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1 coitum (dpc) on the ventromedial side of the mesonephros.
2 1(+) mesenchymal cells that migrate from the mesonephros.
3 by mature structures such as the somites or mesonephros.
4 ds arise on the ventromedial surface of each mesonephros.
5 rs that are repressed by the presence of the mesonephros.
6 y coelomic epithelium invagination along the mesonephros.
7 lockade on the WD cultured in the absence of mesonephros.
8 to become the nephric duct, pronephros, and mesonephros.
9 ation in the absence of a developing pro- or mesonephros.
10 of homeotic transformation of metanephros to mesonephros.
11 ue and Lmx-1 mRNA expression as a marker for mesonephros.
12 umber of endothelial cells from the adjacent mesonephros, a mechanism totally absent in XX gonads.
14 the PGE2-cAMP-PKA pathway in the aorta-gonad-mesonephros (AGM) abolished enhancement in hematopoietic
15 c stem cells (HSCs) arise in the aorta-gonad-mesonephros (AGM) and mature as they transit through the
16 c stem cell emergence, viz., the aorta-gonad-mesonephros (AGM) and the fetal liver at 10.5-11.5 dpc,
19 tive direct Notch targets in the aorta-gonad-mesonephros (AGM) embryonic tissue by chromatin precipit
22 elopment, but which cells in the aorta-gonad-mesonephros (AGM) microenvironment produce these factors
25 ic endothelium (HE) in the aorta- gonads-and mesonephros (AGM) region and reside within Intra-aortic
26 , this effect is specific to the aorta-gonad-mesonephros (AGM) region and shows that the AGM is the m
28 gie Stage (CS) 14 to CS17 in the aorta-gonad-mesonephros (AGM) region is a tightly regulated process.
30 (CD34(+)c-kit(+)) cells from the aorta-gonad-mesonephros (AGM) region of the developing mouse are mul
31 arise during development in the aorta-gonad-mesonephros (AGM) region of the embryo from a population
33 mogenic endothelium within the aorta, gonad, mesonephros (AGM) region of the mammalian embryo is cruc
34 itor cells (HSPCs) emerge in the Aorta-Gonad-Mesonephros (AGM) region of the mid-gestation mouse embr
35 fates concurrently occur in the aorta-gonad-mesonephros (AGM) region prior to haematopoietic stem ce
36 inactivated SCL in yolk sac, the aortagonad-mesonephros (AGM) region, and fetal liver hematopoietic
37 HSCs were not released from the aorta-gonad-mesonephros (AGM) region, as evidenced by the accumulati
38 tem cells (HSCs) emerge from the aorta-gonad-mesonephros (AGM) region, but the molecular regulation o
39 an intraembryonic location, the aorta-gonad-mesonephros (AGM) region, is a site of residence and, po
40 t human HSCs emerge first in the aorta-gonad-mesonephros (AGM) region, specifically in the dorsal aor
41 such as the dorsal aorta of the aorta-gonad-mesonephros (AGM) region, suggesting that signals from t
42 lly dissect HSC emergence in the aorta-gonad-mesonephros (AGM) region, we screened a collection of in
55 were recently identified in the aorta-gonad-mesonephros (AGM) region; however, their role in the hem
56 vested from embryonic day 9 (E9) aorta-gonad-mesonephros (AGM) regions of GATA2 null embryos showed r
57 topoietic stem cells (HSCs) from aorta/gonad/mesonephros (AGM) regions of midgestation mouse embryos
58 Rare endothelial cells in the aorta-gonad-mesonephros (AGM) transition into hematopoietic stem cel
64 mice lacking RA synthesis and signalling in mesonephros and adjacent gonad and reveal that Stra8 exp
65 embryonic day 10.5 by the thickening of the mesonephros and consist of somatic cells and migratory p
66 n the bone marrow, reside in the aorta-gonad-mesonephros and contribute to the development of the dor
71 We also show that the region between the mesonephros and the gonad harbors steroidogenic cell pre
74 ube derived from an epithelial anlage at the mesonephros anterior end, which then segregates from the
