ライフサイエンスコーパス: meningitidis

1 causing bacteria, Neisseria meningitidis (N. meningitidis).

2 or the lack of -35 consensus sequences in N. meningitidis.

3 s (phasevarions), have been identified in N. meningitidis.

4 inhibit the CRISPR-Cas9 system of Neisseria meningitidis.

5 loyment and assessment of vaccines against N meningitidis.

6 lactamica prevents colonization by Neisseria meningitidis.

7 a, Mycobacterium tuberculosis, and Neisseria meningitidis.

8 amic transmission model of group A Neisseria meningitidis.

9 hracis, Neisseria gonorrhoeae, and Neisseria meningitidis.

10 ations frequently fail to identify Neisseria meningitidis.

11 l disease caused by infection with Neisseria meningitidis.

12 explain adolescent/adult colonization by N. meningitidis.

13 ctedly versatile Cas9 protein from Neisseria meningitidis.

14 ance, and virulence in the human pathogen N. meningitidis.

15 mary human meningothelial cells to Neisseria meningitidis.

16 pP from the Gram-negative pathogen Neisseria meningitidis.

17 ence factor and vaccine antigen of Neisseria meningitidis.

18 first observation of heteroresistance in N. meningitidis.

19 PorB from the pathogenic bacterium Neisseria meningitidis.

20 gens including Vibrio cholerae and Neisseria meningitidis.

21 s with Streptococcus pneumoniae or Neisseria meningitidis.

22 tive as conventional methods in detecting N. meningitidis (13.2% versus 5.7%; P < 0.0001).

23 Overall, S. pneumoniae (53.4%), Neisseria meningitidis (13.7%), and Haemophilus influenzae (12.3%)

24 86) of bacterial meningitis cases: Neisseria meningitidis (1350 cases, 22%), Streptococcus pneumoniae

25 hilus influenzae (18 [14.3%]), and Neisseria meningitidis (15 [11.9%]).

26 regulation mechanism observed for Neisseria meningitidis 3-deoxy-d-arabino-heptulosonate 7-phosphate

27 umoniae (84% of positive CSF isolates) and N.meningitidis (4%).

28 firmed in 273 patients: 48% (131/273) was N. meningitidis, 45% (123/273) S. pneumoniae, and 7% (19/27

29 Neisseria meningitidis A epidemics have been eliminated here; howe

30 N. meningitidis abundance was negatively correlated with un

31 N. meningitidis abundance was positively correlated with th

32 H. influenzae and N. meningitidis accounted for 6.8% (5 of 73) and 21.9% (16

33 mbilical vein endothelial cells or Neisseria meningitidis after incubation with human serum was compl

34 obactam, cefepime, and gentamicin, Neisseria meningitidis and ceftriaxone, and Haemophilus influenzae

35 shared function of fHbp in N. cinerea and N. meningitidis and cross-reactive responses elicited by Be

36 resulted in loss of functional traits in N. meningitidis and E. coli Our study indicates that the ex

37 ny gram-negative pathogens such as Neisseria meningitidis and Escherichia coli express acidic capsule

38 arriage prevalence and acquisition of all N. meningitidis and individual disease-causing genogroups.

39 and compares it to studies done on Neisseria meningitidis and Moraxella catarrhalis; the two other or

40 cal isolate described here expresses both N. meningitidis and N. gonorrhoeae 16S rRNA genes, as shown

41 Comparison of PG fragment release from N. meningitidis and N. gonorrhoeae showed that meningococci

42 eisseria contains two pathogenic species (N. meningitidis and N. gonorrhoeae) in addition to a number

43 ons that allow for discrimination between N. meningitidis and N. gonorrhoeae.

