ライフサイエンスコーパス: memory trace

1 in) storing and recalling memories (i.e., a memory trace).

2 rfering with reconsolidation of the original memory trace.

3 proposed to act as a long-lasting molecular memory trace.

4 tioning, a short-lived and branch-restricted memory trace.

5 nsistent with an expression of a stable fear memory trace.

6 hippocampus, experiences disappear without a memory trace.

7 between mossy fibers and DCN neurons in this memory trace.

8 , probably reflecting a fast decaying iconic memory trace.

9 es that are thought to form the basis of the memory trace.

10 C function, depending on the strength of the memory trace.

11 ) neurons and so possess the properties of a memory trace.

12 to many genes comprising the transcriptional memory trace.

13 sistent with retrieval of an activity-silent memory trace.

14 e stimulated for therapy to remove traumatic memory trace.

15 or incorporation of new information into the memory trace.

16 are organized to constitute a corresponding memory trace.

17 as critical components of an olfactory fear memory trace.

18 memory reactivation, on strengthening of the memory trace.

19 to indicate disruption of an existing motor memory trace.

20 destabilization and modification of the fear memory trace.

21 porating new contextual information into the memory trace.

22 t to explain in terms of access to a unitary memory trace.

23 hways to localize definitively the essential memory trace.

24 oral weighted voltage trace, also called the memory trace.

25 al mechanism for the restructuring of neural memory traces.

26 thereby improving the signal:noise ratio of memory traces.

27 t is recruited first into dopamine-dependent memory traces.

28 ather than suppression of no-longer-relevant memory traces.

29 ing attributional biases from the underlying memory traces.

30 ng in REM sleep rather than linking episodic memory traces.

31 ed on the temporal lobe that builds specific memory traces.

32 l amygdala (LA) neurons are assigned to fear memory traces.

33 ing to degradation and forgetting of the old memory traces.

34 etting arises due to inhibition of competing memory traces.

35 the formation and expression of new spatial memory traces.

36 s, indexing the ignition of their underlying memory traces.

37 rontal cortex (PFC), to "stamp in" posttrial memory traces.

38 emporal persistence of transient hippocampal memory traces.

39 critical for the establishment of permanent memory traces.

40 ecific synaptic modifications to consolidate memory traces.

41 retain odors by extending the decay of weak memory traces.

42 formation and retrieval of content-specific memory traces.

43 ortex and the stabilization of cortical fear memory traces.

44 ociated semantic and weakly-encoded episodic memory traces.

45 ciated with stability and incorporation into memory traces.

46 ning by replaying recent and old conflicting memory traces.

47 resentation, as well as in the evaluation of memory traces.

48 ion to slowly degrade molecular and cellular memory traces.

49 stabilization of hippocampal and neocortical memory traces.

50 hippocampal-association cortical transfer of memory traces.

51 means of "updating" or "rewriting" existing memory traces.

52 for the hypothesized transfer of hippocampal memory traces.

53 s of reconsolidation which leads to modified memory traces.

54 t well understood how spindles modify neural memory traces.

55 he environment to determine when to form new memory traces.

56 the molecular components of the long-lasting memory trace?

57 ity and showed that early termination of the memory trace abolished the memory.

58 l improvement was reflected in dentate gyrus memory trace activity.

59 e periodic clearance of outdated hippocampal memory traces after cortical memory consolidation, there

60 ghting of the current interval with previous memory traces after the interval has been perceived.

61 ism for consolidation is the reactivation of memory traces after their initial encoding during subseq

62 and is behaviorally relevant for stabilizing memory traces against interference.

63 d activation of amygdala neurons bearing the memory trace and increased the synaptic exchange from Ca

64 he physiological manifestation of a specific memory trace and is characterized by dynamic changes in

65 the sensory representation of the competitor memory trace and predicted the amount of retrieval-induc

66 chanism that encodes an effector-independent memory trace and uncover a central role for the PPC in i

67 ocampus could coordinate the reactivation of memory traces and direct their reinstatement in cortical

68 inks between intact identity-specific visual memory traces and later semantic face processing stages.

69 lter inhibiting the development of imprecise memory traces and reducing the false memory rate.

70 sing finding that DPMs contain odor-specific memory traces and send integrated information about the

71 estions about the fundamental limits of such memory traces and the properties required of dynamical s

72 Memory traces are believed to be ensembles of cells used

73 o the cerebellum), and putative higher-order memory traces are characterized in the hippocampus.

