ライフサイエンスコーパス: mesentery

1 l muscle, and is unlike other vascular beds (mesentery).

2 rfused venular microvessels of mouse and rat mesentery).

3 vasculature of the allergen-challenged mouse mesentery.

4 or idiopathic inflammatory conditions in the mesentery.

5 y, posterior to the descending colon and its mesentery.

6 n accumulated throughout the interior of the mesentery.

7 and in association with blood vessels in the mesentery.

8 thin, elastic membranous tissue, the dorsal mesentery.

9 iment' or 'anlage' at the injured end of the mesentery.

10 ry lymphoid structure (TLS) formation in the mesentery.

11 ted to lymphatic valve development in murine mesentery.

12 ix hours p.i. in the lungs compared with the mesentery.

13 ic epithelium of the regenerating anlage and mesentery.

14 and the number of macroscopic tumors in the mesentery.

15 ases of the Jamaican fruit bat intestine and mesentery.

16 c vasculature in both the lamina propria and mesentery.

17 ersus unresectable masses in the root of the mesentery.

18 ation of E28 pig pancreatic primordia in the mesentery.

19 penetrated through the muscle wall into the mesentery.

20 symmetries in the architecture of the dorsal mesentery.

21 es involving the pancreatic head and root of mesentery.

22 ital microscopy on IL-1beta-stimulated mouse mesentery.

23 l microscopy in the microvessels in five rat mesenteries.

24 luding a blind gastric cavity partitioned by mesenteries.

25 iver (53%), small bowel (25%), kidney (17%), mesentery (14%), adrenal gland (8%), abdominal wall (8%)

26 dian, 83 v 63 mm; P = .003), and the area of mesentery (19,657 v 11,829 mm(2); P < .0001).

27 P < .001), resulting in a smaller amount of mesentery (8,309 v 17,957 mm(2), P < .001) and nodal yie

28 large inflammatory mass (18%), and thickened mesentery (9%).

29 We used mouse mesentery, a tissue whose numerous microvessels are high

30 he major source of oxidants generated in the mesentery after abdominal irradiation.

31 ure, the large accumulations observed in the mesenteries and ascites fluid of tumor-bearing animals m

32 oth muscle of all major blood vessels in the mesenteries and gut.

33 gehog1 and patched domains in the developing mesenteries and use gene knockdown, knockout and inhibit

34 carcinomas in the colon that spread into the mesentery and adjacent organs.

35 uding lymph node stations in the perigastric mesentery and along the celiac arterial branches).

36 tal and adult peritoneum covering intestine, mesentery and body wall only maintained itself and faile

37 termediate region by surgical lesions of the mesentery and by application of guanethidine (3 microM)

38 and thoracic levels stop short of the dorsal mesentery and do not enter the gut.

39 ue lymphatic channels were identified in the mesentery and followed to the blue-stained SN(s), which

40 t cells from other axial levels to enter the mesentery and gut mesenchyme.

41 n kidneys, lymph nodes, blood vessels, bowel mesentery and intestine.

42 at vasohibin-1 and VEGF are up-regulated, in mesentery and liver, in cirrhotic and precirrhotic porta

43 ctin, with the largest responses observed in mesentery and lung.

44 denopathy, and lymphangiogenesis in both the mesentery and mucosa.

45 dal infiltration occurred selectively in the mesentery and omentum containing resident gp38/podoplani

46 kidneys, liver, musculoskeletal system, and mesentery and pancreas were not altered by diaspirin cro

47 t, spleen, musculoskeletal system, skin, and mesentery and pancreas.

48 oneal cavity, such as abdominal lymph nodes, mesentery and peri-intestinal adipose tissues, demonstra

49 uation of the bowel lumen and wall, adjacent mesentery and soft tissues, as well as a variety of extr

50 opportunity to invade the mesenchyme of the mesentery and the gut, so that earlier arrival assures t

51 lete lymphangion) were isolated from the rat mesentery and tied to glass cannulae capable of independ

52 igns of bowel disease within the surrounding mesentery, and abnormal findings in adjacent structures.

53 denopathy, thickening of the omentum and the mesentery, and ascites.

