ライフサイエンスコーパス: micromol

1 dry weight) was lower than SHAM (13.5+/-1.0 micromol/g dry weight).

2 0.5 to 5.0 micromol/L and for IL-8 up to 1.0 micromol/L.

3 n average tHb of the Tis-T1 group were 102.0 micromol/L +/- 28.5 (standard deviation) and 71.9 microm

4 a linear response range from 5.0 up to 200.0 micromol L(-1), detection limit of 8.25 nmol L(-1) and a

5 nsor has a linear response in the 0.05-250.0 micromol L(-1) concentration range, a very good detectio

6 ncentration-dependent manner from 0.5 to 5.0 micromol/L and for IL-8 up to 1.0 micromol/L.

7 roup were 100.3 micromol/L +/- 26.4 and 67.0 micromol/L +/- 18.3, respectively.

8 ects compared with a decrease of 8.3 +/- 9.0 micromol/L in controls (P < .0001).

9 , -5.31) and plasma CySS (mean change, -3.00 micromol/L; 95% CI: -4.61, -1.40).

10 fter 24 h of high light (approximately 1,000 micromol m(-2) s(-1)), as shown by the decline in maximu

11 centration values ranging from 100 to 10,000 micromol/mol with relative expanded uncertainties (95% c

12 linder gas mixtures: set no. 1, He at 12,000 micromol/mol, H(2) at 600 micromol/mol, Ar at 100 microm

13 at 600 micromol/mol; set no. 2, He at 15 000 micromol/mol, H(2) at 5000 micromol/mol, Ar at 1000 micr

14 (1,200 micromol mol(-1)) and SE CO(2) (4,000 micromol mol(-1)), at two developmental stages early and

15 xide-supported catalyst, yield around 22,000 micromoles of acetic acid per gram of catalyst, or aroun

16 urinary sodium excretion (from 0.07 +/- 0.01 micromol min(-1) at baseline to 0.76 +/- 0.08 micromol m

17 as also markedly increased (0.22 versus 0.01 micromol/L) in the UDCA group compared to the placebo gr

18 de concentration (mean +/- SD: 0.52 +/- 0.02 micromol/250 g) in the meat of the SBs compared with the

19 ith pore size, with a maximum level of 0.037 micromol/m(2) being obtained for 4000 A silica.

20 ong those in the lowest quartile (mean, 0.04 micromol/L [0.89 microg/dL]).

21 +/- 0.05 vs. 0.25 +/- 0.07 vs. 0.32 +/- 0.05 micromol kg adipose lipid(-1) min(-1), respectively (P =

22 +/- 0.06 vs. 0.20 +/- 0.05 vs. 0.31 +/- 0.05 micromol kg adipose lipid(-1) min(-1), respectively (P =

23 changes in the UDCA level (16.86 versus 0.05 micromol/L) and total bile acid level (17.21 versus -0.5

24 was higher (0.12 +/- 0.05 vs. 0.03 +/- 0.06 micromol x g(-1) x h(-1), means +/- SD, P < 0.02, n = 5)

25 an: 0.70 micromol/L, IQR: 0.40-0.84 vs. 0.06 micromol/L; IQR: 0.01-.018; p = 0.002), cobalt (median:

26 centrations had increased from 1.78 +/- 0.07 micromol/L at baseline to 2.85 +/- 0.11 micromol/L for m

27 og/L (3.65 nmol/L) and 1.58 microg/dL (0.076 micromol/L), respectively, in 14,778 adults aged >or=20

28 icromol min(-1) at baseline to 0.76 +/- 0.08 micromol min(-1) at 120 min; P < 0.001).

29 S-methyl-L-thiocitrulline (SMTC, 2.1 +/- 0.1 micromol kg(-1)).

30 ndependent of antifibrotic actions, RLX (0.1 micromol/L) increased Na(+) current density, INa ( appro

31 tion of CaV1.2 channels with nifedipine (0.1 micromol/L) or diltiazem (10 micromol/L) abolished this

32 y relevant concentrations (approximately 0.1 micromol/L) that inhibit RTK targets.

33 10 micromol/L), PD98059 (MEK1 inhibitor, 0.1 micromol/L), and wortmannin (phosphatidylinositol 3-kina

34 otein (20 pmol/L, >8-fold) and bombesin (0.1 micromol/L, 8-fold) through cAMP signaling.

