戻る
「早戻しボタン」を押すと検索画面に戻ります。 [閉じる]

コーパス検索結果 (１語後でソート)

通し番号をクリックするとPubMedの該当ページを表示します
1 und changes that can be better described as "metaplasticity".
2 ion, and triggers a form of learning-induced metaplasticity.
3 ia a(i/o) signaling in a form of presynaptic metaplasticity.
4 duction of a form of NMDA receptor-dependent metaplasticity.
5 ds was sufficient to induce this presynaptic metaplasticity.
6  thereby contributing to a form of cell-wide metaplasticity.
7 GluR5s are necessary for this input-specific metaplasticity.
8 arietal-motor circuit through heterosynaptic metaplasticity.
9 S could enhance stimulation outcomes through metaplasticity.
10 memory and is achieved by mechanisms such as metaplasticity.
11 y to undergo a normal developmental shift in metaplasticity.
12 d is adjusted by overall neural activity via metaplasticity.
13  to their inability to undergo developmental metaplasticity.
14 riming protocol, in a manner consistent with metaplasticity.
15 olds, the slow dynamics of which account for metaplasticity.
16  ethanol represents a form of NMDAR-mediated metaplasticity.
17 d by habituation, and is thus a paradigm for metaplasticity.
18 s important roles in synaptic plasticity and metaplasticity.
19 a novel spatially delimited form of synaptic metaplasticity.
20  adjust the threshold for plasticity, termed metaplasticity.
21 ticity and (2) the history of plasticity, or metaplasticity.
22 portant role in both information storage and metaplasticity.
23 uR7-RIM1alpha interactions underlies MF-SLIN metaplasticity.
24 ty, endocannabinoids (eCBs) act as agents of metaplasticity.
25 ticity, reduction of synaptic crosstalk, and metaplasticity.
26  form of plasticity has been referred to as 'metaplasticity', a modification of synapses reflected as
27                      The devices show unique metaplasticity, a useful feature for generalizable deep
28  learning benchmarks show that probabilistic metaplasticity achieves performance equivalent to state-
29                              Here we studied metaplasticity affecting spike-timing-dependent plastici
30 ified in manners consistent with homeostatic metaplasticity and depotentiation.
31 DA-R function likely play a critical role in metaplasticity and in stabilizing activity levels in neu
32 scriptional coupling provides a mechanism of metaplasticity and may regulate capacity for synaptic mo
33 cetylase (HDAC) inhibition rescues GABAergic metaplasticity and normalizes AKAP signaling in MD anima
34 exhibit properties such as those observed in metaplasticity and synaptic scaling.
35 n forms of non-Hebbian neuronal plasticity: "metaplasticity" and "homeostatic synaptic scaling," whic
36 y), (3) real cTBS150 followed by real tbTUS (metaplasticity), and (4) real tbTUS followed by real cTB
37 elayed expression of synaptic modifications, metaplasticity, and spacing effects.
38      However, the rules that govern synaptic metaplasticity are much less clear.
39      These data provide in vivo evidence for metaplasticity as a mechanism for binocular competition
40 ctions, suggesting that strategies targeting metaplasticity at glutamatergic synapses can be used to
41  behavioural relevance, induces differential metaplasticity at this synapse, attenuating its ability
42 at mossy fiber NMDARs mediate heterosynaptic metaplasticity between mossy fiber and associational-com
43 ortant regulators of synaptic plasticity and metaplasticity, but the exact mechanisms underlying thei
44 n (LTP) in visual cortex by a process termed metaplasticity, but the mechanism is unknown.
45      Authentic stress can induce hippocampal metaplasticity, but whether transmitted stress has the s
46             Furthermore, the enhancement of 'metaplasticity' by both GLYX-13 and ketamine may help ex
47 induces LTP-like processes and suggests that metaplasticity can be harnessed in the therapeutic devel
48                                              Metaplasticity can enhance plasticity induction and clin
49            Overall, our results suggest that metaplasticity can provide a neural substrate for adapti
50 ted doses of ketamine, but not ECS, led to a metaplasticity characterized by an increased magnitude o
51 ines undergo activity-dependent changes with metaplasticity consequences on synaptic regulation.
52  Accordingly, we suggest that stress-induced metaplasticity could disrupt Ca2+ homeostasis and thus e
53          This work offers a new insight that metaplasticity defects are central to synaptic dysfuncti
54                             We conclude that metaplasticity-defined broadly as the adaptive control o
55 nsistent with spine ER regulating structural metaplasticity, delivery of a second stimulus to ER- spi
56 tate dependence of the mechanisms underlying metaplasticity during behavior and pathology.
57 y depend on both the stage of plasticity and metaplasticity during memory formation.
58 n underlying mechanism for the regulation of metaplasticity during this time period.
59  have previously described a heterodendritic metaplasticity effect, whereby strong high-frequency pri
60 L-1beta), were responsible for mediating the metaplasticity effect.
61 ither necessary nor sufficient to induce the metaplasticity effect.
62 ty to induce subsequent synaptic plasticity (metaplasticity) has not been investigated.
63 work by testing predictions of the threshold metaplasticity hypothesis in a second mouse line with en
64                Consistent with the threshold metaplasticity hypothesis, behavioral pre-training desig
65                    One form of regulation is metaplasticity (i.e., the activity-dependent and long-la
66 ptability and precision can be mitigated via metaplasticity, i.e. synaptic changes that do not always
67 ent an important mechanism for bidirectional metaplasticity in BLA circuits and thus modulate the acq
68    Thus, TNFalpha mediates a heterodendritic metaplasticity in healthy rodents that becomes constitut
69 wo optimal protocols for inducing functional metaplasticity in human pharyngeal motor cortex have bee
70 ow that atRA mediates synaptopodin-dependent metaplasticity in mouse dentate granule cells.
71 ts provide evidence for enhanced directional metaplasticity in pharyngeal motor cortex and new insigh
72 pressin (VP) in social buffering of synaptic metaplasticity in stress-responsive corticotropin-releas
73 P and the inability to undergo developmental metaplasticity in Tg mice.
74 y paradigms, perhaps thereby contributing to metaplasticity in the adult brain.
