ライフサイエンスコーパス: microvascular endothelial cell

1 ion at threonine 495 levels in human cardiac microvascular endothelial cells.

2 e, and in supernatants of primary human lung microvascular endothelial cells.

3 engineered HeLa cells and Nox2(-/-) coronary microvascular endothelial cells.

4 avirus (PUUV)-infected patients and in human microvascular endothelial cells.

5 haracteristics different from those of other microvascular endothelial cells.

6 studied using primary fetal human pulmonary microvascular endothelial cells.

7 reted MFAP5 promotes tube formation of human microvascular endothelial cells.

8 cells to induce adhesion molecules on dermal microvascular endothelial cells.

9 tions and prevent virus transit across brain microvascular endothelial cells.

10 insulin and how this occurs in the relevant microvascular endothelial cells.

11 ndothelial cells and primary human pulmonary microvascular endothelial cells.

12 tumor microenvironment with cancer cells and microvascular endothelial cells.

13 rier disruption in LPS-challenged human lung microvascular endothelial cells.

14 ctive macropinocytic entry into human dermal microvascular endothelial cells.

15 ages and the tubule forming of mouse retinal microvascular endothelial cells.

16 es STIM1, thus inhibiting SOCE in human lung microvascular endothelial cells.

17 ion release but not entry into primary human microvascular endothelial cells.

18 ch as Andes virus (ANDV), which targets lung microvascular endothelial cells.

19 wing HPAI virus infection of human pulmonary microvascular endothelial cells.

20 nvestigated the role of P-Rex1 in human lung microvascular endothelial cells.

21 surface of IEs binding to human receptors on microvascular endothelial cells.

22 rvival, and proliferation of human pulmonary microvascular endothelial cells.

23 tyrosine nitration in cultured human cardiac microvascular endothelial cells.

24 lary damage was examined using primary brain microvascular endothelial cells.

25 eability in a dose-dependent manner in human microvascular endothelial cells.

26 which can further stimulate human intestinal microvascular endothelial cells.

27 ensitivity to TGF-beta1 stimulation in human microvascular endothelial cells.

28 ges prior to establishing their infection in microvascular endothelial cells.

29 Similar effects were observed in human brain microvascular endothelial cells.

30 nt, which profoundly alters the phenotype of microvascular endothelial cells.

31 erties of monocyte subsets using human brain microvascular endothelial cells.

32 and survival of BMCs or mature human cardiac microvascular endothelial cells.

33 l NanA had increased invasion of human brain microvascular endothelial cells.

34 sing NTR1 (NCM460-NTR1) and human intestinal microvascular-endothelial cells.

35 Human dermal microvascular endothelial cells 1, human platelets and n

36 esis study was evaluated in vitro with human microvascular endothelial cells-1 and in vivo with the M

37 anner in which they interact with macro- and microvascular endothelial cells, a key enabling componen

38 PCR-array screening of human brain microvascular endothelial cells after ABCD1 silencing re

39 RNA silencing studies in microvascular endothelial cells, along with knockout and

40 uman serum disrupted the barrier function of microvascular endothelial cells, an effect fully neutral

41 ic capacity of CNP was examined in pulmonary microvascular endothelial cells and aortic rings isolate

42 ion across monolayers of primary human brain microvascular endothelial cells and diminished BBB damag

43 Human microvascular endothelial cells and glomerular endotheli

44 ion at threonine 495 levels in human cardiac microvascular endothelial cells and improved BMC angioge

45 s in the barrier function of human pulmonary microvascular endothelial cells and in neutrophil traffi

46 ivo human SCD blood leukocyte recruitment by microvascular endothelial cells and in vivo neutrophil a

47 Y4 nucleotide receptor, expressed on cardiac microvascular endothelial cells and involved in postnata

48 GATA5 is expressed in microvascular endothelial cells and its genetic inactiva

49 deleterious effects induced by IL2 on brain microvascular endothelial cells and may inform the devel