35 te autonomic stimulation (acetylcholine [0.1 micromol/L] or isoproterenol [0.01-0.1 micromol/L]) afte

36 [0.1 micromol/L] or isoproterenol [0.01-0.1 micromol/L]) after termination of atrial tachypacing (5-

37 2, 0.9 +/- 2.1, 8.6 +/- 2.3, and 6.0 +/- 1.1 micromol kg(-1) min(-1) within 5 h in control, LY0.25, L

38 rol subjects (PCr=5.4+/-1.2 versus 9.6+/-1.1 micromol/g wet weight in MI versus control subjects, res

39 lasma (6.9 +/- 1.9 compared with 6.7 +/- 1.1 micromol/L) or RBCs (2068 +/- 50 compared with 2117 +/-

40 tration decreased (from 35 +/- 2 to 14 +/- 1 micromol/L) and the grand median [quartiles] FGF21 conce

41 QR: 111-145; p<0.001) and zinc (median: 23.1 micromol/L, IQR: 12.9-32.6 vs. 5.1 micromol/L; IQR: 4.4-

42 : 0.01-.018; p = 0.002), cobalt (median: 3.1 micromol/L, IQR: 2.62-3.15 vs. 1.17 micromol/L; IQR: 0.9

43 xygen consumption (from 59 +/- 5 to 34 +/- 1 micromol oxygen/min/10(6) cells, P < 0.001); the latter

44 group were 55.1 micromol/L +/- 22.7 and 39.1 micromol/L +/- 14.9, respectively.

45 ian: 23.1 micromol/L, IQR: 12.9-32.6 vs. 5.1 micromol/L; IQR: 4.4-9.4; p = 0.020) when compared with

46 an average tHb of the benign group were 55.1 micromol/L +/- 22.7 and 39.1 micromol/L +/- 14.9, respec

47 KBP12] in intact myocytes is approximately 1 micromol/L (similar to [RyR]), whereas [FKBP12.6] is <or

48 Acute cocaine (1 micromol/L) application significantly decreased GABA rel

49 ent with the parent compound (I3C) or DIM (1 micromol/L) protected against cell killing due to H(2)O(

50 led that the Rho-kinase inhibitor fasudil (1 micromol/L) significantly blunted artery contractions to

51 Isoproterenol infusion (1 micromol/L) increased sinus rate and shifted pacemaking

52 es with the beta-AR agonist isoproterenol (1 micromol/L) or the blockers CGP+ICI (baseline).

53 Tetrodotoxin (1 micromol/L) also reduced cardiac contracture during isch

54 bitors Ran (5 micromol/L) or tetrodotoxin (1 micromol/L).

55 nhibited CDK2 at concentrations lower than 1 micromol/L, with CDK2 being inhibited 35-fold less poten

56 ) cells with VEGF (10 ng/ml), ANP (1 pM to 1 micromol/L), and/or isatin, an ANP receptor antagonist.

57 romol/L) and/or isoproterenol (Iso; 0.2 to 1 micromol/L).

58 81% during isoproterenol infusion (0.01 to 1 micromol/L).

59 Application of U50488 (1 micromol/L) significantly decreased GABAergic transmissi

60 ug was tested at five concentrations (0.1-10 micromol/L) for its effects on YFP expression, cell viab

61 In addition, 10 micromol/L of ZnMP reduced HCV replication by approximat

62 cells at high (100 nmol/L, 1 mmol/L, and 10 micromol/L, respectively) but not at physiologically rel

63 ype 5 inhibitor and adenosine antagonist, 10 micromol/L) and phorbol myristate acetate (phorbol ester

64 ion, approximately 0.28 and approximately 10 micromol/L) at a late stage of entry, (2) induce Niemann

65 Nicotine at 10 micromol/L significantly downregulated the release of TN

66 l/L) and especially P(2)Y(12) (Cangrelor, 10 micromol/L) blunted CXCL16-triggered platelet activation

67 nifedipine (0.1 micromol/L) or diltiazem (10 micromol/L) abolished this effect.

68 phorbol myristate acetate (phorbol ester, 10 micromol/L), and inhibited by H-89 (protein kinase A inh

69 total homocysteine (tHcy) was elevated (>10 micromol/L) in 20% of subjects.

70 ',5'-cyclic monophosphate [cAMP] inducer, 10 micromol/L; >3-fold), potentiated by 3-isobutyl-1-methyl

71 ited by H-89 (protein kinase A inhibitor, 10 micromol/L), PD98059 (MEK1 inhibitor, 0.1 micromol/L), a

72 half maximal inhibitory concentration of <10 micromol/L, a selectivity index>20, and potent activity

73 C-reactive protein <2 mg/L, homocysteine <10 micromol/L, N-terminal pro-brain natriuretic peptide <10

74 were imaged before and after injection of 10 micromol/kg CM-101.

