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1 oss of FGF9 functions after formation of the mesenchymal condensation.
2 l rearrangements and pattern formation, e.g. mesenchymal condensation.
3 some aspects of the developmental process of mesenchymal condensation.
4 ionally active in limb bud mesenchyme and in mesenchymal condensations.
5 cells were unable to contribute chondrogenic mesenchymal condensations.
6 , in turn, leads to the formation of smaller mesenchymal condensations.
7 Sox9 gene is inactivated after chondrogenic mesenchymal condensations.
9 the enamel knot (EK), which maintains dental mesenchymal condensation and epithelial invagination.
10 ly reduced ureteric bud branching but normal mesenchymal condensation and expression of markers indic
12 lopment of the limb bud and formation of the mesenchymal condensation and has key roles in regulating
14 at CRYBP1 mRNA was also downregulated during mesenchymal condensation and that CRYBP1 mRNA was highly
16 To investigate the role of Hif-1alpha in mesenchymal condensations and in early chondrogenesis, w
18 ked by Prx-cre prevents the formation of the mesenchymal condensations as shown by routine histology
19 rogenic cells that will subsequently undergo mesenchymal condensation, cartilage production and, fina
20 primary target of Bmp7 and that formation of mesenchymal condensations characteristic of signaling ce
22 ription factor Barx2 is expressed in primary mesenchymal condensations, digital rays, developing join
23 and Col2a1 RNA in areas of precartilaginous mesenchymal condensations during mouse embryo developmen
24 Embryos, in which Sox9 was deleted after mesenchymal condensations, exhibited a severe generalize
26 f culture, as indicated by the expression of mesenchymal condensation genes and deposition of tenasci
27 ocus resulted in robust expression of CRE in mesenchymal condensations giving rise to both osteoblast
28 lt elastic cartilage progenitors to generate mesenchymal condensation in a self-driven manner, withou
33 timately linked, it is not known whether the mesenchymal condensation is necessary for inducing the i
34 ompaction of the extracellular matrix during mesenchymal condensation is sufficient to drive tissue f
35 mutant mice, chondrogenic differentiation of mesenchymal condensations is delayed and impaired, where
36 enous tissue, in a process designed to mimic mesenchymal condensation leading into chondrogenesis.
37 y evaginations are associated with premature mesenchymal condensation marker, and are reduced by inhi
38 st, an early step presumably at the stage of mesenchymal condensation of cartilage primordia, and sec
39 os abundantly express Mustang, especially in mesenchymal condensations of limbs, vertebral perichondr
40 fa1 transgene to restore Cbfa1 expression in mesenchymal condensations of the Cbfa1-deficient mice.
41 Osf2/Cbfa1 expression is initiated in the mesenchymal condensations of the developing skeleton, is
42 led that BAPX1 is predominantly expressed in mesenchymal condensations of the fetal limb and axial sk
43 ls are present inside rudimentary cartilage (mesenchymal condensation) prior to cartilage maturation.
44 his normally occurs during the physiological mesenchymal condensation response that triggers tooth fo
45 Inactivation of Sox9 in limb buds before mesenchymal condensations resulted in a complete absence
49 for the morphogenesis and patterning of the mesenchymal condensations that serve as primordia of the
50 ily cause disorders associated with abnormal mesenchymal condensation, whereas defects in the transcr
51 scular elastic cartilage from in vitro-grown mesenchymal condensation, which recapitulated the early