50 crosslink HLA I on human aortic, venous, and microvascular endothelial cells and measured the binding

51 and the cellular influences of human retinal microvascular endothelial cells and mouse-derived neutro

52 tained from db/db mice in vivo and in kidney microvascular endothelial cells and podocytes treated wi

53 fering with circulating PCa cell adhesion to microvascular endothelial cells and potentially reducing

54 sis-resistant, and inflammatory phenotype of microvascular endothelial cells and smooth muscle cells

55 s reversed the PAH phenotype in isolated PAH microvascular endothelial cells and smooth muscle cells

56 xes between the central nervous system (CNS) microvascular endothelial cells and the choroid plexus e

57 ions, GD RBCs adhered more strongly to human microvascular endothelial cells and to laminin than CTR.

58 Here we show that Irx3 is expressed in human microvascular endothelial cells, and expression is eleva

59 ures, primary lung fibroblasts, primary lung microvascular endothelial cells, and primary alveolar ty

60 e, hantaviruses induced tPA but not PAI-1 in microvascular endothelial cells, and the induction was d

61 ch assay to assess serum reactivity to human microvascular endothelial cells, and used a combination

62 c (~2-fold vs. ~1.6-fold increase) for human microvascular endothelial cells, and Y2/Y5 receptor anta

63 an lung, human alveolar epithelial cells and microvascular endothelial cells are cultured in the micr

64 an enhanced ability to adhere to human brain microvascular endothelial cells as compared with monocyt

65 WNV-MAD78 replicated in and traversed brain microvascular endothelial cells as efficiently as WNV-NY

66 anistic studies in primary brain and retinal microvascular endothelial cells, as well as occluded rat

67 also inhibits the migration of human dermal microvascular endothelial cells at similar concentration

68 Silencing SIRT7 in pulmonary artery or microvascular endothelial cells attenuated LPS-induced i

69 Cultured human umbilical vein and microvascular endothelial cells avidly engulf BODIPY (4,

70 We focused our study on microvascular endothelial cells because they play a key

71 hosphatidylinositol-anchored glycoprotein of microvascular endothelial cells, binds lipoprotein lipas

72 erived endothelial progenitor cells to brain microvascular endothelial cell (BMEC)-like cells with go

73 arrier (BBB), which mainly consists of brain microvascular endothelial cells (BMEC).

74 brin-Matrigel mixed gel by coculturing brain microvascular endothelial cells (BMECs) and human mesenc

75 The molecular constituents of brain microvascular endothelial cells (BMECs) and pericytes, w

76 Acute Krit1 gene inactivation in mouse brain microvascular endothelial cells (BMECs) changes expressi

77 buted to the restrictive nature of the brain microvascular endothelial cells (BMECs) that comprise th

78 ation and tube formation properties of brain microvascular endothelial cells (BMECs) were analyzed as

79 Primary mouse brain microvascular endothelial cells (BMECs) were cultured an

80 a human in vitro BBB model comprising brain microvascular endothelial cells (BMECs), pericytes, astr

81 in several BBB models of rat and human brain microvascular endothelial cells (BMVEC) using a recyclab

82 ected to sham or stroke surgery and in brain microvascular endothelial cells (BMVECs) from Wistar and

83 usly that GSK3beta inhibition in human brain microvascular endothelial cells (BMVECs) reduced monocyt

84 human brain tissues and primary human brain microvascular endothelial cells (BMVECs), we demonstrate

85 adherin mediated by miRNA-101 in human brain microvascular endothelial cells (BMVECs).

