ライフサイエンスコーパス: memory retrieval

1 campal contribution to contextually mediated memory retrieval.

2 al compression may be a universal feature of memory retrieval.

3 ive state where we treat stimuli as cues for memory retrieval.

4 lude memory consolidation, or to deficits in memory retrieval.

5 for the involvement of awake replay in fear memory retrieval.

6 mPFC circuitry supports post-extinction fear memory retrieval.

7 uited during learning and reactivated during memory retrieval.

8 y be crucial to reinstatement for successful memory retrieval.

9 lations during retrieval supports successful memory retrieval.

10 tion and short-term memory tasks and enhance memory retrieval.

11 taste novelty processing and familiar taste memory retrieval.

12 thing phase enhances fear discrimination and memory retrieval.

13 ve value of elements during autobiographical memory retrieval.

14 ansport impaired memory acquisition, but not memory retrieval.

15 y replacing it with a much weaker one during memory retrieval.

16 it is present during remote, but not recent, memory retrieval.

17 acetylcholine release in the cortex to allow memory retrieval.

18 rain glucose uptake at the time of attempted memory retrieval.

19 GABAergic systems during taste learning and memory retrieval.

20 idline structures that are involved in later memory retrieval.

21 mo- and optogenetic approaches impaired fear memory retrieval.

22 ted a robust consensus: Acute stress impairs memory retrieval.

23 our understanding of the network dynamics of memory retrieval.

24 neural strategies associated with successful memory retrieval.

25 ructures that were also engaged during later memory retrieval.

26 ed AC-CA) is capable of eliciting contextual memory retrieval.

27 player in mediating the impact of stress on memory retrieval.

28 oing protein synthesis is required to enable memory retrieval.

29 techniques, much because it fosters repeated memory retrieval.

30 king stress effects on HPA-axis activity and memory retrieval.

31 lly recruited by the AC-CA projection during memory retrieval.

32 ion and advance a functional role for EMs in memory retrieval.

33 ic levels of fear learning, or altering fear memory retrieval.

34 ng of AMPA receptors that takes place during memory retrieval.

35 f neural encoding patterns is beneficial for memory retrieval.

36 f a larger brain network supporting episodic memory retrieval.

37 licated in place memory and autobiographical memory retrieval.

38 play important roles in successful episodic memory retrieval.

39 ning and conditioned taste aversion, but not memory retrieval.

40 appears to act as a context for learning and memory retrieval.

41 ocampus (dHipp) beta-ARs for drug-associated memory retrieval.

42 en IGF-II treatment is given in concert with memory retrieval.

43 linked by the number of cue presentations at memory retrieval.

44 ons of multiple hubs characterize successful memory retrieval.

45 , as well as how these areas interact during memory retrieval.

46 red in vivo for reduction of the sAHP during memory retrieval.

47 representation that is then deployed during memory retrieval.

48 ergic modulation in the medial PFC (mPFC) in memory retrieval.

49 esses, corresponding to the specific type of memory retrieval.

50 own how these structures implement selective memory retrieval.

51 cell activity was reinstated during episodic memory retrieval.

52 ts irrespective of the cues used to initiate memory retrieval.

53 ement of DMN elements in discrete aspects of memory retrieval.

54 5-HT2AR) could have a role in the control of memory retrieval.

55 rtex (vmPFC) is a key mediator of extinction memory retrieval.

56 e involvement of the striatum in declarative memory retrieval.

57 wer (70-180 Hz) within PMC subregions during memory retrieval.

58 adult-born neurons essential in hippocampal memory retrieval.

59 atum primarily supports cognitive control of memory retrieval.

60 bsequent reactivation of this engram induces memory retrieval.

61 ons to suppress freezing during context fear memory retrieval.

62 mine how memorability influences associative memory retrieval.

63 ortical representations during free episodic memory retrieval.

64 ntribute to long-term fear expression during memory retrieval.

65 er, especially as it pertains to awake human memory retrieval.

66 ys a key role in HC-PFC function during fear memory retrieval.

67 l and subjective reactivity during emotional memory retrieval.

68 both fear memory acquisition and extinction memory retrieval.

69 role of Cdk5 activation in attenuating fear memory retrieval.

70 rioritizes certain information to facilitate memory retrieval.

71 nt reorganization in PL ensembles underlying memory retrieval.

72 rodents and alEC activations during temporal memory retrieval.

73 on, spatial navigation, and autobiographical memory retrieval.

74 ed after long-term, but not short-term, fear memory retrieval.

75 uence that overlapping memories exert during memory retrieval.

76 mation reinstatement in the neocortex during memory retrieval.