44 against the Gram-negative species Neisseria meningitidis and Neisseria gonorrheae and improved activ

45 NrrF, a trans-acting sRNA in Neisseria meningitidis and Neisseria gonorrhoeae, has been shown i

46 while the two pathogenic species, Neisseria meningitidis and Neisseria gonorrhoeae, straddle the bor

47 nts is due largely to interaction between N. meningitidis and other members of the upper respiratory

48 Neisseria meningitidis and S. pneumoniae remain important causes o

49 thogens, Neisseria gonorrhoeae and Neisseria meningitidis, and at least 13 species of commensals that

50 tly increases detection of S. pneumoniae, N. meningitidis, and H. influenzae in CSF, and that applica

51 ing/grouping of Streptococcus pneumoniae, N. meningitidis, and H. influenzae were done.

52 hat were qPCR positive for S. pneumoniae, N. meningitidis, and H. influenzae, only 10 were culture po

53 against Streptococcus pneumoniae, Neisseria meningitidis, and H. influenzae.

54 Streptococcus pneumoniae, Neisseria meningitidis, and Haemophilus accounted for 66% (76/115)

55 ed Streptococcus pneumoniae (Spn), Neisseria meningitidis, and Haemophilus influenzae meningitis with

56 qPCR for Streptococcus pneumoniae, Neisseria meningitidis, and Haemophilus influenzae.

57 -restricted Neisseria species two, Neisseria meningitidis, and Neisseria gonorrhoeae, cause invasive

58 f conjugate vaccines against H influenzae, N meningitidis, and S pneumoniae in England.

59 ction with Haemophilus influenzae, Neisseria meningitidis, and Streptococcus pneumoniae causes substa

60 Neisseria gonorrhoeae and Neisseria meningitidis are closely related organisms that cause th

61 Neisseria gonorrhoeae and Neisseria meningitidis are closely-related bacteria that cause a s

62 r detecting pharyngeal carriage of Neisseria meningitidis are complex.

63 tal colonization and urethritis caused by N. meningitidis are possible across a range of meningococca

64 Several outer membrane proteins of Neisseria meningitidis are subject to phase variation due to alter

65 nses of human tonsil-derived DC to Neisseria meningitidis as a model organism.

66 umerous bacterial pathogens, using Neisseria meningitidis as a model organism.

67 as a ligand for these molecules on Neisseria meningitidis As with N. meningitidis NspA (Nm-NspA), N.

68 This was particularly relevant for N. meningitidis, as only 1/13 cases was culture positive.

69 tructure of an ASBT homologue from Neisseria meningitidis (ASBT(NM)) in detergent was reported recent

70 substrate-bound ClpXP complex from Neisseria meningitidis at 2.3 to 3.3 angstrom resolution.

71 ation to respond to an outbreak of Neisseria meningitidis B at a U.S. university.

72 cases of meningococcal disease caused by N. meningitidis B were reported among vaccinated students.

73 prevalence of carriage of disease-causing N. meningitidis between the vaccination group (2.55%; 326 o

74 This isolate was identified as N. meningitidis by biochemical identification methods but g

75 ulfment of Neisseria gonorrheae or Neisseria meningitidis by human cells and can offer deep understan

76 ive bacterial meningitis caused by Neisseria meningitidis can be prevented by active immunization wit

77 Neisseria meningitidis can be transmitted via asymptomatic nasopha

78 heat-killed cells of Gram-negative Neisseria meningitidis, capsular serogroup C (MenC) or Gram-positi

79 des comprehensive characterization of the N. meningitidis capsule, which is critical for meningococca

80 At baseline, natural N. meningitidis carriage in the control group was 22.4% (36

81 ures grown overnight doubled the yield of N. meningitidis carriage isolates compared with conventiona

82 tions that are more or less favorable for N. meningitidis carriage.