54 articularly advantageous for the peritoneum, mesentery, and bowel.

55 ic fragment to different sites (mediastinum, mesentery, and kidney capsule) of ATX B6 mice treated wi

56 terioles serving heart, brain, kidney, lung, mesentery, and skin; plus aortic, carotid, and mesenteri

57 mural striation, fatty proliferation of the mesentery, and soft-tissue infiltration of pericolonic f

58 ease, large inflammatory mass, and thickened mesentery are all conditions predisposing to a conversio

59 patterns generated by these processes in the mesentery are consistent with experimental observations.

60 on of tertiary lymphoid organs (TLOs) in the mesentery are features of Crohn's disease.

61 The mesenchymal cells of the dorsal mesentery are more condensed on the left side than on th

62 n the current study, we used isolated murine mesentery arterioles and co-cultures of human coronary a

63 tolic blood pressure (SBP) by telemetry, and mesentery artery endothelial function by pressurized myo

64 Using the mesentery as a model tissue, we show that Tbx1 is not re

65 differential expression were made using the mesentery as a reference against 24 h and 3 days regener

66 uced reciprocal tissue stiffness in the left mesentery as mechanical feedback with the right side.

67 ng the early stages of the process using the mesentery as the focal point of intestinal regeneration.

68 vessel walls and to connective tissue in the mesentery as they migrate toward the gonadal ridges.

69 ed to positional changes of the colon in the mesentery, as opposed to true mobility of the polyp.

70 in mesenteric lymph nodes suggests that the mesentery-associated lymphatics may also collect leukocy

71 ion, very few, if any, PGCs found in the gut mesentery at E10.5 migrate into the genital ridges.

72 age dependency and development along the gut-mesentery-blood-brain course of infection, can be replic

73 cine tissue in either the mediastinum or the mesentery, but not in mice grafted under both kidney cap

74 ynovial capillaries, so the results for frog mesentery capillaries cannot be generalised.

75 he majority of cases involve the small bowel mesentery; colorectal MP is rare.

76 ripotent stem cell lines, gonadal ridges and mesenteries containing primordial germ cells (PGCs, 5-9

77 pport the physiological relevance of the rat mesentery culture model as a biomimetic tool for investi

78 Recently, our laboratory introduced the rat mesentery culture model as an ex vivo experimental platf

79 rs, with most staple lines oversewn, and all mesentery defects closed.

80 o be dispensable for Pitx2 expression during mesentery deformation.

81 The dorsal mesentery (DM) connects the gut tube to the body and dir

82 The dorsal mesentery (DM) is the major conduit for blood and lympha

83 symmetrical cellular behaviors in the dorsal mesentery (DM) of the early mid-gut, a structure connect

84 asymmetric cell behaviors within the dorsal mesentery (DM), which suspends the gut tube, and is down

85 ing the phenotype of new microvessels in the mesentery during induction of vascular remodelling by ov

86 ide synthase in the fat pad of the adult rat mesentery during inhibition of angiopoietin signalling w

87 We find that the dorsal mesentery ECM is indeed left-right asymmetric and moreov

88 determined that Bmp2 expressed in the dorsal mesentery establishes differential elongation rates betw

89 teraction in the microcirculation of the rat mesentery exposed to 25 mmol/l D-glucose for 12 h.

90 bstruction, hemorrhage, cancer and thickened mesentery; extensive disease; the presence of short gut;

91 l foster future investigation of the role of mesentery fat in colorectal diseases.

92 lect the larger sizes of rdh1-null mice, but mesentery, femoral, and inguinal fat pads grow dispropor

93 Second, in the E9.0-E9.5 period, before the mesentery forms, PGCs very rapidly exit the gut, but do

94 ral echinoderm tissues, including esophagus, mesenteries, gonads, respiratory trees, hemal system, te

95 re exposure to VEGF and 24 h later after the mesentery had been replaced in the abdominal cavity.

96 Diaphragm disease and large bowel mesentery implants were the only CT predictors of subopt

97 tion of tumors on the peritoneal surface and mesentery in an i.p. ovarian xenograft model.

