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1 observations describe a mechanism for gating membrane fusion.
2 ular invasion and resistance in driving cell membrane fusion.
3 ming the SNARE complex that drives vesicular membrane fusion.
4 an essentially irreversible change, to drive membrane fusion.
5 ains that are critical for the efficiency of membrane fusion.
6 ng the fusogenic potential of gp41 to induce membrane fusion.
7 of a six-helix bundle and viral and cellular membrane fusion.
8 lls, where LY6E predominantly promotes viral membrane fusion.
9 iven disassembly that enables a new round of membrane fusion.
10 exocyst complex during vesicle tethering and membrane fusion.
11 fusin is required after tethering to promote membrane fusion.
12 achment stage, with several inhibiting viral membrane fusion.
13 I viral fusion protein mediating virus-cell membrane fusion.
14 gether offer an evolving perspective on cell membrane fusion.
15 ed to be responsible for host attachment and membrane fusion.
16 tein gB to undergo rearrangements leading to membrane fusion.
17 nveloped virus entry by promoting virus-cell membrane fusion.
18 ion of small molecules that arrest EBOV-host membrane fusion.
19 er refolding of trimeric fusion proteins and membrane fusion.
20 ally to induce cell-to-cell, not virus-cell, membrane fusion.
21 ial for many physiologic processes requiring membrane fusion.
22 ational changes of the S protein, leading to membrane fusion.
23 n protein structures involved in prokaryotic membrane fusion.
24 site exposure and subsequent viral-host cell membrane fusion.
25 e, irreversible conformation change to drive membrane fusion.
26 nd are both required for mitochondrial outer membrane fusion.
27 otein conformational changes associated with membrane fusion.
28 pletion of conformational changes that drive membrane fusion.
29 ssembly and minimize repulsive forces during membrane fusion.
30 dyes used also have the potential to perturb membrane fusion.
31 post-F) state at the time of virus-host cell membrane fusion.
32 e viral F protein that is required to elicit membrane fusion.
33 n, which is required for outer mitochondrial membrane fusion.
34 e concerning the mechanism of virus-mediated membrane fusion.
35 tein binds to host cell receptors to mediate membrane fusion.
36 ein Ov8 can significantly enhance, cell-cell membrane fusion.
37 of hemagglutinin (HA) and blocks HA-mediated membrane fusion.
38 e to both FIP and AS-48 without compromising membrane fusion.
39 cells in early response to virosome-induced membrane fusion.
40 of cellular factors necessary for virus-cell membrane fusion.
41 n for receptor binding and a stem domain for membrane fusion.
42 D63 stimulates pH-activated LUJV GP-mediated membrane fusion.
43 n, and GP2 is responsible for low pH-induced membrane fusion.
44 e the basic mechanism by which they catalyze membrane fusion.
45 hreonine 206-an early molecular event during membrane fusion.
46 efined assembly intermediates on the path to membrane fusion.
47 onformational rearrangements associated with membrane fusion.
48 rotein incorporation, lipid composition, and membrane fusion.
49 that enable homodimerization and subsequent membrane fusion.
50 lfish" genetic element from cell to cell via membrane fusion.
51 s suggest revisions to the SNARE paradigm of membrane fusion.
52 ral changes that mediate viral entry through membrane fusion.
53 ep in SNARE complex assembly, and stimulates membrane fusion.
54 rus entry because it mediates viral and host membrane fusion.
55 glycoprotein that can significantly enhance membrane fusion.
56 s into a parallel four-helix bundle to drive membrane fusion.
57 d in gK-mediated regulation of virus-induced membrane fusion.
58 ered (ld) phase boundary to facilitate viral membrane fusion.
59 nel so as to synchronize calcium influx with membrane fusion.
60 which carries out the process of virus-cell membrane fusion.
61 imum required OvHV-2 glycoproteins to induce membrane fusion.
62 NARE-binding proteins catalyze intracellular membrane fusion.
63 ntributed to the regulation of HSV-1-induced membrane fusion.
64 rearrangements through which the HA mediates membrane fusion.
65 nism by which the syntaxin cluster regulates membrane fusion.
66 embly into a stable four-helix bundle drives membrane fusion.
67 onize HIV-1 Env at gp120 V1V2 to block viral membrane fusion.