77 red to those TRAPed during learning or early memory retrieval.

78 lations are reinstated to support successful memory retrieval.

79 d inhibitory PrL neurons during context fear memory retrieval.

80 bout hippocampal activity during spontaneous memory retrieval.

81 of stress-susceptible male mice after remote memory retrieval.

82 ing context fear conditioning and subsequent memory retrieval 2 days later.

83 opfield published a neural network model for memory retrieval, a model that became a cornerstone in t

84 During memory retrieval, a population of vCA1 neurons became co

85 eports of IPL activity arising from episodic memory retrieval, according to the type of information t

86 We show that, on memory retrieval after prolonged withdrawal, the matured

87 However, nothing is known about its role in memory retrieval after self-administration of cocaine, a

88 On episodic memory retrieval, an external stimulus (or an internal r

89 ve ripples (SWRs) is thought to support both memory retrieval and consolidation in distributed hippoc

90 r cFos activity in dorsal hippocampus during memory retrieval and context generalization, whereas fem

91 and retrospective modes, possibly reflecting memory retrieval and encoding, respectively.

92 es, possibly to prevent interference between memory retrieval and encoding.

93 re-activation is thought to be necessary for memory retrieval and expression of conditioned behavior.

94 f vHC-PrL projectors suppresses context fear memory retrieval and impairs the ability of PrL neurons

95 of hippocampal Cdk5 promoter attenuated fear memory retrieval and increased tau phosphorylation in fe

96 re, but not after, a CPP trial disrupted CPP memory retrieval and induced a persistent deficit in ret

97 diates differential behavioral outcomes upon memory retrieval and may be crucial for survival by prom

98 between these structures occurs during fear memory retrieval and may facilitate synaptic plasticity,

99 le dissociation in the mechanisms underlying memory retrieval and memory destabilization, since AMPAR

100 In particular, memory retrieval and novelty encoding were considered in

101 anding of dopaminergic processes in episodic memory retrieval and offer new perspectives on memory im

102 dynamics of parietal cortex during episodic memory retrieval and provide clear neurophysiological co

103 n the PL-mPFC and BLA for cocaine-associated memory retrieval and reconsolidation in rats.

104 l mechanisms required for cocaine-associated memory retrieval and reconsolidation.

105 ere more pronounced during successful verbal memory retrieval and recover the cortical neural represe

106 can modulate reconstructive processes during memory retrieval and reduce memory errors due to misinfo

107 n can modulate cortical reinstatement during memory retrieval and reduce misinformation errors.

108 and cognitive stress mechanisms may disrupt memory retrieval and restrict prospective planning, with

109 e the first evidence in humans that episodic memory retrieval and scene imagination rely on similar v

110 ttention processing in the right hemisphere; memory retrieval and semantic judgement in the left hemi

111 hanism for flexibly and selectively engaging memory retrieval and show that memory-based choices are

112 bition of competing memories during episodic memory retrieval and suggest that competitive retrieval

113 s that respond to the familiar mouse enabled memory retrieval and the association of these neurons wi

114 Memory retrieval and the molecular mechanisms that are s

115 ed to faulty reconstructive processes during memory retrieval and the reactivation of brain regions i

116 network that supports the ability to control memory retrieval and to understand the neural basis of a

117 Reactivation was contingent on memory retrieval and was not observed when animals were

118 This pattern of results suggests that memory retrieval and ZIP-sensitive maintenance mechanism

119 describe a model for the interaction between memory retrieval and ZIP-sensitive mechanisms, showing t

120 of the fronto-parietal network: declarative memory retrievals and updating of working memory.

121 twork-wide communication during naturalistic memory retrieval, and (ii) understand the causal relatio

122 consolidation, but dispensable for learning, memory retrieval, and reconsolidation.

123 y in PL-mPFC may underlie cocaine-associated memory retrieval, and therefore disruption of this plast

124 genetic activation of these cells results in memory retrieval, and this correlates with retained engr

125 ormation and were replayed during successful memory retrieval, and this replay was associated with ri

126 atomotor hand, fronto-parietal task control, memory retrieval, and visual and dorsal attention system

127 than inducing vulnerability to interference, memory retrieval appeared to aid the preservation of exi

128 rophysiological mechanisms that support such memory retrieval are largely unknown.

129 damentally distinct roles of context in fear memory retrieval are processed by distinct vHC output pa

130 PL neurons TRAPed during later memory retrievals are more likely to be reactivated and

131 Neurophysiological studies focus on memory retrieval as a reproduction of what was experienc

132 to-striatal circuits in item and associative memory retrieval as well as in the stabilization of memo

133 d the cognitive and neural bases of episodic memory retrieval, as well as the extent to which differe

134 CB1R antagonism also inhibited memory retrieval-associated increases in BLA zinc finger

135 the neuronal circuitry participating in fear memory retrieval at both early and late time points foll

136 the 5 d delay, but there was no evidence for memory retrieval at the 25 d delay.

