ライフサイエンスコーパス: mitochondrial protein

1 vivo, suggesting that Arabidopsis COX19 is a mitochondrial protein.

2 d anti-apoptotic Bcl-2 protein and increased mitochondrial proteins.

3 cation and expression, and for the import of mitochondrial proteins.

4 suppressors are pathway intrinsic and encode mitochondrial proteins.

5 rt, depends on the activation of antioxidant mitochondrial proteins.

6 d acetyl-CoA levels, and hyperacetylation of mitochondrial proteins.

7 berrantly elevated phosphorylation on select mitochondrial proteins.

8 The aggresomes appear to triage unimported mitochondrial proteins.

9 in (Dox)-cardiotoxicity via deacetylation of mitochondrial proteins.

10 by transient alterations in contractile and mitochondrial proteins.

11 nction may be the result of dysregulation of mitochondrial proteins.

12 lates thousands of nuclear, cytoplasmic, and mitochondrial proteins.

13 tochondrial function through upregulation of mitochondrial proteins.

14 tochondrion-associated ER membrane (MAM) and mitochondrial proteins.

15 g in simultaneous rescue of the loss of both mitochondrial proteins.

16 s normal transcript levels at genes encoding mitochondrial proteins.

17 scent 2-iron, 2-sulfur clusters to recipient mitochondrial proteins.

18 amphetamine-induced increase in synaptosomal mitochondrial proteins.

19 electron transport chain subunits and other mitochondrial proteins.

20 tation-tolerant tend to encode metabolic and mitochondrial proteins.

21 e cytosol is limited in degrading unimported mitochondrial proteins.

22 ) is a member of a family of nuclear-encoded mitochondrial proteins.

23 increase in 4HNE adduction of metabolic and mitochondrial proteins (16 of 27 identified proteins), i

24 As in BJAB cells, mitochondrial proteins accumulated in BCR-stimulated cel

25 itochondrial function by not only repressing mitochondrial protein acetylation but also enhancing PPA

26 The therapeutic effects of normalizing mitochondrial protein acetylation by expanding the NAD(+

27 kout (cKO) mice showed significantly reduced mitochondrial protein acetylation following a HFD relati

28 d AEC SIRT3 protein expression and increased mitochondrial protein acetylation, including MnSOD(K68)

29 (GCN5L1) protein has been shown to modulate mitochondrial protein acetylation, mitochondrial content

30 ession of nuclear-encoded, TIM23-transported mitochondrial proteins ACO2, TUFM, IDH3A, CLPP and mitoc

31 emical uncoupling of mitochondria, increased mitochondrial-protein aging, and accumulation of p62/SQS

32 hondrial function, following deletion of the mitochondrial protein AIF, OPA1, or PINK1, as well as ch

33 Thus, DGS1 links mitochondrial protein and lipid import with cellular lip

34 rmation process in vitro using only purified mitochondrial proteins and defined DNA templates.

35 not decrease 4HNE adduction of metabolic and mitochondrial proteins and did not improve oxidative pho

36 e the null phenotypes for 21 nuclear-encoded mitochondrial proteins and in-depth characterization of

37 red NADPH oxidoreductase activity, increased mitochondrial proteins and increased superoxide producti

38 under heat shock contain both cytosolic and mitochondrial proteins and interact with the mitochondri

39 complex is the entry gate for virtually all mitochondrial proteins and is essential to build the mit

40 x of Cav-1-sphingolipid particles containing mitochondrial proteins and lipids.

41 brane essential for the function of numerous mitochondrial proteins and processes.

42 nal mechanisms controlling the expression of mitochondrial proteins and suggest novel strategies to t

43 s several novel structures including knotted mitochondrial proteins and the most deeply embedded prot

44 we generated 21 novel antibodies to various mitochondrial proteins and used this resource to charact

45 ase: Dld2, which, as its human homolog, is a mitochondrial protein, and the cytosolic protein Dld3.