75  mGluR7 surface expression governs a form of metaplasticity in the hippocampus, little is known about
76 ich nuclear to synaptic interactions induce "metaplasticity" in NAc MSNs, and we reveal the specific
77 naptic modifications, analogous to "synaptic metaplasticity" in the brain, and how memory is consolid
78  compensation thus involves a novel form of 'metaplasticity' in the adult brain, in which the increas
79                    Plasticity of plasticity (metaplasticity) in the CNS has been linked to group I me
80 y of neuronal response plasticity is called "metaplasticity." In suppressing synaptic inhibition and
81 tion of synaptic plasticity has been called "metaplasticity." In this report, we describe the facilit
82                   Targeting ketamine-induced metaplasticity intermittently (i.e., 24-hour interval) b
83 posed mechanism by integrating probabilistic metaplasticity into a spiking network trained on an erro
84                                              Metaplasticity is a higher order plasticity which regula
85                      This induction of local metaplasticity is a novel mechanism by which endocannabi
86 s stability for individual synaptic weights, metaplasticity is also used.
87                             This hippocampal metaplasticity is bufferable following social interactio
88     In females, however, this stress-induced metaplasticity is buffered by the presence of a naive pa
89  the underlying mechanism for this effect on metaplasticity is caused by caspase cleavage of the APP-
90 disease, we examined whether heterodendritic metaplasticity is dysregulated in a transgenic mouse mod
91                                 This type of metaplasticity is essential for navigation of experience
92                                              Metaplasticity is the process of regulating future capac
93 experience-dependent switch, a novel form of metaplasticity, is not dependent on NMDA receptors but m
94      Many in vitro studies have demonstrated metaplasticity-like effects whereby prior neuronal activ
95 tic homeostasis and is likely to function in metaplasticity, long-term regulation of the ability of a
96          Our results provide examples of how metaplasticity may play a key role in the ongoing modula
97                              This persistent metaplasticity may promote the excessive amplification o
98 l AMPAR subunits GluA1 and GluA2, as well as metaplasticity measured ex vivo using electrically-stimu
99 us, there is an endogenous engagement of the metaplasticity mechanism in this mouse model of AD, supp
100  pull-push control of LTP/LTD form a general metaplasticity mechanism that may contribute to neuromod
101 gether, these results suggest a link between metaplasticity mechanisms in the hippocampus and the for
102                           Thus, the rules of metaplasticity might manifest in opposite directions, de
103 isual experience on NMDAR EPSCs and prevents metaplasticity of LTP and LTD.
104 strongly suggest that group I mGluRs control metaplasticity of spinal learning through a PKC-dependen
105 e resistant to further plasticity, a type of metaplasticity often referred to as saturation.
106 ur NIBS protocol combinations to investigate metaplasticity on tbTUS in humans of either sex.
107 her the underlying mechanisms are related to metaplasticity or depotentiation.
108                                     BCM-like metaplasticity over a shorter timescale has also been ob
109                                          The metaplasticity perspective could inform the development
110 utic mechanisms provides the opportunity for metaplasticity processes to be harnessed as a druggable
111 es in AD mouse models may reflect defects in metaplasticity processes.
112     Recent treatments based on the notion of metaplasticity provide a powerful model for individual b
113         Our finding that the APP-ICD affects metaplasticity provides new insights into the altered re
114        Here we propose that reward-dependent metaplasticity (RDMP) can provide a plausible mechanism
115                                              Metaplasticity refers to activity-dependent changes in n
116                                              Metaplasticity refers to an activity-dependent regulatio
117                                              Metaplasticity regulates the threshold for modification
118                                              Metaplasticity represents activity-dependent synaptic ch
119                              Priming-induced metaplasticity requires mGluR5-mediated mobilization of
120              Hence, eCBs are also objects of metaplasticity, subject to higher levels of physiologica
121                     Derived from elements of metaplasticity (synaptic priming), this review discusses
122 xcitatory postsynaptic signaling and altered metaplasticity (temporal summation of NMDA receptor curr
123 us unreliability of synaptic changes evinces metaplasticity that can provide a robust mechanism for m
124 tes synapse-specific NMDAR-AKT signaling and metaplasticity that contributes to memory updating and i
125 how that cocaine self-administration induces metaplasticity that inhibits further induction of synapt
126 nt CaM-based sliding threshold mechanism for metaplasticity that is governed by the phosphorylation s
127  to inhibit GSK3 and mediates input-specific metaplasticity that protects potentiated synapses from s
128 ephrine exposure mediates a form of synaptic metaplasticity that recalibrates fear memory processing.
129                                              Metaplasticity, the adaptive changes of long-term potent
130 nd impaired mice by inducing a novel form of metaplasticity to regulate circuit function, providing a
131                                              Metaplasticity tunes the synapses to undergo changes tha
132                                          The metaplasticity we discuss acts by co-opting Hebbian mech
133 occluded further reduction by heterosynaptic metaplasticity, whereas LTP was entirely rescued by incu
134 sing depends on sensory experience-dependent metaplasticity, which allows homeostatic maintenance of
135   In this research, we propose probabilistic metaplasticity, which consolidates weights by modulating
136                     This social buffering of metaplasticity, which requires the presence of another i
137 (WT) controls, but there was an inversion of metaplasticity, with increased GluN2B phosphorylation, w

 
Page Top