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1 o polyamide coated crystals (mimicking an RO membrane surface).
2 ACA showed an amorphous protein layer on the membrane surface.
3 e the non-specific binding of antigen on the membrane surface.
4 viscous "suction" at close proximity to the membrane surface.
5 ne headgroup is approximately 20 A above the membrane surface.
6 nsmembrane (TM)5-6 loop on the intracellular membrane surface.
7 in the reversible binding of annexin to the membrane surface.
8 residues of the gangliosides relative to the membrane surface.
9 how a disordered protein can interact with a membrane surface.
10 nates only at distances beyond 10 A from the membrane surface.
11 y fold up into alpha-helical segments on the membrane surface.
12 s stack to form 2D matrix lattices below the membrane surface.
13 ecruit multiple active PI3K molecules to the membrane surface.
14 not have a similar tendency to adhere to the membrane surface.
15 ls and increase of the hydrophilicity of the membrane surface.
16 assemble together at a specific site on the membrane surface.
17 e a helix orientation of K * parallel to the membrane surface.
18 enhances leakage through fiber growth on the membrane surface.
19 d open structure BSA layer was formed on the membrane surface.
20 ted by gentle rubbing of a PVC eraser on the membrane surface.
21 ad no effect on Cav2.2 protein levels on the membrane surface.
22 monomer-monomer interactions of CDCs at the membrane surface.
23 There was less accumulation of BSA on the membrane surface.
24 o organize their spatial distribution at the membrane surface.
25 ons of Ras' orientational preferences at the membrane surface.
26 nteractions between the bacteria and charged membrane surface.
27 n of the applied electrical potential to the membrane surface.
28 l interactions between the N-glycans and the membrane surface.
29 sm and its assembly is away from the crowded membrane surface.
30 packed amphiphilic helices that rest on the membrane surface.
31 ne and increased the roughness factor of the membrane surface.
32 pendent on the anionic charge density of the membrane surface.
33 S34CII-PI3P-PX signaling pathway on a target membrane surface.
34 ter having found a stable orientation at the membrane surface.
35 nds to a specific region of LPS close to the membrane surface.
36 iquid and DAMO organisms attach close to the membrane surface.
37 t interactions with the fluid and functional membrane surface.
38 repeated after the Cu-NPs dissolve from the membrane surface.
39 te flux during bacterial accumulation on the membrane surface.
40 , and peripheral proteins are located at the membrane surface.
41 ction between the C domains of FVIII and the membrane surface.
42 e segment 2 (TM2) and TM3 on the cytoplasmic membrane surface.
43 GPR30 protein levels residing at the plasma membrane surface.
44 membrane and likely embed the helix into the membrane surface.
45 s within a transmembrane segment or near the membrane surface.
46 reduced amounts and was undetectable on the membrane surface.
47 transmembrane segment or at a site near the membrane surface.
48 re by means of complex interactions with the membrane surface.
49 t gases as well as CO2 reactant gases on the membrane surface.
50 of respiratory syncytial virus bound on the membrane surface.
51 ucleotides to anchor proteins to a supported membrane surface.
52 e SAR1B, SAR1A was prone to oligomerize on a membrane surface.
53 a polyethylene glycol chain tethered to the membrane surface.
54 g-Ca and Al-Fe fouling was observed over the membrane surface.
55 ic with a preferential localization near the membrane surface.
56 han the chemistry used to attach ENTH to the membrane surface.
57 Min proteins in the propagating waves on the membrane surface.
58 whereas the FPPR and MPER are exposed to the membrane surface.
59 ride lipids distort and spread over the host membrane surface.
60 etween the nucleotide-binding domain and the membrane surface.
61 allosteric linker lies superficially on the membrane surface.
62 inding as well as dimerization of Btk on the membrane surface.
63 of viral polyproteins Gag and Gag-Pol at the membrane surface.
64 oferlin's C2F domain interacting with a cell membrane surface.