98 ructural evidence in the capillaries of frog mesentery indicates a regularity in the structure of the

99 ellate cells), and splanchnic organs (liver, mesentery, intestine, colon, and spleen) were isolated f

100 neutropenic mice, ATAK cells spread from the mesentery into visceral organs on days 1-3.

101 rt that translocation of gut bacteria to the mesenteries is associated with the formation of creeping

102 e of the vascular anatomy of the root of the mesentery is necessary for the performance of complex su

103 lthough VEGF(165)b is anti-angiogenic in the mesentery, it does signal in endothelial cells in vivo r

104 er previous small bowel resection, thickened mesentery, large inflammatory mass, and extensive diseas

105 afted RV-ATL cells developed lymphoma in the mesentery, liver, thymus, lungs, and spleen.

106 the diaphragm, disease located on the bowel mesentery, located on the bowel serosa, and disease loca

107 y observed, with occasional viremia; tonsil, mesentery lymph nodes, and intestinal mucosa served as m

108 In rat mesentery microvessels after occlusion, circulating leuk

109 ultifunctional intravital videomicroscopy in mesentery microvessels with hydroethidine, an oxidant-se

110 l and Phillips on individually perfused frog mesentery microvessels.

111 ior aspect of the pancreatic head or root of mesentery (mid gut carcinoid) may involve one of the 2 p

112 Using a rat mesentery model of inflammation-induced angiogenesis and

113 of various organs including the heart, lung, mesentery, muscle, and eye of different species.

114 o apply to the vascular systems of the lung, mesentery, muscle, eye, and so on.

115 is (n = 7), hepatoduodenal ligament (n = 3), mesentery (n = 2), mediastinum (n = 4), portal venous sy

116 ty in vascularized structures located in the mesentery near the pancreas, intestines, and spleen.

117 ith control tissue, as well as in livers and mesenteries of rats and mice with cirrhosis or/and porta

118 AF increased IOI to comparable values in the mesenteries of wild-type mice and those lacking the gene

119 ntery of wild-type mice but much less in the mesentery of eNOS-/- mice.

120 al cell lines established from the ovary and mesentery of female H-2K(b)-tsA58 mice were tested for r

121 in individually perfused microvessels in the mesentery of frogs and rats.

122 Xenotransplantation in mesentery of pig pancreatic primordia obtained very earl

123 ent intra-abdominal shunts) of BDL rats, and mesentery of sham and BDL rats.

124 polymers, and implanted into the small bowel mesentery of syngeneic animals.

125 hat typically affects the adipose tissue and mesentery of the small intestine but may also affect the

126 Similarly, PAF increased IOI in the mesentery of wild-type mice but much less in the mesente

127 ndrome, causing selective hypertrophy of the mesentery, omentum and other lymphoid tissue-containing

128 The perigastric ligaments, mesentery, omentum, and potential spaces between the par

129 superior mesenteric vein at the level of the mesentery or head of the pancreas.

130 y disease involvement at the cut edge of the mesentery or nonserosalized portions of the colon.

131 e peritoneum (P < .05), bowel (P < .01), and mesentery (P < .05).

132 unit C fibre afferents innervating duodenum, mesentery, pancreas, portal hepatis, bile duct, gall bla

133 is involved in tumor cell attachment to the mesentery possibly via interaction with tumor cell surfa

134 re performed excising the envelope of rectal mesentery posteriorly and the supporting tissues lateral

135 ed and large-sized vessels of the serosa and mesentery preferentially demonstrated histologic changes

136 orcine thymus into either the mediastinum or mesentery provides earlier and more efficient reconstitu

137 In the mesentery, Prox1 expression is high in valve-forming lym

138 ed from the microbiota maintain inflammatory mesentery remodeling and consequently, transient ablatio

139 performed using an appropriately wide rectal mesentery resection technique if the tumor was high; if

140 fferent vascular beds (lung, heart, stomach, mesentery, small intestine, large intestine, and muscle)

141 volves vessels of the submucosa, serosa, and mesentery, some mucosal alterations have been suggested

142 and MYPT1 phosphorylation was reduced in KO mesenteries suggesting that reduced ROCK activity contri

143 Embryologic development of the dorsal mesenteries suggests the existence of retromesenteric pl

144 ll relative to parenchymal tissue in the rat mesentery suggests that in addition to serving as a cond

145 transmigrated leukocytes (P<0.01 vs. control mesenteries superfused with Krebs-Henseleit buffer).

