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1 mediated by the E1 protein, a class II virus membrane fusion protein.
2 mediated by the E1 protein, a class II virus membrane fusion protein.
3 e, which functions as a receptor binding and membrane fusion protein.
4 which is probably derived from an ancestral membrane fusion protein.
5 f specialized virus envelope proteins termed membrane fusion proteins.
6 he only feature that E2 shares with class II membrane fusion proteins.
7 equently, nonenveloped viruses do not encode membrane fusion proteins.
8 are involved in the activation of some virus membrane fusion proteins.
9 HAP2 that reveal homology to class II viral membrane fusion proteins.
10 ional homology with other well-characterized membrane fusion proteins.
11 s domain has a similar function in different membrane fusion proteins.
12 s a common motif found in many diverse viral membrane-fusion proteins.
13 s in retrovirus, paramyxovirus and filovirus membrane-fusion proteins.
14 dentifying coiled-coil-like regions in viral membrane-fusion proteins.
15 ibed for the haemagglutinin and HIV/SIV gp41 membrane-fusion proteins.
16 ms a cell envelope-spanning complex with the membrane fusion protein AcrA and the outer membrane prot
17 novel classes of EPIs that interact with the membrane fusion protein AcrA, a critical component of th
18 he RND transporter All3143 and the predicted membrane fusion protein All3144, as homologs of E. coli
19 e the cytoplasmic membrane-localized ATPase, membrane fusion protein and outer membrane protein compo
20 in channels tested but necessitated the MexJ membrane fusion protein and the MexK inner membrane RND
21 in order to understand the functionality of membrane fusion proteins and to define key parameters in
23 mary active transporter (IMP), a periplasmic membrane fusion protein, and an outer membrane channel.
24 a central RND proton-substrate antiporter, a membrane fusion protein, and an outer membrane factor.
25 I) associates with AcrA(HI), the periplasmic membrane fusion protein, and the outer membrane channel
26 l, inner membrane transporter; a periplasmic membrane fusion protein; and a beta-barrel, outer membra
27 r membrane, substrate-binding transporter; a membrane fusion protein; and an outer-membrane-anchored
28 poyl/biotin swinging arm domain in bacterial membrane fusion proteins; and a DH domain in the yeast B
30 ehairpin intermediate (PHI) of class I viral membrane-fusion proteins are generally weakly neutralizi
31 are set apart from other viral and cellular membrane fusion proteins by their extensively palmitoyla
32 n, vesicle accumulation at lesion sites, and membrane fusion proteins; Ca(2+) influx also initiates c
34 the outer membrane component, TolC, and the membrane fusion protein component, AcrA, of the major an
36 of this study suggest that LpqN may act as a membrane fusion protein, connecting MmpL transporters wi
37 4830-4919)-GFP, were localized to the plasma membrane; fusion proteins containing the fourth transmem
38 , we found that purified MacA, a periplasmic membrane fusion protein, contains one tightly bound roug
39 the inner membrane transporter CusA and the membrane fusion protein CusB of the CusCBA efflux system
40 both the inner-membrane transporter CusA and membrane fusion protein CusB of the CusCBA tripartite ef
42 ) proteins comprise a unique family of viral membrane fusion proteins dedicated to inducing cell-cell
44 rusion of periplasmic substrate bypasses the membrane fusion protein, enters the RND-transporter dire
45 YegN and YegO produce a complex(es) with the membrane fusion protein family member YegM and pump out
46 E possesses features similar to those of the membrane fusion protein family that facilitates the pass
47 jacent genes yhcQ, encoding a protein of the membrane fusion protein family, and yhcR, encoding a sma
48 owed that AcrA, a periplasmic protein of the membrane fusion protein family, could function with at l
50 Here we review current concepts about viral membrane fusion proteins focusing on how they are trigge
51 cture of Atlas G(C) reveals a class II viral membrane fusion protein fold not previously seen in retr
52 by deletion of mitochondrial outer or inner membrane fusion proteins (Fzo1p or Mgm1p) leads to decre
53 logy with viral trimeric coiled-coil class I membrane fusion proteins, gp26 may represent the membran
55 (Mfn1 and Mfn2) are the mitochondrial outer-membrane fusion proteins in mammals and belong to the dy
56 he coiled-coil motif occurs in several viral membrane-fusion proteins, including HIV-1 gp41 and influ
57 l architecture resembles several other viral membrane-fusion proteins, including those from HIV and i
58 itch model requires substrate binding to the membrane fusion protein, inducing a conformational chang
60 ncreased expression of AcrA, the periplasmic membrane fusion protein, is toxic only in cells lacking
61 eukaryotic cells and has been linked to the membrane-fusion proteins known as soluble N-ethylmaleimi
64 or MF; periplasmic proteins belonging to the membrane fusion protein (MFP) family; and outer membrane
66 orter, an outer-membrane factor (OMF), and a membrane fusion protein (MFP) that spans the periplasmic
70 rters function in complexes with periplasmic membrane fusion proteins (MFPs) that enable antibiotic e
74 of the outer membrane component MtrE and the membrane fusion protein MtrC, obtained by a combination
76 uires adenosine triphosphate and the general membrane fusion protein, N-ethylmaleimide sensitive fact
81 es encode homologues of ABC transporters and membrane fusion proteins of Type I secretion systems, re
86 in the expression of the mitochondrial inner membrane fusion protein optic atrophy type 1, and compon
87 binding transporter (or pump); a periplasmic membrane fusion protein (or adaptor); and an outer-membr
91 ologically different to that mediated by the membrane fusion proteins, SNAREs, as initial fusion is b
94 polyhedrovirus (AcMNPV) is a class III viral membrane fusion protein that is triggered by low pH duri
96 y hydrophobic segments of viral and nonviral membrane fusion proteins that enable these proteins to f
99 shuttling of periplasmic substrate from the membrane fusion protein to the RND transporter and furth
100 the outer membrane protein TolC and cognate membrane fusion proteins to form tripartite transperipla
101 ly P. aeruginosa RND pump which contains two membrane fusion proteins, TriA and TriB, and both are re
102 ehairpin intermediate (PHI) of class I viral membrane-fusion proteins typically show limited neutrali
103 nthesized, and the soluble core of the Visna membrane fusion protein was reconstituted in solution.
104 ce lacking specific vesicle-associated SNARE membrane fusion proteins, we found that VAMP-8-deficient
105 functions together with MacA, a periplasmic membrane fusion protein, which stimulates MacB ATPase.
106 ter PRV infection due to the action of viral membrane fusion proteins, yet it is unclear if such acti
107 plex from Bacillus subtilis, where YknX is a membrane fusion protein, YknY is an ATPase, and YknZ is