65 has been challenging because it occurs on a membrane surface.
66 ance diffusion of the electron donors at the membrane surface.
67 ctively optimizing substrate turnover at the membrane surface.
68 atures of multiprotein signaling pathways on membrane surfaces.
69 s to and assembles clathrin on highly curved membrane surfaces.
70 ert protein or polymer, and by adsorption to membrane surfaces.
71 nd in similar probe volumes near protein and membrane surfaces.
72 y of encounter with target complexes on cell membrane surfaces.
73 ion is a general property of native H-Ras on membrane surfaces.
74 ntly exists for measuring steric pressure at membrane surfaces.
75 and by phycobilisomes situated on thylakoid membrane surfaces.
76 ed by the stiffness of the lateral and basal membrane surfaces.
77 nsity on the extracellular and intracellular membrane surfaces.
78 al different classes of protein which act on membrane surfaces.
79 d full-length Syt1 to interact with opposing membrane surfaces.
80 brane packing defects found on highly curved membrane surfaces.
81 rmembrane space and by the 2D environment of membrane surfaces.
82 lying direct interactions between P2 and two membrane surfaces.
83 e and dynamics of lipid-signaling enzymes on membrane surfaces.
84 s of how lipid-signaling enzymes function on membrane surfaces.
85 ane traffic, and cell motility, originate at membrane surfaces.
86 tidylinositol-4,5-phosphate [PI(4,5)P(2)] on membrane surfaces.
87 ticles uniformly distributed over the entire membrane surfaces.
88 tructured protein sense the structure of the membrane surface?
91 of these extracellular molecules across the membrane surface allows rapid screening of the biofilm c
93 nto two helices, one running parallel to the membrane surface, analogous to the S4-S5 linker of domai
95 the 3E6 epitope faces the negatively charged membrane surface and Arg2320 is poised at the center of
97 associated proteins that laminates the inner membrane surface and attaches to the overlying lipid bil
98 ably, while gypsum crystals grew both on the membrane surface and deep in the membrane matrix, silica
99 domain (EC2) of CD53 protrudes away from the membrane surface and exposes a variable region, which is
100 mately 75 residues that lies parallel to the membrane surface and has been proposed to play a mechani
101 on, which results in its accumulation at the membrane surface and hence in a potential response.
102 relevant membrane, namely attachment to the membrane surface and insertion into the lipid A leaflet.
104 lis (B. subtilis) by bacterial growth on the membrane surface and its exposure to bacterial suspensio
105 electrostatic adsorption of exenatide to the membrane surface and its self-association (K(d) = 46 muM
106 magainin 2, resists self-association at the membrane surface and penetrates further into the hydroph
107 and hydrophobic interactions with the viral membrane surface and that the collective physical proper
108 he attractive charge interaction between the membrane surface and the deprotonated Glu134 residue of
109 an open activation gate at the intracellular membrane surface and the intracellular C-terminal domain
110 s enable these proteins to polymerize on the membrane surface and undergo two-dimensional phase separ
111 binding to concave, cardiolipin-containing, membrane surfaces and compare findings to convex binding
112 s Rab1 and COPII vesicles to create enlarged membrane surfaces and optimal lipid composition within t
113 differences include adsorption on the lipid membrane surfaces and partitioning into the center of li
114 (XPS) were used to characterize foulants on membrane surfaces and rigorously deduce their contributi
115 al chain pressure, low charge density at the membrane surface, and increased salt concentration promo
116 mitigation strategies, hydrodynamics at the membrane surface, and membrane module configuration may
117 in a higher deposition rate of BSA onto the membrane surface, and the formation of a denser BSA laye
118 orces, the adsorption behavior of BSA on the membrane surface, and the structure of the BSA adsorbed
119 tide PI(4,5)P2 attract the protein to acidic membrane surfaces, and myristoylation increases the affi
120 opy demonstrated that cells deposited on the membrane surface are inactivated, resulting in a layer o
121 t lipid bilayer structures and low curvature membrane surface are preferable for CYP2B4-cytb5 complex
122 of common techniques for modification of the membrane surface are reviewed, including surface coating
124 al cells build apical microvilli to increase membrane surface area and enhance absorptive capacity.
