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1 embranoproliferative glomerulonephritis, and membranous glomerulonephritis).
2 ultrastructural, and clinical model of human membranous glomerulonephritis.
3 CD5Rg in a murine model of antibody-mediated membranous glomerulonephritis.
4 s, and one case was associated with atypical membranous glomerulonephritis.
5 hich reproduces features of human idiopathic membranous glomerulonephritis.
7 tis, focal proliferative glomerulonephritis, membranous glomerulonephritis, and mesangial glomerulone
8 tanding the pathogenesis of human idiopathic membranous glomerulonephritis, and proposes future direc
9 sions that involved >or=50% of glomeruli +/- membranous glomerulonephritis (class III >or=50%+/-V) we
10 diffuse proliferative glomerulonephritis +/- membranous glomerulonephritis (class IV+/-V) was less co
11 overexpressed in human kidney epithelium in membranous glomerulonephritis, diabetes mellitus, IgA ne
12 [PCT], vitiligo, and lichen planus); renal (membranous glomerulonephritis [GN] and membranoprolifera
16 sed the earliest manifestations of recurrent membranous glomerulonephritis (MGN) in renal allografts.
18 nts with focal-segmental glomerulosclerosis, membranous glomerulonephritis, minimal change disease, I
19 = 3; psoriasis, n = 2; Graves disease, n 1; membranous glomerulonephritis, n = 2; rheumatoid arthrit
21 diagnosed as having transverse myelitis and membranous glomerulonephritis secondary to systemic lupu
22 us pseudotumor, sclerosing mesenteritis, and membranous glomerulonephritis should all be added to the
23 in mind, particularly in older patients with membranous glomerulonephritis where the possibility of m