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1 tin filaments and the plasma membrane of the microvillus.
2  localized to the intestinal epithelial cell microvillus.
3 anipulation of signal diffusivity within the microvillus.
4 ibers, and taper apically to a single, large microvillus.
5 napses with nerve fibers, and a single large microvillus.
6 ediated by molecular targets within the same microvillus.
7  that increasing signal diffusivity within a microvillus accelerates arrest.
8  components that control the organization of microvillus actin cytoskeleton, leading to severe microv
9  higher doses caused internalization without microvillus activation.
10 mber of catch bonds initially immobilize the microvillus, after which additional bonds accumulate and
11 lus surface of enterocytes that disrupts the microvillus and alters its actin structure to form a dom
12                 The number of gold particles/microvillus and the density of gold particles/microgram
13 yte differentiation, and profoundly disrupts microvillus architecture and essential nutrient transpor
14 ed to HH GBS mutants revealed global loss of microvillus architecture, disruption of cytoplasmic and
15 rface and induce a massive remodeling of the microvillus architecture.
16 al model of cell rolling that focuses on the microvillus as the unit of cell-substrate interaction an
17 tailed analysis describes for the first time microvillus assembly by villin, redefines the actin-bund
18  as being instrumental for brush border (BB) microvillus assembly during differentiation and effaceme
19                                       Apical microvillus assembly is reduced by up to 50%, as measure
20 ndling activities of Eps8 and Eps8L1a during microvillus assembly.
21 (+/- 5 pN), a tether will be formed from the microvillus at a constant velocity, which depends linear
22 villus actin cytoskeleton, leading to severe microvillus atrophy.
23      In contrast to a rigid or nonextendible microvillus, both microvillus extension and tether forma
24 ve found also that, under a pulling force, a microvillus can be extended (microvillus extension) or a
25 ase endocytosis only on the G(M1), integrin, microvillus-containing apical surface.
26 ted that act-5 gene expression is limited to microvillus-containing cells within the intestine and ex
27 rmine domain-specific organization, with the microvillus coordinating release modes along its membran
28    The mRNA for villin, a well-characterized microvillus cytoskeletal protein, was sorted to the basa
29  of villin, an actin-bundling protein of the microvillus cytoskeleton.
30 mber of bonds per cell as well as per single microvillus decreases with increasing membrane stiffness
31 he state diagram for adhesion which includes microvillus deformation, and find four adhesion states-f
32        Thus, villin severs F-actin to ensure microvillus depolarization and enterocyte remodeling upo
33 machinery into the intestinal lumen and that microvillus-derived LVs modulate epithelial-microbial in
34 face of small intestine enterocytes, causing microvillus effacement and rearrangement of the host cel
35                                              Microvillus effacement is inhibited after exposure of ca
36                                        While microvillus effacement was detected in both 388- and 388
37 cytoskeletal structure, loss of readherence, microvillus effacement, and interruption of signal trans
38 on, leading to the loss of cell adhesion and microvillus effacement.
39 ar properties such as cell deformability and microvillus elasticity actively modulate leukocyte rolli
40          Fixation of neutrophils to abrogate microvillus elasticity resulted in rolling behavior simi
41    Moreover, adherence to IVOC, EPEC-induced microvillus elongation and colonization of the murine in
42                                         Each microvillus employs a full G-protein-coupled receptor si
43 n indented nucleus, possess a single, apical microvillus extending through the taste pore, and are ch
44 large round or oval nucleus, a single apical microvillus extending through the taste pore, and specia
45 , and the constitutive equations that govern microvillus extension and tether extraction.
46 o a rigid or nonextendible microvillus, both microvillus extension and tether formation can decrease
47                     Our results suggest that microvillus extension during transient PSGL-1/P-selectin
48               Moreover, the microbes induced microvillus extension from the epithelial cell surface.
49                                              Microvillus extension has been hypothesized in contribut
50 olling by incorporating cortical tension and microvillus extension into a versatile, semianalytical f
51 at glutaraldehyde-fixed neutrophils (without microvillus extension or tether extraction) roll unstabl
52 n individual microvilli will dictate whether microvillus extension or tether formation occurs.