75 nic day 11.5 Nr4a1-/-; Nr4a2-/- aorta-gonads-mesonephros are devoid of in vivo long-term hematopoieti
76 d/or para-aorta-splanchno-pleura/aorta-gonad-mesonephros are hypothesized to colonize the fetal liver
77 anterior structures, the pronephros and the mesonephros, are transitory and largely non-functional,
78 ated embryos cultured for 3 days in ovo, the mesonephros as well as the pronephros failed to develop
79 TH-responsive cells are present at the gonad/mesonephros border and seem to migrate into the XY but n
81 rta and populate the E10.5-E11.5 aorta-gonad-mesonephros but by E13.5 were replaced by neural-crest-d
82 opoietic stem cells (HSC) in the aorta-gonad-mesonephros by abrogating Smad1 expression and the conse
88 ack, unilateral anomalous development of the mesonephros in males causes atresia of the homolateral e
90 elopment, endothelial cells migrate from the mesonephros into the gonad to form a coelomic blood vess
93 geny validated the generation of aorta-gonad-mesonephros-like definitive haematopoietic stem and prog
100 CellComm to investigate how the aorta-gonad-mesonephros microenvironment dictates haematopoietic ste
102 the para-aortic splanchnopleura/aorta-gonads-mesonephros of mouse embryos and that abrogation of nitr
103 -aortic splanchnopleura/aorta-genital ridges-mesonephros (P-Sp/AGM) region are the main sites of haem
104 hes definitive haematopoiesis at aorta-gonad-mesonephros, placenta and foetal liver, but does not imp
105 ting cells from murine yolk sac, aorta-gonad-mesonephros, placenta, fetal liver, and bone marrow with
108 undergoes dramatic growth in the aorta-gonad-mesonephros region and by E11.5 reaches the size that ma
109 e of arterial endothelium in the aorta-gonad-mesonephros region and document novel benchmarks for HSC
110 ls (HSCs) are first found in the aorta-gonad-mesonephros region and vitelline and umbilical arteries
111 itors are first generated in the aorta-gonad-mesonephros region between days 27 and 40 of human embry
112 c stem cells (HSCs) in the mouse aorta-gonad-mesonephros region emerge between embryonic days 10.5 an
114 genitor cells derived from the aorto gonadal mesonephros region of day 11 mouse embryos on the Jagged
116 (HSCs) that first appear in the aorta-gonado-mesonephros region of the fetus on embryonic day (E) 10.
118 ived during embryogenesis in the aorta-gonad-mesonephros region subsequently colonize fetal and adult
119 on and atria earlier than in the aorta-gonad-mesonephros region, and is transient and definitive in n
120 CX3CR1(+) cells localized to the aorta-gonad-mesonephros region, and visualized at embryonic day (E)9
121 they are first generated in the aorta-gonad-mesonephros region, but at later developmental stages, i
122 h other already in the embryonic aorta-gonad mesonephros region, but it is still unknown how their di
123 od vessel walls in the yolk sac, aorta-gonad-mesonephros region, embryonic liver, and fetal bone marr
125 os developed cd41(+) HSCs in the aorta-gonad-mesonephros region, which later migrated to the kidney,
129 poietic cluster formation in the aorta-gonad-mesonephros region; embryonic-to-adult transplantation s
131 endent induction of Stra8, but only when the mesonephros remains attached, pointing to a non-RA signa
132 ized human-pig chimeric middle-stage kidney (mesonephros) structures up to embryonic day 28 inside ne
133 pression pattern of the transgene in the pro/mesonephros suggest an intraembryonic site of developmen
134 tached, pointing to a non-RA signal from the mesonephros that induces Stra8 in the adjacent gonad.
135 sult from failure of cell migration from the mesonephros, thought to be a possible source of Leydig c
139 d11 is ectopically activated in the anterior mesonephros, we observe a partial transformation to a me
141 by ex vivo culture of dissected aorta-gonads-mesonephros with SCF, IL3 and FLT3L, which may bypass No