83 IC3 proteins were found to inhibit Neisseria meningitidis Cas9 (NmeCas9) activity in bacterial and hu

84 show that these proteins bind directly to N. meningitidis Cas9 (NmeCas9) and can be used as potent in

85 Cas9 orthologs (including Neisseria meningitidis Cas9 [NmeCas9]) have also been adopted for

86 We were unable to serogroup the N. meningitidis cases by PCR.

87 NmX increased from 0.6% of N. meningitidis cases in 2015 to 27% in 2017.

88 Of N. meningitidis cases, 53% were NmC, 30% NmW, and 13% NmX.

89 Neisseria meningitidis causes 500 000 cases of septicemia and meni

90 Neisseria meningitidis causes bacterial meningitis and septicemia.

91 The pathogen Neisseria meningitidis causes disease amongst infants and adolesce

92 The bacterium Neisseria meningitidis causes life-threatening meningitis and seps

93 Among 25 serogroup B Neisseria meningitidis clinical isolates, we identified four (16%)

94 several lines of supporting evidence that N. meningitidis colonization is correlated with propionic a

95 entification of at least 90% of the 1,605 N. meningitidis core genome loci for 50% of the specimens.

96 distinct protospacer adjacent motif, the N. meningitidis CRISPR-Cas machinery increases the sequence

97 osphoethanolamine transferase from Neisseria meningitidis, determined to 2.75-A resolution.

98 ards a promising application in the field of meningitidis diagnosis.

99 sed biosensor for the detection of Neisseria meningitidis DNA employing Kretschmann configuration.

100 ly enrich, sequence, and de novo assemble N. meningitidis DNA from clinical specimens with low bacter

101 or exhibits a linear response towards target meningitidis DNA over the concentration range from 10 to

102 the fitness of the human pathogen Neisseria meningitidis during infection.

103 l disease (IMD) due to serogroup Y Neisseria meningitidis emerged in Europe during the 2000s.

104 Neisseria meningitidis encodes up to five TpsA proteins that are s

105 c nature and the high diversity of Neisseria meningitidis, epidemiological surveillance incorporating

106 nts with Streptococcus pneumoniae, Neisseria meningitidis, Escherichia coli, and Pseudomonas aerugino

107 raised against sheaths presenting Neisseria meningitidis factor H binding protein (fHbp) antigen wer

108 ecombinant proteins, including the Neisseria meningitidis factor H binding protein (fHbp), a lipoprot

109 a are at odds with this proposal and that N. meningitidis fits the criteria that we have proposed for

110 keleton linker, were more pronounced when N. meningitidis formed larger colonies on HBMEC under physi

111 to hospital and identification of Neisseria meningitidis from a sterile site.

112 (fHbp), a virulence factor which protects N. meningitidis from innate immunity by binding the human c

113 s for PorA (encoding porin protein A) and N. meningitidis genogroups.

114 ty in the Campylobacter jejuni and Neisseria meningitidis genomes encoded hypothetical proteins.

115 e class III Fic protein NmFic from Neisseria meningitidis gets autoadenylylated in cis, thereby auton

116 itis incidence and carriage due to Neisseria meningitidis group A (MenA).

117 s launched using a newly developed Neisseria meningitidis group A (NmA) polysaccharide-tetanus toxoid

118 ng protein, are essential for survival of N. meningitidis group B strain H44/76 in normal human serum

119 yngeal carriage of disease-causing Neisseria meningitidis (group A, B, C, W, X, or Y) in students in

120 hilus influenzae cases were confirmed and N. meningitidis/H. influenzae were serogrouped/serotyped by

121 athogens such as Escherichia coli, Neisseria meningitidis, Haemophilus influenzae, and Pasteurella mu

122 ingitis, which is caused mainly by Neisseria meningitidis, Haemophilus influenzae, and Streptococcus

123 argeted meningitis cases caused by Neisseria meningitidis, Haemophilus influenzae, and Streptococcus

124 ections (Streptococcus pneumoniae, Neisseria meningitidis, Haemophilus influenzae, S suis) and O tsut

125 Group A Neisseria meningitidis has been a major cause of bacterial meningi

126 N. meningitidis has been considered a paradigmatic case of

127 Neisseria meningitidis has several strategies to evade complement-

128 eria, including the human pathogen Neisseria meningitidis, have evolved means to preferentially take

129 protein (fHbp) is a lipoprotein of Neisseria meningitidis important for the survival of the bacterium

130 ach for fast instrument-free diagnosis of N. meningitidis in resource-limited settings.

