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1 all subtypes of mesothelioma, but not normal mesothelium.
2 uscle differentiation in the developing lung mesothelium.
3 anced cell-cell adhesion to the lipid-loaded mesothelium.
4 r smooth muscle cells arise from the surface mesothelium.
5 in spleen morphogenesis and expressed in the mesothelium.
6 gene expression to a specific region of the mesothelium.
7 ed in a coelomic cavity and are covered by a mesothelium.
8 ontransformed ovarian surface epithelium and mesothelium.
9 re cells in the early allantois and never in mesothelium.
10 hages, and the visceral and parietal pleural mesothelium.
11 and posterior tissues such as mesenchyme and mesothelium.
12 ty through exfoliated regions of the pleural mesothelium.
13 contained within cellular aggregates on the mesothelium.
14 gesting that intestinal vSMCs arise from the mesothelium.
15 that adhesion myofibroblasts arise from the mesothelium.
16 ize by attaching to and invading through the mesothelium, a single layer of mesothelial cells lining
17 The developmentally regulated competency of mesothelium and a localized inductive signal might play
18 le gene expression program in the developing mesothelium and allow appropriate cell fate decisions to
19 of allantoic mesoderm into an outer layer of mesothelium and an inner vascular network begins in the
20 es emphasize the importance of examining the mesothelium and chest cavity as a whole, rather than foc
21 that specification of allantoic endothelium, mesothelium and chorio-adhesive cells does not occur by
22 signaling molecule that is expressed in lung mesothelium and epithelium and is required for lung deve
26 VEGF-A was immunolocalized to peritoneal mesothelium and TGF-beta1 increased VEGFA mRNA (P < 0.05
27 sitional (or junction) area between the OSE, mesothelium and tubal (oviductal) epithelium, as a previ
29 senting endogenous antigen in the peritoneal mesothelium and vessels led to the local recruitment of
30 ee cases, cells derived from lineage-labeled mesothelium are found inside the lung and as smooth musc
33 gnals are ablated in the single cell-layered mesothelium at the periphery, neonatal growth is disturb
34 fic developmental stage, a large area of the mesothelium becomes competent to express proepicardial m
35 s show that the gut is initially devoid of a mesothelium but that serosal mesothelial cells expressin
37 , the inner endoderm and the outer jacket of mesothelium, coordinately regulate the proliferation and
38 ogene-driven fast growth of tumor nodules on mesothelium covered surfaces, causing ascites, bowel obs
42 Interaction of these MCAs with peritoneal mesothelium disrupts mesothelial integrity, exposing the
44 mechanism by which neutrophils adhere to the mesothelium during their transmigration into the inflame
45 -1 in monocyte transmigration across pleural mesothelium during tuberculous inflammation was investig
46 pression remained elevated in smooth muscle, mesothelium, endothelium, and fibroblasts in regions of
47 including epithelium, mesenchyme, pericytes, mesothelium, endothelium, and immune cells in both organ
48 inflammatory cells, smooth muscle cells, and mesothelium exhibited increased TGF-beta1 expression and
49 of zebrafish hand2 reporter embryos captures mesothelium formation including pericardium, visceral, a
52 whilst the distal outer layer of presumptive mesothelium gradually acquires vascular cell adhesion mo
53 We investigated the role of the pulmonary mesothelium in dysregulated lung growth noted in the Wt1
56 ribute to the regeneration of the peritoneal mesothelium, indicating an inherent difference between p
57 de Kupffer cells grew and escaped across the mesothelium into the peritoneal cavity and immediately i
59 dings further support a paradigm wherein the mesothelium is a source of progenitors for mesenchymal l
60 tochemistry, we demonstrate that the serosal mesothelium is the major source of vasculogenic cells in
62 is caused by malignant transformation of the mesothelium, is incurable, and can be categorized into t
64 atures: 60-90% pure liver septum transversum/mesothelium-like, 70-80% pure liver-like fibroblasts and
67 s fluid phase and subsequent adhesion to the mesothelium lining covering abdominal organs to establis
69 uring heart development, epicardial cells (a mesothelium) move to and over the heart, undergo epithel
70 ranous VCAM-1 expression was observed on the mesothelium of 13 of 14 women with ovarian cancer compar
71 dherent spheroids and their adherence to the mesothelium of distant organs lead to cancer progression
73 ar adhesion molecule (ICAM)-1 in the pleural mesothelium of patients with active pleural tuberculosis
74 ecules and is expressed predominantly in the mesothelium of the diaphragm during embryonic developmen
75 growing metastases that breach the visceral mesothelium of the liver via the "find me signal", ATP.
76 t interfacing of the beta1-fragment with the mesothelium of the peritoneal membrane via a biomaterial
77 vivo that macrophages adhere specifically to mesothelium overlying draining lymphatics and that their
79 introducing caHIF1alpha into the epicardial mesothelium prevented EPDCs from proper migration into t
82 impaired bacterial clearance, and defective mesothelium repair, suggesting a critical role of TNF to
83 Loss of Ezh2 specifically in the developing mesothelium reveals a mesothelial cell-autonomous role f
84 sitive cells of mesothelial origin and human mesothelium shows an increase of mesothelial EGFR expres
85 VTE proportion was highest in cancer of the mesothelium/soft tissue (RR: 19.35 [17.44-21.47]) and lo
90 blast growth factor (FGF) 9 signals from the mesothelium (the future pleura) to sub-mesothelial mesen
91 rowth factor-sensitive events in the ovarian mesothelium, the tissue source of ovarian epithelial can
92 hat PDGF receptors cooperate in the yolk sac mesothelium to direct blood vessel maturation and sugges
93 ; rather, they migrate across the peritoneal mesothelium to the lymphatics, through which they furthe
94 lopment, cells of the superficial epicardial mesothelium undergo EMT to give rise to precursor cells
96 This tissue is a simple, poorly committed mesothelium which exhibits characteristics of epithelial
97 responsiveness of endometriosis ESCs to the mesothelium, which induces migration and gap junction co
98 her VEGF isoforms and/or the outer sheath of mesothelium, whose maintenance did not appear to be depe