46 stoma cells, isolated mitochondria, isolated mitochondrial proteins, and planar lipid bilayer membran

47 hether caspase activation, apoptotic-related mitochondrial proteins, and regulators of ER stress sens

48 Other mitochondrial proteins are also affected, resulting in i

49 Most of over a thousand mitochondrial proteins are encoded by nuclear genes and

50 Nearly all mitochondrial proteins are encoded by the nuclear genome

51 Most mitochondrial proteins are encoded by the nuclear genome

52 We have previously shown that heart mitochondrial proteins are hyperacetylated in OVE26 mice

53 Most mitochondrial proteins are imported through the TIM23 tr

54 Most mitochondrial proteins are nuclearly encoded and are imp

55 Mitochondrial proteins are replete with phosphorylation,

56 Most of the >1,000 different mitochondrial proteins are synthesized as precursors in

57 Most mitochondrial proteins are synthesized as precursors tha

58 Mutations in CHCHD10, a gene coding for a mitochondrial protein, are implicated in ALS-FTD spectru

59 target of miR-379, and identified the DAPIT mitochondrial protein as a translational target of EIF4G

60 t of structurally and functionally unrelated mitochondrial proteins as substrates of the SUMO pathway

61 Tom22 forms a 500-kDa complex with mitochondrial proteins associated with 3betaHSD2.

62 We also observed increased acetylation of mitochondrial proteins associated with decreased NAD+/NA

63 uggest that PC2 normally serves to limit key mitochondrial proteins at the ER-mitochondrial interface

64 Our proteomic analysis identifies mitochondrial protein ATAD3A as an interactor of mitocho

65 The mitochondrial protein Atg32 is the yeast SAR that mediat

66 A mitochondrial protein, BbOhmm, is demonstrated to limit

67 haliana) OXIDATION RESISTANCE2 (AtOXR2) is a mitochondrial protein belonging to the Oxidation Resista

68 ational modification that is also present on mitochondrial proteins, but the mitochondrial lysine-spe

69 the synthesis of a subset of nuclear-encoded mitochondrial proteins by cytosolic ribosomes on the mit

70 ithin mitochondria, modulate the activity of mitochondrial proteins by protein processing, and mediat

71 Here, we describe a putative human mitochondrial protein, C6orf203, that contains an S4-lik

72 These data indicate that cardiac mitochondrial proteins can be target autoantigens in myo

73 es between hESCs and hiPSCs, we identified a mitochondrial protein, CHCHD2, whose expression seems to

74 We analyzed partial sequences of three mitochondrial protein-coding genes (COI, ND2 and CytB) a

75 A Bayesian dated phylogeny, based on the 13 mitochondrial protein-coding genes, supports a mid-Pleis

76 depletion results in impaired translation of mitochondrial protein-coding mRNAs and decreases mitocho

77 ) and identified 27 differentially expressed mitochondrial proteins compared with tagged Col-0 contro

78 tochondrial biogenesis and expression of the mitochondrial proteins Complex III and IV, consistent wi

79 r X1)-TUFM (Tu translation elongation factor mitochondrial) protein complex, promoting autophagic flu

80 and Hsp70 and downregulation of a cluster of mitochondrial protein components of complexes III, IV, a

81 Many mitochondrial proteins contain N-terminal presequences t

82 as analysed by citrate synthase activity and mitochondrial protein content by Porin expression, whils

83 eatment, we observed no change in markers of mitochondrial protein content.

84 Three sperm mitochondrial proteins copurified with the recombinant,

85 ease caused by the deficiency of frataxin, a mitochondrial protein crucial for iron-sulfur cluster bi

86 l oxidation and, in particular, oxidation of mitochondrial protein cysteine residues.

87 We propose that cumulative distal mitochondrial protein damage results in impaired protein

88 ance from the soma correlates with increased mitochondrial protein damage, PINK1 accumulation, reacti

89 These findings demonstrate a role for a mitochondrial protein deacetylase in hippocampal neurons

90 twork and behavioral adaptations require the mitochondrial protein deacetylase SIRT3 as they are abol

91 insulin deficiency were related to increased mitochondrial protein degradation and decreased protein

92 Therefore, CLPP2 contributes to the mitochondrial protein degradation network through suppor

93 We show that SUMO modification of mitochondrial proteins does not rely on mitochondrial ta

94 Analyses of the nuclear ITS2 region and the mitochondrial protein-encoding loci allowed accurate tax

95 upregulated 66 endoplasmic reticulum and 193 mitochondrial proteins, enhancing several processes and

96 Lineage-specific analysis of mitochondrial protein evolution revealed a significant e

97 to endoplasmic reticulum stress and aberrant mitochondrial protein expression in autophagy-deficient

98 clear base excision repair (BER) protein, in mitochondrial protein extracts derived from mammalian ti

99 levels of reactive oxygen species (ROS), or mitochondrial protein folding stress, a percentage of AT

100 nd transcription, and TRAP1, which regulates mitochondrial protein folding.