86 events, inhibit VEGF-induced angiogenesis in microvascular endothelial cells by both (a) cleavage and

87 ed individuals, as controls) and human brain microvascular endothelial cells by using quantitative po

88 ned from patients during remission, to human microvascular endothelial cells caused vascular endothel

89 Silencing of ABCD1 in human brain microvascular endothelial cells causes accumulation of v

90 transporter 1 (GLUT-1) levels in human brain microvascular endothelial cells, causing disruption of b

91 protects cultured HT22 neuronal and primary microvascular endothelial cells co-cultured with primary

92 a set of differentially expressed miRNAs in microvascular endothelial cells co-cultured with tumour

93 man bronchial epithelial cells and pulmonary microvascular endothelial cells, compared with the abili

94 involved in lipid metabolism, in a cerebral microvascular endothelial cell culture system (hCMEC/D3)

95 16-1 cluster in primary mouse or human brain microvascular endothelial cell cultures enhanced in vitr

96 n, leukotriene generation, leukocyte-induced microvascular endothelial cell death, and retinal functi

97 ombinant proteinase-3 applied to human brain microvascular endothelial cells degraded both the tight

98 eproduce a directed differentiation of brain microvascular endothelial cells (dhBMECs) from two fluor

99 In cytokine-treated human lung microvascular endothelial cells, disruption of B1R-CPM h

100 respectively, in 10-day KSHV-infected dermal microvascular endothelial cells (DMVEC).

101 ating leukocytes with IL-1- or TNF-activated microvascular endothelial cells (ECs) and pericytes (PCs

102 Microvascular endothelial cells (ECs) are increasingly r

103 Microvascular endothelial cells (ECs) display a high deg

104 We demonstrate that microvascular endothelial cells (ECs) from Anxa2(-/-) mi

105 ed neurotrophic factor (BDNF) is secreted by microvascular endothelial cells (ECs) in the brain, func

106 Dermal microvascular endothelial cells (ECs) isolated from this

107 Knockdown of ZNF24 by siRNA in human primary microvascular endothelial cells (ECs) led to significant

108 TG microvascular endothelial cells (ECs) treated with AngII

109 Microvascular endothelial cells (ECs) within different t

110 he disassembly of junctional proteins within microvascular endothelial cells (ECs).

111 ns cells (LCs) to T cells through actions on microvascular endothelial cells (ECs).

112 We found that both human and murine brain microvascular endothelial cells express constituents of

113 Human lung microvascular endothelial cells expressed the TJ protein

114 We showed that acute stimulation of murine microvascular endothelial cells expressing the tumor nec

115 PAH pericytes seeded with healthy pulmonary microvascular endothelial cells failed to associate with

116 ue ABCD2, is highly expressed in human brain microvascular endothelial cells, far exceeding its expre

117 firmed by protection of cultured human brain microvascular endothelial cells from hydrogen peroxide-i

118 combination of human samples from PAH, human microvascular endothelial cells from lung, and Arrb knoc

119 vitro, angiotensin-(1-7) protected pulmonary microvascular endothelial cells from thrombin-induced ba

120 s of our genome-scale metabolic model of the microvascular endothelial cell function.

121 In human pulmonary microvascular endothelial cells, G was 20.4 +/- 12 Pa an

122 ) and chondroitin sulfate (CS) to glomerular microvascular endothelial cell (GEnC) glycocalyx and exa

123 ulin]) and AP components in human glomerular microvascular endothelial cells (GMVECs) and in HUVECs,

124 tion technology, we interlaced Human Adipose Microvascular Endothelial Cells (HAMEC) with hiPSCs, lea

125 sis across individual, primary human adipose microvascular endothelial cells (HAMECs), involving insu

126 our recently described role for human brain microvascular endothelial cells (HBEC) in modulating imm

127 of high therapeutic dosage on a human brain microvascular endothelial cell (HBMEC) model of the BBB.

128 mutant resulted in disruption of human brain microvascular endothelial cell (hBMEC) monolayer integri

129 tion and permeability of primary human brain microvascular endothelial cell (HBMEC) monolayers.

