ライフサイエンスコーパス: memory disorder

1 vocabulary development were arrested by his memory disorder.

2 hat is needed to develop therapies for human memory disorders.

3 s, a key region associated with epilepsy and memory disorders.

4 lities for degenerative and neuropsychiatric memory disorders.

5 H2A.Z as a potential therapeutic target for memory disorders.

6 ble to probe the biological underpinnings of memory disorders.

7 , offering promise for targeted treatment of memory disorders.

8 t clinical implications for the treatment of memory disorders.

9 tion as treatment strategies for maladaptive memory disorders.

10 with those working primarily in the area of memory disorders.

11 it may be useful for treating patients with memory disorders.

12 s received little attention in patients with memory disorders.

13 uld contribute to FAS and ethanol-associated memory disorders.

14 tially relevant to synaptic malplasticity or memory disorders.

15 understanding of human cognition and related memory disorders.

16 single imaging session in participants with memory disorders.

17 ributions to neurologic sequelae, especially memory disorders.

18 mise of brain stimulation-based treatment of memory disorders.

19 of interest for the treatment of age-related memory disorders.

20 d at 3 US outpatient clinics specializing in memory disorders.

21 diagnostic subtypes of MCI have an episodic memory disorder (amnestic MCI) occurring either alone [s

22 overall pathophysiological understanding of memory disorders and AD.

23 plications for developing therapies to treat memory disorders and cognitive impairment using DBS.SIGN

24 patients consulting their GP with suspected memory disorders and proportion of those referred to mem

25 hold the key to understanding the variety of memory disorders associated with aging and disease.

26 e inconsistent with Mendelian segregation of memory disorders both in families of affected probands w

27 The hypothalamus is implicated in memory disorders, but how its rapidly changing activity

28 tool for developing strategies to ameliorate memory disorders by targeting brain regions involved in

29 We studied patients who attended the Duke Memory Disorders Clinic and were diagnosed as having pro

30 shape our identities, and many debilitating memory disorders currently lack effective treatments.

31 evidence that the pattern of transmission of memory disorders differs in nuclear families in which th

32 In humans, the most conspicuous memory disorder following vmPFC damage is confabulation;

33 ral lobe seizures in 16 patients (88.9%) and memory disorders in 17 patients (94.4%) from the cohort.

34 Leucettinib-21 corrects memory disorders in the Down syndrome mouse model Ts65Dn

35 s in the hippocampus may conceivably lead to memory disorders in the mammalian brain.

36 P consultations with patients with suspected memory disorders increased in intervention versus contro

37 e huge motivation to treat age-related human memory disorders, interaction between human CA3 and dent

38 It thus appears that, if this selective memory disorder is a special syndrome related to the ear

39 ship of gene expression changes to memory or memory disorders is not well understood.

40 ble to probe the biological underpinnings of memory disorders marked by impairments in source memory.

41 with 21 consecutively recruited neurological memory-disordered patients and 14 healthy control subjec

42 of outcome, or comparisons with neurological memory-disordered patients.

43 chemic and hemorrhagic stroke, cognition and memory disorders, peripheral nervous system disorders, e

44 the fundamental substrates of age-associated memory disorders related to hippocampal dysfunction.

45 r time, and women have an increased risk for memory disorders relative to men later in life.

46 ue is critical for developing treatments for memory disorders such as post-traumatic stress disorder(

47 prominent sex differences in stress-related memory disorders, such as post-traumatic stress disorder

48 oring response to treatment in patients with memory disorders, such as those with voltage-gated potas

49 he probands is modified by family history of memory disorders, suggesting gene-by-gene interactions.

50 ive impairment (MCI) is an isolated episodic memory disorder that has a high likelihood of progressin

51 mory and for their use in the development of memory disorder treatments.

52 n children aged 51/2 to 6 years with working memory disorders who were born very preterm.