131 k so far and shows the continued threat of N meningitidis in sub-Saharan Africa.

132 p genes provides a metabolic advantage to N. meningitidis in the adult oral cavity, which is rich in

133 nses except that made with group C Neisseria meningitidis; in the latter case, only peptides generate

134 N. meningitidis incidence was 1.5 in Burkina Faso, 2.7 in C

135 vel aspects of the methylcitrate cycle in N. meningitidis include a propionate kinase which was purif

136 k factors for carriage of disease-causing N. meningitidis included later year of schooling (adjusted

137 had positive abundance correlations with N. meningitidis, including Aggregatibacter aphrophilus, Cam

138 vity potentiator (rMIP) protein of Neisseria meningitidis induces significant serum bactericidal anti

139 The incidence of Neisseria meningitidis infection decreased from 0.721 per 100 000

140 understanding the epidemiology of Neisseria meningitidis infection.

141 rotection against capsular group B Neisseria meningitidis infections, but the full breadth of the imm

142 ake via GltT-GltM plays multiple roles in N. meningitidis internalization into HBMEC.

143 We previously reported that Neisseria meningitidis internalization into human brain microvasoc

144 Neisseria meningitidis is a commensal microbe that colonizes the h

145 Neisseria meningitidis is a commensal of humans that can colonize

146 Neisseria meningitidis is a frequent colonizer of the human nasoph

147 Neisseria meningitidis is a human commensal that can also cause li

148 Neisseria meningitidis is a human pathogen causing bacterial menin

149 Neisseria meningitidis is a human-specific bacterium that varies i

150 Neisseria meningitidis is a human-specific pathogen and leading ca

151 Neisseria meningitidis is a leading cause of bacterial meningitis

152 GNA2091 of Neisseria meningitidis is a lipoprotein of unknown function that i

153 Neisseria meningitidis is a major cause of bacterial meningitis an

154 Neisseria meningitidis is a major cause of bacterial meningitis wo

155 The capsular polysaccharide surrounding N. meningitidis is a major virulence factor.

156 Neisseria meningitidis is a strict human pathogen that closely int

157 The limit of detection of N. meningitidis is about 3 copies per LAMP zone within 45 m

158 Neisseria meningitidis is an encapsulated pathogen, and antibodies

159 Neisseria meningitidis is an important cause of invasive bacterial

160 Neisseria meningitidis is an important human pathogen that is capa

161 Neisseria meningitidis is an obligate human commensal bacterium th

162 lthough the opportunistic pathogen Neisseria meningitidis is enriched for colonization in the throat,

163 Although Neisseria meningitidis is naturally competent and porB genetic mos

164 Neisseria meningitidis is one of the few commensal bacteria that c

165 re commonly found in dental plaque, while N. meningitidis is primarily found in the pharynx, suggesti

166 An apparently rare Neisseria meningitidis isolate containing one copy of a Neisseria

167 The N. meningitidis isolate described must have obtained N. gon

168 Probe Aptima assays cross-react with this N. meningitidis isolate.

169 re, we analyzed the genomes of 39 diverse N. meningitidis isolates associated with urethritis, collec

170 Almost all invasive Neisseria meningitidis isolates express capsular polysaccharide.

171 report a 1.44 A crystal structure of the N. meningitidis major pilin PilE and a approximately 6 A cr

172 N. meningitidis MC58 NMB0419 encodes a Sel1-like repeat (SL

173 h glyco-conjugate capsular group C Neisseria meningitidis (Men C) vaccines in infancy.

174 vel protection against serogroup A Neisseria meningitidis (MenA) are unknown.

175 largely attributed to serogroup A Neisseria meningitidis (MenA).

176 Serogroup B Neisseria meningitidis (MenB) is a major cause of severe sepsis an

177 NmC is the predominant serogroup causing N. meningitidis meningitis.

178 esponsible for 59% (10/17) of serogrouped N. meningitidis meningitis.