74 widely assumed to be the mechanism by which memory traces are encoded and stored in the central nerv

75 Mounting evidence suggests neural memory traces are formed by auditory learning in utero.

76 5) solved the long-standing puzzle of where memory traces are formed in the brain when the CS is ele

77 According to two-stage accounts, memory traces are gradually translocated from hippocampu

78 regions of these subtypes, we conclude that memory traces are guided to target regions of the activa

79 functional account in which forward-looking memory traces are informationally and computationally tu

80 during the encoding phase, and whether such memory traces are less affected by an intervening maskin

81 These studies suggest that distinct memory traces are located in the DG and in CA3 but that

82 These findings also suggest that fear memory traces are partially erased after extinction.

83 ultineuronal activity suggestive of episodic memory traces are reactivated during REM sleep.

84 Newly acquired memory traces are spontaneously reactivated during slow-

85 authors point to findings that suggest that memory traces are susceptible to modification.

86 Over time, memory traces are thought to undergo a neural reorganiza

87 During these fast oscillations, memory traces are transferred from the hippocampus to th

88 e therefore uncovered a mechanism by which a memory trace at one synapse could alter signal handling

89 ior to slice preparation, provides a lasting memory trace at synapses.

90 xperiences are represented by collections of memory traces at the cellular level.

91 role in trace conditioning is to maintain a memory trace between the offset of the CS+ and the delay

92 memory, but rather led to disruption of the memory trace, breaking down the link between memory reac

93 ting that masking did not erase or overwrite memory traces but limited perception.

94 ential to update the reactivated recognition memory trace, but not to consolidate or maintain an inac

95 ty are more likely to be integrated into the memory trace, but that competitive synaptic interactions

96 Notably, extending the memory trace by transiently disinhibiting taste cortical

97 ity that occurs during the creation of a new memory trace can be observed using functional magnetic r

98 Our model revealed that an activity-silent memory trace can be realized by facilitation of inter-ar

99 ade, a large body of research has shown that memory traces can become labile upon retrieval and must

100 Memory-trace cells in the entorhinal cortex, in particul

101 We identified 'memory-trace cells' with activity that was spatially tun

102 , pathway tracing) to identify the essential memory trace circuit for a given form of learning and me

103 e responses as a model system, the essential memory trace circuit is identified, the basic memory tra

104 p participates in the consolidation of fresh memory traces come from a wide range of experimental obs

105 ng stimulation cues associated with specific memory trace could selectively augment replay and enhanc

106 Such memory traces could be stabilised from short- to long-te

107 However, a model that elucidates how these memory traces could emerge through spike-timing-dependen

108 such SWR-directed reactivation of brain-wide memory traces could underlie memory consolidation.

109 nt synaptic reinforcement of the hippocampal memory traces created during initial learning.

110 namics, it remains largely unknown how those memory traces decay in different contexts and whether an

111 hat maintenance of subsequent reconsolidated memory trace depends on CaMKII, and these results also s

112 ation of synaptic circuits to retain salient memory traces despite the noise of daily experience.

113 A cellular memory trace, detected as increased calcium influx into

114 However, the mechanisms by which motor memory traces develop during sleep remain controversial

115 el experiences, suggesting that a persistent memory trace develops with experience.

116 We suggest that reactivation of memory traces distributed across modality-specific brain

117 ngth of neural reinstatement of the original memory trace during reactivation, driven by the hippocam

118 attributed to an alteration of the original memory trace during reactivation-dependent reconsolidati

119 t SWS, suggesting a lack of strengthening of memory traces during REM sleep, at least in the case of

120 rack the reactivation of lateralized sensory memory traces during retrieval.

121 ggers the reactivation and reorganization of memory traces during sleep, a systems-level process that

122 proposed to be involved in the processing of memory traces during sleep.

123 rgeted memory reactivation (TMR) of specific memory traces during slow-wave sleep promotes the emerge

124 nce learning to investigate how newly-formed memory traces evolve dynamically over time.

125 een for genes comprising the transcriptional memory trace, finding 16 positive hits whose disruption

126 The cellular memory traces first appear at 30 min after conditioning

127 Hippocampal memory traces followed by novelty were more dominant by

128 we have succeeded in localizing an essential memory trace for a basic form of associative learning an

129 fantile amnesia period is stored as a latent memory trace for a long time; indeed, a later reminder r

130 players form a more robust visual short-term memory trace for coherent moving stimuli during the enco

131 OFC neuronal ensembles store a memory trace for newly learned information, which appear

132 dimensional dynamical systems could retain a memory trace for past inputs in their current state.