137 of this molecular change was evident only as memory retrieval became PKA-dependent over time.

138 With the passage of time, memory retrieval becomes independent of alpha'/beta' and

139 on the track again and displayed an apparent memory retrieval behavior: avoidance of the shock zone.

140 embles selectively interferes with odor fear memory retrieval but does not compromise basic odor dete

141 l memory activation with impaired subsequent memory retrieval but have not provided an account of thi

142 brain regions have been linked with episodic memory retrieval, but limited progress has been made in

143 tal cortex (PPC) is thought to contribute to memory retrieval, but little is known about its specific

144 ergic receptor antagonist propranolol before memory retrieval, but not after (during memory reconsoli

145 eta-AR blockade had no effect on initial CPP memory retrieval, but prevented CPP expression during su

146 induce long-term deficits in drug-associated memory retrieval by reducing neuronal excitability, prov

147 enetic mechanisms during initial learning or memory retrieval can lead to persistent memory.

148 servation that mechanisms underlying cocaine memory retrieval change depending on the age of the memo

149 ke larger behavioral contributions to remote memory retrieval compared to those TRAPed during learnin

150 The meta-analysis showed that declarative memory retrievals correlated with activity in the inferi

151 Recollection is constructive, the product of memory retrieval cues, the information stored in memory,

152 d amygdala while neurosurgical patients made memory retrieval decisions together with a confidence ju

153 maze, produces an as large strategy shifting/memory retrieval deficit as mPFC or dHip inactivation in

154 s more affected in pure DLB than in AD while memory retrieval deficit was more affected in AD than in

155 lications for understanding visuospatial and memory-retrieval deficits in patients with parietal lobe

156 l on proteasome activity, relative to saline memory retrieval, depend on withdrawal time.

157 hippocampal endocannabinoid system modulates memory retrieval depending on the training-associated ar

158 erve to facilitate either memory encoding or memory retrieval, depending on which type of gamma rhyth

159 at the relationship between UPS activity and memory retrieval depends on training paradigm, brain reg

160 f hippocampal memory engram cells results in memory retrieval despite the fact that these mice are am

161 tant, the neural underpinnings of successful memory retrieval differ when remembering life events and

162 sses how the neural substrates of successful memory retrieval differed as a function of the targeted

163 s-context preexposure or interference during memory retrieval-differentially affected trace dominance

164 cating that dHipp beta-AR blockade induces a memory retrieval disruption.

165 f age, individuals who were able to suppress memory retrieval exhibited tighter coupling between key

166 lapse prevention characteristics than the CS memory retrieval-extinction procedure and could be a pro

167 ly reported that a conditioned stimulus (CS) memory retrieval-extinction procedure decreases reinstat

168 The UCS memory retrieval-extinction procedure has superior relap

169 Unlike the CS-based memory retrieval-extinction procedure, the UCS memory re

170 y assigned to receive either smoking-related memory retrieval followed by extinction training (the R-

171 These findings suggest that memory retrieval followed by new learning does not relia

172 two subregions differentially contribute to memory retrieval for an association between temporally d

173 awake replay in learning from reward and in memory retrieval for decision making.

174 h was evidenced through multiple measures of memory retrieval function.

175 enial cortex (RSC) in human autobiographical memory retrieval has been confirmed by functional brain

176 namic processes requiring protein synthesis, memory retrieval has long been considered a passive read

177 Theoretical models of memory retrieval have focused on processes of recollecti

178 g episodic memory formation; during episodic memory retrieval, however, hippocampal "slow" gamma (40

179 nhibitory peptide, ZIP) and those induced by memory retrieval (i.e., reconsolidation).

180 nction training following brief cue-elicited memory retrieval (ie, retrieval-extinction [R-E] trainin

181 h it is known that glucocorticoid effects on memory retrieval impairment depend on rapid interactions

182 Here we show that practising memory retrieval improves long-term retention in a nonhu

183 es have shown a negative correlation between memory retrieval in alpha and beta power, and a positive

184 ent of Context technique appears to scaffold memory retrieval in an age-appropriate manner during a p

185 pocampus and examined the regulation of fear memory retrieval in male and female mice.

186 s while stimulating AC-CA projections during memory retrieval in mice behaving in virtual-reality env

187 receptor antagonist pyrilamine disrupted IA memory retrieval in rats, thus strongly supporting an ac

188 al cortex have been linked with context fear memory retrieval in rodents, but the mechanisms by which

189 ality-independent network supporting spatial memory retrieval in the human brain.