75 thelin-1- (10 nmol/L), or phenylephrine- (10 micromol/L) induced hypertrophic cultured neonatal ventr

76 For microarrays, cells were exposed to 10 micromol/L of tamoxifen and genes with expression change

77 Responses to 10 micromol/L papaverine, a direct smooth muscle dilator, w

78 Incubation with 10 micromol/L mexiletine rescued the trafficking defect of

79 4) revealed that adenosine perfusion (10-100 micromol/L) produced more significant shortening of acti

80 N omega-nitro-L-arginine (100 micromol/L; nitric oxide antagonist) blocked the reducti

81 mol/mol, H(2) at 600 micromol/mol, Ar at 100 micromol/mol, and O(2) at 600 micromol/mol; set no. 2, H

82 purinergic receptors P(2)Y(1) (MRS2179, 100 micromol/L) and especially P(2)Y(12) (Cangrelor, 10 micr

83 erm N2-fixation rates were higher, up to 100 micromol N m(-2)d(-1), and detected N2fixation at all si

84 l/mol, H(2) at 5000 micromol/mol, Ar at 1000 micromol/mol, O(2) at 5000 micromol/mol; and set no. 3,

85 um creatinine levels falling from 315 to 105 micromol/L by day 7.

86 median creatinine at 3 and 12 months was 107 micromol/L (range 72-222) and 121 micromol/L (range 63-1

87 0.07 micromol/L at baseline to 2.85 +/- 0.11 micromol/L for men and from 1.64 +/- 0.04 to 3.32 +/- 0.

88 t follow-up in the lifestyle arm (beta=-0.11 micromol/L per genetic risk scores risk allele; 95% conf

89 2 carriers; 11.53 micromol/l/h, versus 12.11 micromol/l/h, P = 0.036) and after adjustment for age an

90 with high homocysteine (above the median, 11 micromol/L) and that, in these participants, a causal Ba

91 y acid: 8.97 + or - 1.39 to 4.53 + or - 1.12 micromol/min; glucose: 1.31 + or - 0.52 to 6.86 + or - 1

92 line control group (102 +/- 8 and 104 +/- 12 micromol min(-1), respectively).

93 and cervicovaginal human tissues and at 3-12 micromol/L in CD4(+) T cells.

94 bits a specific decarboxylase activity of 12 micromol pyruvate min(-)(1) mg(-)(1) protein using benzy

95 group of 104 patients with tHcy level of 12 micromol/L or greater revealed that those subjects with

96 as defined as total serum homocysteine of 12 micromol/L or greater.

97 hs was 107 micromol/L (range 72-222) and 121 micromol/L (range 63-157), respectively.

98 median: 311 micromol/L, IQR: 289-329 vs. 129 micromol/L; IQR: 111-145; p<0.001) and zinc (median: 23.

99 SssI with comparable IC(50) (6-13 micromol/L) by competing with S-adenosylmethionine in th

100 dase enzymatic activity than controls (11.14 micromol/l/h versus 11.85 micromol/l/h, P = 0.011).

101 ing concentrations of L-ascorbic acid (-4.15 micromol/L; 95% CI: -0.49, -7.81 micromol/L; P = 0.03 re

102 -2.23) and elevated homocysteine levels (>15 micromol/L: OR = 2.24; 95% CI = 1.38-3.63) were independ

103 of daily oral doses of sulforaphane (50-150 micromol) for 18 wk, followed by 4 wk without treatment,

104 up MGd with the optimal concentration at 150 micromol/L.

105 153, PCB138, and PCB180; total PCB dose, 150 micromol/kg) for 2 days.

106 og/L) (high) and 2.9 mg per 24 hours (15.167 micromol/d) (normal), respectively.

107 malondialdehyde concentration (1.79 +/- 0.17 micromol/250 g) in the meat of the control burgers (CBs)

108 ian: 3.1 micromol/L, IQR: 2.62-3.15 vs. 1.17 micromol/L; IQR: 0.95-1.27; p<0.001), iron (median: 311

109 eir NHGU was blunted (68 +/- 9 and 16 +/- 17 micromol.100 g liver(-)(1).min(-)(1), respectively).

110 n level of 62 micromol/L (normal range, 3-17 micromol/L).