179 Neisseria meningitidis (meningococcus) is a symbiont of the human

180 ccus), Haemophilus influenzae, and Neisseria meningitidis (meningococcus) was performed by rapid diag

181 ococcus pneumoniae (pneumococcus), Neisseria meningitidis (meningococcus), and Haemophilus influenzae

182 ococcus pneumoniae (pneumococcus), Neisseria meningitidis (meningococcus), and Haemophilus influenzae

183 ococcus pneumoniae (pneumococcus), Neisseria meningitidis (meningococcus), and Haemophilus influenzae

184 ccus), Haemophilus influenzae, and Neisseria meningitidis (meningococcus).

185 entify the recognition site for three key N. meningitidis methyltransferases: ModA11 (exemplified by

186 orrhea and increases the probability that N. meningitidis might exit the oral cavity to produce syste

187 y, PilE is structurally similar to Neisseria meningitidis minor pilins PilXNm and PilVNm, recently su

188 lipopolysaccharide-null mutants in Neisseria meningitidis, Moraxella catarrhalis, and most recently i

189 neumoniae, Haemophilus influenzae, Neisseria meningitidis, Mycoplasma pneumoniae, Mycobacterium tuber

190 on pathogen detected (n = 17) followed by N. meningitidis (n = 13).

191 available, of which 26% were confirmed as N. meningitidis (n = 2433; 56%), S. pneumoniae (n = 1758; 4

192 Neisseria meningitidis (N. meningitidis), Streptococcus pneumoniae

193 main meningitis-causing bacteria, Neisseria meningitidis (N. meningitidis).

194 Last, we revisit the species status of N. meningitidis, N. gonorrheae, and N. lactamica in the lig

195 ding those of three well-defined species (N. meningitidis; N. gonorrhoeae; and Neisseria polysacchare

196 The three species Neisseria meningitidis, Neisseria gonorrheae, and Neisseria lactam

197 pathogens Haemophilus influenzae, Neisseria meningitidis, Neisseria gonorrhoeae, Helicobacter pylori

198 Neisseria meningitidis (Nm) clonal complex 11 (cc11) lineage is a

199 Neisseria meningitidis (Nm) is a Gram-negative diplococcus that no

200 The human pathogen Neisseria meningitidis (Nm) is a leading cause of bacterial mening

201 Neisseria meningitidis (Nm) is a leading cause of bacterial mening

202 Neisseria meningitidis (Nm) is a nasopharyngeal commensal carried

203 The human pathogen Neisseria meningitidis (Nm) is known to possess several mechanisms

204 Neisseria meningitidis (Nm) strains infecting these patients are p

205 Clusters of Neisseria meningitidis (Nm) urethritis among primarily heterosexua

206 pathogens, 16 (73%) of which were Neisseria meningitidis (Nm).

207 f meningitis, 5590 were confirmed: Neisseria meningitidis ([Nm] 85%), Streptococcus pneumoniae ([Sp]

208 due to a large epidemic of group A Neisseria meningitidis (NmA) meningitis.

209 jugate vaccine against serogroup A Neisseria meningitidis (NmA), MenAfriVac, was first introduced in

210 The Cas9 RNA-guided endonuclease from N. meningitidis (NmCas9) recognizes a 5'-NNNNGATT-3' protos

211 o identify a compact ortholog from Neisseria meningitidis-Nme2Cas9-that recognizes a simple dinucleot

212 Serogroup X N. meningitidis (NmX) dominated in both vaccinated and unva

213 While vaccines are routinely used for N. meningitidis, no vaccine is available for N. gonorrhoeae

214 nactivated, unencapsulated, intact Neisseria meningitidis nor Streptococcus agalactiae inhibited the

215 lecules on Neisseria meningitidis As with N. meningitidis NspA (Nm-NspA), N. gonorrhoeae NspA (Ng-Nsp

216 of the upper respiratory tract by Neisseria meningitidis occurs despite elicitation of adaptive immu

217 lonized by Neisseria lactamica and Neisseria meningitidis One HGT event resulted in the acquisition o

218 th S. pneumoniae, S. agalactiae, E. coli, N. meningitidis, or H. influenzae in combination with cefot

219 Carriage for Neisseria meningitidis (P < 0.05) and Neisseria lactamica (P < 0.0

220 typing and vaccine antigen profile of the N. meningitidis pathogen, thus enabling thorough characteri

221 ptomatic oropharyngeal carriage of Neisseria meningitidis peaks in adolescence and young adulthood.

