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1 between IL-1beta and 5-HT2 receptors in the medial hypothalamus.
2 taining glutamatergic neurons in the ventral medial hypothalamus.
3 PAG neurons whose axons project back to the medial hypothalamus.
4 ed defensive rage behavior elicited from the medial hypothalamus.
5 um, infundibulum/median eminence, and dorsal medial hypothalamus.
6 putative GABAergic pathway projecting to the medial hypothalamus.
7 to the suprachiasmatic nucleus and adjacent medial hypothalamus.
8 were equally distributed in the lateral and medial hypothalamus.
9 l limbic cortices were more prevalent in the medial hypothalamus.
10 to suppress defensive rage elicited from the medial hypothalamus.
11 ensive rage behavior elicited from the cat's medial hypothalamus.
12 ce P pathway from the medial amygdala to the medial hypothalamus.
13 in doses of 0.05, 0.5 and 2.5 nmol) into the medial hypothalamus.
14 OCT3 mRNA, but not OCT1 or OCT2 mRNA, in the medial hypothalamus.
15 tor agonist (+/-)-DOI hydrochloride into the medial hypothalamus (0.5, 1.0, and 3.0 nmol) facilitated
17 have identified neuronal populations in the medial hypothalamus and brainstem that encode defensive
18 ulation over the rostro-caudal extent of the medial hypothalamus and dorsolateral aspect of the peria
19 ron projects from the medial amygdala to the medial hypothalamus and its functions are mediated by su
20 - and progesterone-dependent signal from the medial hypothalamus and results in heightened sexual mot
21 selectively blocks hissing elicited from the medial hypothalamus and that the suppressive effects of
22 iated over reciprocal pathways that link the medial hypothalamus and the dorsolateral quadrant of the
25 ncluding the arcuate nucleus and the ventral medial hypothalamus, areas implicated in regulation of b
26 tissue mass in a part of the right and left medial hypothalamus associated functionally with physiol
27 ddition, pretreatment with CP96,345 into the medial hypothalamus blocked the suppressive effects of s
28 ctions associated with either the lateral or medial hypothalamus, but not both, can be activated at a
31 of 5-HT(1A) and 5-HT(2) receptors within the medial hypothalamus exert differential modulatory effect
32 indings indicate that NK(1) receptors in the medial hypothalamus facilitate defensive rage elicited f
33 stimulating electrodes were implanted in the medial hypothalamus for elicitation of defensive rage be
34 , cannula-electrodes were implanted into the medial hypothalamus for elicitation of defensive rage be
35 ctrodes were implanted into sites within the medial hypothalamus from which defensive rage behavior c
36 imulating electrodes were implanted into the medial hypothalamus from which defensive rage behavior c
37 imulating electrodes were implanted into the medial hypothalamus from which defensive rage behavior c
38 ed following activation of the region of the medial hypothalamus from which defensive rage behavior i
39 d placement of monopolar electrodes into the medial hypothalamus from which defensive rage could be e
40 oinjected through a cannula-electrode in the medial hypothalamus from which defensive rage had been e
43 pothalamus (LH), and lateral portions of the medial hypothalamus, have widespread projections and inf
46 aspect which receives major inputs from the medial hypothalamus in association with defensive rage b
51 e extracellular fluid compartment within the medial hypothalamus (MH), the OCT blocker, decynium 22 (
52 tested the hypothesis that perfusion of the medial hypothalamus (MH), which includes the DMH, with t
55 ression in the lateral hypothalamus (LH) and medial hypothalamus (MH, including perifornical and dors
57 and function of OCTs in the periventricular medial hypothalamus of male Sprague Dawley rats using re
59 ed by 5-HT(1A) and 5-HT(2C) receptors in the medial hypothalamus on the expression of defensive rage
60 ndings show that activation of IL-1RI in the medial hypothalamus potentiates defensive rage behavior
61 ain-derived cytokine, microinjected into the medial hypothalamus, potentiates defensive rage behavior
63 ron, which also projects from the lateral to medial hypothalamus, serves to suppress defensive rage e
64 eciprocal connections included the thalamus, medial hypothalamus, substantia nigra/ventral tegmental
65 st 8-OHDPAT (0.1, 1.0 and 3.0 nmol) into the medial hypothalamus suppressed PAG-elicited hissing in a
66 scimol (0.3-30 pmol), microinjected into the medial hypothalamus, suppressed defensive rage elicited
68 ip between IL-1RI and 5-HT2 receptors in the medial hypothalamus that is consistent with the previous
69 the hypothesis that descending fibers of the medial hypothalamus that supply the dorsal aspect of the
70 , identified large numbers of neurons in the medial hypothalamus that were labeled positively for bot
71 ck-out mice restored 5-HT1A receptors in the medial hypothalamus; this effect was accompanied by elim
72 gdala activates substance P receptors in the medial hypothalamus, thus triggering an inhibitory mecha
73 othesis that the descending pathway from the medial hypothalamus to the dorsal periaqueductal gray (P
74 mediated over a descending pathway from the medial hypothalamus to the dorsolateral midbrain periaqu
75 mediated over a descending pathway from the medial hypothalamus to the dorsolateral quadrant of the
76 of 0.5, 1.0 and 2.0 nmol), directly into the medial hypothalamus upon lateral hypothalamically elicit
77 fects of dual stimulation of the lateral and medial hypothalamus upon response latencies were compare
78 amygdala, lateral hypothalamus (LH), ventral medial hypothalamus (VMH) and arcuate nucleus (ARC).
79 g glutamatergic neurons, or into the ventral medial hypothalamus (VMH), which contains predominantly
80 and the function of 5-HT1A receptors in the medial hypothalamus were significantly reduced in Ad-1AP
81 nt on the same neurons within regions of the medial hypothalamus where IL-1beta and the IL-1beta rece
82 by a pathway from the medial amygdala to the medial hypothalamus which utilizes substance P as a neur
83 ct inhibitory projection from the lateral to medial hypothalamus whose functions are mediated by GABA