111 The genetic score raised uric acid by 17 micromol/L (95% CI 15, 18) per SD increase and explained

112 d HHcy and elevated Hcy levels to 53 and 173 micromol/L in Cbs(+/+) and Cbs(-/+) mice fed an HM diet,

113 ents in the highest BLL quartile (mean, 0.19 micromol/L [3.95 microg/dL]) compared with 1.76% (CI, 1.

114 5% confidence interval: [CI]: -2.51 to -1.19 micromol/L, Z17 = 5.45, p < .00001).

115 for samples containing approximately 0.1-0.2 micromol of (13)C,(15)N- or (2)H,(13)C,(15)N-labeled pro

116 men and from 1.64 +/- 0.04 to 3.32 +/- 0.1.2 micromol/L for women.

117 y reduced in patients with AMD (median: 16.2 micromol/L, IQR: 11.4-31.3 vs. 49.9 micromol/L; IQR: 32.

118 ile (12.4 +/- 1.8 compared with 13.0 +/- 2.2 micromol/L; P < 0.001).

119 In contrast, N(G)-monomethyl-L-arginine (2 micromol/min) infusion reduced radial blood flow to a si

120 half maximal effective concentration of 0.20 micromol/L.

121 kinase inhibitors and Akt inhibitor SH-6 (20 micromol/L) further diminished CXCL16-induced platelet a

122 0.001) and arginine levels (mean: 68 +/- 20 micromol/l vs. 74 +/- 24 micromol/l, p < 0.001) than tho

123 es revealed a UDP EC(50) of approximately 20 micromol/L and an ATP IC(50) of approximately 5 micromol

124 Greater IPA (20 micromol/L ADP, final extent) occurred with ticagrelor t

125 when glucose infusion rates (GIRs) were ~20 micromol kg(-1) min(-1) in all groups, was higher in con

126 ficantly increased, and when treated with 20 micromol/L (-)-epigallocatechin gallate (green tea) was

127 nd during pharmacological uncoupling with 20-micromol/L carbenoxolone.

128 400 micromol mol(-1)), elevated CO(2) (1,200 micromol mol(-1)) and SE CO(2) (4,000 micromol mol(-1)),

129 known that super-elevated (SE) CO(2) (>1,200 micromol mol(-1)) induces physiological responses differ

130 at of moderately elevated CO(2) (up to 1,200 micromol mol(-1)), but little is known about the molecul

131 Plants grown at 200 micromol m(-2)s(-1) light were compared with plants accl

132 ux of (13)NH3/(13)NH4(+), which exceeded 200 micromol g(-1) h(-1), was not commensurate with membrane

133 l concentration (Km = 152 microm, Vmax = 205 micromol g(-1) h(-1)).

134 Blood lead levels (BLLs) less than 1.21 micromol/L (<25 microg/dL) among adults are considered a

135 nt total bile acid levels (34.99 versus 9.21 micromol/L, P < 0.08) in comparison with those who did n

136 micromol/hr-cm(2)) and low (0.33 + or - 0.22 micromol/hr-cm(2)) fluxes that coincide with therapeutic

137 Mild (22 micromol/L) and moderate (88 micromol/L) HHcy were induc

138 six stations, and rates ranged from 0 to 23 micromol N m(-2)d(-1)based on sediment trap fluxes.

139 tic acid per gram of catalyst, or around 230 micromoles of methanol per gram of catalyst, respectivel

140 36 micromol/l) and fish eaters (227, 221-233 micromol/l).

141 ol/l) and lower in vegetarians (230, 224-236 micromol/l) and fish eaters (227, 221-233 micromol/l).

142 ls (mean: 68 +/- 20 micromol/l vs. 74 +/- 24 micromol/l, p < 0.001) than those without significantly

143 oM and specific activity of approximately 24 micromol of porphobilinogen/mg of protein/h.

144 phosphate (P) was found to be highest (~0.24 micromoles per kilogram per year) in the vicinity of the

145 icromol/l) than in meat eaters (237, 231-242 micromol/l) and lower in vegetarians (230, 224-236 micro

146 were slightly higher in vegans (241, 234-247 micromol/l) than in meat eaters (237, 231-242 micromol/l

147 A detection limit of about 25 micromol/L of Tm-DOTMA was calculated from in vitro MR m

148 NOS inhibitor N(G)-monomethyl-L-arginine (25 micromol/min) also reduced basal coronary flow (by 22.3+

149 micromol/mol, H(2), Ar, and O(2) each at 25 micromol/mol with a balance gas of N(2).