101 isease, promotes mitophagy by ubiquitinating mitochondrial proteins for efficient engagement of the a

102 isease caused by inherited deficiency of the mitochondrial protein Frataxin (FXN), which has no appro

103 isorder caused by recessive mutations in the mitochondrial protein frataxin (FXN).

104 t is caused by systemic insufficiency of the mitochondrial protein frataxin.

105 XN) gene, resulting in loss of the essential mitochondrial protein frataxin.

106 onal interactions for further exploration of mitochondrial protein function.

107 ource should provide molecular insights into mitochondrial protein functions.

108 mitochondria and ER interplay, and how this mitochondrial protein gains access to the proteasome.

109 ochondria and ER, and shed light on how this mitochondrial protein gains access to the proteasome.SIG

110 Previously, mutation in three genes encoding mitochondrial proteins has been implicated in autosomal

111 to the mitochondria and its interaction with mitochondrial proteins has not been explored.

112 quality control (IMQC) system is central to mitochondrial protein homeostasis and cellular health.

113 overed a conserved, robust mechanism linking mitochondrial protein homeostasis and the cytosolic fold

114 (UPR(mt)), which includes genes that promote mitochondrial protein homeostasis and the recovery of de

115 , loss of OMA1 results in alterations in the mitochondrial protein homeostasis, as reflected by enhan

116 n its binding partner, Cdc48, contributes to mitochondrial protein homeostasis.

117 terol receptor that recruits Vms1 to support mitochondrial protein homeostasis.

118 emain, including: which nuclear genes encode mitochondrial proteins; how their expression varies with

119 ced levels of posttranslationally lipoylated mitochondrial proteins, hyperaccumulation of photorespir

120 lating the NAD(+) salvage pathway suppressed mitochondrial protein hyperacetylation and cardiac hyper

121 We show that mitochondrial protein hyperacetylation due to NAD(+) red

122 Among the mitochondrial proteins identified by gene ontology analy

123 In particular, mitochondrial proteins implicated in H2S detoxification

124 nalyses establish an unexpected link between mitochondrial protein import and inner membrane protein

125 in vivo for the bacterial Sec system and the mitochondrial protein import apparatus.

126 sent in the intermembrane space and inhibits mitochondrial protein import by interacting with TIM23,

127 19) describe an unexpected role for Porin in mitochondrial protein import by regulating the oligomeri

128 complex, resulting in inhibition of synaptic mitochondrial protein import first detected in presympto

129 dentifies conserved and modified features of mitochondrial protein import in apicomplexan parasites.

130 versatility and dynamic organization of the mitochondrial protein import machineries.

131 23 as a novel regulator or stabilizer of the mitochondrial protein import machinery that is specifica

132 interacting with TIM23, a major component of mitochondrial protein import machinery, but evidence for

133 a central, membrane-embedded subunit of the mitochondrial protein import machinery.

134 tasis network, emphasizing the importance of mitochondrial protein import processes for development,

135 and enters the mitochondria through TOM20, a mitochondrial protein import receptor.

136 oteases, such as HtrA2 and Lon protease, and mitochondrial protein import significantly aggravates al

137 , pATOM36 has a dual function and integrates mitochondrial protein import with mitochondrial DNA inhe

138 significance of mHTT-mediated inhibition of mitochondrial protein import, a mechanism likely broadly

139 complex, is essential for parasite survival, mitochondrial protein import, and assembly of the TOM co

140 known about the cytosolic events regulating mitochondrial protein import, partly due to the lack of

141 patients, supporting the lack of deficit in mitochondrial protein import.

142 TOMM70 is a receptor that assists mainly in mitochondrial protein import.

143 that is important for parasite survival and mitochondrial protein import.

144 hat mHTT may interact with TIM23 and inhibit mitochondrial protein import.

145 nd scavenging, cardiolipin peroxidation, and mitochondrial protein import.

146 The level of soluble matrix mitochondrial proteins imported through the TIM23 comple

147 KO) abrogates lysine methylation of a single mitochondrial protein in human cells.

148 usly unknown pathway can selectively degrade mitochondrial proteins in aged and stressed cells withou

149 A-tagged nuclear, cytoplasmic, membrane, and mitochondrial proteins in diverse cell types.

150 nges in expression of nuclear genes encoding mitochondrial proteins in human skeletal muscle cells fo

151 d immunofluorescent labelling of neurons and mitochondrial proteins in mouse and human brain tissues

152 Thus, the incomplete lipoylation of mitochondrial proteins in mtacp mutants, particularly Gl

153 ial bioenergetics as well as the increase in mitochondrial proteins in Nox4-deficient lung fibroblast

