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1 he existence of developmental differences in megakaryocytopoiesis.
2 to the pathogenesis of disorders of neonatal megakaryocytopoiesis.
3 opoiesis/monocytopoiesis, lymphopoiesis, and megakaryocytopoiesis.
4 ked stimulus to drive the observed excessive megakaryocytopoiesis.
5 strate that TRIB3 is a negative modulator of megakaryocytopoiesis.
6 , functioning as a thermostat to "fine-tune" megakaryocytopoiesis.
7  is presumably caused by an up-regulation in megakaryocytopoiesis.
8 nd may contribute to the local regulation of megakaryocytopoiesis.
9 ietic progenitor cell development and during megakaryocytopoiesis.
10               The lack of B-Raf also impairs megakaryocytopoiesis.
11 ikely to affect cell cycle regulation during megakaryocytopoiesis.
12 ansduction protein 14-3-3xi and to influence megakaryocytopoiesis.
13 tudied the effects of ITP plasma on in vitro megakaryocytopoiesis.
14 d to directly determine the role of Raf-1 in megakaryocytopoiesis.
15 ly and indirectly to play a critical role in megakaryocytopoiesis.
16 ly to regulate lineage-specific genes during megakaryocytopoiesis.
17 ay play important roles in the regulation of megakaryocytopoiesis.
18 level, selective gene transcription early in megakaryocytopoiesis.
19 red delivery of platelets despite stimulated megakaryocytopoiesis.
20 RF-dependent and SRF-independent activity in megakaryocytopoiesis.
21 d SDF-1alpha may modulate several aspects of megakaryocytopoiesis.
22  and is the primary physiologic regulator of megakaryocytopoiesis.
23 poietic factor involved in the regulation of megakaryocytopoiesis.
24 volunteers PEG-rHuMGDF transiently increases megakaryocytopoiesis 2-fold.
25   In studying the role of Tie-2 and Ang-1 in megakaryocytopoiesis, 3 alternatively spliced species of
26  (CD31) knockout (KO) mice exhibit excessive megakaryocytopoiesis accompanied by increased numbers of
27 t that B-Raf plays a cell-autonomous role in megakaryocytopoiesis and a permissive role in myeloid pr
28 o the relationship between the regulation of megakaryocytopoiesis and bone mass.
29 ukopenia because of significant increases in megakaryocytopoiesis and concomitant blockage of erythro
30 taining the potential to selectively promote megakaryocytopoiesis and enhance stress-associated plate
31  high level of TPO overexpression stimulates megakaryocytopoiesis and myelopoiesis leading to thrombo
32 control of membrane remodeling, critical for megakaryocytopoiesis and normal platelet production and
33 ted regulatory effects of miRNAs relevant to megakaryocytopoiesis and platelet biology by analyzing e
34 xpression of a GP Ib-IX-V complex and normal megakaryocytopoiesis and platelet morphogenesis.
35                                              Megakaryocytopoiesis and platelet production can be asse
36 y have evidence of underlying impaired fetal megakaryocytopoiesis and platelet production following p
37  hematopoietic growth factor that stimulates megakaryocytopoiesis and platelet production in vivo and
38 sk of bleeding, but its biological effect on megakaryocytopoiesis and platelet production is unknown.
39 rovide evidence for a novel pathway by which megakaryocytopoiesis and platelet production may be regu
40 es an excellent in vivo model to study human megakaryocytopoiesis and platelet production.
41  been shown to be the principal regulator of megakaryocytopoiesis and platelet production.
42 delineate the expression of microRNAs during megakaryocytopoiesis and suggest a regulatory role of mi
43  suggest that VIP may have direct effects on megakaryocytopoiesis and support our earlier observation
44 ine kinase phosphatase PTP-RO is involved in megakaryocytopoiesis and that its function is mediated b
45 o, it has provided both unique insights into megakaryocytopoiesis and the means to stimulate platelet
46                            The regulation of megakaryocytopoiesis and thrombopoiesis appears to be un
47 t novel approaches to monitor the DMS during megakaryocytopoiesis and thrombopoiesis.
48 e thrombopoietin, the principal regulator of megakaryocytopoiesis and thrombopoiesis.
49 c tail sequestered signaling proteins during megakaryocytopoiesis and, as such, became a critical reg
50 t) to conditions associated with exaggerated megakaryocytopoiesis and/or proplatelet formation.