133 ptive field plasticity, and could serve as a memory trace for stimuli or episodes that have acquired

134 t produced initial retention impairment, the memory trace for the aversive event was reactivated (i.e

135 ould result in a long-lasting and meaningful memory trace for the event but, at the same time, make i

136 ausal processes, and to create and reinforce memory traces for better recall and application over tim

137 An important feature of our task is that memory traces for contextual information were not access

138 elease may interfere with the laying down of memory traces for incidents of childhood abuse.

139 ial information, the creation and storage of memory traces for spatial information, and the use of sp

140 rtex engages in the formation and storage of memory traces for spatial information.

141 We predict that memory traces for various stimuli may "merge," such that

142 optical imaging have revealed that cellular memory traces form in different areas of the insect brai

143 These memory traces form in only one of the two branches of th

144 Large spines are stable and important for memory trace formation.

145 pattern we have found may play a key role in memory trace formation.

146 elationship between chromatin plasticity and memory trace formation.

147 Notably, a memory trace formed in the APL neuron by pairing odor wi

148 on inverted, consistent with transfer of the memory trace from one hemisphere to the other.

149 ethylation promoted the transfer of the fear memory trace from the hippocampus to the cortex and the

150 an instructive role in the communication of memory traces from the hippocampus to the cerebral corte

151 ear memories by facilitating the transfer of memory traces from the hippocampus to the cortex and cor

152 ult dentate gyrus may serve to clear out old memory traces from the hippocampus, thus leaving the hip

153 ern classification, resolves why an enduring memory trace has proven elusive in previous human studie

154 e cellular architecture supporting long-term memory traces has also substantially improved.

155 ion reservoir computers with reasonably long memory traces have an error probability that is at least

156 nsistent with the idea that these particular memory traces have strengthened with time, and therefore

157 brain substrates of memory is the nature of memory traces, how memories are formed, stored, and retr

158 ing memory trace, reward creates a competing memory trace, impairing expression of the original rewar

159 mation and strengthening of neural long-term memory traces, improving discrimination skills, in parti

160 nd maintain synaptic specificity of a labile memory trace in a recurrent DPM and MB alpha'beta' neuro

161 ildren, indicating qualitative difference in memory trace in children.

162 e, whereas the capacity to form a short-term memory trace in the alpha'/beta' mushroom body neurons r

163 ifying to their relevance for establishing a memory trace in the PL.

164 r, only rewarded extinction created a stable memory trace in the vmPFC, identified through overlappin

165 pendent behavioral changes to the underlying memory trace in this marine mollusk.

166 A new study of memory traces in an invertebrate challenges convention i

167 tic events required for the consolidation of memory traces in cortical networks.

168 and on consistent processing of associative memory traces in midline structures that are involved in

169 esized to allow for the transient storage of memory traces in neuronal networks.

170 excessive DA may prevent storage of lasting memory traces in PFC networks and impair executive funct

171 Together with few episodic memory traces in REM sleep, and REM sleep deprivation af

172 n, a process that enables updating of stored memory traces in response to novelty.

173 ent findings on the cellular architecture of memory traces in rodents and how the application of new

174 r generalization and alter the corresponding memory traces in TBI mice.

175 ression (LTD) underlie at least a portion of memory traces in the brain, but the exact cellular locus

176 f fear and extinction are stored as distinct memory traces in the brain.

177 imply simultaneous reactivation of coherent memory traces in the cortex and hippocampus during sleep

178 However, early memory traces in the MB remain elusive.

179 hat avoidance training produces two opposing memory traces in these regions.

180 from our distant past, despite evidence that memory traces in this region vanish over time.

181 ly recognition ('retrieval') and matching of memory traces in working memory.

182 cal response reliability, but also leaves a 'memory trace' in subsequent spontaneous activity.

183 night sleep correlate with changes in neural memory traces, including enhanced functional connectivit

184 he persistence and breadth of the DPM neuron memory trace influences the duration of behavioral memor

185 ry consolidation transforms initially labile memory traces into more stable representations.

186 incremental integration of recently acquired memory traces into neocortical schemas through the inter

187 y consolidation-the transformation of labile memory traces into stable long-term representations-is f

188 , it remains unknown whether a given sensory memory trace is being maintained as a unitary item to as

189 This time window for the gamma neuron memory trace is displaced relative to the modifications

190 gh classical associative learning, but which memory trace is eligible for modification depends on a s

191 ing spatial learning an experience-dependent memory trace is formed in this structure.