154 large structures, we were able to visualise mitochondrial proteins in passively cleared tissues to r

155 In particular, damage to mitochondrial proteins in skeletal muscle, which is a lo

156 hts the importance of correct trafficking of mitochondrial proteins in the cell and the potential imp

157 P techniques resulted in high purity of >90% mitochondrial proteins in the lysate.

158 ease, investigating the distribution of nine mitochondrial proteins in thousands of single muscle fib

159 fy 2,427 cross-linked peptide pairs from 327 mitochondrial proteins in whole, respiring murine mitoch

160 , the much lower level of basal synaptosomal mitochondrial proteins in WT mice showed a rapid increas

161 of PIGBOS reveals an undiscovered role for a mitochondrial protein, in this case a microprotein, in t

162 We highlight organism-wide differences in mitochondrial proteins including consistent increases in

163 ith many proteins but with a predominance of mitochondrial proteins including CYP2E1.

164 We report the localization of 12 new mitochondrial proteins, including 6 putative mitoribosom

165 organelle, along with a subset of authentic mitochondrial proteins, including Ant4, Suox, and Spata1

166 g Skd3 exhibit reduced solubility of various mitochondrial proteins, including anti-apoptotic Hax1.

167 intracellular domain interacts with multiple mitochondrial proteins, including critical factors assoc

168 This coincides with the repression of mitochondrial proteins, including many proteins of the i

169 NAcase inhibition on translation of specific mitochondrial proteins, including superoxide dismutase 2

170 Proteomic analysis showed that clusters of mitochondrial proteins, including voltage-dependent anio

171 derived l-[1-13C]-phenylalanine into de novo mitochondrial protein increased dose-dependently after i

172 Mitochondrial proteins increased by 32% from P1 to P42.

173 ficient cells exhibited decreased GFP-tagged mitochondrial proteins inside the vacuole and decreased

174 Mitochondrial protein interactions and complexes facilit

175 -acyl carrier protein transacylase (MCAT), a mitochondrial protein involved in fatty acid biosynthesi

176 This mitochondrial protein is a vital energy regulator that,

177 me analyses, we show that the translation of mitochondrial proteins is highly down-regulated in yeast

178 The data suggest that the import of mitochondrial proteins is saturable and that the cytosol

179 Expression of mitochondrial proteins is tightly regulated in response

180 observed for many different nuclear-encoded mitochondrial protein knockouts hints that distinct ener

181 in vivo studies in mice and humans, that the mitochondrial protein LACTB potently inhibits the prolif

182 teomic analysis validated similar changes in mitochondrial protein levels in the isolated tumor tissu

183 To enable the assessment of mitochondrial protein levels, we have developed a quadru

184 a comprehensive central resource for data on mitochondrial protein localisation.

185 lated with hyperacetylation of IDH2 and SOD2 mitochondrial proteins, lowered enzymatic activities, an

186 out in HCV replicon cells, we identified the mitochondrial protein LRPPRC as an NS5A binding factor.

187 with specialized/enriched functions, such as mitochondrial protein maturation, thermotolerance, senes

188 al protein Vps13 or by overexpression of the mitochondrial protein Mcp1.

189 These largely consisted of mitochondrial proteins, metabolic regulators, and sarcom

190 Here we report that human mitochondrial protein Miner2 [2Fe-2S] clusters can bind

191 oxide in the CDGSH-type [2Fe-2S] clusters in mitochondrial protein Miner2 may represent a new nitric

192 Here, we show that the mitochondrial protein mitofusin 2 (Mfn2) protects agains

193 t mutations in the nuclear gene encoding the mitochondrial protein mitofusin-2 (MFN2).

194 Titrating expression levels of the mitochondrial protein mitoNEET is a powerful approach to

195 ed large scale loss of the oxidant-sensitive mitochondrial protein Mpv17L.

196 These mtROS damage mitochondrial proteins, mtDNA, and membrane lipids and r

197 Nuclear-encoded mitochondrial proteins need to be imported, processed, f

198 mologue (CLUH) regulates the expression of a mitochondrial protein network supporting key metabolic p

199 MitoQ also ameliorated alterations in mitochondrial proteins observed in obese rats: increases

200 A51 (also known as MCART1)-an essential(6,7) mitochondrial protein of previously unknown function-as

201 ect, it differs from all other characterized mitochondrial proteins of baker's yeast.

