ライフサイエンスコーパス: microvesicular

1 developed hepatic steatosis characterized by microvesicular and macrovesicular lipid accumulation and

2 deep neural network-based model to quantify microvesicular and macrovesicular steatosis in the liver

3 to recognize and differentiate the areas of microvesicular and macrovesicular steatosis, and to quan

4 frozen-section biopsy to distinguish between microvesicular and macrovesicular steatosis.

5 s showed confluent parenchymal necrosis with microvesicular and macrovesicular steatosis.

6 rtal mononuclear infiltrates, hepatocellular microvesicular changes, cytoplasmic lipid droplets, and

7 ne COS7 and found that a cellugyrin-positive microvesicular compartment was present in all cell types

8 n fat synthesis were strongly activated, and microvesicular fat accumulated.

9 g almost exclusively in the third trimester; microvesicular fatty infiltration of hepatocytes causes

10 isomal beta-oxidation, development of severe microvesicular fatty liver, peroxisome assembly, cell de

11 e, the livers of ACOX -/- mice reveal severe microvesicular fatty metamorphosis of hepatocytes.

12 s, including reduced liver weight, transient microvesicular fatty metamorphosis, prolonged extramedul

13 r cell hyperplasia, erythrophagocytosis, and microvesicular fatty metamorphosis.

14 NI1/hSNF5 is completely absent from purified microvesicular fractions, it is specifically incorporate

15 a short-term treatment (5 days) that induces microvesicular hepatic steatosis and marked hypercholest

16 exposed larvae, including macrovesicular and microvesicular hepatic steatosis, as well as focal liver

17 We found that VLCAD-deficient hearts have microvesicular lipid accumulation, marked mitochondrial

18 ion by fibroblasts is controlled through the microvesicular release of EMMPRIN from tumor cells.

19 mal beta-oxidation system, exhibit extensive microvesicular steatohepatitis, leading to hepatocellula

20 ngly reduced (macrovesicular steatosis -34%; microvesicular steatosis -100%; inflammation -74%) and w

21 There was a significant increase in microvesicular steatosis accompanied by a marked reducti

22 al liver histology consisting of significant microvesicular steatosis and fatty Kupffer cells but no

23 s in the serum and induced chronic low-level microvesicular steatosis and inflammation in GWI vs Naiv

24 CV, there was an inverse correlation between microvesicular steatosis and level of autophagy (r = -0.

25 nodeficiency virus infection, which includes microvesicular steatosis and more severe hepatic injury

26 cholestasis and cirrhosis in the former and microvesicular steatosis and oncocytic transformation (m

27 tical information including the detection of microvesicular steatosis and quantitation of liver lipid

28 Livers with even severe microvesicular steatosis can be reliably used for transp

29 ced biliary and liver damage and may promote microvesicular steatosis development during NAFLD progre

30 considered macrovesicular and 50% considered microvesicular steatosis important.

31 at-free diet and converted macrovesicular to microvesicular steatosis in B6.V-Lep(ob) obese mice as d

32 id screening of mitochondrial toxins-induced microvesicular steatosis in primary hepatocyte cultures.

33 Necropsy analysis revealed hepatic microvesicular steatosis in pubescent male homozygous mi

34 Loss of MCs prominently reduced microvesicular steatosis in zone 1 hepatocytes.

35 d liver damage and specifically up-regulated microvesicular steatosis in zone 1 hepatocytes.

36 Hepatic microvesicular steatosis is a hallmark of drug-induced h

37 transplanting livers with moderate to severe microvesicular steatosis is unknown.

38 Liver histology showed widespread, microvesicular steatosis on light-microscopic examinatio

39 acts/delays rapid progression of the hepatic microvesicular steatosis to the characteristic macrovesi

40 Microvesicular steatosis was the only ethanol-induced ch

41 ectron-microscopic examination showed severe microvesicular steatosis with severe mitochondrial injur

42 reated bcl-2 (-/+) mice displayed only early microvesicular steatosis without progression to extensiv

43 this virus developed diffuse hepatocellular microvesicular steatosis, an abnormal accumulation of in

44 higher degrees of necrosis, fibrosis stage, microvesicular steatosis, and ductular reaction among ot

45 rmal liver histology with macrovesicular and microvesicular steatosis, fatty Kupffer cells, extensive

46 itis with changes in hepatic tissues such as microvesicular steatosis, likely caused by an increase i

47 may progress to NASH when the liver displays microvesicular steatosis, lobular inflammation, and peri

48 The liver showed diffuse microvesicular steatosis, marked periportal nuclear glyc

49 ocyte ballooning, which can be confused with microvesicular steatosis, whereas demonstration of an in

50 Blunting of microvesicular steatosis, which is restricted to few liv

51 he detection and characterization of hepatic microvesicular steatosis.

52 quate for the clinical evaluation of hepatic microvesicular steatosis.

53 han Oil Red O histology for the detection of microvesicular steatosis.

54 tool for the clinical evaluation of hepatic microvesicular steatosis.

55 ess that includes the development of hepatic microvesicular steatosis.

56 xacerbated hepatocyte damage, with extensive microvesicular steatosis.

57 e, progressive hepatocellular ballooning and microvesicular steatosis.

58 th were commonly identified risk factors for microvesicular steatosis.

59 ion of 40 allografts with moderate or severe microvesicular steatosis.

60 which the donor liver contained at least 30% microvesicular steatosis.

61 terized by liver inflammation, fibrosis, and microvesicular steatosis.

62 mers in microvesicles/exosomes and show that microvesicular TDP-43 is preferentially taken up by reci

63 n of tubular injury in all biopsy specimens: microvesicular tubular epithelial cytoplasmatic vacuoliz

64 ique, we provide evidence for preferentially microvesicular uptake as well as both soma-to-soma "hori