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1 spiration of a right axillary node confirmed metastatic carcinoma.
2 asive adenocarcinoma to castration-resistant metastatic carcinoma.
3  paradigm for clinical intervention of liver metastatic carcinoma.
4  of the three sentinel lymph nodes contained metastatic carcinoma.
5  differentiation from cholangiocarcinoma and metastatic carcinoma.
6  carcinoma, and in metastatic cell lines and metastatic carcinoma.
7 ary for active cell migration in invasive or metastatic carcinoma.
8 Two of the 11 axillary lymph nodes contained metastatic carcinoma.
9 ken for an intraparotid lymph node harboring metastatic carcinoma.
10 erts preneoplastic lesions into invasive and metastatic carcinomas.
11 plasms that progress to locally invasive and metastatic carcinomas.
12 r-reaching implications for the treatment of metastatic carcinomas.
13 or macrophage-activating factors secreted by metastatic carcinomas.
14 adhesion kinase (FAK) are expressed in colon metastatic carcinomas.
15  of non-invasive tumor cells into malignant, metastatic carcinomas.
16 protease associated with rapidly growing and metastatic carcinomas.
17 ents a testable therapeutic approach against metastatic carcinomas.
18 s but delayed their development to invasive, metastatic carcinomas.
19 mas, 18 of 28 primary carcinomas, and 5 of 5 metastatic carcinomas.
20 A1, a human gene with elevated expression in metastatic carcinomas.
21 derstood and unleveraged in the treatment of metastatic carcinomas.
22 n reaction in 30 of 33 EBVaGC cases, 8 of 10 metastatic carcinomas, 14 non-neoplastic tissues from 27
23 ary tumor growth and lung dissemination of a metastatic carcinoma (4T1), but not of a nonmetastatic c
24 nulosa cell (GC) tumors that can progress to metastatic carcinoma and thus provide a model system for
25 on of BKV large T antigen in the primary and metastatic carcinoma, but not in the nonneoplastic uroth
26 concurrently with the transition to invasive metastatic carcinoma, but they were expressed in differe
27 ntous G proteins, which are overexpressed in metastatic carcinomas, but their functions in epithelial
28  silenced alphaB-crystallin in two different metastatic carcinoma cell lines.
29 r actin polymerization in the invadopodia of metastatic carcinoma cells and define four distinct stag
30                                              Metastatic carcinoma cells exhibit at least two differen
31  In addition, silencing alphaB-crystallin in metastatic carcinoma cells reduced the number of viable
32                               The ability of metastatic carcinoma cells to survive matrix detachment
33 ular mechanisms of invadopodium formation in metastatic carcinoma cells.
34 he splenic lesion was performed and revealed metastatic carcinoma, consistent with the lung primary.
35 epithelial neoplasia (HGPIN) to invasive and metastatic carcinoma could facilitate study of the regul
36                             slanDCs surround metastatic carcinoma deposits in close proximity to dead
37  hyperplasias, papillomas, and dysplasias to metastatic carcinomas developed in squamous epithelia of
38  distinguish adenocarcinoma of the NPCE from metastatic carcinoma, especially metastatic clear cell r
39 6 per million in Asians) from hemangioma and metastatic carcinoma following the clinical practice.
40  patients in the radical group, only 10% had metastatic carcinoma in the resected retroperitoneal lym
41 ation, lymphoproliferative inflammation, and metastatic carcinomas in the lung after a period of late
42 e been linked to various diseases, including metastatic carcinomas in which the 9 carbon sialic acid
43 ), whereas 10 cholangiocarcinomas and 8 of 9 metastatic carcinomas lacked this staining.
44  were maintained when stratifying tumor into metastatic carcinoma (N = 57) and meningioma (N = 17).
45                                              Metastatic carcinoma of a renal allograft is a rare but
46  biopsy of the gastric antrum later showed a metastatic carcinoma of breast origin with typical tumor
47 approach to determine tumor type in cases of metastatic carcinoma of clinically unknown origin.
48 18 years) diagnosed with locally advanced or metastatic carcinoma of the pancreas confirmed by cytolo
49     Identifying the primary site in cases of metastatic carcinoma of unknown origin has profound clin
50 lerable, but is unlikely to have activity in metastatic carcinomas of the biliary tree.
51 d 18 years or older initially diagnosed with metastatic carcinoma or who subsequently developed metas
52           Patients with multiple myeloma and metastatic carcinoma to the skeleton who are receiving i
53                                              Metastatic carcinoma was identified in 10 of 13 axillary
54 clinical diagnosis of verrucous carcinoma or metastatic carcinoma was made.
55 promotes cell migration and is a hallmark of metastatic carcinomas, we asked whether it increases FA
56 biopsies from patients with various advanced metastatic carcinomas, wherein these cells are transitio
57 hat develop extensive HGPIN and invasive and metastatic carcinoma with neuroendocrine (NE) differenti
58 t inhibitor, in 41 patients with progressive metastatic carcinoma with or without mismatch-repair def