202 tent, judged by increased levels of numerous mitochondrial proteins, of the mitochondrial structural

203 ork captured a profound effect of unimported mitochondrial proteins on cytosolic proteostasis and rev

204 osylation of various nuclear, cytosolic, and mitochondrial proteins on serine/threonine amino acid re

205 We identified a so far uncharacterized mitochondrial protein (open reading frame YDR381C-A) as

206 ges in mass-specific respiratory capacities, mitochondrial protein or antioxidant content were found.

207 aperones, meaning that the overproduction of mitochondrial proteins or the limited availability of ch

208 Reduced mitochondrial protein oxidation in FABP4/aP2(-/-) macrop

209 Moreover, SUMOylated forms of mitochondrial proteins particularly accumulate in HSP70-

210 ilitate direct visualization and labeling of mitochondrial protein PARylation.

211 Remarkably, the majority of cycling mitochondrial proteins peaked during the early light pha

212 PGAM5 is a mitochondrial protein phosphatase whose genetic ablation

213 adation in yeast via involvement of the Aup1 mitochondrial protein phosphatase, as well as 2 known ma

214 Here we demonstrate that the mitochondrial protein phosphoglycerate mutase family mem

215 Thus, succinylation of UCP1 and other mitochondrial proteins plays an important role in BAT an

216 te tethering is mechanistically regulated by mitochondrial proteins promoting Rab7 GTP hydrolysis, an

217 volves binding of the ER protein VAPB to the mitochondrial protein PTPIP51, which act as scaffolds to

218 ic protease P (ClpP) plays a central role in mitochondrial protein quality control by degrading misfo

219 A deeper understanding of how mitochondrial protein quality control mechanisms are coo

220 ther resolubilization nor degradation by the mitochondrial protein quality control system were observ

221 ring stability to the NEFs, helped fine-tune mitochondrial protein quality control, and regulated cru

222 in 70 (mtHsp70), a chaperone contributing to mitochondrial protein quality control.

223 ADMA impaired protein synthesis and reduced mitochondrial protein quality.

224 The yeast mitochondrial proteins Rcf1 and Rcf2 are associated with

225 drial DNA (mtDNA) mutations and oxidation of mitochondrial proteins, reactive oxygen species (ROS) le

226 quality control (QC) pathways for misfolded mitochondrial proteins remain poorly defined.

227 Mitochondrial proteins remain the subject of intense res

228 (OR) = 0.78, P = 4.05 x 10(-11)) encoding a mitochondrial protein required for redox homeostasis; rs

229 rotein-derived amino acid incorporation into mitochondrial protein respond to increasing protein inta

230 om patients with obesity show alterations in mitochondrial proteins similar to those observed in obes

231 We find that levels of mitochondrial protein succinylation and malonylation are

232 possibly, other mitochondrial proteins such as ADP/ATP carrier proteins.

233 [4Fe-4S] cluster insertion into a subset of mitochondrial proteins such as lipoate synthase and succ

234 hese changes reflect selective inhibition of mitochondrial protein synthesis (probably translation) w

235 Defects of mitochondrial protein synthesis account for the largest

236 hich do not display an overall inhibition in mitochondrial protein synthesis but rather have a proble

237 an essential role in determining the rate of mitochondrial protein synthesis by regulating the level

238 e functional rescue of mt-RNA processing and mitochondrial protein synthesis defects after lentiviral

239 lasts, skeletal and cardiac muscle, although mitochondrial protein synthesis defects were confined to

240 Inhibition of mitochondrial protein synthesis induces acetylation and

241 ly, mS38 is necessary to sustain the overall mitochondrial protein synthesis rate, despite an adaptiv

242 and consequently, maintenance of the overall mitochondrial protein synthesis rate.

243 ctron transport, mitochondrial dynamics, and mitochondrial protein synthesis were dysregulated.

244 rs metabolic changes in protein translation, mitochondrial protein synthesis, and posttranslational r

245 s was selective and did not adversely affect mitochondrial protein synthesis.

246 of impaired mt-RNA processing and defective mitochondrial protein synthesis.

247 ed levels of the 55S monosome and attenuated mitochondrial protein synthesis.

248 iciency, causing a decrease in cytosolic and mitochondrial protein synthesis.

