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1 s the predominate cause of canine hereditary methemoglobinemia.
2  associated with 3-AP administration include methemoglobinemia.
3  of gastrointestinal cancer and, in infants, methemoglobinemia.
4 nt systemic vasoconstriction without causing methemoglobinemia.
5 cytochrome b(5) reductase mutant that causes methemoglobinemia.
6 ons that give rise to the hereditary disease methemoglobinemia.
7 e and health conditions, such as malaria and methemoglobinemia.
8 s a compensatory response to nitrate-induced methemoglobinemia.
9 ead to clinically significant hypotension or methemoglobinemia.
10  municipal tap water as a potential cause of methemoglobinemia and monitor for excessive levels of ox
11 eath in APAP-treated snakes was likely acute methemoglobinemia and respiratory failure due to severe
12 polycythemias, including hemoglobin mutants, methemoglobinemias and congenital 2,3-bisphosphoglycerat
13 relations of humans and dogs with hereditary methemoglobinemia are not yet well characterized.
14 ysis systems resulted in no further cases of methemoglobinemia at our institution.
15 sion and activity is associated with type II methemoglobinemia, cancer, neurodegenerative disorders,
16    To evaluate the root cause of a series of methemoglobinemia cases in a medical ICU.
17                                   Congenital methemoglobinemia caused by an erythrocytic deficiency o
18                         The positions of the methemoglobinemia-causing mutations are scattered throug
19 to alter the clinical approach to hereditary methemoglobinemia in dogs, it demonstrates the possibili
20 subacute toxicity testing, 4 did not produce methemoglobinemia in rats (400 mg/kg po daily for 9 days
21 tact in human whole blood and did not induce methemoglobinemia in the dark.
22 the first description of familial idiopathic methemoglobinemia in the United Kingdom was reported in
23 lated cardiac glycosides, local anesthetics, methemoglobinemia, opioids, organophosphates and carbama
24                         Recessive congenital methemoglobinemia (RCM) is caused by a deficiency of red
25                 We report a sentinel case of methemoglobinemia that was associated with dialysis sess
26                                              Methemoglobinemia, the first hereditary disease to be id
27 described for the human enzyme or any of the methemoglobinemia variants.
28          S127P, a mutant that causes type II methemoglobinemia, was the first to be positively identi
29  interfering factors, including pigment from methemoglobinemia, we found the assay had to be modified
30 ta indicate the t-BuBpT ligands may minimize methemoglobinemia, which is a marked advantage over 3-AP
31 on of four additional patients who developed methemoglobinemia while undergoing portable dialysis.