150 ors wortmannin (100 nmol/L) and LY294002 (25 micromol/L).

151 determined using a normal curve (0.78 to 25 micromol l(-1) ATP).

152 ibroblasts were exposed to various FFAs (250 micromol/l) in either 5 or 25 mmol/l (high) glucose for

153 m sample of 36 anonymous individuals was 277 micromol/L erythrocyte lysate/hour with a standard devia

154 ely, P<0.001; ATP=3.4+/-1.1 versus 5.5+/-1.3 micromol/g wet weight, P<0.001).

155 8.8, and those of the T2-T4 group were 100.3 micromol/L +/- 26.4 and 67.0 micromol/L +/- 18.3, respec

156 cMR(glc) value was, therefore, 40.6 +/- 13.3 micromol/100 g/min (lumped constant = 0.6).

157 ession with CsCl (5 mmol/L, n=3), BaCl(2) (3 micromol/L, n=3), and low extracellular potassium (1 mmo

158 yme A recycling, in nontransgenic (4.5+/-2.3 micromol/min per gram dry weight) was 3.75-fold faster t

159 cGMP peak concentrations of approximately 3 micromol/L.

160 eak concentrations of approximately 1 to > 3 micromol/L could also be monitored in blood vessels of t

161 Isoproterenol up to 3 micromol/l increased heart rate to 100 +/- 6.8 beats/min

162 /AFL induced by acetylcholine (ACh; 0.3 to 3 micromol/L) and/or isoproterenol (Iso; 0.2 to 1 micromol

163 [quartiles 30.8, 61.8] vs. 51.9 [41.0, 77.3] micromol glucose/microU insulin . mL(-1) . min(-1); P <

164 phan were 0.26/0.72 micromol/L and 3.39/4.30 micromol/mmol creatinine, respectively.

165 concentrations of BCAA ranging from 8 to 30 micromol/liter, which is 10 to 17% of the concentration

166 with UGT2B17 exhibiting the lowest K(M) (300 micromol/L) against SAHA of any UGT in vitro.

167 IQR: 0.95-1.27; p<0.001), iron (median: 311 micromol/L, IQR: 289-329 vs. 129 micromol/L; IQR: 111-14

168 18 micromol/l) and vegetarians (303, 294-312 micromol/l).

169 6-324 micromol/l), fish eaters (309, 300-318 micromol/l) and vegetarians (303, 294-312 micromol/l).

170 ol/l), followed by meat eaters (315, 306-324 micromol/l), fish eaters (309, 300-318 micromol/l) and v

171 gh concentration of active vinyl groups (330 micromol g(-1)).

172 paclobutrazol (PAC) at ambient [CO(2)] (350 micromol CO(2) mol(-1)) was reverted by elevated [CO(2)]

173 ration (340, 95% confidence interval 329-351 micromol/l), followed by meat eaters (315, 306-324 micro

174 7 micromol/L [9.4 mg/dL]; range, 34.2 to 356 micromol/L [2.0 to 20.8 mg/dL]).

175 [K(+)]ext and yielding fluxes as high as 36 micromol g (root fresh weight)(-1) h(-1) at 5 mm [K(+)]e

176 ive in the Chung model, with an ED(50) of 38 micromol/kg after intraperitoneal administration.

177 CM components increased A(sat) from 24 to 38 micromol m(-)(2) s(-)(1).

178 amin C deficient (serum concentrations <11.4 micromol/L).

179 quine (4 micromol/L, n=7) or flecainide (2-4 micromol/L, n=5).

180 etine (Prozac) inhibited mK(ATP) (IC(50)=2.4 micromol/L).

181 ssociated with blood GSH (mean change, -25.4 micromol/L; 95% CI: -45.3, -5.31) and plasma CySS (mean

182 root transformed (SM) concentration was 51.4 micromol/L (95% CI: 48.4, 54.6).

183 R, 1.35 [CI, 1.14 to 1.60] per 1 mg/dL [88.4 micromol/L]), and lower triage systolic blood pressure (

184 Administration of A-889425 (4 micromol/kg, i.v.) significantly increased T(r) by 0.42+

185 in patients with isolated endarteritis, 96.4 micromol/L (95% CI, 48.6 to 143.2) in positive controls

186 nd during perfusion of either chloroquine (4 micromol/L, n=7) or flecainide (2-4 micromol/L, n=5).