53 ulling force, a microvillus can be extended (microvillus extension) or a long thin membrane cylinder
54  exhibit a 2.5-fold reduction in the rate of microvillus extension.
55 have been identified, the molecular basis of microvillus formation is largely undefined.
56 act-5 gene function revealed that intestinal microvillus formation requires a specific actin isoform.
57 s on the photoreceptor apical surface before microvillus formation, and at the time of microvillus in
58  dynamics in processes such as phagocytosis, microvillus formation, and tumor cell metastasis.
59  had distinctive foot process effacement and microvillus formation.
60      These results provide a new context for microvillus function in the host-pathogen relationship,
61                                              Microvillus function was additionally altered as lymphoc
62 are the first to offer a temporally resolved microvillus growth mechanism and highlight factors that
63 at the cell surface and mark future sites of microvillus growth.
64 r vacuolar structures containing microvilli (microvillus inclusion bodies) in epithelial enterocytes,
65  nonconventional myosin Vb (Myo5b) result in microvillus inclusion disease (MVID) and massive secreto
66                                              Microvillus inclusion disease (MVID) is a disorder of in
67                                              Microvillus inclusion disease (MVID) is a rare congenita
68                                              Microvillus inclusion disease (MVID) is a rare intestina
69                                              Microvillus inclusion disease (MVID) is a severe form of
70                                              Microvillus inclusion disease (MVID) is caused by inacti
71 nterocytes, a phenotype reminiscent of human microvillus inclusion disease (MVID), a devastating cong
72 (Stx3) disturb epithelial polarity and cause microvillus inclusion disease (MVID), a lethal hereditar
73                                              Microvillus inclusion disease (MVID), caused by loss-of-
74                           Some patients with microvillus inclusion disease due to myosin 5B (MYO5B) m
75 nclusions in small-intestinal enterocytes in microvillus inclusion disease is currently unclear.
76 alfunction causes the congenital enteropathy microvillus inclusion disease, underlining its importanc
77 rved in duodenal biopsies from patients with microvillus inclusion disease.
78  VB and that UNC45A loss causes a variant of microvillus inclusion disease.
79 ce appeared similar to that of patients with microvillus inclusion disease.
80  in Cdc42 signaling could be associated with microvillus inclusion disease.
81                                     Finally, microvillus inclusions and shortened microvilli were evi
82 cal and basolateral trafficking; however, no microvillus inclusions were observed in MYO5B-KD cells.
83 utant forms of MYO5B, we observed that early microvillus inclusions were positive for the sorting mar
84 an be diagnosed based on loss of microvilli, microvillus inclusions, and accumulation of subapical ve
85 ence of apical microvilli, the appearance of microvillus inclusions, and enlarged intercellular space
86  diarrhea, loss of microvilli, occurrence of microvillus inclusions, and subapical secretory granules
87 of secretory granules precedes occurrence of microvillus inclusions, indicating involvement of MYO5B
88 ds displayed altered luminal development and microvillus inclusions, while 2D cultures revealed Rab11
89  MYO5B-KD phenotype but induced formation of microvillus inclusions.
90 interaction between RAB11A and MYO5B induced microvillus inclusions.
91 re microvillus formation, and at the time of microvillus initiation WASp colocalizes with amphiphysin
92                                         Each microvillus is a cylindrical membrane protrusion that is
93                                         Each microvillus is capable of generating elementary response
94                                       When a microvillus is extended, it acts like a spring with a sp
95 ceptor-ligand bonds impact the motion of the microvillus, leading to feedback between binding and mic
96 o examine the combined effect of gravity and microvillus length heterogeneity on tip contact force (F
97                                          The microvillus length necessary to reconcile dissociation c
98  a novel ezrin-interacting partner, controls microvillus length through its capping activity.