125 ice, which relieves contraction of the model membrane surface area and eventual lipid bilayer collaps
126 of microvilli that serves to amplify apical membrane surface area and increase functional capacity.
128 physical properties, specifically available membrane surface area and the membrane area expansion mo
129 of cristae creates more mitochondrial inner membrane surface area and thus more protonic capacitance
130 generated by membrane stretching, when both membrane surface area and volume are variable parameters
131 he structure provides a significantly higher membrane surface area for loading of membrane proteins a
132 icle fusion is directly related to the total membrane surface area of the sample added, and a calibra
133 an LC elasticity-induced expansion of lipid membrane surface area of up to 3% and conservation of th
134 rifugation conditions, as well as by varying membrane surface area or membrane fouling, the filtrate
136 llars each cell faces varies with its plasma membrane surface area, despite their large population an
137 ically the quantification of the total lipid membrane surface area, found in a sample using Forster r
139 that collective switching can spread on the membrane surface as a traveling wave of Rab5 activation.
140 have a high potential to cause biofouling on membrane surface as the bacteria still maintain the cell
141 rength affects the structure of pHLIP at the membrane surface as well as the acidity needed for diffe
142 isfolded in a beta-sheet conformation at the membrane surface, as detected by in situ synchrotron gra
143 low insertion peptide (pHLIP) adsorbs to the membrane surface at a neutral pH, but it inserts into th
144 e the hydration water structure at the lipid membrane surface at XDMSO <0.1, lower the energetic barr
145 lement approach to model diffusion on curved membrane surfaces based on solutions to Fick's law of di
146 Cryo-EM has revealed the structures of the membrane-surface bound pre-pore and inserted-pore oligom
147 tional to the alpha-syn concentration on the membrane surface but found to be sensitive to the specif
148 hat acidification of the local extracellular membrane surface by a light-activated proton pump recrui
149 the ability of Sec7 to activate Arf1 on the membrane surface by facilitating membrane insertion of t
150 individual pH-sensitive fluorophores at the membrane surface by fluorescence correlation spectroscop
151 ressure, which if unbalanced on the opposite membrane surface can dramatically increase membrane curv
153 es since accumulation of contaminants to the membrane surface can lead to fouling, performance declin
155 oduce an extended conformation of Ail at the membrane surface, cause thickening and rigidification of
156 nium species valence and size in relation to membrane surface charge and pore size) and (ii) concentr
157 osomes, which demonstrated the importance of membrane surface charge and the presence of the glucosyl
158 face roughness were also maintained, and the membrane surface charge became positive after functional
160 stigate the effects of lipid chain disorder, membrane surface charge, and intrinsic negative curvatur
162 we show that by controlling the liquid cell membrane surface chemistry and electron beam conditions,
163 However, increasing the mitochondrial inner membrane surface comprises an alternative mechanism for
164 id bilayer in order to elucidate the role of membrane surface curvature in modulating the peptide str
166 species signifying initiation of biofilms on membrane surfaces, demonstrated by specific DESI MS sign
168 supersaturated conditions develop along the membrane surface due to the passage of water through the
173 form of active deformation, we find that the membrane surface exceeds by a factor of two the amount o
174 onal diffusion of the PH domain on the lipid membrane surface exhibit transient subdiffusion, with an
175 nstrate that active transport to the capture membrane surface expedites antibody-antigen binding.