130 218 activates Rac1 (GTP-Rac1) of human brain microvascular endothelial cells (HBMEC) in a time-depend

131 m for inflammatory activation of human brain microvascular endothelial cells (HBMEC) in response to i

132 tococcus binding and invasion of human brain microvascular endothelial cells (HBMEC) is a prerequisit

133 am and interact with specialized human brain microvascular endothelial cells (hBMEC), which constitut

134 utes to type III GBS invasion of human brain microvascular endothelial cells (HBMEC), which constitut

135 egress in infected, nonpolarized human brain microvascular endothelial cells (HBMECs) and observed on

136 el to mimic the BBB by culturing human brain microvascular endothelial cells (HBMECs) in transwell in

137 Escherichia coli K1 infection of human brain microvascular endothelial cells (HBMECs) induces the exp

138 xidase (GlpO), was cytotoxic for human brain microvascular endothelial cells (HBMECs) via generation

139 Here, we found that infection of human brain microvascular endothelial cells (hBMECs) with GBS and ot

140 f neonatal meningitis, can enter human brain microvascular endothelial cells (hBMECs), but the host r

141 strates that CnMVs can fuse with human brain microvascular endothelial cells (HBMECs), the constituen

142 ivirus that persistently infects human brain microvascular endothelial cells (hBMECs), the primary ba

143 ic to neurons and persistently infects brain microvascular endothelial cells (hBMECs), which normally

144 hibitor of E. coli invasion into human brain microvascular endothelial cells (HBMECs).

145 acterized for binding to primary human brain microvascular endothelial cells (HBMECs).

146 er-endothelial cell junctions in human brain microvascular endothelial cells (HBMECs).

147 Human brain microvascular endothelial cells (hBMVECs) that constitut

148 using Heme-treated and untreated human brain microvascular endothelial cells (HBVEC), and determined

149 adhere to and migrate through human cerebral microvascular endothelial cells (HCMEC/D3), in a manner

150 bacteriophage (phage) K1F and human cerebral microvascular endothelial cells (hCMECs).

151 In human dermal microvascular endothelial cells (HDMECs), exposure to hy

152 VEC-derived EA.hy926 cells, and human dermal microvascular endothelial cells (HDMECs), we found that

153 d Rac1 in a sustained manner in human dermal microvascular endothelial cells (HDMVECs).

154 Microvascular endothelial cell heterogeneity and its rel

155 Human intestinal microvascular endothelial cells (HIMEC) and human intest

156 We investigated whether human intestinal microvascular endothelial cells (HIMEC) undergo EndoMT a

157 a (Caco-2) and human non-malignant intestine microvascular endothelial cells (HIMEC) was assessed.

158 e induces EndoMT in primary human intestinal microvascular endothelial cells (HIMECs) and whether End

159 ucosal biopsies and primary human intestinal microvascular endothelial cells (HIMECs) isolated from s

160 , and characterized human kidney peritubular microvascular endothelial cells (HKMECs) and reconstitut

161 ), as well as for GHRH itself, in human lung microvascular endothelial cells (HL-MVEC).

162 cal resistance (TEER) of human or mouse lung microvascular endothelial cells (HLMVEC or MLMVEC), and

163 In normal human lung microvascular endothelial cells (HLMVEC), bradykinin (BK

164 lipodia formation and motility of human lung microvascular endothelial cells (HLMVECs) via PI3K/Akt s

165 pre-requisite for migration using human lung microvascular endothelial cells (HLMVECs).

166 diated intracellular signaling of human lung microvascular endothelial cells (HLMVECs).

167 gs and CS extract-exposed primary human lung microvascular endothelial cells (HLMVECs).

168 n Tg(fli1:EGFP) zebrafish and inhibits human microvascular endothelial cell (HMEC-1) proliferation, t

169 Guided by cDNA microarray analysis of human microvascular endothelial cells (HMEC-1 line) subjected

170 In line with these data, human microvascular endothelial cells (HMEC-1) displayed an OM

171 Human microvascular endothelial cells (HMEC-1) stimulated with

172 its than control serum on unstimulated human microvascular endothelial cells (HMEC-1).

173 thelial cells (HUVEC) and immortalized human microvascular endothelial cells (HMEC-1).

