patho
108                                  Phyllachora maydis is a fungal pathogen causing tar spot of corn (Ze
109                                     Ustilago maydis is a fungal pathogen of maize, some strains of wh
110                                     Ustilago maydis is a haploid basidiomycete with single genes for
111  (GLS) caused by Cercospora zeina or C. zeae-maydis is a major maize disease throughout the world.
112                                     Ustilago maydis is a phytopathogenic fungus exhibiting extreme re
113        The ascomycete fungus Cercospora zeae-maydis is an aggressive foliar pathogen of maize that ca
114          Knowledge of the epidemiology of P. maydis is limited but could be useful in developing tar
115                                     Ustilago maydis is the causal agent of maize smut disease.
116 tive of the well studied grass smut Ustilago maydis is the only smut fungus adapted to Brassicaceae h
117 These results indicate that gap repair in U. maydis is unlikely to proceed by the mechanism envisione
118 hliobolus heterostrophus (anamorph Bipolaris maydis), is a major foliar disease which causes signific
119                                       The U. maydis killer toxin KP6 contains two polypeptide chains,
120                                     Ustilago maydis killer toxins are small polypeptides (7-14 kDa) w
121 three well-characterized killer toxins in U. maydis-KP1, KP4, and KP6-which are secreted by the P1, P
122 articularly been driven forward using the U. maydis-maize pathosystem.
123 slocation of a number of effectors in the U. maydis-maize system and show data that suggest that the
124 ucky moments when major advances made the U. maydis-maize system what it is now-a well-established mo
125  homologous recombination system of Ustilago maydis, mediating delivery of Rad51 to single-stranded D
126 NPC motility required F-actin, whereas in U. maydis, microtubules, kinesin-1, and dynein drove pore m
127      We highlight the contribution of the U. maydis model system but also discuss the differences fro
128 henotypes mirror previous observations of U. maydis mutants deficient in Brh2 or Rad51.
129                   Phenotypic screening of U. maydis mutants deleted for genes encoding secreted prote
130                      In S. cerevisiae and U. maydis, NPC motility prevented NPCs from clustering.
131                         Recapitulation in U. maydis of defects in DNA repair and genome stability ass
132                         Brh2 is the Ustilago maydis ortholog of the BRCA2 tumor suppressor.
133 ce the discovery of this process in Ustilago maydis, our understanding of its molecular basis and bio
134 DNA from downy mildews that attack maize (P. maydis & P. philippinensis), sugar cane (P. sacchari), p
135        Brh2, the BRCA2 homologue in Ustilago maydis, plays a crucial role in homologous recombination
136    Infection of maize by corn smut (Ustilago maydis) provides an agronomically important model of bio
137  and pathogenesis were identified in C. zeae-maydis, providing specific targets for characterization
138                   A gene encoding a Ustilago maydis Rad51 orthologue has been isolated, rad51-1, a mu
139 ces pombe rad1+ gene product and to Ustilago maydis Rec1, a known 3'->5'exonuclease.
140 A repair and recombination proficiency in U. maydis requires both Rec2 and Rad51.
141 ox3 mutation-based resistance of maize to U. maydis requires functional Rip1.
142        Mutation in the REC1 gene of Ustilago maydis results in extreme sensitivity to killing by ultr
143 ains of the plant-pathogenic fungus Ustilago maydis secrete toxins (killer toxins) that are lethal to
144                       In the fungus Ustilago maydis, sexual pheromones elicit mating resulting in an
145 oter and iron-regulatory sequences of the U. maydis sid1 gene were defined by fusing restriction and
146 ty, are very similar in P. flocculosa and U. maydis, Sporisorium reilianum, and Ustilago hordei.
147 uch motifs affects peroxisome function in U. maydis strains challenged with fatty acids.
148               A small proportion of Ustilago maydis strains produce killer toxins, to which they are
149  maize plants and on engineering of Ustilago maydis strains to secrete Avitagged effectors.
150 double-stranded RNA (dsRNA) virus in each U. maydis subtype.
151                                       The U. maydis TER (UmTER) contains a 5'-monophosphate, distinct
152 reted by the phytopathogenic fungus Ustilago maydis that inhibits the growth of sensitive target stra
153 cess occurs for the appressorium of Ustilago maydis, the agent responsible for corn smut disease.
154                                     Ustilago maydis, the causal agent of corn smut disease, acquires
155                                     Ustilago maydis, the causal agent of corn smut disease, displays
156 nds of Saccharomyces cerevisiae and Ustilago maydis, the initial association of helicase genes with f
157             In the corn smut fungus Ustilago maydis, the myosin-chitin synthase Mcs1 moves to the pla
158 ologous recombination in the fungus Ustilago maydis through interaction with Rad51.
159 binding affinity of purified native Ustilago maydis topoisomerase I enzyme for radiolabeled DNA subst
160 uranosidases from the basidiomycete Ustilago maydis (UmAbf62A) and ascomycete Podospora anserina (PaA
161                             Moreover, the U. maydis ump2 gene, initially detected as an upregulated g
162 tion of a system enabling the survival of U. maydis under such conditions could be a secondary conseq
163              Our findings suggest that in U. maydis, unprotected telomeres arising from Ku depletion
164 FU1 that encodes a homologue of the Ustilago maydis URBS1, a transcriptional repressor of siderophore
165 unrelated antifungal toxin KP4 from Ustilago maydis, whereas structurally similar MtDef2 and the radi
166 m formation and cell cycle progression in U. maydis, which serves as a "toggle switch" to control the
167 atin (CC9) is induced upon penetration by U. maydis wild type.
168 pression and a hypersensitive response to U. maydis wild-type infection.
169 ant plants showed increased resistance to U. maydis wild-type strains, rip1 deletion strains infectin
170 og of the BRCA2 tumor suppressor in Ustilago maydis, works hand in hand with Rad51 to promote repair
171 related to the model plant pathogen Ustilago maydis yet is not a phytopathogen but rather a biocontro

 
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