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1 protein is identical to CD46 (also known as membrane cofactor protein).
2 ent inhibitors decay-accelerating factor and membrane cofactor protein.
3 atory proteins decay-accelerating factor and membrane cofactor protein.
4 -binding protein, complement receptor 1, and membrane cofactor protein.
5 6 and increased production of interleukin-6, membrane cofactor protein-1, and tumor necrosis factor-a
7 (Crry), a membrane C3 inhibitor with DAF and membrane cofactor protein activities, were rapidly elimi
8 hway, normal membrane regulators of C (e.g., membrane cofactor protein and CD59) prevent cytotoxicity
9 ell as other complement-regulatory proteins (membrane cofactor protein and CD59), were analyzed follo
10 statin, and decay-accelerating factor (DAF), membrane cofactor protein, and CD59 expression was measu
11 lement regulators decay accelerating factor, membrane cofactor protein, and CD59, or to reduced compl
12 HV-6) employs the complement regulator CD46 (membrane cofactor protein) as a receptor for fusion and
14 or the human complement regulatory proteins, membrane cofactor protein (CD46) and decay-accelerating
18 the complement regulatory proteins factor H, membrane cofactor protein (CD46), and factor I predispos
21 caused by mutations in complement factor H, membrane cofactor protein, factor I or factor B, or by a
22 lerating factor (DAF), CD59 (protectin), and membrane cofactor protein in frozen samples of human bre
24 at least three members of this family confer membrane cofactor protein-like activity (MCP or CD46) up
25 n decay-accelerating factor (DAF [CD55]) and membrane cofactor protein (MCP [CD46]), and rat inhibito
26 Decay-accelerating factor (DAF or CD55) and membrane cofactor protein (MCP or CD46) function intrins
28 teins, including factor H (FH) in plasma and membrane cofactor protein (MCP) on the cell surface.
29 s decay-accelerating factor (DAF), CD59, and membrane cofactor protein (MCP) protect endothelial cell
31 egulatory proteins including serum factor H, membrane cofactor protein (MCP), and membrane decay-acce
32 ctor H (FH), C4 binding protein alpha chain, membrane cofactor protein (MCP), decay accelerating fact
33 ll as by studying the effect of depletion of membrane cofactor protein (MCP), decay-accelerating fact
34 xpression of complement regulatory proteins, membrane cofactor protein (MCP), decay-acceleration fact
35 rdin, decay accelerating factor (DAF, CD55), membrane cofactor protein (MCP, CD46), complement recept
36 an decay accelerating factor (DAF, CD55) and membrane cofactor protein (MCP, CD46), in two models of
41 osylated isoform of the complement regulator membrane cofactor protein (MCP; CD46) is expressed on th
44 ion with the control proteins factor H (FH), membrane cofactor protein (MCP; CD46), and complement re
45 finity for factor B and a reduced binding to membrane cofactor protein (MCP; CD46), but a normal regu
47 s of the human complement regulatory protein membrane cofactor protein (MCP;CD46) has been utilized t
48 s demonstrate that heterozygous mutations of membrane cofactor protein (MCP;CD46) predispose to atypi
49 t purified C-regulators of the pig (CD59 and membrane cofactor protein [MCP]) are efficient regulator
50 greater than that of soluble forms of either membrane cofactor protein or decay-accelerating factor o
51 chor of DAF with the transmembrane anchor of membrane cofactor protein or HLA-B44 resulted in aboliti
53 ording to the crystal structure of CCP1-2 of membrane cofactor protein, the essential residues formed
54 periments in which the equivalent regions of membrane cofactor protein were swapped into decay-accele
55 nomas of the lung usually expressed CD59 and membrane cofactor protein with variable DAF immunoreacti