174 Induction of miR-199a-5p in human dermal microvascular endothelial cells (HMECs) blocked angiogen

175 cape of R. conorii during infection of Human Microvascular Endothelial Cells (HMECs) by strand-specif

176 on protein (STAT) signaling pathway in human microvascular endothelial cells (HMECs), the most releva

177 Using human microvascular endothelial cells (HMECs), we examined inf

178 tions present in the RA joint, induced human microvascular endothelial cell (HMVEC) migration that wa

179 P-EA exerted antiangiogenic effects in human microvascular endothelial cells (HMVEC) and vasodilatory

180 d associated signaling to enter human dermal microvascular endothelial cells (HMVEC-d), an in vivo ta

181 iated herpesvirus (KSHV) enters human dermal microvascular endothelial cells (HMVEC-d), its naturalin

182 During de novo infection of human dermal microvascular endothelial cells (HMVEC-d), Kaposi's sarc

183 ociated herpesvirus (KSHV) into human dermal microvascular endothelial cells (HMVEC-d), natural in vi

184 ngiogenic potential of neonatal dermal human microvascular endothelial cells (HMVEC-dNeo).

185 formation and chemotaxis assays using human microvascular endothelial cells (HMVECs) transfected wit

186 PS mediate Ang2 signaling in human pulmonary microvascular endothelial cells (HPMECs) remain understu

187 an macrophages, HSAEpCs, and human pulmonary microvascular endothelial cells (HPMECs) significantly a

188 We found that in human retinal microvascular endothelial cells (HRECs) vitreous activat

189 To this end, human retinal microvascular endothelial cells (HRMEC) transfected with

190 In vitro studies using human retinal microvascular endothelial cells (HRMECs) showed increase

191 d in the vasculature, while in human retinal microvascular endothelial cells (HRMECs), TNF-alpha stim

192 ive agonist, beta-LGND2, using human retinal microvascular endothelial cell (HRMVEC) cultures and a m

193 s Pyk2 activation in mediating human retinal microvascular endothelial cell (HRMVEC) migration, sprou

194 Western blots of human retinal microvascular endothelial cells (hRMVECs) stimulated wit

195 induced Kdr phosphorylation in human retinal microvascular endothelial cells (HRMVECs).

196 educes the angiogenic potential of pulmonary microvascular endothelial cells, human umbilical vein en

197 In human primary retinal microvascular endothelial cells, hypoxia induces the exp

198 2) stimulated migration and tubulogenesis of microvascular endothelial cells, implicating a proangiog

199 mononuclear cells and in primary human lung microvascular endothelial cells in a concentration- and

200 otypic adhesion were further confirmed using microvascular endothelial cells in a static condition.

201 n imaging reveal that the plasma membrane of microvascular endothelial cells in caveolin 1(-/-) mice

202 igrated and proliferated less than wild-type microvascular endothelial cells in response to vascular

203 all, these data show for the first time that microvascular endothelial cells in the bone marrow and s

204 ced adherence to and invasion of human brain microvascular endothelial cells in vitro, demonstrating

205 lated gremlin secretion from human pulmonary microvascular endothelial cells in vitro, which inhibite

206 mechanism in pulmonary arterial and lung MV (microvascular) endothelial cells in response to DNA dama

207 of neurons, pericytes, astrocytes, and brain microvascular endothelial cells, in brain-like tissues a

208 were abundantly expressed in cultured mouse microvascular endothelial cells, including NLRP3, apopto

209 d caspase-1 activity and IL-1beta release in microvascular endothelial cells, indicating an activatio

210 MC(TC)LUVA potentiated fetal human pulmonary microvascular endothelial cell interactions, inhibited t

211 catabolism is disrupted in human intestinal microvascular endothelial cells isolated from patients w

212 CSE expression was also increased in cardiac microvascular endothelial cells, isolated from endotheli

213 , increased miR-19b expression in human lung microvascular endothelial cells, leading to a decrease i

214 4var09) cytoadhere in vitro to a human brain microvascular endothelial cell line (HBEC-5i).