190 d association cortex may underlie successful memory retrieval in the human brain.

191 tigating the molecular mechanisms underlying memory retrieval in the rat.

192 ich reduced performance without obliterating memory retrieval in the water maze, produces an as large

193 e mechanisms can be generalized to selective memory retrieval in which competing memories interfere w

194 circuit activity and context discrimination memory retrieval, in a within-subjects design, of male r

195 aged across trials, in regions implicated in memory retrieval, including the hippocampus and angular

196 perception, auditory perception, and visual memory retrieval, indicating that this phenomenon transc

197 s by which memories are maintained following memory retrieval-induced destabilization.

198 Consideration of how memory retrieval influences the addiction process sugges

199 , converging evidence suggests that episodic memory retrieval involves the reinstatement of neural ac

200 own that vmPFC drives the hippocampus during memory retrieval, irrespective of how old the recalled m

201 sible model of human memory, suggesting that memory retrieval is a hierarchical, multi-layered proces

202 These results suggest that memory retrieval is associated with uncertain cue proces

203 Episodic memory retrieval is assumed to rely on the rapid reactiv

204 hippocampus.SIGNIFICANCE STATEMENT Episodic memory retrieval is characterized by a dialog between hi

205 ]; however, it remains elusive whether human memory retrieval is driven by attractor dynamics and wha

206 ongoing protein synthesis is required before memory retrieval is engaged, and suggest that this prote

207 Episodic memory retrieval is increasingly influenced by schematic

208 Thus, cocaine-associated memory retrieval is mediated by beta-AR activity within

209 Episodic memory retrieval is reliant upon cognitive control syste

210 nt behavioural paths, at specific times when memory retrieval is required, and in a manner that could

211 ur findings reveal that post-extinction fear memory retrieval is supported by local and interregional

212 Detailed memory retrieval is thought to involve the reinstatement

213 Episodic memory retrieval is thought to rely on the replay of pas

214 previous studies have suggested that verbal memory retrieval leads to the reinstatement of activity

215 s could not be attributed to changes in fear memory retrieval, learned safety, or memory interference

216 e possibility is that neural activity during memory retrieval, like replay of spiking neurons in the

217 mory retrieval-extinction procedure, the UCS memory retrieval manipulation decreases renewal and rein

218 esults suggest that the effects of stress on memory retrieval may be contingent on the strength of th

219 However, previous findings suggest that memory retrieval may be more dynamic than previously tho

220 We show here that memory retrieval may benefit from this non-specific plas

221 These findings reveal a sparsely implemented memory retrieval mechanism in the hippocampus that opera

222 This study demonstrates that repeated memory retrieval might strengthen memory by inducing mor

223 ment effects within a posterior midline core memory retrieval network during all phases of the experi

224 ecently-experienced events throughout a core memory retrieval network.

225 AIY memory retrieval neurons sense tyramine via the SER-2 re

226 es, with the most robust reactivation during memory retrieval occurring in mid-CA3 (CA3b), the region

227 fects on noradrenergic activity in impairing memory retrieval of emotionally arousing experiences.

228 show the greatest developmental changes for memory retrieval of scenes.

229 role in the selective regulation of spatial memory retrieval of stressful experience, shedding light

230 Memory retrieval, often termed reconsolidation, can rend

231 ation prevented stress-induced impairment of memory retrieval on both the object-recognition and the

232 in regions confirmed that effects of cocaine memory retrieval on proteasome activity, relative to sal

233 ce is particularly important during episodic memory retrieval or when remembering specific events in

234 rontal cortex, plays a critical role in fear memory retrieval over time.

235 th a 7% increase in correct responses during memory retrieval (P = .01).

236 o that commonly found in the classic old/new memory retrieval paradigms, suggesting that the recognit

237 ments in the cognitive neuroscience of human memory retrieval, pinpointing the neural chronometry und

238 ons, such as introspection, autobiographical memory retrieval, planning the future, and predicting so

239 The cortical reinstatement hypothesis of memory retrieval posits that content-specific cortical a

240 thalamus, whereas metacognitive ability for memory retrieval predicted greater connectivity between

241 activation during episodic autobiographical memory retrieval predicted the degree of suppression dur

242 he role of attribution mechanisms in shaping memory retrieval, prior work examining implicit memory s

243 in these cells critically contribute to the memory retrieval process.

244 ck, which may echo an increased influence of memory retrieval processes in CA3 on firing in CA1.