68 glycoprotein, which mediates cell entry and membrane fusion.
69 ing of cell entry, especially at the step of membrane fusion.
70 initiating conformational changes that drive membrane fusion.
71 d-induced conformational change required for membrane fusion.
72 on of the SNARE complex is a key step during membrane fusion.
73 vacuolar membrane and downregulates vacuolar membrane fusion.
74 virion to cell-surface receptors and mediate membrane fusion.
75 broad anti-coronavirus activity by blocking membrane fusion.
76 ng antibodies that potently block gB-induced membrane fusion.
77 ultiple viral entry glycoproteins to trigger membrane fusion.
78 ) to engage host cell receptors and catalyze membrane fusion.
79 s by interfering with spike protein-mediated membrane fusion.
80 in the fusion (F) glycoprotein that mediates membrane fusion.
81 then activates the fusogen gB, resulting in membrane fusion.
82 ne sperm into the ooplasm, thereby bypassing membrane fusion.
83 ns for attachment, receptor interaction, and membrane fusion.
84 receptor-mediated endocytosis and endosomal membrane fusion.
85 peptide (FP), which initiates the process of membrane fusion.
86 to reveal events leading up to PSI-mediated membrane fusion.
87 mation during the early events of virus-cell membrane fusion.
89 the residue level, which helps to understand membrane fusion, a fundamental transport and communicati
90 ted GTPases that mediate mitochondrial outer-membrane fusion, a process that is required for mitochon
91 e strategies and review the strategy of cell membrane fusion, a recent strategy for direct delivery o
93 y decreasing the threshold for activation of membrane fusion activity triggered by the host factors c
94 at combines optogenetics with single-vesicle membrane fusion, aiming to gain a better understanding o
96 he conformational changes of DNA cages drive membrane fusion and bending with predictable outcomes, o
98 ceptor (SNARE) proteins play a major role in membrane fusion and contribute to cell expansion, signal
100 the late endosome(9), are necessary for the membrane fusion and delivery of RNA from exo-HAV into th
101 roduce a synthetic fusogen that can modulate membrane fusion and equivalently prime lipid membranes f
102 (HA) by host proteases is a prerequisite for membrane fusion and essential for virus infectivity.
103 e findings support our current model for HeV membrane fusion and expand our knowledge of the TMD and
104 Dynamin-like proteins (DLPs) mediate various membrane fusion and fission processes within the cell, w
106 ctive than WQ in blocking HIV-1 Env-mediated membrane fusion and had higher levels of binding affinit
108 s suggested that the PR contributes to HIV-1 membrane fusion and infectivity; however, the precise ro
110 try into host cells via receptor binding and membrane fusion and is a validated target for drug disco
112 nserted specific lipids into the cell PM via membrane fusion and studied their acute effects on caveo
113 oprotein 2 (GP2) which facilitate host-viral membrane fusion and subsequent release of the viral geno
114 (AS-48) have similar efficacies in blocking membrane fusion and syncytium formation mediated by meas
115 that network maintenance requires continuous membrane fusion and that Yop1p favours the generation of
117 ere, we investigate in detail the process of membrane fusion and the role of opposite charges in a pr
118 ailed view of Serinc restriction of HIV-cell membrane fusion and thus extend current structural and f
119 d the ubiquitous process of protein-mediated membrane fusion and to reveal novel mechanisms of nonenv
120 surface-exposed viral protein, GP, mediates membrane fusion and undergoes major structural rearrange
122 F conformational change needed to facilitate membrane fusion and virus infection, and the epitope rec
123 nctions, including metabolism, mitochondrial membrane fusion and/or fission dynamics, and apoptosis.
