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1 ight persulfides using the alkylating agent, monobromobimane.
2 pha by measuring the thiol reactivities with monobromobimane.
4 ants were labeled with the fluorescent probe monobromobimane and subjected to an array of fluorescenc
5 known "turn on" fluorescence probe, that is, monobromobimane, and the trapping agent, that is, 2,4-di
7 tivity with 100 microM mycothiol or with the monobromobimane derivative of 1-D-myo-inosityl-2-(L-cyst
8 ity of the NDA method with an assay based on monobromobimane derivatization and HPLC analysis with fl
9 tion of mitochondria with tamoxifen, DTT, or monobromobimane did not reverse the diminished calcium l
10 ith the small, solvent-sensitive fluorophore monobromobimane, exhibit spectral changes only upon bind
12 e show that the steady-state emission from a monobromobimane fluorophore in Loop V-VI is quenched by
14 ling the cysteines with a fluorescent label (monobromobimane) followed by fluorescence spectroscopic
15 ed cystine for varying periods, treated with monobromobimane, harvested, and extracted with perchlori
16 Wild-type Arabidopsis plants treated with monobromobimane (mBB) form a fluorescent GSH-mBB conjuga
20 maleimidylphenyl)-4-methylcoumarin (CPM), or monobromobimane (mBBr) were used to study the interactio
21 he resulting SonoCage construct reacted with monobromobimane (mBBr), a fluorogenic, thiol-reactive mo
22 gmatis was treated with the alkylating agent monobromobimane (mBBr), the cellular mycothiol was conve
24 The persulfides had higher reactivity with monobromobimane than analogous thiols and putative thiol
26 te and hydrogen peroxide, and with the probe monobromobimane, were studied and compared with those of
27 thod uses a very small cysteine-reactive dye monobromobimane, with virtually no linker, and various t