187 e grown under ambient atmospheric CO(2) (400 micromol mol(-1)), elevated CO(2) (1,200 micromol mol(-1

188 30) were grown and monitored at ambient (400 micromol mol(-1)) or elevated (800 micromol mol(-1)) [CO

189 methylarginine concentration in plasma (1.41 micromol/L in the endo-DDAH1(-/-) versus 0.69 micromol/L

190 atmosphere at 20 cm above ground level (412 micromol mol(-1)).

191 calculated from microsensor profiles (31-437 micromol CH4m(-2)d(-1)) were sufficiently high to preven

192 creatinine greater than 25% or 0.5 mg/dL (44 micromol/L) from baseline to peak value within the first

193 RBCs (2068 +/- 50 compared with 2117 +/- 48 micromol/L).

194 o (HR) of 1.20 (95% CI 1.11, 1.30) per 59.48 micromol/L (1 mg/dL) uric acid.

195 e unconfounded effect, we found that a 59.48 micromol/L higher uric acid concentration did not have a

196 y slightly lower than that of ZD2-Cy5.5 (0.5 micromol kg(-1)) in fluorescence imaging.

197 own with and without supplementary UV-B (1.5 micromol m(-2) s(-1)).

198 was blocked in rat hearts perfused with 2.5 micromol/L TAT-HK2 before ischemia or at the onset of re

199 lux were suppressed to 23 +/- 3 and 26 +/- 5 micromol L(-1) (P = 0.91) and 44 +/- 4 and 39 +/- 5 micr

200 l L(-1) (P = 0.91) and 44 +/- 4 and 39 +/- 5 micromol min(-1) (P = 0.41) in the insulin and niacin gr

201 The maximum yield is approximately 5.5 micromol H(2) h(-1) mg(-1) chlorophyll and is estimated

202 romol/L and an ATP IC(50) of approximately 5 micromol/L.

203 a selective AMPK inhibitor (compound C, at 5 micromol/L) reversed the effects of metformin on [Ca(2+)

204 ith the late Na(+) current inhibitors Ran (5 micromol/L) or tetrodotoxin (1 micromol/L).

205 At a concentration of 2.5 to 5 micromol/L (similar to that of decitabine), complete deg

206 xylic acids have the potential to supply 4-5 micromoles C hr(-1)g(-1) fresh weight to the soil soluti

207 after the XO inhibitor allopurinol (ALLO, 50 micromol/L).

208 At 48 hours, cells exposed to 10 and 50 micromol/L of tamoxifen were 85.6% and 48.4% viable, res

209 t 5000 micromol/mol; and set no. 3, He at 50 micromol/mol, H(2), Ar, and O(2) each at 25 micromol/mol

210 A pharmacologic dose (50 micromol/L) decreased proliferation, invasion, and MMP-9

211 rothrombin time <50% and serum bilirubin >50 micromol/L on postoperative day 5) and the International

212 sulfide-donor sodium hydrosulfide (NaHS) (50 micromol/kg, twice daily) or vehicle (n=7 per group).

213 Cells exposed to 0, 10, or 50 micromol/L of tamoxifen for 48 hours were evaluated for

214 In VH rats, Ro25-6981 (500 micromol/L) inhibited ACC neuronal firing, evoked by 30

215 elial cells (HAECs) treated with DL-Hcy (500 micromol/L) and d-glucose (25 mmol) for 48 h.

216 Addition of l-homocysteine (100 to 500 micromol/L), but not l-cysteine, maintained the Ly-6C(hi

217 . 2, He at 15 000 micromol/mol, H(2) at 5000 micromol/mol, Ar at 1000 micromol/mol, O(2) at 5000 micr

218 l/mol, Ar at 1000 micromol/mol, O(2) at 5000 micromol/mol; and set no. 3, He at 50 micromol/mol, H(2)

219 clusion of all GBA and LRRK2 carriers; 11.53 micromol/l/h, versus 12.11 micromol/l/h, P = 0.036) and

220 nd total bile acid level (17.21 versus -0.55 micromol/L) were significantly higher in the UDCA group

221 the ranges of 0.01-250, 1.0-500, and 3.0-550 micromol L(-)(1), with detection limits of 0.007, 0.6, a

222 Growth of soybean at elevated [CO(2)] (550 micromol x mol(-1)) under field conditions stimulated th

223 ations in the range of 0.004-340 and 0.5-550 micromol L(-1), with detection limits of 1.0 nmol L(-1)

224 levels rose from 246 +/- 288 to 561 +/- 596 micromol/L (P < 0.01).

225 corresponded with normalized TAG (14.9+/-0.6 micromol/g dry weight) and improved contractility.