192 emory trace circuit is identified, the basic memory trace is localized (to the cerebellum), and putat

193 in interacting with pre-existing reactivated memory traces is critical for successful modification of

194 However, such reinstatement of memory traces is not trivial because it goes against the

195 ional imaging study has revealed a long-term memory trace manifested as an increase in the Ca(2+) act

196 tute memory recall, whereas consolidation of memory traces may be revealed and served by correlated f

197 The transient memory traces may support behavior across the time windo

198 gets suggest that the persistent form of the memory trace might be comprised of molecules that mainta

199 Forming memory traces might be how the brain is able to provide

200 rning model, whereas a simple fragment-based memory-trace model that counts occurrence summary statis

201 cation, according to which modification of a memory trace occurs through classical associative learni

202 ntion can reach into the past, acting on the memory trace of a stimulus that has disappeared before b

203 generations to establish a transgenerational memory trace of ancestral environments and distinguish s

204 uding the striatum, which shapes an abnormal memory trace of drug consumption that virtually highjack

205 short-term threat relapse and stabilize the memory trace of extinction in the ventromedial prefronta

206 at CaMKIIalpha accumulation at synapses is a memory trace of past synaptic activity.

207 y, we show that the dFB appears to contain a memory trace of prior exposure to metabolic challenges i

208 hypothesis is that the hippocampus stores a memory trace of the conditioned stimulus (CS) during the

209 Our analyses show that the intrinsic memory trace of the fractional derivative provides a neg

210 This occurs due to the memory trace of the fractional-order dynamics.

211 join neural networks, and may constitute the memory trace of the imprinted stimulus.

212 ments gives rise to a kinematically specific memory trace of the observed motions in M1.

213 layer 5 pyramidal tract neurons contained a memory trace of the previous trial's outcome.

214 arge offset transient response may reflect a memory trace of the stimulus when it is no longer visibl

215 depends on the synaptic connectivity and the memory trace of the system.

216 biological systems must retain a short-term memory trace of their recent inputs.

217 and demonstrate that there are two parallel memory traces of a novel taste: a short-duration robust

218 ndent (autonomous) activity, thereby leaving memory traces of calcium ion stimuli beyond their durati

219 t to reflect the activation of stored visual memory traces of known individual faces.

220 or if this phenomenon generalizes to working memory traces of other visual features.

221 thus maintains concurrent stimulus-specific memory traces of past input, enabling the visual system

222 Individuals' memory traces of post-event information in the hippocamp

223 on a mechanism of aversive learning based on memory traces of recently encountered stimuli, reflectin

224 quiring, storing, and retrieving associative memory traces of repeatedly co-occurring neural events i

225 In the dark, network gain maintained a 'memory trace' of the previously displayed landmark.

226 This Bayesian analysis revealed the 'memory traces' of the chemical network.

227 s that retrieval can destabilize an existing memory trace, opening a time-dependent window during whi

228 to processes involved in the formation of a memory trace or, more probably, involves both trace form

229 y of a cocaine-cue association (the "cocaine memory trace" or "cocaine engram").

230 anges in neuronal physiology that encode the memory trace, or engram.

231 for initial encoding, for the expression of memory traces, or for both encoding and expression.

232 'spontaneously' in CA3 and propagate recent memory traces outward to the neocortex to facilitate mem

233 low but continuous accumulation of long-term memory traces over repetitions [e.g., Page & Norris, Phi

234 then, we find no anatomical substrate for a memory trace persisting from larva to adult.

235 on in the study of long-term memory is how a memory trace persists for years when the proteins that i

236 spindles support the network distribution of memory traces, potentially restructuring memory represen

237 honological encoding and/or by strengthening memory traces rather than by fundamentally subserving co

238 Although memory trace reactivation is correlated with low-voltage

239 To investigate the types of memory traces recovered by the medial temporal lobe (MTL

240 the influence of sleep discharge patterns on memory traces remains fragmentary.

241 During rest and sleep, memory traces replay in the brain.

242 Because detection and quantification of memory-trace replay depends critically on analysis metho

243 mpal memory space, with consolidation of the memory traces representing repeated paired associates in

244 amic compensatory response only when initial memory traces required consolidation.