146 immunofluorescence staining of ear skin and mesentery that lymphatic vessels in Ang2(-/-) mice fail

147 echanism of VEGF overexpression in liver and mesentery that promotes pathologic, but not physiologic,

148 s in the cellular architecture of the dorsal mesentery, the structure that connects the primitive gut

149 al elongation rates between the gut tube and mesentery, thereby regulating the compressive forces tha

150 force interstitial osmotic pressure in frog mesenteries, this was assessed in synovium.

151 In vivo, in the rat mesentery, thrombin (0.5 U/ml) or N(G)-nitro-L-arginine-

152 ological materials--including blood vessels, mesentery tissue, lung parenchyma, cornea and blood clot

153 a indicate that, at least in the CD-affected mesentery, TLOs are positioned along collecting lymphati

154 fferences in cellular organization cause the mesentery to assume a trapezoidal shape, tilting the pri

155 ntravital video microscopy in exposed rabbit mesentery to investigate the potential role of fibrinoge

156 sly through the embryonic hindgut and dorsal mesentery to reach the gonads.

157 e proximal-to-distal decrease and a moderate mesentery-to-antimesentery decrease in villus innervatio

158 1.2 to 35 dyne/cm2) of the exteriorized rat mesentery using high-resolution intravital microscopy.

159 tra- or retroperitoneal solid organs, bowel, mesentery, vascular structures, diaphragm, and urinary t

160 Similar phosphorylations were observed when mesentery was exposed to VPF/VEGF in vitro, or when mese

161 e tumor was in the middle or low rectum, all mesentery was resected.

162 deletion, the total number of valves in the mesentery was significantly (P < 0.01) increased in the

163 In this model, the exteriorized mesentery was superfused with ferric chloride and the ac

164 Using intravital microscopy of the rat mesentery, we measured leukocyte-endothelium interaction

165 sly with E28 pig pancreatic primordia in the mesentery, we show normalization of glucose tolerance in

166 ry was exposed to VPF/VEGF in vitro, or when mesenteries were harvested from mice bearing the mouse o

167 At intervals after injecting VPF/VEGF i.p., mesenteries were harvested, extracted, and immunoprecipi

168 served due to idiopathic inflammation of the mesentery were identified.

169 g the diseased bowel segment and surrounding mesentery were manually delineated and used to develop a

170 f lymph nodes inside the intussusception and mesentery were noted.

171 TA3/St cells that initially attached to the mesentery were strongly CD44 positive.

172 dually perfused venular microvessels in frog mesentery when the perfusate contained albumin.

173 single perfused venular microvessels in rat mesentery, which enabled direct observation of permeabil

174 begins with tissue deformation of the dorsal mesentery, which is dependent on left-sided expression o

175 ing ventral to the dorsal aorta and into the mesentery, which is the portal to the gut.

176 rats and vasorelaxation in the isolated rat mesentery, which was blocked by the NO synthase inhibito

177 isappear from the regenerating intestine and mesentery, while fibronectin labeling and 4G7 (an echino

178 Concomitant superfusion of the rat mesentery with 10 nmol/liter of each of three lipoxin an

179 -glucose was inhibited by superfusion of the mesentery with 30 nmol/l bisindolylmaleimide-I, a potent

180 Superfusion of the rat mesentery with 50 micromol/liter NG-nitro-L-arginine met

181 Superfusion of IL-1beta-stimulated rabbit mesentery with C3a resulted in a rapid and stable adhesi

182 Superfusion of the rat mesentery with either 0.5 U/ml thrombin or 50 microM L-N

183 Superfusion of the rat mesentery with either thrombin or L-NAME consistently an

184 We demonstrated that superfusion of rat mesentery with LPS resulted in significant increases in

185 Thus, in vivo superfusion of the rat mesentery with stable lipoxin analogs at 10 nmol/liter r

186 e segments of collecting lymphatics from rat mesentery, with preserved or inhibited NO signalling.