226 betic CTGF+/+ group (11.7+/-1.2 vs 7.9+/-0.6 micromol/L p<0.01), while urinary albumin excretion and

227 1 month after rejection treatment was 132.6 micromol/L (95% confidence interval [95% CI], 78.7 to 18

228 3.2) in positive controls (P=0.32), and 18.6 micromol/L (95% CI, 1.8 to 35.4) in untreated negative c

229 Up to 1.6 micromoles of rcSso7d-CBD was found to adsorb per gram o

230 our different levels (100, 200, 400, and 600 micromol m(-2) s(-1)) by imaging chlorophyll fluorescenc

231 o. 1, He at 12,000 micromol/mol, H(2) at 600 micromol/mol, Ar at 100 micromol/mol, and O(2) at 600 mi

232 mol, Ar at 100 micromol/mol, and O(2) at 600 micromol/mol; set no. 2, He at 15 000 micromol/mol, H(2)

233 wth irradiance was increased from 100 to 600 micromol m(-2) s(-1) after 24 d of growth.

234 -wise increase in irradiance from 100 to 600 micromol m(-2) s(-1).

235 8-11.17 mmol/L]) and a bilirubin level of 62 micromol/L (normal range, 3-17 micromol/L).

236 eries, intracoronary infusion of SMTC (0.625 micromol/min) reduced basal coronary blood flow by 34.1+

237 99.0+/-40.1 mg.dL(-1)) and Lp(a) 3.74+/-1.63 micromol.L(-1) (104.9+/-45.7 mg.dL(-1)), respectively.

238 e amniotic fluid embolism group (234 134-635 micromol/L) compared with the non-amniotic fluid embolis

239 between the soil surface and the leaves (638 micromol mol(-1)) and the atmosphere at 20 cm above grou

240 ssfully switch between high (1.3 + or - 0.65 micromol/hr-cm(2)) and low (0.33 + or - 0.22 micromol/hr

241 centrations for pseudouridine were 2.89/5.67 micromol/L and 39.7/33.9 micromol/mmol creatinine.

242 icromol/L in the endo-DDAH1(-/-) versus 0.69 micromol/L in the control mice), kidney, lung, and liver

243 enzymatic activity than non-carriers (13.69 micromol/l/h versus 11.93 micromol/l/h, P = 0.002).

244 TAG content in HYP (9.7+/-0.7 micromol/g dry weight) was lower than SHAM (13.5+/-1.0 m

245 ive breast cancer in mice at a low dose (1.7 micromol kg(-1), 1 T), but not in oestrogen receptor-pos

246 n had a decrease in homocysteine level (-1.7 micromol/L; p = 0.005).

247 U/L), and serum bilirubin (mean peak, 160.7 micromol/L [9.4 mg/dL]; range, 34.2 to 356 micromol/L [2

248 ansplantation, high serum creatinine (>291.7 micromol/L), or retinal photocoagulation or vitrectomy (

249 queous humor levels of cadmium (median: 0.70 micromol/L, IQR: 0.40-0.84 vs. 0.06 micromol/L; IQR: 0.0

250 ions for C-mannosyltryptophan were 0.26/0.72 micromol/L and 3.39/4.30 micromol/mmol creatinine, respe

251 id of -5.98 micromol/L (95% CI: -8.23, -3.73 micromol/L; P = 2.0 x 10(-7) per minor allele).

252 l(-1)) was reverted by elevated [CO(2)] (750 micromol CO(2) mol(-1)).

253 lucose: 1.31 + or - 0.52 to 6.86 + or - 1.78 micromol/min).

254 micromol/L, 65 39-91 micromol/L and 49 30-78 micromol/L, respectively (p < .001).

255 Creatinine concentration (79 micromol/L [IQR, 70-87 micromol/L] vs 79 micromol/L [IQR

256 (79 micromol/L [IQR, 70-87 micromol/L] vs 79 micromol/L [IQR, 72-89 micromol/L], P = .61) and glycate

257 halved in MI [to 1.7+/-0.5 versus 3.3+/-0.8 micromol(g . s)(-1); P<0.001].

258 rs, serum iron had increased by 15.9 +/- 9.8 micromol/L from baseline in lexaptepid-treated subjects

259 ient (400 micromol mol(-1)) or elevated (800 micromol mol(-1)) [CO2] for 120 d and subjected to droug

260 acid (-4.15 micromol/L; 95% CI: -0.49, -7.81 micromol/L; P = 0.03 reduction per minor allele).

