ライフサイエンスコーパス: membrane resistance

1 d instead likely mediated by lowering of the membrane resistance.

2 he intrinsic AP threshold and increasing the membrane resistance.

3 iring rate but as a secondary action reduces membrane resistance.

4 llular resistivity, membrane capacitance, or membrane resistance.

5 able phase of the action potential with high membrane resistance.

6 erning signal dissipation through changes in membrane resistance.

7 s in spontaneous IPSCs (sIPSCs), sEPSCs, and membrane resistance.

8 t decrease in spontaneous AP firing rate and membrane resistance.

9 microM), with a concomitant net decrease in membrane resistance.

10 eus resulted in depolarization and increased membrane resistance.

11 ns by inducing depolarization and increasing membrane resistance.

12 rization and hence a decrease in basolateral membrane resistance.

13 eover, it did not produce sizable changes in membrane resistance.

14 cells a hyperpolarization and a reduction of membrane resistance.

15 cause of both uphill transport and increased membrane resistance.

16 :0 being the crucial lipid species affecting membrane resistance.

17 as negatively correlated with their cellular membrane resistance.

18 le for these cannabinoid-produced changes in membrane resistances.

19 urons and a relative decrease in the somatic membrane resistance (0.7-8.1 M omega) was detected durin

20 sport resistance for P(m)>1 cm s(-1) whereas membrane resistance accounts for 50-75% of total transpo

21 We show that voltage-dependent changes in membrane resistance amplify synaptic activity, whereas t

22 e appears to depend upon both an increase in membrane resistance and a decrease in total cell surface

23 Warming led to a significant decrease in membrane resistance and a shunting of the dendritic sign

24 increased spike duration, and reductions in membrane resistance and amplitude of the Ih current.

25 ride was associated with a decrease in ileal membrane resistance and an increase in inducible nitric

26 ne excitability, as indexed by a decrease in membrane resistance and an increase in the stimulus thre

27 ane region includes local models for passive membrane resistance and capacitance, nonlinear active so

28 depend on the membrane time constant, set by membrane resistance and capacitance.

29 on/dehydration induces reversible changes of membrane resistance and effective capacitance.

30 abilizing it while synergistically elevating membrane resistance and electrotonic spread.

31 (Kir) and I (h) synergistically elevate the membrane resistance and favor dendritic integration whil

32 Ventral midbrain astrocytes have very low membrane resistance and inward-rectifying potassium chan

33 n its resting membrane properties, including membrane resistance and potential.

34 ubthreshold sensory stimuli due to increased membrane resistance and reduced membrane capacitance, re

35 ability after stimulation, with increases in membrane resistance and reductions in spike threshold.

36 tic potential integration by influencing the membrane resistance and resting membrane potential.

37 filaments have to be bundled to overcome the membrane resistance and that the filopodial length is li

38 often indicate voltage-dependent changes in membrane resistance and time constant values that can am

39 ng synaptic inputs minimally altered somatic membrane resistance and time constant values.

40 The neutral salt is used to reduce membrane resistance and to ensure reversibility of the s

41 ere also associated with a small increase in membrane resistance, and in voltage-clamp recordings ore

42 s of membrane potential, severe reduction of membrane resistance, and influx of Na+, Ca2+, Cl- and wa

43 ntial (RMP), spontaneous AP firing rate, and membrane resistance are cyclically regulated as a functi

44 d motor outputs while many neurons with high membrane resistances are still maturing.

45 r at infinite dilution, and an area-specific membrane resistance as low as 0.17 Omega cm(2).

46 llate cells, a voltage-dependent increase in membrane resistance at sub-threshold voltages mediated b

47 with a substantial increase in the specific membrane resistance between somatic and dendritic compar

48 ance of the nerve cell and by increasing its membrane resistance, but little is known about the latte

49 ptic current numbers, were scaled with their membrane resistances, but their own synaptic outputs wer

50 eurons (7.8 +/- 0.6 mV; n = 16), reduced the membrane resistance by 33 +/- 3%, and could convert the

51 rectification and steady-state components of membrane resistance by 37 and 38 %, respectively, in 66

52 dels was accompanied with decreased specific membrane resistance by approximately 25% and efficacy of

53 No differences were found in the membrane resistance, capacitance, or kinetic and voltage

54 Neither membrane resistance changes nor spine density changes we

55 hannels in S-D muscle produced high specific membrane resistance, comparable to similarly treated con

56 olarized resting potentials and an increased membrane resistance compared with age-matched control ce

57 to L-glutamate without significant change in membrane resistance, consistent with the well-establishe

58 Over the next 2 weeks, membrane resistance decreased and resting membrane poten

59 partial depolarization, to about -40 mV; the membrane resistance decreased by only 37%.