249 In this way, we identify SMIM10, a mitochondrial protein that in melanoma cells selectively

250 MTCH2, which encodes a mitochondrial protein that regulates mitochondrial metab

251 Here, 127 cytosolic and 20 mitochondrial proteins that are components of essential

252 e in basal level of synaptosomal hnRNP H and mitochondrial proteins that decreased in response to met

253 Mmt1 and Mmt2 are nuclearly encoded mitochondrial proteins that export iron from the mitocho

254 Carefully orchestrated interactions between mitochondrial proteins that facilitate cell death remain

255 nt optic atrophy (ADOA) are caused by mutant mitochondrial proteins that lead to defects in mitochond

256 inducing post-translational modifications of mitochondrial proteins that regulate mitochondrial dynam

257 ylome analysis identified a subpopulation of mitochondrial proteins that was sensitive to changes in

258 and functional studies demonstrate HPDL is a mitochondrial protein, the loss of which causes a clinic

259 sulfur protein family that also includes two mitochondrial proteins: the type II diabetes-related mit

260 Several mitochondrial protein thiols exposed to the mitochondria

261 PfLipL2) that are responsible for activating mitochondrial proteins through the covalent attachment o

262 itochondrial function to a 'fixed' amount of mitochondrial protein, thus allowing for intrinsic mitoc

263 in 3 (SIRT3) deacetylates and regulates many mitochondrial proteins to maintain health, but its funct

264 tion and presentation of ubiquitinated sperm mitochondrial proteins to the 26S proteasome, explaining

265 Most nuclear-encoded mitochondrial proteins traffic from the cytosol to mitoc

266 Here, we identify a mitochondrial protein trafficking pathway in Drosophila

267 The mitochondrial protein trafficking route mediated by Dosm

268 cles, including decreased levels of specific mitochondrial protein transcripts (RNA) and progressive

269 ent tumors showed elevated expression of the mitochondrial protein translation (MPT) gene pathway rel

270 thymidine monophosphate (dTMP) biosynthesis, mitochondrial protein translation, and methionine regene

271 mal proteins or mitoribosome assembly impair mitochondrial protein translation, causing combined OXPH

272 and tigecycline, an antibiotic that inhibits mitochondrial protein translation, selectively eradicate

273 ms remove arrested import intermediates from mitochondrial protein translocases, stabilize protein ho

274 Elucidating the molecular mechanisms of mitochondrial protein translocation is crucial for under

275 The mitochondrial protein, translocator protein (TSPO), is a

276 rs homologues of proteins from all the major mitochondrial protein translocons present in yeast, sugg

277 ally affect the TIM22 and TIM23 complexes in mitochondrial protein transport.

278 The mitochondrial proteins TRAP1 and HTRA2 have previously b

279 reased levels of reactive oxygen species and mitochondrial protein ubiquitination.

280 m implicates a role for calcium signaling in mitochondrial protein ubiquitylation, protein turnover,

281 s an important role in clearing mislocalized mitochondrial proteins upon cell stimulation, and its ab

282 ss spectrometry, we identified enrichment of mitochondrial proteins upon immunoprecipitation of p62.

283 The majority of mitochondrial proteins use N-terminal presequences for t

284 of a custom panel including genes coding for mitochondrial proteins was performed in patients with co

285 For the mitochondrial proteins, we discuss the folding problem f

286 83 nuclear mRNAs encoding mitochondrial proteins were changed following 1alpha,25(

287 Expression levels of representative mitochondrial proteins were compared from harvested tiss

288 UBQLN1 expression was acutely inhibited, 120 mitochondrial proteins were enriched in the cytoplasm, s

289 iated methylation changes in nuclear-encoded mitochondrial proteins were involved in regulating cellu

290 In total, 371 mitochondrial proteins were quantified, and of these 76

291 that SUMO serves as a mark for nonfunctional mitochondrial proteins, which only sporadically arise in

292 t the Obg family protein GTPBP5 or MTG2 is a mitochondrial protein whose absence in a TALEN-induced H

293 of USP30, the E3 ligase March5 ubiquitinates mitochondrial proteins whose eventual import depends on

294 SCO1 is a ubiquitously expressed, mitochondrial protein with essential roles in cytochrome

295 ins including consistent increases in NNT, a mitochondrial protein with essential roles in influencin

296 Here, we identified NaSIPP, a mitochondrial protein with phosphate transporter activit

297 dampens the transcription of genes encoding mitochondrial proteins with no change to transcript half

298 s MitoMiner a unique platform to investigate mitochondrial proteins, with application in mitochondria

299 Hundreds of nuclear, cytoplasmic, and mitochondrial proteins within multicellular eukaryotes h

300 FMMS produced 3x more synaptic mitochondrial protein yield compared to UC from the same