261 ymatic activity than GBA heterozygotes (0.85 micromol/l/h versus 7.88 micromol/l/h, P < 0.001), and G

262 Zinc concentrations were approximately -1.85 micromol/L lower in depressed subjects than control subj

263 an controls (11.14 micromol/l/h versus 11.85 micromol/l/h, P = 0.011).

264 ine concentration (79 micromol/L [IQR, 70-87 micromol/L] vs 79 micromol/L [IQR, 72-89 micromol/L], P

265 tivity than GBA and LRRK2 non-carriers (7.88 micromol/l/h versus 11.93 micromol/l/h, P < 0.001).

266 heterozygotes (0.85 micromol/l/h versus 7.88 micromol/l/h, P < 0.001), and GBA heterozygotes had lowe

267 Mild (22 micromol/L) and moderate (88 micromol/L) HHcy were induced in cystathionine beta-synt

268 Stoichiometry showed that 0.89 micromol of inorganic phosphate was produced for each mi

269 -87 micromol/L] vs 79 micromol/L [IQR, 72-89 micromol/L], P = .61) and glycated hemoglobin (5.9% [IQR

270 es calculated from cell densities (660-4,890 micromol CH4m(-2)d(-1)) and actual rates calculated from

271 with detection limits of 0.007, 0.6, and 0.9 micromol L(-)(1), respectively.

272 thin 30 min; mean +/- SEM NHGU was 105 +/- 9 micromol.100 g liver(-)(1).min(-)(1).

273 dine were 2.89/5.67 micromol/L and 39.7/33.9 micromol/mmol creatinine.

274 an: 16.2 micromol/L, IQR: 11.4-31.3 vs. 49.9 micromol/L; IQR: 32.0-.142.0; p = 0.022) when compared t

275 mol/L +/- 28.5 (standard deviation) and 71.9 micromol/L +/- 18.8, and those of the T2-T4 group were 1

276 lysate/hour with a standard deviation of 90 micromol/L erythrocyte lysate/hour.

277 r groups, which had serum levels of 56 36-91 micromol/L, 65 39-91 micromol/L and 49 30-78 micromol/L,

278 erum levels of 56 36-91 micromol/L, 65 39-91 micromol/L and 49 30-78 micromol/L, respectively (p < .0

279 non-carriers (7.88 micromol/l/h versus 11.93 micromol/l/h, P < 0.001).

280 on-carriers (13.69 micromol/l/h versus 11.93 micromol/l/h, P = 0.002).

281 participants), fibrinogen by 10% (mean -0.96 micromol/l, three trials, 159 participants), ADP platele

282 g concentrations of L-ascorbic acid of -5.98 micromol/L (95% CI: -8.23, -3.73 micromol/L; P = 2.0 x 1

283 ols (median L-arginine level, 65, 66, and 98 micromol/mL, respectively [P = .0001]; median L-arginine

284 y (reciprocal of glucose rate of appearance [micromol x kg fat-free mass(-1) x min(-1)] x insulin [mU

285 sure brain glucose metabolism (quantified as micromol/100 g/min), which serves as a marker of brain f

286 When the concentrations of biomarkers are at micromoles per liter, such as for sarcosine, which was r

287 3% and a specific activity of 2.2 +/- 0.2 Ci/micromol.

288 Urinary malondialdehyde concentrations (micromol/g creatinine) decreased by 49% (P = 0.021) in s

289 reatinine value in milligrams per deciliter (micromoles per liter).

290 simple amperometric biosensors for detecting micromoles per liter of analytes in serum or urine.

291 Hepatic glucose and intermediate fluxes (micromol . kg(-1) . min(-1)) were measured with and with

292 moker samples (mean +/- SD, 179 +/- 205 fmol/micromol dAdo).

293 ofile the higher-concentration metabolites (>micromol/L concentrations).

294 ery good detection sensitivity (0.4177 muA L micromol(-1)), and a low detection limit of 1x10(-2) mum

295 .25 nmol L(-1) and a sensitivity of 120 nA L micromol(-1).

296 MMA concentration expressed as the ratio of micromoles of MMA to millimoles of creatinine (uMMA rati

297 The catalase activity, in units of micromoles hydrogen peroxide decomposed per minute over

298 rane potentials the channel requires several micromoles of intracellular Ca(2+) for activation.

299 rane potentials the channel requires several micromoles of intracellular Ca2+ for activation.

300 4 (192.0) ng/mL, respectively (to convert to micromoles per liter, multiply by 0.00489; P = .07).