222 Neisseria meningitidis phasevarions regulate genes including virul

223 eriments demonstrate that the E. coli and N. meningitidis protein homologs are functionally conserved

224 nd in urogenitally-adapted versus typical N. meningitidis, providing evidence for a model in which ex

225 erial pathogenic strains including Neisseria meningitidis, Pseudomonas aeruginosa and Escherichia col

226 SWGA increased the mean proportion of N. meningitidis reads by 2 to 3 orders of magnitude, enabli

227 We found that N. meningitidis recycles PG fragments via the selective per

228 st time, that PorB2-expressing strains of N. meningitidis regulate the AP of baby rabbits and rats.

229 The decreased release of PG monomers by N. meningitidis relative to N. gonorrhoeae is partly due to

230 ility of antibiotics and vaccines, Neisseria meningitidis remains a major cause of meningitis and sep

231 gens, Streptococcus pneumoniae and Neisseria meningitidis, revealed both previously identified and no

232 ion of a few DNA copies per LAMP zone for N. meningitidis, S. pneumoniae and Hib were achieved within

233 Average annual incidences for N. meningitidis, S. pneumoniae, and H. influenzae, respecti

234 alciparum infections, and virulent Neisseria meningitidis samples.

235 te sequences were obtained; 92 (76%) were N. meningitidis sequences, and 29 (24%) were N. gonorrhoeae

236 The number of N. meningitidis serogroup A (NmA) among confirmed bacterial

237 0, the burden of meningitis due to Neisseria meningitidis serogroup A (NmA) has substantially decreas

238 l correlates of protection against Neisseria meningitidis serogroup A (NmA) in Burkina Faso before th

239 To combat Neisseria meningitidis serogroup A epidemics in the meningitis bel

240 the African meningitis belt where Neisseria meningitidis serogroup A historically caused large-scale

241 ries of the meningitis belt, where Neisseria meningitidis serogroup A historically caused large-scale

242 Vaccination campaign against Neisseria meningitidis serogroup A was carried out in 2010-2011.

243 protection against infection with Neisseria meningitidis serogroup A, we use an assumed SBA titre of

244 Neisseria meningitidis serogroup B (MnB) is a leading cause of bac

245 recently licensed vaccines against Neisseria meningitidis serogroup B (NmB) will depend partly on dis

246 neumoniae, Listeria monocytogenes, Neisseria meningitidis serogroup B, Candida albicans, and P. bergh

247 Neisseria meningitidis serogroup C (NmC) was responsible for 59% (

248 f the Nm-positive specimens, 14 (88%) were N meningitidis serogroup C (NmC), 1 was NmW, and 1 was non

249 luding 1147 (71.5%) that were positive for N meningitidis serogroup C (NmC).

250 Because the N. meningitidis serogroup L capsule polymer consists of a t

251 yze the synthesis of the complex trimeric N. meningitidis serogroup L capsule polymer repeating unit.

252 of the pathophysiologically less relevant N. meningitidis serogroup L, is one of the smallest known S

253 sialic acid has been found in the Neisseria meningitidis serogroup W (NmW) capsular polysaccharide (

254 , Togo experienced its first major Neisseria meningitidis serogroup W (NmW) outbreak.

255 Kingdom due to a sublineage of the Neisseria meningitidis serogroup W ST-11 clonal complex (hereafter

256 ingococcal disease (IMD) caused by Neisseria meningitidis serogroup Y has increased in Europe, especi

257 ombinant capsular polymerases from Neisseria meningitidis serogroups A (CsaB) and X (CsxA) were chara

258 Neisseria meningitidis serogroups A and X are among the leading ca

259 consists of polyhexosamine phosphates in N. meningitidis serogroups A and X.