245 Prospective coding is believed to reflect memory retrieval processes, whereas retrospective coding

246 induced by immutable bottlenecks in people's memory retrieval processes.

247 t medial temporal lobe-dependent declarative memory retrieval processes.SIGNIFICANCE STATEMENT Interi

248 rk interactions as being central to episodic memory retrieval, providing insight into how multiple co

249 formed auditory Pavlovian fear memory; fear memory retrieval (reactivation) and postreactivation (PR

250 val.SIGNIFICANCE STATEMENT We show that cued memory retrieval reinstates neural activity on a faster

251 regation-is markedly reduced during episodic memory retrieval relative to closely matched analogical

252 reduced when individuals engage in episodic memory retrieval, relative to other cognitive tasks, and

253 Episodic memory retrieval relies on the recovery of neural repres

254 noninvasive, content-specific indicators of memory retrieval remains a central challenge.

255 tional significance of LPC activation during memory retrieval remains a subject of active debate.

256 How these cells participate in memory retrieval remains unclear.

257 the contribution of striatum to declarative memory retrieval remains unknown.

258 ether the addition of new information during memory retrieval required GluR2-endocytosis to modify th

259 rapid succession, to ensure that successful memory retrieval required them to disambiguate multiple

260 Similar to aging patterns in memory retrieval, results also showed that older adults

261 Intentionally suppressing memory retrieval (retrieval stopping) reduces hippocampa

262 Choices requiring memory retrieval selectively engaged phase-locking of MF

263 It is known that ZIP sensitivity and memory retrieval share at least some molecular targets (

264 ically and temporally with histamine-induced memory retrieval, showing the active involvement of hist

265 sion of valence-specific behavior upon taste memory retrieval.SIGNIFICANCE STATEMENT In the present s

266 for the role of oscillatory reinstatement in memory retrieval.SIGNIFICANCE STATEMENT Recent neurobiol

267 nt of cortical activity is a feature of cued memory retrieval.SIGNIFICANCE STATEMENT We show that cue

268 indow after either fear conditioning or fear memory retrieval significantly impairs the consolidation

269 based on general cognitive processes such as memory retrieval, similarity-based partial matching, and

270 ping of action and the stopping of long-term memory retrieval (stopping thoughts), where increased pr

271 l to dissociate three components of episodic memory: retrieval success, precision, and vividness.

272 CRF(+) neuron inhibition impairs extinction memory retrieval, supporting the notion that CRF(+) neur

273 Participants, ages 19-85, took part in a memory retrieval task with a protracted retrieval trial

274 ivity and poorer performance on a subsequent memory retrieval task, strongly implicating sex steroids

275 ajor deficits in a strategy shifting/spatial memory retrieval task.

276 g, then 28 days later, they underwent a fear memory retrieval test.

277 nd that male mice exhibit stronger long-term memory retrieval than do female mice, and this finding w

278 for neurally inspired cognitive modeling of memory retrieval that has no biologically unconstrained

279 nd show that this substrate predicts working memory retrieval times on a trial-by-trial basis.

280 Animals need to optimize the efficacy of memory retrieval to adapt to environmental circumstances

281 Together, these findings link memory retrieval to stabilization and provide a powerful

282 e relevance of cognitive processes governing memory retrieval to substance use disorder.

283 or anisomycin) in the amygdala 10 min before memory retrieval transiently impaired auditory fear memo

284 ssed by using a reinforced or non-reinforced memory retrieval trial before extinction, compared to a

285 increases in connectivity during successful memory retrieval typified network topology, with individ

286 nses in which the participant's awareness of memory retrieval was absent.

287 l activity during the period 1-hour prior to memory retrieval was associated with superior memory per

288 Successful memory retrieval was characterized by greater global con

289 In initial experiments, memory retrieval was elicited via a cue-induced seeking/

290 agged CA1 cells were silenced, we found that memory retrieval was impaired and representations in the

291 ished event-related potential (ERP) index of memory retrieval was present for both trials when the st

292 heart rate and skin conductance level during memory retrieval were measured before, directly after, a

293 re, two levels of functional organization of memory retrieval were shown.

294 e, but effectively suppressed aversive taste memory retrieval when applied either during or before th

295 Off-line processing also occurs after memory retrieval when memories are destabilized and then

296 ipants generally had difficultly suppressing memory retrieval when negative stimuli were presented.

297 tween these two alternative outcomes of fear memory retrieval, when neither process is engaged.

298 bilization, since AMPAR antagonism prevented memory retrieval while still allowing the destabilizatio

299 erm memory and reconcile current theories of memory retrieval with classic notions about the memory m

300 between these MTL regions during successful memory retrieval, with reversible signal flow from the c