178 rientate both substrate cavities towards the membrane surface for efficient substrate transit between
179 l, may pave the way to generate an increased membrane surface for interaction with monocytes and neut
184 l droplets or in two-dimensional lattices on membrane surfaces, have emerged as another important org
185 ilic MAG2 helix was found to lie flat on the membrane surface in 1,2-dimyristoyl-sn-glycero-3-phospha
186 At physiological pH, ATRAM binds to the membrane surface in a largely unstructured conformation,
187 t allows cells to be maintained on the upper membrane surface in a thin layer of fluid while media is
189 e membrane matrix, silica only formed on the membrane surface in the form of a relatively thin film c
190 ory subunit, factor Va (fVa), assembled on a membrane surface in the presence of divalent metal ions.
193 at Gea1 and Gea2 prefer neutral over anionic membrane surfaces in vitro, consistent with their locali
194 tions for processes that take place at lipid membrane surfaces, including molecular recognition, bind
195 ng by developing a killing and self-cleaning membrane surface incorporating antibacterial silver nano
196 on changes in cholesterol reactivity at the membrane surface independently of the overall cholestero
197 ns are linked by a helix that runs along the membrane surface interacting with the phospholipid head
199 that eukaryotic cells are able to convert a membrane surface into a high-definition lipid-signaling
201 The localization of His-379 on the lumenal membrane surface is consistent with a role for this inva
202 ments confirm that the zeta-potential of the membrane surface is converted from negative (non-functio
204 we present evidence that adhesion to the UF membrane surface is mediated by cell-surface macromolecu
207 er chlorine uptake, and (vi) scission at the membrane surface is unrepresentative of volume-averaged
209 studies showing that proton diffusion along membrane surfaces is time- and length-scale dependent.
210 Added electrostatic repulsion between the membrane surfaces is used to identify the formation of s
211 previously believed to be exposed above the membrane surface, is also membrane associated, suggestin
213 Intriguingly, oligomerization of HIV-Tat on membrane surfaces leads to the formation of membrane por
215 ein levels, reduced glycosylation, and lower membrane surface levels of hCaV3.3 when expressed in hum
216 ng stresses as the filament elongates on the membrane surface likely contributes to the driving force
217 voltage-gated currents from channels on the membrane surface (membrane clock) with rhythmic Ca(2+) r
219 by making the electrostatic potential at the membrane surface more negative, while decreasing the pen
220 ber and large size of milk fat globules, the membrane surface needed for their release might exceed t
221 to generate ratiometric images of the plasma membrane surface of Bright Yellow 2 tobacco (Nicotiana t
222 t cells innervate the entire somato-denditic membrane surface of principal neurons, the spike control
224 fer protein plastocyanin (Pc) to the lumenal membrane surface of the cytb6f complex using a Pc-functi
225 ophobic extension that lies on the cytosolic membrane surface of the lysosome, where it interacts wit
229 ioned about 65 +/- 35 angstrom away from the membrane surface, offering the first estimations of the
231 or building penetrated channels on vesicular membrane surface often involve regulating the solvent po
232 of surface morphology and surface energy on membrane surface omniphobicity were systematically inves
233 DAMTS-4_v1 was found to bind to the synovial membrane surface on cryosections, and the protein was de
234 able permeants dynamically neutralize at the membrane surface or permeate in their charged form.