260 coccal disease is mainly caused by Neisseria meningitidis serogroups A, B, C, X, W, and Y.

261 accharide synthesis (cps) locus, classify N. meningitidis serogroups, and identify mechanisms for non

262 ce determinants of disease causing Neisseria meningitidis species are their extracellular polysacchar

263 e resolution the primary transcriptome of N. meningitidis strain 8013.

264 Neisseria meningitidis strain H44/76 was modified by expression of

265 the transcriptome of adherent serogroup B N. meningitidis strain MC58 was determined at intervals dur

266 much lower than that within the wild-type N. meningitidis strain only upon HBMEC infection and was co

267 ratio of species-specific sequences, the N. meningitidis strain seems to have replaced one of its fo

268 tion against antigenically diverse Neisseria meningitidis strains and to compare this protection to a

269 allelic patterns in urethritis-associated N. meningitidis strains may reflect genetic diversity in th

270 s-like and distinguishing them from other N. meningitidis strains.

271 henotypes and hence invasive potential of N. meningitidis strains.

272 Neisseria meningitidis (N. meningitidis), Streptococcus pneumoniae (S. pneumoniae),

273 Neisseria meningitidis, Streptococcus pneumoniae, or Haemophilus i

274 nfluenzae, Listeria monocytogenes, Neisseria meningitidis, Streptococcus pneumoniae, Streptococcus ag

275 E6 thioesterase from the bacterium Neisseria meningitidis Structural analysis with X-ray crystallogra

276 N. meningitidis subverts immune responses by hijacking a ho

277 In all infected mice, N. meningitidis targeted the human vasculature, leading to

278 uenzae type b and capsular group C Neisseria meningitidis tetanus toxoid conjugate vaccine (Hib-MenC-

279 genomic island (the prp gene cluster) in N. meningitidis that enables this species to utilize propio

280 oniae, Haemophilus influenzae, and Neisseria meningitidis, the 3 most common bacteria causing meningi

281 land is absent from the close relative of N. meningitidis, the commensal Neisseria lactamica, which c

282 In E. coli and N. meningitidis, the predominant lipid is a lysophosphatidy

283 on has been studied extensively in Neisseria meningitidis, the specific subset of genes that CrgA tar

284 complement attack achieved by circulating N. meningitidis therefore depends on the relative levels of

285 ty of the human bacterial pathogen Neisseria meningitidis to cause invasive disease depends on surviv

286 Neisseria meningitidis, typically a resident of the oro- or nasoph

287 Neisseria meningitidis use Type IV pili (T4P) to adhere to endothe

288 Neisseria meningitidis utilizes capsular polysaccharide, lipooligo

289 tive bacterial pathogens including Neisseria meningitidis, Vibrio cholerae, and Salmonella enterica h

290 ing protein (FHbp) is an important Neisseria meningitidis virulence factor that binds a negative regu

291 Carriage of N. meningitidis was investigated by using three different m

292 Meningitis caused by N. meningitidis was more common in West-Central than South-

293 ture of an ADEP-ClpP complex derived from N. meningitidis was solved.

294 beta1-4 galactosyltransferase from Neisseria meningitidis we demonstrate fast and robust access to th

295 thogens Haemophilus influenzae and Neisseria meningitidis We hypothesized that activation of compleme

296 a distinct CRISPR-Cas system from Neisseria meningitidis, we demonstrate efficient targeting of an e

297 95.7% and 85.7%, and L. monocytogenes and N. meningitidis were not observed in the study.

298 Hyperinvasive lineages of Neisseria meningitidis, which persist despite extensive horizontal

299 ressed the gene encoding LpxA from Neisseria meningitidis, which specifically attaches 3OH-C12 chains

300 s and 25 isolates from carriers of Neisseria meningitidis without disease.