51 gs demonstrate that Raf-1 is dispensable for megakaryocytopoiesis, and for thrombopoietin-induced ERK
52 vironment is also the site of hematopoiesis, megakaryocytopoiesis, and platelet production.
53  shown that rhIL-11-induced murine and human megakaryocytopoiesis are not mediated by thrombopoietin
54    Although the growth factors that regulate megakaryocytopoiesis are well known, the molecular deter
55 our understanding of normal versus malignant megakaryocytopoiesis, as well as aberrant mitosis in ane
56  hinted that TRIB3 could be also involved in megakaryocytopoiesis but its role in this process has so
57 pond to Mpl ligand, the pivotal regulator of megakaryocytopoiesis, by increasing their expression of
58                   During the later stages of megakaryocytopoiesis, CD31KO megakaryocytes exhibited ab
59 hrombopoietic cytokines regulating, in part, megakaryocytopoiesis during states of acute thrombocytop
60 creased PLT survival, but intact bone marrow megakaryocytopoiesis, endogenous IL-11 levels were signi
61 ed in the hepatic sinusoids support in vitro megakaryocytopoiesis from murine hematopoietic stem cell
62                                 The study of megakaryocytopoiesis has been based largely on in vitro
63                      Thus, PECAM-1 modulates megakaryocytopoiesis in a hierarchic manner, functioning
64                            The evaluation of megakaryocytopoiesis in culture has remained unsatisfact
65                     During earlier stages of megakaryocytopoiesis in KO marrow, an expanded Lin(-)Sca
66  reports have defined the in vivo changes in megakaryocytopoiesis in response to a single injection o
67  of hematopoietic stem cell self-renewal and megakaryocytopoiesis in the bone marrow microenvironment
68  Recombinant human interleukin-11 stimulates megakaryocytopoiesis in vitro and platelet production in
69 or and shows agonist activity by stimulating megakaryocytopoiesis in vitro.
70 combinant human interleukin-11 (rhIL-11), on megakaryocytopoiesis in vitro.
71 g pathways and is important for Tpo-mediated megakaryocytopoiesis in vivo.
72 n of cytokine-dependent cell lines and basal megakaryocytopoiesis in vivo.
73 ess the role of P-selectin and E-selectin in megakaryocytopoiesis, in vitro assays were performed in
74                               This increased megakaryocytopoiesis is a cell-intrinsic defect of c-Myc
75                                              Megakaryocytopoiesis is a complex differentiation proces
76                            Second, increased megakaryocytopoiesis is not sufficient for development o
77 nvolvement of specific Raf family members in megakaryocytopoiesis is unknown.
78 o further investigate the role of Lyn during megakaryocytopoiesis, Lyn-deficient mice (lyn(-/-)) were
79 d significant inhibitory effects on in vitro megakaryocytopoiesis (P <.001).
80  growth and development factor (rHu-MGDF) on megakaryocytopoiesis, platelet function, and thrombogene
81 The effects of thrombopoietic stimulation on megakaryocytopoiesis, platelet production, and platelet
82  effects of PEG-rHuMGDF on pharmacokinetics, megakaryocytopoiesis, platelet production, and platelet
83 h likely compensates for the impaired marrow megakaryocytopoiesis, resulting in normal peripheral pla
84  these cytokines play a critical role in the megakaryocytopoiesis seen in patients who develop reacti
85 fication of TRIB3 as a negative regulator of megakaryocytopoiesis suggests that in-vivo this gene cou
86 thrombopoietin (TPO) and its contribution to megakaryocytopoiesis, the exact mechanisms and sites of
87 ated bioenergetic pathway governing terminal megakaryocytopoiesis; these observations also define a m
88  ligand for c-mpl and the major regulator of megakaryocytopoiesis, TPO deficient mice were generated
89 mportance of stathmin down-regulation during megakaryocytopoiesis, we used a lentiviral-mediated gene
90 akaryocytic lineage and the role of SDF-1 in megakaryocytopoiesis were investigated.
91 s exhibited excessive splenic extramedullary megakaryocytopoiesis, which likely compensates for the i
92  miR-150 and miR-155 play divergent roles in megakaryocytopoiesis, with the former promoting developm