60 onstrates for the first time that increasing membrane resistance decreases the efficiency of this res

61 ined depolarization up to -27 mV and reduced membrane resistance (EC50 140-170 pm).

62 Ang II depolarized and increased membrane resistance equally in both TNs (n = 8) and PNs

63 ee Hb due to transport resistances including membrane resistance, extra- and intra-cellular resistanc

64 Furthermore, hydrogel membrane resistances extracted from equivalent circuit m

65 a passive current-voltage relationship, low membrane resistance, high capacitance, and dye-coupling

66 increased membrane capacitance and decreased membrane resistance in the absence of PMP22, which was c

67 elation between input synaptic strengths and membrane resistances in modeling broke down swimming rhy

68 e contrast is due to the decrease in tip and membrane resistance, in the vicinity of the pore opening

69 Also, the membrane resistance increased quite strongly at high pol

70 On average, the membrane resistance is 14 times lower and the effective

71 er of G-protein trimers required to overcome membrane resistance is 3 to 5, within a contact zone bet

72 1) for 10-45% Hct, respectively, below which membrane resistance is more significant and above which

73 d depend on the number of bundled filaments, membrane resistance, lamellipodial protrusion rate, and

74 take by intracellular hemoglobin or a unique membrane resistance mechanism.

75 The phasic membrane resistance modulation in relation to the gill r

76 Gram-negative bacteria utilize dual membrane resistance nodulation division-type efflux syst

77 In contrast, a decreased membrane resistance of DCN granule cells (multisensory i

78 This strategy exploits the change in the membrane resistance of the powered system, comprising a

79 In contrast, the resting potential and membrane resistance of the recorded cells remained uncha

80 P>0.05) in T-type current kinetics or in the membrane resistance of the thalamic cells between the tw

81 ing voltage clamp, we found that the passive membrane resistance of VS cells was reduced during fligh

82 ir own synaptic outputs were correlated with membrane resistances of their postsynaptic partners.

83 ough strophanthidin did not alter either the membrane resistance or the Na(+) reversal potential, the

84 ated inhibitory postsynaptic potentials, the membrane resistance, or the holding current, whereas it

85 se effects on resting membrane potential and membrane resistance persisted in the presence of TTX.

86 n ORNs (4.4 +/- 0.4 pF) and a lower apparent membrane resistance (R(m)) (160 +/- 11 MOmega versus 664

87 increased the ratio of apical-to-basolateral membrane resistance (R:(A)/R:(B)).

88 T, excitability is determined by the resting membrane resistance, R(m).

89 Their membrane resistance reaches ~10(4) Omega cm(2) in 1 m Cl

90 ntrinsic membrane excitability, increases in membrane resistance, reductions in the rheobase, and red

91 larized, with spontaneous AP firing rate and membrane resistance remaining stable.

92 pharmacological agent that blocks changes in membrane resistance reverted this effect.

93 We show that RF decreases the membrane resistance (Rm) and depolarizes the membrane po

94 in fibres of young HSA(LR) mice, the resting membrane resistance (Rm) at -90 mV is only slightly larg

95 resting membrane potential (Vm), IK,ADO, and membrane resistance (Rm) in rabbit isolated AV nodal myo

96 lant conductive systems, such as conduit pit membrane resistance, should scale in exact harmony with

97 e inexcitable despite resting potentials and membrane resistances similar to those of control denerva

98 ices taken from MeA-trained birds had higher membrane resistances than did cells from water-trained c

99 hibits a varying leak constant and a varying membrane resistance that are both dependent on the magni

100 ime constant is attributed to an increase in membrane resistance; the increase in input resistance ap

101 The upper limit of resting membrane resistance, then, is 6 GOmega.

102 e element method (FEM) is used to model cell membrane resistance to deformation.

103 In addition, a more limited decrease in membrane resistance upon reduction of extracellular calc

104 n transfected cells was 43.7 +/- 13.8 pF and membrane resistance was 458 +/- 123 Mohms.

105 Additionally, membrane resistance was found to be an important factor

106 n = 18) and PNs (-48 +/- 1 mV, n = 23) while membrane resistance was significantly higher in TNs.

107 Apical and basolateral membrane resistances were determined by nonlinear curve-

108 Image contrast also becomes independent of membrane resistance when an electrical shunt is used, al

109 The membrane resistances, which were similar for the TFC mem