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1  controls and nontransfected cells in either microsatellite sequence.
2 specific DNA molecules covering the complete microsatellite sequence.
3 ector sensitive to frameshift mutations in a microsatellite sequence.
4 ntegral numbers of dinucleotide repeats in a microsatellite sequence.
5 very two consecutive GGAA-repeats on shorter microsatellite sequences.
6 ucleotide, dinucleotide, and tetranucleotide microsatellite sequences.
7 , RFX knockdown cells display instability in microsatellite sequences.
8 d markers by characterization of Xiphophorus microsatellite sequences.
9 NA polymerase errors in vitro using template microsatellite sequences.
10 haracterized by repeat length alterations at microsatellite sequences.
11 omyces cerevisiae contains numerous unstable microsatellite sequences.
12 cleotide mismatches and slippage mistakes at microsatellite sequences.
13  of many vertebrate genomes are comprized of microsatellite sequences.
14 e a major factor in promoting instability of microsatellite sequences.
15 ave been found to exhibit instability of DNA microsatellite sequences.
16 tor phenotype in these tumors extends beyond microsatellite sequences.
17 enced neutral markers (mitochondrial DNA and microsatellites), sequenced an expressed MHC class IIB g
18 es, including the well-studied P. falciparum microsatellite sequences, are commonly classified as VNT
19 air deficiency causes genetic instability at microsatellite sequences because of the cell's inability
20 nes were shown to be modified genetically in microsatellite sequences by MNU and are believed to have
21            The target DNA to be screened for microsatellite sequences can be from YAC, P1, cosmid, ba
22  mechanism different from the instability in microsatellite sequences caused by defects in mismatch r
23 pression required preservation of the TGFBR2 microsatellite sequence; cells in which this sequence wa
24 nes displayed segments of the tk gene and/or microsatellite sequences copied into the DSB.
25  contains a previously unmapped, polymorphic microsatellite sequence, D8S421.
26                                  In general, microsatellite sequence data is in agreement with data o
27 ool for accurate genotyping of the resulting microsatellite sequencing data that uses an ensemble app
28                            Lack of repair of microsatellite sequence errors, created during replicati
29 dually increased to include up to 67% of the microsatellite sequences examined after 19 to 20 weeks o
30             The potential usefulness of this microsatellite sequence for population-level studies is
31 ome oxidase I gene, nine polymorphic nuclear microsatellites, sequences from 11 single-copy nuclear m
32            Instability in the repeat size of microsatellite sequences has been described in both here
33                                              Microsatellite sequences have an enhanced susceptibility
34                              Size changes in microsatellite sequences have been detected in many type
35 lustrate (i) the reduction of instability at microsatellite sequences, (ii) a significant decrease in
36 en used to determine mutation rates within a microsatellite sequence in human cancer cell lines with
37 surement of a spontaneous mutation rate of a microsatellite sequence in normal human cells.
38 nstability (MSI-H) accumulate mutations at a microsatellite sequence in the gene encoding transformin
39                 We sequenced the 10-adenines microsatellite sequence in the TGFBR2 gene of 32 MSI-H c
40 ted for the ability to detect alterations in microsatellite sequences in colon tumor samples.
41 t DSB repair can contribute to the spread of microsatellite sequences in mammalian genomes.
42                           EWS/FLI binds GGAA-microsatellite sequences in vivo and in vitro.
43 ed by widespread insertions and deletions in microsatellite sequences, including those comprised of m
44  [GT/CA](10), [TC/AG](11) and [TTCC/AAGG](9) microsatellite sequences inserted in the herpes simplex
45 idenced by the associated instability of DNA microsatellite sequences (MSI).
46 t strains, the vector containing the (A)(10) microsatellite sequence of TGFBR2 had a mutation rate (m
47 lbeta) error frequencies were quantitated in microsatellite sequences, relative to frame-shift error
48 the in vitro polymerase error frequencies at microsatellite sequences representative of those found i
49 f TGFBR2 with a 1-nucleotide deletion at its microsatellite sequence still produced a full-length TGF
50 ultiple human tumor suppressor genes include microsatellite sequences that are prone to mutations.
51               Exposure of plasmid containing microsatellite sequences to hydrogen peroxide resulted i
52 r pZCA29 and further promoted instability of microsatellite sequences under H(2)O(2) stress.
53   The mutational expansion of triplet repeat microsatellite sequences underlies the transmission of a
54 polymerase-DNA interactions as a function of microsatellite sequence, using polypyrimidine/polypurine
55 had deletions of a single CA-repeat from the microsatellite sequence, whereas repair-proficient cells
56 tions that were localized exclusively to the microsatellite sequences, whereas DNA damage by UV or N-
57 ave utilized this site to insert a synthetic microsatellite sequence within the beta-lactamase gene a