124 dependent Rab7 activation and Rab7-dependent membrane fusion, and (iii) that this process is regulate
125 nt protein receptors (SNAREs), that catalyze membrane fusion, and homotypic fusion and vacuole protei
126 ant to understand how these assays report on membrane fusion, and recent studies with yeast vacuolar
127 es directly contributes to the efficiency of membrane fusion, and suggest that nanoscale organization
129 o study OvHV-2 glycoproteins responsible for membrane fusion as a part of the entry mechanism, we dev
130 ding of the conformation of gB that promotes membrane fusion as well as the identification of structu
131 ate a myriad of cellular functions including membrane fusion, as exemplified by the yeast vacuole, wh
132 nism, we developed a virus-free cell-to-cell membrane fusion assay to identify the minimum required O
133 tations were validated in a luciferase-based membrane fusion assay, using transfected fusion and hema
138 veloped viruses enter cells via a process of membrane fusion between the viral envelope and a cellula
139 AMP721, which assembles with SYP121 to drive membrane fusion, binds to the KAT1 K(+) channel via two
140 e in intracellular trafficking by catalyzing membrane fusion, but assigning SNAREs to specific intrac
141 3 (RHD3) has been demonstrated to mediate ER membrane fusion, but how exactly RHD3 is involved in the
142 t with the SSP to regulate GP processing and membrane fusion, but its biological role in the context
144 a virus entry where cells restrict or permit membrane fusion by changing deformability, for instance,
145 analysis suggested that 10E8 inhibits viral membrane fusion by lifting the MPER N-terminal region ou
146 ting mechanistic model for the triggering of membrane fusion by synaptotagmin-1 reconciles many exper
152 tion, paramyxoviruses cause a second type of membrane fusion, cell-cell fusion (syncytium formation),
154 s (SNAREs) constitute the core machinery for membrane fusion during eukaryotic cell vesicular traffic
157 We also discuss the elusive mechanism of membrane fusion during nuclear pore complex (NPC) biogen
159 virus glycoprotein complex gB/gH-gL mediates membrane fusion during virion entry and cell-cell fusion
160 /G dissociation model of henipavirus-induced membrane fusion, even in the context of heterologous gly
162 rocess, autophagosomes are formed de novo by membrane fusion events leading to phagophore formation i
170 hereas the molecular machineries involved in membrane fusion/fission have been dissected, regulation
172 anti-gB MAbs and suggests that blocking the membrane fusion function of gB could be one mechanism of
173 or the pH at which HA is activated to cause membrane fusion, has been associated with the replicatio
174 poorly understood mechanism of Mfn-mediated membrane fusion, here we characterize a Mitofusin mutant
175 d altered endosomal trafficking affect viral membrane fusion.IMPORTANCE Many enveloped viruses infect
177 e same mutations abolished GP processing and membrane fusion in a plasmid-based protein expression sy
180 41 has been proposed to participate in HIV-1 membrane fusion in biochemical analyses, but its role in
181 s that are essential components required for membrane fusion in eukaryotic intracellular membrane tra
182 unc18 (SM)-family proteins are essential for membrane fusion in exocytic and endocytic trafficking.
186 formation during homotypic vacuolar lysosome membrane fusion in Saccharomyces cerevisiae Using cell-f
188 ch lacks a transmembrane domain, can support membrane fusion in vivo is uncertain, as is the precise
189 s to track synaptic vesicle localization and membrane fusion in zebrafish during developmental myelin
190 nts were defective in negative regulation of membrane fusion, increasing the number of prominent vacu
192 HIV entry are approved for clinical use: the membrane fusion-inhibitor T20 (Fuzeon, enfuvirtide) and
201 ing of mechanisms governing alphaherpesvirus membrane fusion is expected to inform the rational desig
202 e tethering of the secretory vesicles before membrane fusion is mediated by the exocyst, an essential
203 her this clustering plays a critical role in membrane fusion is poorly understood in live cells.
204 ribe a mechanism for how mitochondrial inner-membrane fusion is regulated by the ratio of two forms o
205 indicate that while autophagosomal-lysosomal membrane fusion is sensitive to inhibition of SNARE prim
206 ssays that require Munc18-1 and Munc13-1 for membrane fusion is stimulated by the D326K mutation and
211 virus (EBOV) envelope glycoprotein (GP) is a membrane fusion machine required for virus entry into ce
212 critical and conserved element of the viral membrane fusion machinery, and neutralize viral entry by
213 ication of OvHV-2 glycoproteins that mediate membrane fusion may help identify viral and/or cellular
216 The homeostasis of most organelles requires membrane fusion mediated by soluble N -ethylmaleimide-se
217 were shown to interfere with low pH-induced membrane fusion mediated by the H1 and H5 (group 1) hema
224 amin-related GTPase atlastin (ATL) catalyzes membrane fusion of the endoplasmic reticulum and thus es
226 c18c's role in mediating ectopic basolateral membrane fusion of ZGs contributes to the initiation of
228 in may regulate the function of syntaxins in membrane fusion or may suggest additional functions of t
229 ions and structural transformations, such as membrane fusion or protein and nucleic acid folding.