236 es that peptide aggregation on the bacterial membrane surface plays a decisive role in the degree of
237 he control of biomolecular phase separation: membrane surfaces, post-translational modifications, and
238 e quantitatively explained by the changes in membrane surface potential due to exclusion of kosmotrop
240 that phenolics studied here are bounded to a membrane surface predominantly via hydrogen bonds, while
241 holipid surfaces, it is instead inhibited by membrane surfaces prepared directly from the plasma memb
242 Gea2 toggle roles in the cytosol and at the membrane surface, preventing membrane binding in the abs
243 ions of electrochemically produced HP on the membrane surface prevents bacterial attachment, thus ens
244 s, and prevents the biofilm formation on the membrane surface, producing excellent antimicrobial acti
245 ize the importance of hydrodynamic shear and membrane surface properties on the initially deposited a
248 places the cytoplasmic domains of PLN at the membrane surface proximal to the calcium entry funnel of
249 reased amount of the enzyme localized to the membrane surface rather than with a loss of activity or
253 e-stranded oligoprobes functionalized on the membrane surface resulting in the formation of a cation
254 s to negatively charged phosphatidylglycerol membrane surfaces results in a higher ordered membrane s
256 parallel fusion pathway, DNA aligns with the membrane surface, showing very quick release of genetic
257 microscopic ligand concentration within the membrane surface solvation layer may exceed that in bulk
259 ing diverse biochemical events that occur on membrane surfaces, such as membrane remodeling, ligand-r
260 olecular dynamics simulations of P2 on model membrane surfaces suggested that Arg-88 is critical for
261 to have maximal catalytic activity near the membrane surface suggests that these conformational chan
262 on analysis approach, we show a reduction in membrane surface tension and increased membrane undulati
263 al. (2019) provide evidence that asymmetric membrane surface tension determines the directionality o
264 , we predict regions of osmotic pressure and membrane-surface tension that produce successful engulfm
265 kness is minimal, Ca(2+) stays longer on the membrane surface than K(+) or Na(+), consequently leadin
266 peptide epitopes naturally presented on the membrane surface, the final formulation contains the nec
267 rp1 self-assembly and GTPase activity at the membrane surface, the mechanism by which CL functions in
268 Specifically, when BAR scaffolds assemble at membrane surfaces, their bulky disordered domains become
270 and association of the F3 subdomain with the membrane surface through a large, interdomain conformati
271 initial stages of recruiting protein to the membrane surface through non-specific electrostatic inte
272 cated that Ras undergoes dimerization at the membrane surface through protein-protein interactions.
273 Here, we report that H-Ras forms a dimer on membrane surfaces through a protein-protein binding inte
274 thic helix is aligned nearly parallel to the membrane surface (tilt angle approximately 77 degrees )
277 ne tubes displaying planar and highly curved membrane surfaces to analyze intrinsic membrane curvatur
278 inity-mapping AFM method directly correlates membrane surface topography with Pc-cytb6f interactions,
279 Combined with a new method to visualize membrane surface topology, we map the molecular landscap
282 the number of live bacteria attached to the membrane surface was over 90% less than that of control
283 ribution in the digestive vacuole and at its membrane surface, we deduce that the excess bromoquine f
284 LC searches for GPI-anchored proteins on the membrane surface, we measured residence times of single
285 disrupting the water network near the lipid membrane surface, weakening the cohesion between water a
288 e active site cleft and pull it close to the membrane surface, where cytidyl transfer can occur effic
289 ns (including FYVE, PX, and PROPPINS) to the membrane surface, where they initiate key cell processes
290 h cytoplasmic domain of CLS from the anionic membrane surface, which enabled subsequent association o
291 elles) and adherence of graft-polymer to the membrane surface, which facilitates grafting and reduces
292 ble positive charge, is left dangling at the membrane surface, which is likely to alter the surface e
293 imply interactions between tr-CYP2B4 and the membrane surface, which might assist in CYP2B4-cytb5 com
294 r flux maximized bacterial attachment to the membrane surface, which was 60 times greater (6 x 10(3)
295 nd irreversible binding of the Cu-NPs to the membrane surface with SEM and XPS after exposing the mem
296 transglutaminase (TG2), thereby endowing the membrane surfaces with anti-inflammatory properties.
297 in which the sheets are connected by twisted membrane surfaces with helical edges of left- or right-h
298 nd uninfected T cells contain interdigitated membrane surfaces, with T cell filopodia extending towar
299 equestering the amyloid fold of curli on the membrane surface without significant accumulation of tox
300 antibody-antigen interactions at the sensing membrane surface without the need to add a label or a re