215 Experiments in a human microvascular endothelial cell line demonstrated that TN

216 ABSTRACT: The human cerebral microvascular endothelial cell line hCMEC/D3 was used to

217 onolayers of the immortalized human cerebral microvascular endothelial cell line hCMEC/D3.

218 Using the human microvascular endothelial cell line, human mast cell lin

219 ther rat alveolar macrophages (AMs) and lung microvascular endothelial cells (LMVECs) support Seoul v

220 Upon KSHV infection, primary dermal microvascular endothelial cells lost expression of endot

221 Here we employ LEC, microvascular endothelial cells (MEC), and human umbilic

222 d the lipoma-preferred partner (LPP) gene in microvascular endothelial cells (MECs) and that LPP expr

223 The relevance of tissue specificity of microvascular endothelial cells (MECs) in the response t

224 We utilised isogenic keratinocytes, microvascular endothelial cells, melanocytes and fibrobl

225 EphrinB2 DeltaV primary kidney microvascular endothelial cells migrated and proliferate

226 The studies conducted in polarized human microvascular endothelial cell monolayers (hCMEC/D3) in

227 hibited leukocyte adherence to human retinal microvascular endothelial cell monolayers and leukostasi

228 In these studies, rat brain microvascular endothelial cell monolayers exposed to cal

229 Permeability of human brain microvascular endothelial cell monolayers was increased

230 bilical vein endothelial cell and human lung microvascular endothelial cell monolayers were treated w

231 induce changes in the permeability of brain microvascular endothelial cell monolayers.

232 bilical vein endothelial cell and human lung microvascular endothelial cell monolayers.

233 demonstrated with lysates of mouse pulmonary microvascular endothelial cells (MPMVECs) that were stim

234 Microvascular endothelial cells (MVEC) were developed fr

235 th factor signaling at the receptor level in microvascular endothelial cells (MVEC), and CD36 has bee

236 We found that the CLL stroma included microvascular endothelial cells (MVECs) expressing BAFF

237 g, migration, and sprouting of primary brain microvascular endothelial cells (MVECs) in a dose-depend

238 Microvascular endothelial cells (MVECs) injury is a crit

239 ction in all forms of PAH and tested whether microvascular endothelial cells (MVECs) or pulmonary art

240 down-regulation of their cognate receptor on microvascular endothelial cells (MVECs), CD36.

241 d functions of ICAM-1 in cerebral and dermal microvascular endothelial cells (MVECs).

242 phasic and concentration-dependent manner in microvascular endothelial cells of the blood-brain barri

243 /-BQ788) was given to cultured rat pulmonary microvascular endothelial cells overexpressing ETB recep

244 However, in human dermal microvascular endothelial cells, P3 did not affect BMP9-

245 e utilised a high TEER primary porcine brain microvascular endothelial cell (PBMEC) culture to assess

246 ascular unit organoid containing human brain microvascular endothelial cells, pericytes, astrocytes,

247 GF, IL8, and CXCL12 leading to chemotaxis of microvascular endothelial cells, phosphorylation of VE-c

248 In cultured microvascular endothelial cells, Piezo1 channel activati

249 ved by the disease process, with luminal and microvascular endothelial cells playing a critical role

250 agonist-induced NOX2 activation in pulmonary microvascular endothelial cells (PMVEC) and that the eff

251 of NADPH oxidase type 2 (NOX2) in pulmonary microvascular endothelial cells (PMVECs), alveolar macro

252 ro transmigration model with human pulmonary microvascular endothelial cells (PMVECs).