230 ted that these compartments are connected by membrane-fusion points, through which mature virions are
231 The formation, expansion, and closing of the membrane fusion pore during exocytosis was found to be s
233 al. visualize intermediates of the HIV-cell membrane fusion process and demonstrate how Serinc prote
234 marize our latest understanding of the HIV-1 membrane fusion process and discuss possible pathways fo
236 he F HR3, improving our understanding of the membrane fusion process for NiV and likely for the relat
238 ormation on the viral spike proteins and the membrane fusion process to provide plausible explanation
241 novel classes of EPIs that interact with the membrane fusion protein AcrA, a critical component of th
244 in the expression of the mitochondrial inner membrane fusion protein optic atrophy type 1, and compon
245 of this study suggest that LpqN may act as a membrane fusion protein, connecting MmpL transporters wi
251 alovirus (HCMV), which are known to use cell membrane fusion rather than endocytosis to enter fibrobl
252 nd mediate virtually all known intracellular membrane fusion reactions on which exocytosis and traffi
257 and NiV-F, mediate host-cell attachment and membrane fusion, respectively, and are targets of the ho
260 dered incapable of pH-induced triggering for membrane fusion, resulting in lysosomal degradation.
261 ts suggest a previously unsuspected role for membrane fusion, similar to nuclear repair, in the forma
262 important to the regulation of HSV-1-induced membrane fusion since mutating N58 to alanine (N58A) cau
264 though both otoferlin and synaptotagmin bind membrane fusion SNARE proteins, only otoferlin interacts
267 g space, we identified genes associated with membrane fusion that could have important roles in the m
268 CoRA-dependent decrease in the rate of viral membrane fusion that extends the lifetime of the interme
269 segregation provides an energetic driver for membrane fusion that increases the efficiency of DNA-med
270 ing and extracellular vesicles for efficient membrane fusion, the resulting ligand-displaying extrace
271 are known to be indispensable for sperm-egg membrane fusion: the sperm proteins IZUMO1 and SPACA6, a
272 eability barrier has been established before membrane fusion, thereby avoiding a major threat to comp
275 ion of alkyne lipids significantly increases membrane fusion to enhance mRNA release, leading to syne
276 r machines that mediate receptor binding and membrane fusion to facilitate entry into host cells.
277 rions, nonenveloped viruses have no need for membrane fusion to gain access to intracellular replicat
279 ORTANCE Enveloped viruses require virus-cell membrane fusion to release the viral genome and replicat
281 , and L (gB, gH, and gL) were able to induce membrane fusion together but not when expressed individu
282 liposomal membrane destabilization involving membrane fusion upon incubation with activated platelets
284 xpression of TMPRSS2, which increases plasma membrane fusion versus endosome fusion of SARS-CoV-2, at
285 2 domain (Doc2) families regulate exocytotic membrane fusion via direct interactions with Ca(2+) and
286 using single-virus measurements of influenza membrane fusion, we show that fluorescent membrane probe
287 c bipolar tetraether lipids can also undergo membrane fusion, which is commonly accompanied by conten
288 requires glycoproteins H (gH) and L (gL) for membrane fusion, which is in contrast to requirements of
289 The formation of the ER requires homotypic membrane fusion, which is mediated by a family of Dynami
290 and shed light on activation of coronavirus membrane fusion, which takes place through a receptor-dr
291 to enhance GRASP65 oligomerization and Golgi membrane fusion, while adding purified DjA1 enhanced GRA
292 HV-2 gB, gH, and gL are sufficient to induce membrane fusion, while glycoprotein Ov8 plays an enhanci
294 ass II, indicating that its ability to block membrane fusion with B cells represents a defect in gB a
296 i-gHgL antibodies, CL40 and CL59, that block membrane fusion with both B cells and epithelial cells.
297 inhibit virus infections by preventing virus membrane fusion with cells and by inhibiting fusion of i
299 d productive infection at the level of viral membrane fusion, with a range of inhibitory activities a
300 meric complex with SLC7A5 and a regulator of membrane fusion, YKT6, to promote leucine uptake and cel