245 t that, rather than reinforcing the existing memory trace, reward creates a competing memory trace, i

246 rmediate SOAs due to the decay of the iconic memory trace, stabilizing at a low asymptote at long SOA

247 es of amnesia result from eradication of the memory trace (storage impairment) or if the trace is pre

248 but not to maintain the inactive recognition memory trace stored over time, in adult male Wistar rats

249 ing that the mPFC controls the expression of memory traces stored in the hippocampus biasing retrieva

250 that both factors modulate a unidimensional memory trace strength.

251 y when there is competition between opposing memory traces, such as that which occurs during the acqu

252 ctive forgetting also involves modulation of memory trace synaptic strength by altering AMPA receptor

253 me-target pairs, a subliminal pair leaves no memory trace that can be observed in response to the nex

254 A1-dependent plasticity appears to leave a memory trace that can be retrieved, facilitating adaptat

255 m of plasticity leaves a hidden postsynaptic memory trace that enables fast relearning of previously

256 alog of the spinal stretch reflex, creates a memory trace that includes changes in the spinal cord.

257 e activity of the Purkinje cell allows for a memory trace that is resistant to ongoing activity in th

258 TMS seems to have specifically affected the memory trace that leads to serial dependence, as we foun

259 yed, branch-specific, and amnesiac-dependent memory trace that may guide behavior after acquisition.

260 , but only some episodes will leave detailed memory traces that can be recollected after weeks and mo

261 formation of distinct yet flexible emotional memory traces that confer an ability to recall extinctio

262 Here we contrast the cellular memory traces that form in the dorsal paired medial (DPM

263 imaging of living flies have identified six memory traces that form in the olfactory nervous system

264 ly, to the modification of retrieval-related memory traces that impact future remembering.

265 o represent more integrated, or overlapping, memory traces that prioritize commonalities across relat

266 experience and embed critical cue-associated memory traces that promote cocaine relapse.

267 lular sites where cocaine experience creates memory traces that subsequently promote cocaine craving

268 ts on the consolidation of retrieval-related memory traces that support future remembering.

269 aging to monitor two different calcium-based memory traces that underlie olfactory classical conditio

270 ared that "consciousness arises instead of a memory-trace." The aim of reconsolidation, and of psycho

271 in the progressive strengthening of cortical memory traces through reactivation of cortical NMDA rece

272 zation; and second, by localizing a critical memory trace to neurons located outside the behavioral c

273 ippocampus has enabled real-time patterns of memory traces to be mathematically described, directly v

274 their ability to form and utilize transient memory traces to guide behavior.

275 use the integrated floral traits from their memory traces to mediate future foraging decisions.

276 ew memory traces with reconsolidation of old memory traces to minimize interference.

277 literature on the potential vulnerability of memory traces to modification and on the effects of stre

278 deep sleep can underlie mapping hippocampal memory traces to persistent cortical representation.

279 ncies, with a directionality consistent with memory trace transfer.

280 lying the characteristic labilization of the memory trace triggered by retrieval.

281 These findings suggest that a memory trace undergoes rapid modifications, and that the

282 Our results demonstrate that the memory traces underlying cortical deviance detection for

283 Thus, the memory traces underlying cortical deviance detection may

284 as administered 24 h after training, when IA memory trace was already formed.

285 This memory trace was defective in all 26 of the LTM mutants.

286 Rather, the potentials appeared whenever a memory trace was observed behaviorally.

287 To visualize memory traces, we created a transgenic line that allows

288 Fear memory traces were affected in the dorsal dentate gyrus

289 he alpha/beta and the gamma neuron long-term memory traces were both blocked by expressing a represso

290 contextual fear discrimination paradigm and memory traces were quantified in numerous brain regions.

291 that stimulation preferentially strengthens memory traces when delivered at a specific phase of the

292 and may act by weakening previously encoded memory traces when new information is learned.

293 anges reflected the context attribute of the memory trace, which has been envisioned as an essential

294 leads to both associative and nonassociative memory traces, which can be preferentially accessed by e

295 ed reactivation of distributed components of memory traces while the cortex is "offline," i.e., not e

296 we tested the impact of reinforcing a skill memory trace with monetary reward following memory react

297 tions reconcile classical theories of stable memory traces with neural drift.

298 l learning by combining consolidation of new memory traces with reconsolidation of old memory traces

299 a nonuniform pattern of reactivation of fear memory traces, with the most robust reactivation during

300 ble inactivation can be used to localize the memory traces within this circuit.

301 it interactions between short- and long-term memory traces, yielding predictions that our experiments