253 lood-brain barrier, mainly composed of brain microvascular endothelial cells, poses an obstacle to dr

254 endothelial cell adhesion molecules in brain microvascular endothelial cell proliferation and apoptos

255 monary veins associated with foci of intense microvascular endothelial-cell proliferation of the capi

256 rent human blood monocytes and in human lung microvascular endothelial cells, providing a mechanism f

257 Isolated cardiomyocytes and microvascular endothelial cells responded to AngII with

258 Graft microvascular endothelial cells responded to DSA in vivo

259 RNA-mediated knockdown of SRSF2 in pulmonary microvascular endothelial cells resulted in elevated lev

260 3 gain and loss of function studies in human microvascular endothelial cells resulted in the modulati

261 T bone marrow-derived macrophages with renal microvascular endothelial cells results in increased lev

262 We isolated primary renal microvascular endothelial cells (RMEC) and aortic endoth

263 formed antibody reactivity against DKO renal microvascular endothelial cells (RMEC) in vitro.

264 In cultured brain microvascular endothelial cells, Sema3E down-regulated D

265 We show that keratinocytes and microvascular endothelial cells show greatest NO release

266 Cell-binding studies using microvascular endothelial cells showed receptor-specific

267 Gene transfection of human pulmonary microvascular endothelial cells showed that C242T p22(ph

268 In vitro experiments using human lung microvascular endothelial cells showed that exogenous S1

269 clinically available BET inhibitor.Methods: Microvascular endothelial cells, smooth muscle cells iso

270 macrovascular endothelial cells and retinal microvascular endothelial cells that C-1-P induces invas

271 ximum wall shear stress, whereas low-density microvascular endothelial cells that lack cell-cell cont

272 major entry pathway of KSHV in human dermal microvascular endothelial cells, the natural target cell

273 Using monolayers of mouse primary brain microvascular endothelial cells, the permeability coeffi

274 flammatory mediators by primary human dermal microvascular endothelial cells through a signaling path

275 e-mediated induction of ALCAM in human brain microvascular endothelial cells through the translocatio

276 this device, we investigated the response of microvascular endothelial cells to shear-stress gradient

277 In vitro exposure of human microvascular endothelial cells to Shiga toxin recapitul

278 hese exosomes alone can activate human brain microvascular endothelial cells to stimulate adhesion mo

279 In TNFalpha-stimulated primary human retinal microvascular endothelial cells, total levels of epoxyei

280 Human microvascular endothelial cells transfected with hHO-1 d

281 S, we performed microarray analysis of human microvascular endothelial cells treated with TNF-alpha i

282 In retinal microvascular endothelial cells, TRPV4 channels regulate

283 In human central nervous system microvascular endothelial cells, VEGFA and the TYMP prod

284 in modulating proliferation and apoptosis of microvascular endothelial cells via its modulation of CD

285 ithelial cell wound healing, maintained lung microvascular endothelial cell viability, and proliferat

286 ial electrical resistance of human pulmonary microvascular endothelial cells was analyzed.

287 of human bronchial epithelial cells and lung microvascular endothelial cells was exposed to immunosup

288 VEGF, transendothelial migration through CNS microvascular endothelial cells was regulated by VEGF.

289 ICAM-1(null)/ICAM-2(-/-) primary mouse brain microvascular endothelial cells, we demonstrate that neu

290 In human microvascular endothelial cells, we found that GPx-1 def

291 Human pulmonary microvascular endothelial cells were also used to examin

292 horylation and barrier disruption, pulmonary microvascular endothelial cells were engineered for the

293 Rat brain microvascular endothelial cells were exposed to various

294 , motility and polarization toward pulmonary microvascular endothelial cells were reduced, whereas wi

295 lical vein endothelial cells or adult dermal microvascular endothelial cells were transduced with the

296 ger annexin A1 (ANXA1) is expressed in brain microvascular endothelial cells, where it regulates BBB

297 (WNV-NY) strains in neurons, astrocytes, and microvascular endothelial cells, which comprise the neur

298 nificantly induced CX3CL1 production in lung microvascular endothelial cells, which was blocked by in

299 Treatment of brain microvascular endothelial cells with AGE caused a simila

300 Transcriptomic analysis of human microvascular endothelial cells with GATA5 knockdown rev