99 , we have obtained two comparable neutrophil microvillus lengths, both approximately 0.3 microm in av
100  outer segments in rods and (ii) between the microvillus-like calyceal processes and the outer segmen
101 ng fertilization occurs via an actin-filled, microvillus-like cell protrusion.
102 lectron microscopy revealed the formation of microvillus-like extensions around adherent bacteria fol
103  P. gingivalis wild-type strain 381 revealed microvillus-like extensions around adherent bacteria; th
104  to C. neoformans triggered the formation of microvillus-like membrane protrusions within 15 to 30 mi
105      This resulted in disappearance of their microvillus-like protrusions accompanied by SPRY2-depend
106 ense features identified as ruffles and with microvillus-like protrusions from the cell's dorsal surf
107 of cell-substrate interaction and integrates microvillus mechanics, receptor clustering, force-depend
108  findings demonstrate that PKCalpha promotes microvillus membrane DMT1 expression and intestinal iron
109                       Hyperglycemia promotes microvillus membrane expression of DMT1 in intestinal ep
110 a brush border actin-binding protein) in the microvillus membrane fraction of rabbit ileum; this pool
111                                          Two microvillus membrane proteins (130 and 140 kDa) were ind
112 in complexes isolated from solubilized renal microvillus membrane vesicles.
113 ocity occurs at an intermediate value of the microvillus membrane viscosity, remarkably close to the
114                     Western blot analyses of microvillus membranes and immunoelectron microscopy of k
115 l epithelial cells with reduced microvilli ("microvillus-minus," or MVM) but retaining normal tight j
116 ich CLIC4 is important for luminal delivery, microvillus morphogenesis, and endolysosomal biogenesis.
117 e protein (WASp) is necessary for rhabdomere microvillus morphogenesis.
118 dgut enterocyte, Myo1B is present within the microvillus (MV) of the apical brush border (BB) where i
119 adhesion and uptake of particles compared to microvillus-positive cells.
120 ns of F with cellular proteins, resulting in microvillus projections necessary for the formation of f
121 e phosphoproteomics identified Ser775 in the microvillus protein Eps8 as a bona fide target of NleH1
122 achment through both Pf-IRBC knobs and HBMEC microvillus protrusions.
123  The apparent elastic spring constant of the microvillus ranged from 1340 to 152 pN/microm at 0.4 and
124                      We also explore how the microvillus rheology itself controls the dynamics of adh
125 crovilli themselves, these results suggest a microvillus-specific function for act-5, and further, th
126 ulated the dependence of rolling velocity on microvillus stiffness.
127     Unexpectedly, DP is important for proper microvillus structure.
128 tes a receptor (type III secretion) into the microvillus surface of enterocytes that disrupts the mic
129 ins, and actin are also located on or in the microvillus, this organelle has many of the major elemen
130 S2 and GPI-anchor-dependent release from the microvillus tip in mice.
131 li, shedding restricts alpha-tectorin to the microvillus tip, compartmentalizing collagen-binding sit
132 of L-selectin, and sequestered away from the microvillus tips in the case of LFA-1, Mac-1, and PSGL-1
133 ) refractory period distribution (time for a microvillus to recover after a QB).
134 merulosclerosis and extensive effacement and microvillus transformation of podocyte foot processes.
135 peptide, and caused a dramatic elongation of microvillus-type parallel actin bundles in transfected e
136 ing capabilities of the actin filament-rich, microvillus-type specializations that mediate sensory tr
137                 The number of gold particles/microvillus was also increased in complement-treated rab
138  protein was confined to the membrane of the microvillus where it was in close association with brush
139 ce is </=34 pN (+/- 3 pN), the length of the microvillus will be extended; if the force is >61 pN (+/

 
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